Search results for "regulator"

showing 10 items of 1009 documents

Polycomb-like 2 Associates with PRC2 and Regulates Transcriptional Networks during Mouse Embryonic Stem Cell Self-Renewal and Differentiation

2010

SummaryPolycomb group (PcG) proteins are conserved epigenetic transcriptional repressors that control numerous developmental gene expression programs and have recently been implicated in modulating embryonic stem cell (ESC) fate. We identified the PcG protein PCL2 (polycomb-like 2) in a genome-wide screen for regulators of self-renewal and pluripotency and predicted that it would play an important role in mouse ESC-fate determination. Using multiple biochemical strategies, we provide evidence that PCL2 is a Polycomb Repressive Complex 2 (PRC2)-associated protein in mouse ESCs. Knockdown of Pcl2 in ESCs resulted in heightened self-renewal characteristics, defects in differentiation, and alte…

Pluripotent Stem CellsCellular differentiationGene regulatory networkDown-RegulationPolycomb-Group Proteinsmacromolecular substancesMethylationBiochemistryArticleCell LineHistonesSelf-RenewalMice03 medical and health sciences0302 clinical medicineEmbryonic Stem CellHistone methylationPolycomb-group proteinsGeneticsAnimalsGene Regulatory NetworksEpigeneticsInduced pluripotent stem cellEmbryonic Stem Cells030304 developmental biologyGenetics0303 health sciencesbiologyurogenital systemGene Expression ProfilingPolycomb Repressive Complex 2Cell DifferentiationCell BiologyCellular ReprogrammingSTEMCELLPRC2Embryonic stem cellRepressor ProteinsOncologyDifferentiation030220 oncology & carcinogenesisembryonic structuresbiology.proteinMolecular MedicineTranscriptional NetworkPRC2Genome-Wide Association StudyProtein BindingCell Stem Cell
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Trade and Interlegality

2019

The idea that the WTO legal system and its institutional bodies (especially the judicial branch) have developed a single-sided ides of what is good and would attempt to impose a deep-rooted liberalization agenda on the rest of world is hardly corroborated by an empirical analysis. Quite the contrary, recourse to a series of legal techniques has made it possible to leave open a space for reciprocal understanding and debate with other circuits of legality.

Political scienceWTO reciprocity direct effect deep liberalization contestation interpretation domestic regulatory autonomy interlegalityIUS/13 - DIRITTO INTERNAZIONALEPrinciple of legalityWTO interlegality Appellate Body interpretation direct effectComputingMilieux_MISCELLANEOUSLaw and economics[SHS]Humanities and Social Sciences
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Effects of indole-3-acetic acid on Sinorhizobium meliloti survival and on symbiotic nitrogen fixation and stem dry weight production

2009

We evaluated the effects of the main auxin phytohormone, indole-3-acetic acid (IAA), on the central metabolism of Sinorhizobium meliloti strain 1021. We either treated the Sinorhizobium meliloti 1021 strain with 0.5 mM IAA (1021+) or use a derivative, RD64, of the same strain harbouring a pathway for IAA biosynthesis converting tryptophan into IAA via indoleacetamide. We assayed the activity of key enzymes in the major energy-yielding pathways (Entner-Doudoroff, Embden-Meyerhof-Parnas, pentose phosphate, glyoxylate bypass and tricarboxylic acid cycle). We found that activity of two main regulative tricarboxylic acid (TCA) cycle enzymes was increased. Citrate synthase (CS) activity, as compa…

PolyestersHydroxybutyratesDehydrogenaseCitrate (si)-SynthaseApplied Microbiology and BiotechnologyCell survival . PHB . TCA . Nitrogen fixationchemistry.chemical_compoundBacterial ProteinsPlant Growth RegulatorsAcetyl Coenzyme AAuxinNitrogen FixationMedicago truncatulaCitrate synthaseKetoglutarate Dehydrogenase ComplexBiomasschemistry.chemical_classificationSinorhizobium melilotiMicrobial ViabilityIndoleacetic AcidsPlant StemsbiologyTryptophanfood and beveragesGeneral MedicineMetabolismbiology.organism_classificationCitric acid cycleBiochemistrychemistrybiology.proteinIndole-3-acetic acidSinorhizobium melilotiBiotechnologyApplied Microbiology and Biotechnology
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Inorganic Polyphosphates: Biologically Active Biopolymers for Biomedical Applications

2013

Inorganic polyphosphate (polyP) is a widely occurring but only rarely investigated biopolymer which exists in both prokaryotic and eukaryotic organisms. Only in the last few years, this polymer has been identified to cause morphogenetic activity on cells involved in human bone formation. The calcium complex of polyP was found to display a dual effect on bone-forming osteoblasts and bone-resorbing osteoclasts. Exposure of these cells to polyP (Ca2+ complex) elicits the expression of cytokines that promote the mineralization process by osteoblasts and suppress the differentiation of osteoclast precursor cells to the functionally active mature osteoclasts dissolving bone minerals. The effect o…

PolyphosphateRegulatorBiological activityBiologyBone morphogenetic protein 2Proinflammatory cytokineCell biologychemistry.chemical_compoundMediatormedicine.anatomical_structurechemistryOsteoclastPrecursor cellmedicine
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The Regulatory State vs. the Networked Polity: confronting narratives of change

2014

The aim of the article is to summarize and reassess the innovations brought about by governance theory. It is argued that the notion of governance is a conceptual device that could help rationalise and articulate the changes undertaken by liberal democracies since the late 1970s. The article suggests the need to distinguish between two distinct research programmes composing governance studies, which are influenced by alternative epistemic traditions—political economy and economic sociology. Rationalizations of change influenced by political economy support the idea that the outcome of recent political change is a market-oriented “Regulatory State”, while those influence by economic sociolog…

Power (social and political)Economic sociologyPolitical scienceCorporate governanceRegulatory stategovernance regulation regulatory state networked polityNormativeNarrativeGeneral MedicinePolityLiberal democracyPositive economicsSocial science
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Exploring a new incubation model for FinTechs: Regulatory sandboxes

2021

Research on incubation models indicates that incubators and accelerators are crucial catalysts for the development of start-ups. To facilitate start-ups in financial markets, several regulatory authorities have adopted a new incubation model called a ‘regulatory sandbox’. Regulatory sandboxes enable eligible applicants to test their technology-enabled financial solutions for a certain period of time (subject to conditions the regulator imposes). As such, these instruments allow innovation while preventing severe instability in financial markets caused by systemic risk. Despite their importance, management research has devoted little attention to studying how sandboxes operate as a new incub…

Process managementBusiness accelerators05 social sciencesFinancial marketGeneral EngineeringDesign elements and principles512 Business and managementBusiness incubatorsIncubation models050905 science studiesFinancial technology (FinTech)Qualitative analysisActivity system frameworkManagement of Technology and Innovation0502 economics and businessVDP::Teknologi: 500::Maskinfag: 570Systemic riskManagement researchSandbox (software development)System frameworkBusinessRegulatory sandbox0509 other social sciencesIncubation050203 business & management
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Polyglutamine toxicity induces rod photoreceptor division, morphological transformation or death in Spinocerebellar ataxia 7 mouse retina

2010

In neurodegenerative disorders caused by polyglutamine (polyQ) expansion, polyQ toxicity is thought to trigger a linear cascade of successive degenerative events leading to neuronal death. To understand how neurons cope with polyQ toxicity, we studied a Spinocerebellar ataxia 7 (SCA7) mouse which expresses polyQ-expanded ATXN7 only in rod photoreceptors. We show that in response to polyQ toxicity, SCA7 rods go through a range of radically different cell fates, including apoptotic and non-apoptotic cell death, cell migration, morphological transformation into a round cell or, most remarkably, cell division. The temporal profile of retinal remodeling indicates that some degenerative pathways …

Programmed cell deathCell divisionProliferationPopulationMice TransgenicNerve Tissue ProteinsBiologylcsh:RC321-571Mice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineCell MovementRetinal Rod Photoreceptor CellsmedicineAnimalsSpinocerebellar AtaxiasNeurodegenerationeducationlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryCell ShapeComputingMilieux_MISCELLANEOUSSpinocerebellar ataxia 7030304 developmental biologyAtaxin-7Mice Knockout0303 health sciencesRetinaeducation.field_of_studyPhotoreceptorCell DeathRetinal DegenerationNeurodegenerationRetinalmedicine.diseaseRemodelingMice Inbred C57BLmedicine.anatomical_structureNeurologyProteotoxicitychemistryNerve DegenerationSpinocerebellar ataxia[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Apoptosis Regulatory ProteinsPeptidesPolyglutamineNeuroscience030217 neurology & neurosurgery
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Bax inhibitor-1 is likely a pH-sensitive calcium leak channel, not a H+/Ca2+ exchanger.

2014

The endoplasmic reticulum (ER) plays a key role in the synthesis, folding, and sorting of proteins, and disturbances of this delicate system can cause cell death. The ER also serves as the major intracellular calcium (Ca(2+)) store, and release of Ca(2+) from this store controls diverse cellular functions. At the interface of both these functions of the ER is Bax inhibitor-1 (BI-1), an evolutionarily conserved multifunctional protein that mediates Ca(2+) efflux from the ER and protects against ER stress. Several mechanisms have been proposed to explain how BI-1 might mediate Ca(2+) efflux from the ER. Chang et al. present structural evidence that a bacterial homolog of BI-1, BsYetJ, is a pH…

Programmed cell deathProtein familyProteolipidschemistry.chemical_elementCalciumBiologyEndoplasmic ReticulumBiochemistryModels BiologicalCalcium in biologySpecies SpecificityHumansMolecular BiologyBAX inhibitor 1Endoplasmic reticulumCell MembraneMembrane ProteinsCell BiologyHydrogen-Ion ConcentrationCell biologychemistryUnfolded protein responseCalciumEffluxCalcium ChannelsApoptosis Regulatory ProteinsBacillus subtilisScience signaling
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MYC and EGR1 synergize to trigger tumor cell death by controlling NOXA and BIM transcription upon treatment with the proteasome inhibitor bortezomib

2014

The c-MYC (MYC afterward) oncogene is well known for driving numerous oncogenic programs. However, MYC can also induce apoptosis and this function of MYC warrants further clarification. We report here that a clinically relevant proteasome inhibitor significantly increases MYC protein levels and that endogenous MYC is necessary for the induction of apoptosis. This kind of MYC-induced cell death is mediated by enhanced expression of the pro-apoptotic BCL2 family members NOXA and BIM. Quantitative promoter-scanning chromatin immunoprecipitations (qChIP) further revealed binding of MYC to the promoters of NOXA and BIM upon proteasome inhibition, correlating with increased transcription. Both pr…

Programmed cell deathTranscription GeneticEGR1ApoptosisBiologyBortezomibProto-Oncogene Proteins c-mycMicehemic and lymphatic diseasesCell Line TumorProto-Oncogene ProteinsGeneticsmedicineAnimalsPromoter Regions GeneticTranscription factorCells CulturedEarly Growth Response Protein 1Zinc finger transcription factorBinding SitesOncogeneBcl-2-Like Protein 11Genes p16Gene regulation Chromatin and EpigeneticsMembrane ProteinsPromoterGenes p53Boronic AcidsChromatinddc:Gene Expression Regulation NeoplasticProto-Oncogene Proteins c-bcl-2PyrazinesCancer researchProteasome inhibitorApoptosis Regulatory ProteinsProteasome Inhibitorsmedicine.drug
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Death of mitochondria during programmed cell death of leaf mesophyll cells

2005

The role of plant mitochondria in the programmed cell death (PCD) is widely discussed. However, spectrum and sequence of mitochondrial structural changes during different types of PCD in leaves are poorly described. Pea, cucumber and rye plants were grown under controlled growing conditions. A part of them were sprinkled with ethylene releaser to accelerate cell death. During yellowing the palisade parenchyma mitochondria were attracted to nuclear envelope. Mitochondrial matrix became electron translucent. Mitochondria entered vacuole by invagination of tonoplast and formed multivesicular bodies. Ethephon treatment increased the frequency of sticking of mitochondria to the nuclear envelope …

Programmed cell deathfood and beveragesApoptosisCell BiologyGeneral MedicineVacuoleMitochondrionPeroxisomeGolgi apparatusBiologyMitochondriaCell biologyPlant LeavesChloroplastsymbols.namesakeOrganophosphorus CompoundsPlant Growth RegulatorsApoptosissymbolsUltrastructurePlant Physiological PhenomenaCell Biology International
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