Search results for "reverse transcriptase"

showing 10 items of 715 documents

Mitochondria from females exhibit higher antioxidant gene expression and lower oxidative damage than males

2003

We have investigated the differential mitochondrial oxidative stress between males and females to understand the molecular mechanisms enabling females to live longer than males. Mitochondria are a major source of free radicals in cells. Those from female rats generate half the amount of peroxides than those of males. This does not occur in ovariectomized animals. Estrogen replacement therapy prevents the effect of ovariectomy. Mitochondria from females have higher levels of reduced glutathione than those from males. Those from ovariectomized rats have similar levels to males, and estrogen therapy prevents the fall in glutathione levels that occurs in ovariectomized animals. Oxidative damage…

Malemedicine.medical_specialtyAntioxidantOvariectomymedicine.medical_treatmentMitochondria LiverMitochondrionBiologymedicine.disease_causeDNA MitochondrialBiochemistryAntioxidantsGene Expression Regulation EnzymologicSuperoxide dismutasechemistry.chemical_compoundRNA Ribosomal 16SPhysiology (medical)Internal medicinemedicineAnimalsRats WistarDNA Primerschemistry.chemical_classificationGlutathione PeroxidaseReverse Transcriptase Polymerase Chain ReactionSuperoxide DismutaseGlutathione peroxidaseEstrogensGlutathioneGlutathionePeroxidesRatsOxygenOxidative StressEndocrinologychemistryOvariectomized ratbiology.proteinRNAFemaleDismutaseReactive Oxygen SpeciesOxidation-ReductionOxidative stressFree Radical Biology and Medicine
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Upregulation of Phospholipase D Expression and Activation in Ventricular Pressure-Overload Hypertrophy

2005

Evidence for a role of phospholipase D (PLD) in cellular proliferation and differentiation is accumulating. We studied PLD activity and expression in normal and hypertrophic rat and human hearts. In rat heart, abdominal aortic banding (constriction to 50% of original lumen) caused hypertrophy in the left ventricle (as shown by weight index and ANP expression) by about 15% after 30 days without histological evidence of fibrosis or signs of decompensation and in the right ventricle after 100 days. The hypertrophy was accompanied by small increases of basal PLD activity and strong potentiation of stimulated PLD activity caused by 4β-phorbol-12β,13α-dibutyrate (PDB) and by phenylephrine. The mR…

Malemedicine.medical_specialtyBlotting WesternCardiomegalyBiologyGene Expression Regulation EnzymologicMuscle hypertrophyRats Sprague-DawleyDownregulation and upregulationInternal medicinePhospholipase DVentricular PressuremedicineAnimalsHumansRNA MessengerPhenylephrineProtein Kinase CProtein kinase CPharmacologyReverse Transcriptase Polymerase Chain ReactionPhospholipase DPLD2lcsh:RM1-950Body WeightRatsReceptors AdrenergicUp-RegulationEnzyme ActivationIsoenzymeslcsh:Therapeutics. Pharmacologymedicine.anatomical_structureEndocrinologyVentricleVentricular pressureMolecular Medicinelipids (amino acids peptides and proteins)Signal Transductionmedicine.drugJournal of Pharmacological Sciences
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BCL-2 UPREGULATION AFTER 3-NITROPROPIONIC ACID PRECONDITIONING IN WARM RAT LIVER ISCHEMIA

2008

We aimed to determine whether 3-nitropropionic acid (3-NPA) preconditioning protects rat livers against warm ischemia/reperfusion injury. We hypothesized that 3-NPA mediates its protective effects by Bcl-2 upregulation. Brown-Norway rats (200 g) were injected with 3-NPA (10 mg/kg intraperitoneally) 24 h before 90 min of selective warm in situ ischemia. In additional experiments, 30-day survival was studied after 90 min of warm liver ischemia and resection of nonischemic liver tissue. We demonstrate increased mRNA and protein levels of Bcl-2 by real-time polymerase chain reaction, immunohistochemistry, and Western blot analysis in 3-NPA-pretreated rats. All treated animals survived, whereas …

Malemedicine.medical_specialtyBlotting WesternIschemiaCritical Care and Intensive Care MedicineLipid peroxidationchemistry.chemical_compoundstomatognathic systemWestern blotDownregulation and upregulationInternal medicinemedicineAnimalsWarm IschemiaIschemic PreconditioningCaspasechemistry.chemical_classificationReactive oxygen speciesmedicine.diagnostic_testbiologyCaspase 3Reverse Transcriptase Polymerase Chain ReactionNitro Compoundsmedicine.diseaseImmunohistochemistryCaspase 9RatsEndocrinologyLiverProto-Oncogene Proteins c-bcl-2chemistryApoptosisReperfusion InjuryEmergency Medicinebiology.proteinPropionatesReperfusion injuryShock
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Effects of levosimendan on hemodynamics, local cerebral blood flow, neuronal injury, and neuroinflammation after asphyctic cardiac arrest in rats.

2014

Despite advances in cardiac arrest treatment, high mortality and morbidity rates after successful cardiopulmonary resuscitation are still a major clinical relevant problem. The post cardiac arrest syndrome subsumes myocardial dysfunction, impaired microcirculation, systemic inflammatory response, and neurological impairment. The calcium-sensitizer levosimendan was able to improve myocardial function and initial resuscitation success after experimental cardiac arrest/cardiopulmonary resuscitation. We hypothesized that levosimendan exerts beneficial effects on cerebral blood flow, neuronal injury, neurological outcome, and inflammation 24 hours after experimental cardiac arrest/cardiopulmonar…

Malemedicine.medical_specialtyCardiotonic AgentsHemodynamicsGene ExpressionEnzyme-Linked Immunosorbent AssayCritical Care and Intensive Care MedicineHippocampusRats Sprague-DawleyInternal medicineMedicineAnimalsNeuroinflammationSimendanCerebral CortexNeuronsAnalysis of Variancebusiness.industryInterleukin-6Reverse Transcriptase Polymerase Chain ReactionHemodynamicsHydrazonesLevosimendanCardiopulmonary ResuscitationHeart ArrestRatsSprague dawleyPyridazinesCerebral blood flowAnesthesiaCerebrovascular CirculationCardiologybusinessmedicine.drugCritical care medicine
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Angiotensin II contractile effects in mouse colon: role for pre- and post-junctional AT1A receptors

2013

Aim This study investigates whether a local renin–angiotensin system (RAS) exists in mouse colon and whether angiotensin II (Ang II) may play a role in the regulation of the contractile activity. Methods Isometric recordings were performed in vitro on the longitudinal muscle of mouse proximal and distal colon. Transcripts encoding for RAS components were investigated by RT-PCR. Results Ang II caused, in both preparations, a concentration-dependent contractile effect, antagonized by losartan, AT1 receptor antagonist, but not by PD123319, AT2 receptor antagonist. The combination of losartan plus PD123319 caused no change on the Ang II-induced contraction than losartan alone. Tetrodotoxin, neu…

Malemedicine.medical_specialtyColonPhysiologymedicine.drug_classMuscarinic AntagonistsBiologyReceptor Angiotensin Type 1Renin-Angiotensin SystemMicechemistry.chemical_compoundOrgan Culture TechniquesInternal medicineMuscarinic acetylcholine receptormedicineAnimalsReceptorAngiotensin II receptor type 1Reverse Transcriptase Polymerase Chain ReactionAngiotensin IIAntagonistMuscle Smoothangiotensin II AT1 receptors AT2 receptors enteric neurones mouse colon muscle contraction.Receptor antagonistAngiotensin IIElectrophysiologyMice Inbred C57BLEndocrinologyLosartanchemistryHexamethoniumhormones hormone substitutes and hormone antagonistsMuscle Contractionmedicine.drug
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An experimental comparative study of dexamethasone, melatonin and tacrolimus in noise-induced hearing loss.

2008

The calcineurin inhibitor tacrolimus (TCR) and the pineal gland hormone and antioxidant melatonin (MLT) have been shown to possess otoprotective properties against noise-induced hearing loss (NIHL). In contrast, dexamethasone (DXM) was not effective as an otoprotective agent against NIHL. Further studies are needed to understand the exact molecular mechanisms involved.Exposure to noise pollution and use of audio devices for long periods of time at high volume is known to cause hearing loss or NIHL. Our goal was to evaluate the effectiveness of various known compounds such as the anti-inflammatory DXM, the antioxidant MLT and the immunosuppressant TCR against NIHL.Thirty-two Wistar rats were…

Malemedicine.medical_specialtyHearing lossOtoacoustic Emissions SpontaneousAnti-Inflammatory AgentsAntioxidantsDexamethasoneTacrolimusMelatoninPineal glandInternal medicineotorhinolaryngologic diseasesmedicineEvoked Potentials Auditory Brain StemAnimalsRats WistarDexamethasoneMelatoninbusiness.industryReverse Transcriptase Polymerase Chain ReactionTumor Necrosis Factor-alphaGeneral Medicinemedicine.diseaseTacrolimusRatsCalcineurinHair Cells Auditory OuterEndocrinologymedicine.anatomical_structureOtorhinolaryngologyHearing Loss Noise-Inducedmedicine.symptombusinessProto-Oncogene Proteins c-fosNoise-induced hearing lossImmunosuppressive AgentsHormonemedicine.drugActa oto-laryngologica
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Hepatic steatosis is not due to impaired fatty acid oxidation capacities in C57BL/6J mice fed the conjugated trans-10,cis-12-isomer of linoleic acid.

2004

Decreased body fat mass and liver steatosis have been reported in mice fed diets containing the conjugated linoleic acid trans-10,cis-12-C18:2 (CLA2), but not in those fed diets containing cis-9,trans-11-C18:2 (CLA1). Because the decrease in fatty acid (FA) oxidation may cause fat accumulation, we questioned whether the effects of both CLAs on enzyme activities and mRNA expression were related to liver FA oxidation. To address this question, 7-wk-old male C57BL/6J mice were fed for 4 wk a diet supplemented with 1% CLA1, CLA2, or cis-9-C18:1 (control) esterified as triacylglycerols. In CLA2-fed mice, the proportions of CLA2 in the total FA of liver lipids were substantially lower than those …

Malemedicine.medical_specialtyLinoleic acidConjugated linoleic acidMedicine (miscellaneous)Mitochondria LiverBiologychemistry.chemical_compoundMiceDietary Fats UnsaturatedInternal medicinemedicineAnimalsLinoleic Acids ConjugatedCarnitineRNA MessengerEnzyme InhibitorsUnsaturated fatty acidTriglycerideschemistry.chemical_classificationNutrition and DieteticsCarnitine O-PalmitoyltransferaseEsterificationReverse Transcriptase Polymerase Chain ReactionFatty liverFatty AcidsFatty acidmedicine.diseaseFatty LiverMalonyl Coenzyme AMice Inbred C57BLEndocrinologychemistryBiochemistryLiverCarnitine palmitoyltransferase IOxidation-ReductionPolyunsaturated fatty acidmedicine.drug
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Bioavailability of nevirapine in rats after oral and subcutaneous administration, in vivo absorption from gastrointestinal segments and effect of bil…

2011

Abstract Nevirapine is a non-nucleoside reverse transcriptase inhibitor of human immunodeficiency virus type-1. The usual dosing regimen is 200 mg twice/day. Reducing the dosing frequency would significantly improve treatment adherence and quality of life of patients. To study new forms of administration, it is necessary to do pre-clinical studies and know the absorption characteristics of nevirapine in laboratory animals. However, there are no studies about its bioavailability in rats and hardly any about its pharmacokinetic. The objectives of this study were to describe the pharmacokinetics of nevirapine in rats after intravenous, oral and subcutaneous administration, to assess its absorp…

Malemedicine.medical_specialtyNevirapineDuodenumInjections SubcutaneousPharmaceutical ScienceAdministration OralBiological AvailabilityIleumAbsorption (skin)PharmacologyGastroenterologyIntestinal absorptionPharmacokineticsSpecies SpecificityOral administrationInternal medicinemedicineAnimalsBileHumansNevirapineRats Wistarbusiness.industrydigestive oral and skin physiologyBioavailabilityRatsGastrointestinal Tractmedicine.anatomical_structureIntestinal AbsorptionInjections IntravenousDuodenumReverse Transcriptase Inhibitorsbusinessmedicine.drugInternational journal of pharmaceutics
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Cannabinoid type 1 receptor blockade promotes mitochondrial biogenesis through endothelial nitric oxide synthase expression in white adipocytes

2008

OBJECTIVE—Cannabinoid type 1 (CB1) receptor blockade decreases body weight and adiposity in obese subjects; however, the underlying mechanism is not yet fully understood. Nitric oxide (NO) produced by endothelial NO synthase (eNOS) induces mitochondrial biogenesis and function in adipocytes. This study was undertaken to test whether CB1 receptor blockade increases the espression of eNOS and mitochondrial biogenesis in white adipocytes. RESEARCH DESIGN AND METHODS—We examined the effects on eNOS and mitochondrial biogenesis of selective pharmacological blockade of CB1 receptors by SR141716 (rimonabant) in mouse primary white adipocytes. We also examined eNOS expression and mitochondrial biog…

Malemedicine.medical_specialtyNitric Oxide Synthase Type IIIEndocrinology Diabetes and MetabolismAdipocytes WhiteImmunoblottingCitrate (si)-SynthaseWhite adipose tissueAMP-Activated Protein KinasesProtein Serine-Threonine KinasesMitochondrionDNA MitochondrialMicechemistry.chemical_compoundAdenosine TriphosphatePiperidinesReceptor Cannabinoid CB1AMP-activated protein kinaseMultienzyme ComplexesEnosAdipocyteInternal medicineInternal MedicinemedicineAnimalsPhosphorylationRNA Small InterferingReceptorCells CulturedDose-Response Relationship DrugbiologyReverse Transcriptase Polymerase Chain ReactionFlow Cytometrybiology.organism_classificationMitochondriaMice Inbred C57BLNitric oxide synthaseMetabolismEndocrinologychemistryMitochondrial biogenesisbiology.proteinSettore BIO/14 - FarmacologiaPyrazolesRimonabant
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Resveratrol Reverses Endothelial Nitric-Oxide Synthase Uncoupling in Apolipoprotein E Knockout Mice

2010

A crucial cause of the decreased bioactivity of nitric oxide (NO) in cardiovascular diseases is the uncoupling of the endothelial NO synthase (eNOS) caused by the oxidative stress-mediated deficiency of the NOS cofactor tetrahydrobiopterin (BH(4)). The reversal of eNOS uncoupling might represent a novel therapeutic approach. The treatment of apolipoprotein E knockout (ApoE-KO) mice with resveratrol resulted in the up-regulation of superoxide dismutase (SOD) isoforms (SOD1-SOD3), glutathione peroxidase 1 (GPx1), and catalase and the down-regulation of NADPH oxidases NOX2 and NOX4 in the hearts of ApoE-KO mice. This was associated with reductions in superoxide, 3-nitrotyrosine, and malondiald…

Malemedicine.medical_specialtyNitric Oxide Synthase Type IIISOD3SOD2ResveratrolAntioxidantsSuperoxide dismutaseMicechemistry.chemical_compoundApolipoproteins ESuperoxidesEnosMalondialdehydeInternal medicineStilbenesmedicineAnimalsGTP CyclohydrolaseMice KnockoutPharmacologychemistry.chemical_classificationReactive oxygen speciesbiologyReverse Transcriptase Polymerase Chain ReactionSuperoxide DismutaseChemistrySuperoxideMyocardiumTetrahydrobiopterinbiology.organism_classificationBiopterinIsoenzymesOxidative StressEndocrinologyBiochemistryResveratrolbiology.proteinRNATyrosineMolecular Medicinemedicine.drugJournal of Pharmacology and Experimental Therapeutics
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