Search results for "reverse transcriptase"

showing 10 items of 715 documents

Molecular analysis of the erythropoietin receptor system in patients with polycythaemia vera

1994

Summary Erythropoietin (EPO) is a potent regulator of the viability, proliferation and differentiation of erythroid progenitor cells. Its effect is mediated by binding to the erythropoietin receptor (EPO-R), a member of a new cytokine receptor family. Alterations of the EPO/EPO-R system have recently been shown to be involved in the pathogenesis of familial erythrocytosis and polycythaemia vera (PV). In order to define whether genetic changes in the EPO-R gene and its ligand play a role in the development of PV, the structure and expression levels of the EPO-R and EPO genes were examined in samples from bone marrow and/or peripheral blood mononuclear cells of 24 patients with PV. As expecte…

AdultMalePolycythaemiaTranscription GeneticMolecular Sequence DataBiologyPolymerase Chain ReactionPeripheral blood mononuclear cellPolycythemia veraBone Marrowhemic and lymphatic diseasesReceptors ErythropoietinmedicineHumansRNA MessengerErythropoietinPolycythemia VeraPolymorphism Single-Stranded ConformationalAgedBase SequenceHematologyMiddle Agedmedicine.diseaseMolecular biologyReverse transcriptaseErythropoietin receptormedicine.anatomical_structureErythropoietinFemaleBone marrowOligonucleotide ProbesCytokine receptormedicine.drugBritish Journal of Haematology
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Defects in the Human Leukocyte Antigen Class I Antigen Processing Machinery in Head and Neck Squamous Cell Carcinoma: Association with Clinical Outco…

2005

AbstractPurpose: Human leukocyte antigen (HLA) class I antigen defects, which are frequently present in head and neck squamous cell carcinoma (HNSCC) cells may provide the tumor with an escape mechanism from immune surveillance. Scanty information is available about mechanisms underlying HLA class I antigen defects in both lesions and cell lines from HNSCC. In this study, we investigate the role of antigen processing machinery (APM) component abnormalities in the generation of deficient HLA class I surface expression of HNSCC cells.Experimental Design: Using immunohistochemistry, Western blot, and RT-PCR analyses we correlated the expression of the IFN-γ inducible proteasome subunits and of…

AdultMaleProteasome Endopeptidase ComplexCancer ResearchPathologymedicine.medical_specialtyBlotting WesternDown-RegulationHuman leukocyte antigenBiologyCell LineInterferon-gammaATP Binding Cassette Transporter Subfamily B Member 3HLA AntigensMultienzyme ComplexesCell Line TumorTumor Cells Culturedotorhinolaryngologic diseasesmedicineCarcinomaHumansATP Binding Cassette Transporter Subfamily B Member 2AgedReverse Transcriptase Polymerase Chain ReactionAntigen processingHistocompatibility Antigens Class ICancerMiddle Agedmedicine.diseaseImmunohistochemistrySurvival AnalysisHead and neck squamous-cell carcinomaGene Expression Regulation NeoplasticCysteine Endopeptidasesstomatognathic diseasesOncologyHead and Neck NeoplasmsCarcinoma Squamous Cellbiology.proteinImmunohistochemistryTAP2ATP-Binding Cassette TransportersFemaleTAP1Clinical Cancer Research
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Variations in genes regulating neuronal migration predict reduced prefrontal cognition in schizophrenia and bipolar subjects from mediterranean Spain…

2005

Both neural development and prefrontal cortex function are known to be abnormal in schizophrenia and bipolar disorder. In order to test the hypothesis that these features may be related with genes that regulate neuronal migration, we analyzed two genomic regions: the lissencephaly critical region (chromosome 17p) encompassing the LIS1 gene and which is involved in human lissencephaly; and the genes related to the platelet-activating-factor, functionally related to LIS1, in 52 schizophrenic patients, 36 bipolar I patients and 65 normal control subjects. In addition, all patients and the 25 control subjects completed a neuropsychological battery. Thirteen (14.8%) patients showed genetic varia…

AdultMalePsychosisBipolar DisorderAdolescentLissencephalyNeuropsychological TestsCognitionCell MovementPredictive Value of TestsmedicineHumansBipolar disorderPlatelet Activating FactorPrefrontal cortexMolecular BiologyNeuronsAnalysis of VarianceReverse Transcriptase Polymerase Chain ReactionGeneral NeuroscienceMiddle Agedmedicine.diseaseLogistic ModelsSpainSchizophreniaEndophenotype1-Alkyl-2-acetylglycerophosphocholine EsteraseSchizophreniaFemaleAnalysis of variancePsychologyMicrotubule-Associated ProteinsNeuroscienceNeural developmentChromosomes Human Pair 17Neuroscience
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Efficacy and safety of rituximab treatment in early primary Sjögren's syndrome: a prospective, multi-center, follow-up study.

2013

Introduction Primary Sjögren’s syndrome (pSS) is an autoimmune disorder affecting exocrine glands; however, a subgroup of pSS patients experience systemic extra-glandular involvement leading to a worsening of disease prognosis. Current therapeutic options are mainly empiric and often translated by other autoimmune diseases. In the last few years growing evidence suggests that B-cell depletion by rituximab (RTX) is effective also in pSS. Patients with early active disease appear to be those who could benefit the most from RTX. The aim of this study was to investigate the efficacy and safety of RTX in comparison to disease modifying anti-rheumatic drugs (DMARDs) in early active pSS patients. …

AdultMaleReceptors CXCR5musculoskeletal diseasesReceptors CXCR4Salivamedicine.medical_specialtySjogren's syndrome RituximabTime FactorsBiopsyImmunologyGene ExpressionDiseaseSalivary GlandsAntibodies Monoclonal Murine-Derivedstomatognathic systemRheumatologyInternal medicineBiopsyHumansImmunology and AllergyMedicineProspective StudiesProspective cohort studyAdverse effectFatiguePain Measurementmedicine.diagnostic_testSalivary glandReverse Transcriptase Polymerase Chain Reactionbusiness.industryMiddle AgedChemokine CXCL13Chemokine CXCL12Rheumatologystomatognathic diseasesTreatment Outcomemedicine.anatomical_structureSjogren's syndromeAntirheumatic AgentsImmunologyFemaleRituximabSelf ReportRituximabbusinessFollow-Up StudiesResearch Articlemedicine.drug
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Detection of human immunodeficiency virus-1 DNA, RNA and antibody, and occult blood in inactivated saliva: Availability of the filter paper disk meth…

1996

Eighty three samples of saliva were collected from 60 subjects who were asymptomatic carriers of AIDS related complex (ARC) and AIDS. They included hemophiliacs, homosexuals and heterosexuals. Occult blood, human immunodeficiency virus-1 (HIV-1) antibody and DNA were assayed after heat treatment at 56 degrees C by strip method, particle agglutination and polymerase chain reaction (PCR), respectively. HIV-1 RNA was assayed by reverse transcription (RT)-PCR after heat treatment at 56 degrees C or 90 degrees C, or after application to filter paper disks with drying and heat treatment at 90 degrees C. Positive results were found in 53% (occult blood), 73% (HIV-1 antibody), 23% (HIV-1 DNA, 56 de…

AdultMaleSalivaAIDS-related complexHIV InfectionsBiologyPolymerase Chain Reactionlaw.inventionchemistry.chemical_compoundAIDS-Related ComplexRisk FactorslawHIV SeropositivitymedicineHumansChildSalivaPolymerase chain reactionAgedAcquired Immunodeficiency SyndromeAIDS SerodiagnosisInfantvirus diseasesRNAmedicine.diseaseVirologyMolecular biologyReverse transcriptaseReverse transcription polymerase chain reactionchemistryOccult BloodDNA ViralPediatrics Perinatology and Child HealthHIV-1biology.proteinRNA ViralFemaleAntibodyDNAPediatrics International
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Variability of reverse transcriptase and overlapping S gene in hepatitis B virus isolates from untreated and lamivudine-resistant chronic hepatitis B…

2009

Background The high degree of diversity of the hepatitis B virus (HBV) quasispecies in chronically infected individuals raises the possibility that HBV genetic variants favouring resistance to nucleoside/nucleotide analogues (NAs) might pre-exist to treatment. The aim of this study was to investigate the genetic variability of the entire HBV reverse transcriptase (RT) domain and of the overlapping S gene in a large series of untreated hepatitis B surface antigen carriers and in lamivudine (3TC)-resistant patients. Methods Sequencing analysis of the entire HBV RT domain of isolates from 100 untreated (treatment- naive group) and 59 3TC-resistant (3TC-resistant group) consecutive patients wit…

AdultMaleSettore MED/07 - Microbiologia E Microbiologia ClinicaHepatitis B virusAdult; Aged; Drug Resistance; Viral; Female; Genetic Variation; Hepatitis B Surface Antigens; Hepatitis B virus; Hepatitis B; Chronic; Humans; Lamivudine; Male; Middle Aged; Mutation; RNA-Directed DNA Polymerase; Reverse Transcriptase Inhibitors; Sequence Analysis; DNA; Treatment OutcomeDrug ResistanceViral quasispeciesmedicine.disease_causeVirusHepatitis B ChronicOrthohepadnavirusDrug Resistance ViralmedicineHumansPharmacology (medical)ViralChronicAgedPharmacologyHepatitis B virusSettore MED/12 - GastroenterologiaHepatitis B Surface AntigensbiologyReverse-transcriptase inhibitorLamivudineGenetic VariationRNA-Directed DNA PolymeraseSequence Analysis DNADNAMiddle Agedbiology.organism_classificationHepatitis BVirologyReverse transcriptaseInfectious DiseasesTreatment OutcomeHepadnaviridaeLamivudineMutationReverse Transcriptase InhibitorsHBV reverse transcriptase gene S lamivudine resistantFemaleSequence Analysismedicine.drug
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Naturally Occurring Telomerase-Specific CD4 T-Cell Immunity in Melanoma.

2020

CD4 T cells play a key role in anticancer immunity. In this study, we investigate the clinical relevance of circulating CD4 T helper type 1 (Th1) response against telomerase (anti-TERT Th1 response) in patients with melanoma. The spontaneous anti-TERT Th1 response was detected in 54.5% (85/156) of patients with melanoma before treatment. The prevalence of this systemic response was inversely related to Breslow thickness >1 mm and American Joint Committee on Cancer stage ≥II (P = 0.001 and 0.032, respectively). In contrast to patients treated with targeted therapies, the anti-TERT Th1 immunity was associated with an objective response after immune checkpoint inhibitors treatment. Hence, 86% …

AdultMaleTelomeraseSkin Neoplasmsmedicine.medical_treatmentDermatologyBiochemistryBreslow ThicknessImmunitymedicineHumansTelomerase reverse transcriptaseProgression-free survivalProspective StudiesMolecular BiologyImmune Checkpoint InhibitorsMelanomaTelomeraseAgedNeoplasm Stagingbusiness.industryMelanomaCell BiologyImmunotherapyMiddle AgedTh1 Cellsmedicine.diseaseProgression-Free SurvivalDrug Resistance NeoplasmCancer researchBiomarker (medicine)FemalebusinessFollow-Up StudiesThe Journal of investigative dermatology
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COMT genotype predicts BOLD signal and noise characteristics in prefrontal circuits.

2006

Abstract Objective: Prefrontal dopamine (DA) is catabolized by the COMT (catechol- O -methyltransferase) enzyme. Literature suggests that the Val/Met single nucleotide polymorphism (SNP) in the COMT gene predicts executive cognition in humans with Val carriers showing poorer performance due to less available synaptic DA. Recent fMRI studies are thought to agree with these studies having demonstrated prefrontal hyperactivation during n -back and attention-requiring tasks. This was interpreted as “less efficient” processing due to impaired signal-to-noise ratio (SNR) of neuronal activity. However, electrophysiological studies of neuronal SNR in primates and humans imply that prefrontal cortex…

AdultMaleVisual perceptiongenetic structuresGenotypeCognitive NeurosciencePrefrontal CortexCatechol O-Methyltransferasebehavioral disciplines and activitiesDopaminemedicineImage Processing Computer-AssistedPremovement neuronal activityHumansPrefrontal cortexOddball paradigmReverse Transcriptase Polymerase Chain ReactionSMA*Magnetic Resonance ImagingDorsolateral prefrontal cortexOxygenElectrophysiologymedicine.anatomical_structurenervous systemNeurologyRegression AnalysisFemalePsychologyNeurosciencePhotic StimulationPsychomotor Performancemedicine.drugNeuroImage
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Differential regulation of Fas-mediated apoptosis in both thyrocyte and lymphocyte cellular compartments correlates with opposite phenotypic manifest…

2001

Several mechanisms are probably involved in determining the evolution of autoimmune thyroid disease (AITD) towards either hypothyroidism and the clinical syndrome known as Hashimoto's thyroiditis (HT) or toward hyperthyroidism and the symptoms of Graves' disease (GD). To gain further insight into such mechanisms we performed an exhaustive comparative analysis of the expression of key molecules regulating cell death (Fas, Fas ligand [FasL], Bcl-2) and apoptosis in both thyrocytes and thyroid infiltrating lymphocytes (TILs) from patients with either GD or HT. GD thyrocytes expressed less Fas/FasL than HT thyrocytes, whereas GD TILs had higher levels of Fas/FasL than HT TILs. GD thyrocytes exp…

AdultMaleendocrine systemmedicine.medical_specialtyProgrammed cell deathFas Ligand Proteinendocrine system diseasesEndocrinology Diabetes and MetabolismLymphocyteThyroid Glandchemical and pharmacologic phenomenaApoptosisThyroiditisFas ligandAutoimmune DiseasesEndocrinologyInternal medicinemedicineHumansLymphocytesRNA Messengerfas ReceptorCellular compartmentAgedMembrane GlycoproteinsChemistryReverse Transcriptase Polymerase Chain ReactionThyroidThyroiditis Autoimmunehemic and immune systemsMiddle Agedmedicine.diseasePhenotypeThyroid DiseasesGraves DiseaseEndocrinologymedicine.anatomical_structurePhenotypeGene Expression RegulationProto-Oncogene Proteins c-bcl-2ApoptosisFemaleThyroid : official journal of the American Thyroid Association
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Treatment-associated polymorphisms in protease are significantly associated with higher viral load and lower CD4 count in newly diagnosed drug-naive …

2012

Background: The effect of drug resistance transmission on disease progression in the newly infected patient is not well understood. Major drug resistance mutations severely impair viral fitness in a drug free environment, and therefore expected to revert quickly. Compensatory mutations, often already polymorphic in wild-type viruses, do not tend to revert after transmission. While compensatory mutations increase fitness during treatment, their presence may also modulate viral fitness and virulence in absence of therapy and major resistance mutations. We previously designed a modeling technique that quantifies genotypic footprints of in vivo treatment selective pressure, including both drug …

AdultMalelcsh:Immunologic diseases. AllergyAnti-HIV AgentseducationVirulenceHIV InfectionsDrug resistanceBiologySettore MED/42 - Igiene Generale E ApplicataViruspolymorphism03 medical and health sciencesViral ProteinsSDG 3 - Good Health and Well-beingVirologyGenotypeDrug Resistance Viraldrug-naivemedicineHumansProspective Studies030304 developmental biology0303 health sciencesPolymorphism Genetic030306 microbiologyResearchproteaseViral LoadVirologyReverse transcriptase3. Good healthCD4 Lymphocyte CountDrug-naïveInfectious Diseases3121 General medicine internal medicine and other clinical medicineImmunologybiology.proteinHIV-1FemaleAntibodylcsh:RC581-607Viral loadHIV-1 infected patientmedicine.drugPeptide HydrolasesRetrovirology
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