Search results for "respiratory tract diseases"

showing 10 items of 1015 documents

Short term associations of ambient nitrogen dioxide with daily total, cardiovascular, and respiratory mortality: multilocation analysis in 398 cities

2021

Objective To evaluate the short term associations between nitrogen dioxide (NO2) and total, cardiovascular, and respiratory mortality across multiple countries/regions worldwide, using a uniform analytical protocol. Design Two stage, time series approach, with overdispersed generalised linear models and multilevel meta-analysis. Setting 398 cities in 22 low to high income countries/regions. Main outcome measures Daily deaths from total (62.8 million), cardiovascular (19.7 million), and respiratory (5.5 million) causes between 1973 and 2018. Results On average, a 10 μg/m3 increase in NO2 concentration on lag 1 day (previous day) was associated with 0.46% (95% confidence interval 0.36% to 0.5…

OzoneNitrogen DioxideRespiratory Tract Diseases610 Medicine & healthAir Pollutants; Air Pollution; Cardiovascular Diseases; Cities; Developed Countries; Developing Countries; Environmental Exposure; Global Health; Humans; Linear Models; Nitrogen Dioxide; Respiratory Tract Diseases; Urban HealthRespiratory MortalityHealth benefitsGlobal HealthArbetsmedicin och miljömedicinchemistry.chemical_compound360 Social problems & social servicesCardiovascular MortalityAir PollutionHumansMedicineAmbient Nitrogen DioxideNitrogen dioxideCitiesRespiratory system610 Medicine & healthDeveloping CountriesMCCAir Pollutantsbusiness.industryResearchDeveloped CountriesUrban HealthEnvironmental ExposureOccupational Health and Environmental HealthGeneral MedicineEnvironmental exposureConfidence intervalTerm (time)Increased riskchemistryambient nitrogen dioxideCardiovascular DiseasesLinear ModelsRespiratory mortalityDeterminantes da Saúde e da Doençabusiness360 Social problems & social servicesDaily MortalityDemography
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Phospho-p38 MAPK expression in COPD patients and asthmatics and in challenged bronchial epithelium

2015

<b><i>Background:</i></b> The role of mitogen-activated protein kinases (MAPK) in regulating the inflammatory response in the airways of patients with chronic obstructive pulmonary disease (COPD) and asthmatic patients is unclear. <b><i>Objectives:</i></b> To investigate the expression of activated MAPK in lungs of COPD patients and in bronchial biopsies of asthmatic patients and to study MAPK expression in bronchial epithelial cells in response to oxidative and inflammatory stimuli. <b><i>Methods:</i></b> Immunohistochemical expression of phospho (p)-p38 MAPK, p-JNK1 and p-ERK1/2 was measured in bronchial mucosa in pat…

P38 MAPKMaleMAPK/ERK pathwayAsthma phenotypeSMOKERespiratory SystemMitogen-activated protein kinases; p65; Pathology of chronic obstructive pulmonary disease; Chronic obstructive pulmonary disease phenotypes; Asthma phenotypesPathology of chronic obstructive pulmonary diseasep38 Mitogen-Activated Protein KinasesChronic obstructive pulmonary disease phenotypePulmonary Disease Chronic ObstructiveOXIDATIVE STRESSMACROPHAGESRespiratory systemMitogen-activated protein kinasesChronic obstructive pulmonary disease phenotypesMitogen-activated protein kinases; p65; pathology of chronic obstructive pulmonary disease phenotypes; asthma phenotypesCOPDp65KinaseAsthma phenotypes; Chronic obstructive pulmonary disease phenotypes; Mitogen-activated protein kinases; p65; Pathology of chronic obstructive pulmonary disease; Pulmonary and Respiratory MedicineACTIVATED PROTEIN-KINASEInterleukinMiddle AgedImmunohistochemistrypathology of chronic obstructive pulmonary disease phenotypesAsthma phenotypesFemaleLife Sciences & BiomedicinePulmonary and Respiratory Medicinep38 mitogen-activated protein kinasesBlotting WesternINHIBITIONSocio-culturaleBronchiRespiratory MucosaOBSTRUCTIVE PULMONARY-DISEASE1102 Cardiovascular Medicine And HaematologyCell LinemedicineHumansLymphocyte CountInterleukin 8AgedAsthmaScience & Technologybusiness.industryInterleukin-8Transcription Factor RelAPATHWAYSMitogen-activated protein kinasemedicine.diseaseAsthmarespiratory tract diseasesSEVERITYCase-Control StudiesCELLSImmunologybusiness
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Bronchiolitis: The New American Academy of Pediatrics Guidelines

2008

Respiratory Syncytial Virus (RSV) is the leading cause of hospitalization for lower respiratory tract infections (LRTI) in young children worldwide. RSV is an important cause of LRTI in infants and a variety of factors, such as gender, age at admission, gestational age, birth weight, and exposure to tobacco smoke and breast feeding may affect the prevalence of RSV-related disease and, possibly, the risk of developing asthma-like symptoms during the school years. AAP in 2006 formulated the new guidelines on diagnosis and management of bronchiolitis, giving recommendations on different conditions requesting clinical decisions also in terms of prevention.

PalivizumabPediatricsmedicine.medical_specialtyBirth weightRespiratory Syncytial Virus InfectionsDiseaseAntibodies Monoclonal HumanizedAntiviral AgentsTobacco smokeRisk FactorsPrevalencemedicineBronchiolitis ViralHumansPharmacology (medical)PalivizumabPharmacologyEvidence-Based MedicineRespiratory tract infectionsbusiness.industryAntibodies MonoclonalGestational agemedicine.diseaserespiratory tract diseasesInfectious DiseasesOncologyBronchiolitisPractice Guidelines as TopicBronchiolitisbusinessBreast feedingmedicine.drugJournal of Chemotherapy
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Interstitial lung disease associated with drug therapy

2004

Drug-associated interstitial lung disease (ILD) is not uncommon, with diverse patterns ranging from benign infiltrates to the potentially fatal acute respiratory distress syndrome. As acute respiratory failure due to drug-associated ILD has an unpredictable onset and rapid time course, establishing a diagnosis is often difficult. An accurate diagnosis is based on clinical, radiological (including high-resolution computed tomography) and histological manifestations, although is often only possible by exclusion. Cancer chemotherapy is commonly associated with acute disease that, on pathology, is often diffuse alveolar damage. Furthermore, a combination of drugs with or without radiotherapy ca…

PaperCancer Researchmedicine.medical_specialtyPathologyAntimetabolites AntineoplasticLung Neoplasmsmedicine.medical_treatmentDiseaseNSCLCchemotherapybehavioral disciplines and activitiesPharmacotherapyGefitinibJapanRisk FactorsCarcinoma Non-Small-Cell LungEpidemiologyAntineoplastic Combined Chemotherapy ProtocolsmedicineHumansIntensive care medicineDiffuse alveolar damageLung cancerAgedinterstitial lung diseaseAged 80 and overRespiratory Distress Syndromegefitinib (‘Iressa’)business.industryInterstitial lung diseaseGefitinibrespiratory systemMiddle Agedmedicine.diseaserespiratory tract diseasesRadiation therapyOncologyQuinazolinesOral PresentationbusinessLung Diseases Interstitialmedicine.drugBritish Journal of Cancer
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Lung myofibroblasts are characterized by down-regulated cyclooxygenase-2 and its main metabolite, prostaglandin E2.

2013

Background: Prostaglandin E2 (PGE(2)), the main metabolite of cyclooxygenase (COX), is a well-known anti-fibrotic agent. Moreover, myofibroblasts expressing alpha-smooth muscle actin (alpha-SMA), fibroblast expansion and epithelial-mesenchymal transition (EMT) are critical to the pathogenesis of idiopathic pulmonary fibrosis (IPF). Our aim was to investigate the expression of COX-2 and PGE(2) in human lung myofibroblasts and establish whether fibroblast-myofibroblast transition (FMT) and EMT are associated with COX-2 and PGE(2) down-regulation. Methods: Fibroblasts obtained from IPF patients (n = 6) and patients undergoing spontaneous pneumothorax (control, n = 6) and alveolar epithelial ce…

PathologyPulmonologyMetaboliteImmunofluorescencelcsh:MedicineBiochemistrychemistry.chemical_compoundIdiopathic pulmonary fibrosisMolecular Cell BiologyPulmonary fibrosisProstaglandin E2Myofibroblastslcsh:ScienceLungCells CulturedFisiologia cel·lularMultidisciplinarybiologyFibrosi pulmonarrespiratory systemExtracellular Matrixmedicine.anatomical_structureCytokinesMedicinelipids (amino acids peptides and proteins)Immunohistochemical AnalysisMyofibroblastResearch ArticleSignal Transductionmedicine.drugmedicine.medical_specialtyEpithelial-Mesenchymal TransitionImmunologyInterstitial Lung DiseasesDinoprostonePulmonary fibrosisTransforming Growth Factor beta1ImmunofluorescènciaGrowth FactorsCell Line TumormedicineHumansEpithelial–mesenchymal transitionFibroblastBiologyCell Proliferationlcsh:RProteinsEpithelial Cellsmedicine.diseaseActinsIdiopathic Pulmonary Fibrosisrespiratory tract diseasesGene Expression RegulationchemistryCyclooxygenase 2Immune SystemCase-Control StudiesImmunologic Techniquesbiology.proteinCancer researchClinical Immunologylcsh:QCyclooxygenaseBiomarkersPLoS ONE
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IL-22 is produced by innate lymphoid cells and limits inflammation in allergic airway disease

2011

Interleukin (IL)-22 is an effector cytokine, which acts primarily on epithelial cells in the skin, gut, liver and lung. Both pro- and anti-inflammatory properties have been reported for IL-22 depending on the tissue and disease model. In a murine model of allergic airway inflammation, we found that IL-22 is predominantly produced by innate lymphoid cells in the inflamed lungs, rather than TH cells. To determine the impact of IL-22 on airway inflammation, we used allergen-sensitized IL-22-deficient mice and found that they suffer from significantly higher airway hyperreactivity upon airway challenge. IL-22-deficiency led to increased eosinophil infiltration lymphocyte invasion and production…

PathologyPulmonologymedicine.medical_treatmentT-LymphocytesIntracellular Spacelcsh:Medicine10263 Institute of Experimental ImmunologyInterleukin 22Mice0302 clinical medicineLymphocytesPhosphorylationlcsh:ScienceLung0303 health sciencesMultidisciplinaryInterleukin-13T CellsAllergy and HypersensitivityInnate lymphoid cellInterleukinrespiratory systemInnate ImmunityRecombinant Proteins3. Good healthCytokinemedicine.anatomical_structureInterleukin 13CytokinesMedicineTumor necrosis factor alphaBiological Markersmedicine.symptomResearch ArticleSTAT3 Transcription Factormedicine.medical_specialtyImmune CellsImmunologyAntigen-Presenting CellsImmunoglobulinsInflammation610 Medicine & health1100 General Agricultural and Biological SciencesBiology03 medical and health sciences1300 General Biochemistry Genetics and Molecular BiologymedicineRespiratory HypersensitivityAnimalsBiology030304 developmental biologyInflammation1000 MultidisciplinaryTumor Necrosis Factor-alphaInterleukinslcsh:RImmunityEpithelial CellsEosinophilAllergensAsthmaImmunity Innaterespiratory tract diseasesImmune SystemImmunology570 Life sciences; biologylcsh:QImmunizationBiomarkers030215 immunology
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Involvement of distal airways in a chronic model of experimental asthma.

2005

Summary Background Bronchial asthma is characterized by chronic airway inflammation and airway remodelling which occurs in both proximal and distal airways. These changes are associated with development of airway hyper-responsiveness and airflow limitation. Objective This study was aimed to analyse whether chronic inhalative allergen challenges in mice lead to morphological and physiological changes comparable with this phenotype. Methods For this purpose, BALB/c mice were systemically sensitized to ovalbumin (OVA) followed by aerosol allergen challenges on 2 consecutive days per week for 12 weeks. Results In chronically challenged mice, tissue inflammation in proximal as well as distal air…

Pathologymedicine.medical_specialtyAllergyOvalbuminImmunologyInflammationBronchiMiceTransforming Growth Factor betaAdministration InhalationmedicineImmunology and AllergyAnimalsRespiratory systemAsthmaMice Inbred BALB CMucous MembraneInhalationbusiness.industryRespiratory diseaserespiratory systemAllergensmedicine.diseaseAsthmarespiratory tract diseasesDisease Models Animalmedicine.anatomical_structureImmunologyChronic DiseaseDisease ProgressionCytokinesFemalemedicine.symptomBronchial HyperreactivityAirwaybusinessBronchoalveolar Lavage FluidRespiratory tractClinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology
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FGF-9 overexpression prevents pleural fibrosis induced by intra-pleural adenovirus injection in mice

2015

Introduction: Lung fibrosis is associated with the reactivation of molecular pathways involved in lung development. Mesothelial cells are involved in the fibrotic lung process but their exact role is debated. Fibroblast Growth Factor-9 (FGF-9) is expressed by epithelial cells and visceral pleura during embryonic lung development. Mice homozygous for a targeted disruption of FGF-9 exhibit lung hypoplasia with reduced mesenchyme and early postnatal death. The aim of this study was to evaluate the role of FGF-9 expression by mesothelial cells in the adult lung. We used adenovirus-mediated (FGF-9) gene transfer in mesothelial cells in vivo. Material and Methods: AdFGF9 or a control adenovirus (…

Pathologymedicine.medical_specialtyLungbiologybusiness.industryMesenchymeInflammationrespiratory systemFibroblast growth factorrespiratory tract diseasesCTGFFibronectinmedicine.anatomical_structuremedicinebiology.proteinmedicine.symptombusinessFibroblastMesothelial Cell1.5 Diffuse Parenchymal Lung Disease
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Visuelle Abschätzung der funktionellen Lungenanteile in der HRCT und 3He-MRT bei Patienten nach Einzel-Lungentransplantation: Vergleich mit der absol…

2005

Purpose: Visual assessment of the ventilation using HRCT and 3 He-MRI in patients after single lung transplantation (SLTX). Analysis of specific ventilation defects found with 3 He-MRI and morphological changes found with HRCT. Materials and Methods: We evaluated 8male patients (54 ′ 6 years) suffering from emphysema and six patients (3males and 3 females, 58 ′ 9.5 years) suffering from idiopathic pulmonary fibrosis (IPF) after SLTX. The morphological changes at HRCT were classified and localized. In 3 He-MRI (2D FLASH), 10 to 14 slices (slice thickness 10 mm, gap 5 mm) were acquired in coronal orientation to cover the whole lung. Ventilation defects were localized and characterized. The vi…

Pathologymedicine.medical_specialtyLungbusiness.industryDouble thresholdSlice thicknessrespiratory systemmedicine.diseaserespiratory tract diseasesIdiopathic pulmonary fibrosismedicine.anatomical_structureCoronal planeVisual assessmentBreathingMedicineRadiology Nuclear Medicine and imagingIn patientbusinessNuclear medicineRöFo - Fortschritte auf dem Gebiet der Röntgenstrahlen und der bildgebenden Verfahren
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IL-11 promotes pulmonary vascular remodeling and lung fibrosis through the activation of endothelial to mesenchymal transition

2020

Background: Pulmonary hypertension (PH) in idiopathic pulmonary fibrosis (IPF) portends a poor prognosis and currently there is no approved therapy for hypoxemic PH. IL-11 is a novel lung fibrosis mediator but its role on vascular function is unknown. Objective: To analyze the role of IL-11 on pulmonary artery remodeling and lung fibrosis in vitro and in vivo. Methods: IL-11 expression was evaluated in pulmonary arteries and lung sections of control subjects and patients with IPF, IPF+PH and idiopathic PH (PAH). Human pulmonary artery endothelial cells and smooth muscle cells were stimulated with IL-11. Endothelial to mesenchymal transition was evaluated measuring the increase of mesenchyma…

Pathologymedicine.medical_specialtyLungbusiness.industryMesenchymal stem cellrespiratory systemmedicine.diseasePulmonary hypertensionrespiratory tract diseasesIdiopathic pulmonary fibrosismedicine.anatomical_structureFibrosismedicine.arteryPulmonary fibrosisParenchymaPulmonary arteryMedicinebusinessIdiopathic interstitial pneumonias
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