Search results for "script"

showing 10 items of 5143 documents

The complex interplay between Notch signaling and Snail1 transcription factor in the regulation of epithelial–mesenchymal transition (EMT)

2015

Background The epithelial–mesenchymal transition (EMT) is a highly coordinated process observed during embryonic development and adult tissue repair. It is characterized by the loss of cell–cell adhesion and apicobasal polarity, and the transition to a cell type with a spindle-like phenotype able to migrate through the basal membranes. Methods This review article includes available date from peer-reviewed publications associated with the role of Notch signaling and Snail1 transcription factor in activation and regulation of EMT. Results Growing evidences in the past few years demonstrated a significant role of Notch in EMT activation. It is not surprising because this pathway is the nexus o…

Cell typeNotchSnail1business.industryEMTNotch signaling pathwayAnatomyPhenotypeCell biologyTGFβDownregulation and upregulationCompartment (development)Mesenchymal–epithelial transitionMedicineSurgeryEpithelial–mesenchymal transitionHypoxiabusinessTranscription factorCancerEuropean Surgery
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Lineage-reprogramming of Pericyte-derived Cells of the Adult Human Brain into Induced Neurons

2014

Direct lineage-reprogramming of non-neuronal cells into induced neurons (iNs) may provide insights into the molecular mechanisms underlying neurogenesis and enable new strategies for in vitro modeling or repairing the diseased brain. Identifying brain-resident non-neuronal cell types amenable to direct conversion into iNs might allow for launching such an approach in situ, i.e. within the damaged brain tissue. Here we describe a protocol developed in the attempt of identifying cells derived from the adult human brain that fulfill this premise. This protocol involves: (1) the culturing of human cells from the cerebral cortex obtained from adult human brain biopsies; (2) the in vitro expansio…

Cell typePatch-Clamp TechniquesGeneral Chemical EngineeringCell Culture TechniquesBiologyGeneral Biochemistry Genetics and Molecular BiologySOX2Transduction GeneticmedicineHumansCell LineageCerebral CortexNeuronsGeneral Immunology and MicrobiologyGeneral NeuroscienceSOXB1 Transcription FactorsNeurogenesisHuman brainCell sortingCellular ReprogrammingFlow CytometryImmunohistochemistrymedicine.anatomical_structureRetroviridaeCell culturePericytePericytesNeuroscienceReprogrammingNeuroscience
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Timing of identity: spatiotemporal regulation of hunchback in neuroblast lineages of Drosophila by Seven-up and Prospero.

2006

Neural stem cells often generate different cell types in a fixed birth order as a result of temporal specification of the progenitors. In Drosophila, the first temporal identity of most neural stem cells(neuroblasts) in the embryonic ventral nerve cord is specified by the transient expression of the transcription factor Hunchback. When reaching the next temporal identity, this expression is switched off in the neuroblasts by seven up (svp) in a mitosis-dependent manner, but is maintained in their progeny (ganglion mother cells). We show that svpmRNA is already expressed in the neuroblasts before this division. After mitosis, Svp protein accumulates in both cells, but the downregulation of h…

Cell typeReceptors Steroidanimal structuresTranscription GeneticMitosisNerve Tissue ProteinsNeuroblastAnimalsDrosophila ProteinsCell LineageProgenitor cellMolecular BiologyMitosisGeneticsNeuronsbiologyStem CellsfungiGene Expression Regulation DevelopmentalNuclear ProteinsProsperobiology.organism_classificationEmbryonic stem cellNeural stem cellCell biologyDNA-Binding ProteinsDrosophila melanogasterGanglion mother cellDevelopmental BiologyTranscription FactorsDevelopment (Cambridge, England)
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Multiple signal transduction pathways regulate clusterin (gp 80) gene expression in MDCK cells

1996

ABSTRACT Clusterin (gp 80, apolipoprotein J, TRPM-2) is a widely expressed multifunctional glycoprotein. Its demonstrated and proposed functions include the transport of lipids and membrane fragments, the inhibition of the cytolytic action of the terminal complement complex and the modulation of cell—cell interactions. The expression of the gene is enhanced during tissue injury and remodelling and by hormone-withdrawal-induced apoptosis of prostate and mammary cells. We show here that, in the kidney-derived epithelial cell line MDCK, clusterin mRNA is repressed by glucocorticoids and by progesterone. Treatment with epidermal growth factor also represses clusterin gene expression in MDCK cel…

Cell typeTranscription GeneticKidneyDexamethasoneEpitheliumCell LineAlkaloidsDogsEndocrinologyEpidermal growth factor1-Methyl-3-isobutylxanthineGene expressionCyclic AMPAnimalsRNA MessengerEnzyme InhibitorsAldosteroneMolecular BiologyProgesteroneProtein Kinase CProtein kinase CGlycoproteinsBenzophenanthridinesMessenger RNAEpidermal Growth FactorClusterinbiologyChemistryMolecular biologyeye diseasesPhenanthridinesCell biologyKineticsClusterinCell culturebiology.proteinTetradecanoylphorbol Acetatesense organsSignal transductionMolecular ChaperonesSignal TransductionJournal of Molecular Endocrinology
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Differential expression levels of Sox9 in early neocortical radial glial cells regulate the decision between stem cell maintenance and differentiation

2021

ABSTRACTRadial glial progenitor cells (RGCs) in the dorsal forebrain directly or indirectly produce excitatory projection neurons and macroglia of the neocortex. Recent evidence shows that the pool of RGCs is more heterogeneous than originally thought and that progenitor subpopulations can generate particular neuronal cell types. Using single cell RNA sequencing, we have studied gene expression patterns of two subtypes of RGCs that differ in their neurogenic behavior. One progenitor type rapidly produces postmitotic neurons, whereas the second progenitor remains relatively quiescence before generating neurons. We have identified candidate genes that are differentially expressed between thes…

Cell typeTranscription GeneticNeurogenesisEpendymoglial CellsGenetic VectorsNeocortexNerve Tissue ProteinsBiologyMiceradial glia cellsprogenitors diversityGenes ReporterPregnancyGene expressionmedicineAnimalscortical developmentProgenitors diversityCell Self RenewalProgenitor cellPromoter Regions GeneticTranscription factorResearch ArticlesInjections IntraventricularProgenitorNeuronsNeocortexCortical developmentGeneral NeuroscienceCell CycleGene Expression Regulation DevelopmentalSOX9 Transcription FactorEmbryonic stem cellCell biologyMice Inbred C57BLCorticogenesisElectroporationmedicine.anatomical_structureCerebral cortexForebrainFemalesense organsSingle-Cell AnalysisStem cellNeuroscienceNeurogliaRadial glia cellsCellular/MolecularSox9
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Two mutations in gB-1 and gD-1 of herpes simplex virus type 1 are involved in the "fusion from without" phenotype in different cell types.

1996

Previous studies have shown that certain strains of herpes simplex viruses type 1 (HSV-1) are able to induce “fusion from without” (FFWO) which means no transcription or translation of the viral genome happens. The main determinants for FFWO in BHK cells are mutations in the C-terminal part of gB-1. But single mutations in this part of the genome are not sufficient to transfer the FFWO phenotype also to Vero cells. Here, we report that FFWO of HSV strains indeed need additional mutations in the N-terminal part of gD in order to produce the FFWO phenotype in BHK and Vero cells. By marker transfer we are able to show that loss of mutations in the N-terminal part of gD influences the ability t…

Cell typevirusesCellMolecular Sequence DataGenome ViralHerpesvirus 1 HumanBiologymedicine.disease_causeTransfectionGiant CellsVirusCell LineViral Envelope ProteinsTranscription (biology)VirologyCricetinaeChlorocebus aethiopsGeneticsmedicineBaby hamster kidney cellAnimalsHumansAmino Acid SequenceMolecular BiologyVero CellsBase SequenceGeneral MedicineVirologyPhenotypemedicine.anatomical_structureHerpes simplex virusPhenotypeDNA ViralMutationVero cellVirus genes
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Saccharomyces cerevisiae Rds2 transcription factor involvement in cell wall composition and architecture

2008

Although the cell wall is very important in yeasts, relatively little is known about the relationship between its structure and function. In Saccharomyces cerevisiae, a family of 55 transcription factor proteins unique to fungi, so-called zinc cluster proteins, has been described. Of these, Rds2 has been identified as an activator/inhibitor of gluconeogenesis. However, previous studies have pointed out additional roles for this protein, specifically, in the modulation of cell-wall architecture and drug sensitivity. In this work, evidence regarding the role of Rds2 as a regulator of cell-wall architecture and composition is presented based on phenotypical analysis of the cell walls prepared …

Cell wall:CIENCIAS DE LA VIDA::Microbiología [UNESCO]Gene RDS2Transcription factorsSaccharomyces cerevisiaeSaccharomyces cerevisiae; Transcription factors; Gene RDS2; Cell wallSaccharomyces cerevisiae ; Transcription factors ; Gene RDS2 ; Cell wallUNESCO::CIENCIAS DE LA VIDA::Microbiología
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PTHrP [67-86] regulates the expression of stress proteins in breast cancer cells inducing modifications in urokinase-plasminogen activator and MMP-1 …

2003

It was previously reported that a midregion domain of parathyroid hormone-related protein (PTHrP), that is, [67-86]-amide, is able to restrain growth and promote matrigel penetration by the 8701-BC cell line, derived from a biopsy fragment of a primary ductal infiltrating carcinoma of the human breast, and that cell invasion in vitro is drastically impaired by inactivation of urokinase-plasminogen activator (uPa). In this study we started a more detailed investigation of the possible effects on gene expression arising from the interaction between PTHrP [67-86]-amide and 8701-BC breast cancer cells by a combination of conventional-, differential display-and semi-quantitative multiplex-polyme…

CellBreast NeoplasmsBiologyHeat Shock Transcription FactorsDownregulation and upregulationCell Line TumorHeat shock proteinmedicineHumansNeoplasm InvasivenessHSP90 Heat-Shock ProteinsEnzyme InhibitorsHSF1Heat-Shock ProteinsMatrigelActivator (genetics)CarcinomaParathyroid Hormone-Related ProteinCell BiologyOligonucleotides AntisenseUrokinase-Type Plasminogen ActivatorMolecular biologyPeptide FragmentsProtein Structure TertiaryUp-RegulationDNA-Binding ProteinsGene Expression Regulation NeoplasticHeat shock factormedicine.anatomical_structureCell cultureCancer researchFemaleQuercetinMatrix Metalloproteinase 1Transcription FactorsJournal of Cell Science
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Hypoxia and HIF Signaling: One Axis with Divergent Effects

2020

The correct concentration of oxygen in all tissues is a hallmark of cellular wellness, and the negative regulation of oxygen homeostasis is able to affect the cells and tissues of the whole organism. The cellular response to hypoxia is characterized by the activation of multiple genes involved in many biological processes. Among them, hypoxia-inducible factor (HIF) represents the master regulator of the hypoxia response. The active heterodimeric complex HIF α/β, binding to hypoxia-responsive elements (HREs), determines the induction of at least 100 target genes to restore tissue homeostasis. A growing body of evidence demonstrates that hypoxia signaling can act by generating contrasting res…

CellInflammationReviewBiologyCatalysislcsh:ChemistryInorganic ChemistryImmune systemSettore BIO/13 - Biologia ApplicataOxygen homeostasisBasic Helix-Loop-Helix Transcription FactorsmedicineHumansRNA MessengerAcute and chronic diseasesPhysical and Theoretical ChemistryHypoxialcsh:QH301-705.5Molecular BiologySpectroscopyTissue homeostasisInflammationKidneyImmune cellsOrganic ChemistryHIF-αNuclear ProteinsGeneral MedicineHypoxia (medical)Cell HypoxiaComputer Science ApplicationsCell biologyDNA-Binding ProteinsOxygenmedicine.anatomical_structurelcsh:Biology (General)lcsh:QD1-999Hypoxia-Inducible Factor 1medicine.symptomSignal transductionSignal TransductionTranscription FactorsInternational Journal of Molecular Sciences
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Dihydrocucurbitacin B Inhibits Delayed Type Hypersensitivity Reactions by Suppressing Lymphocyte Proliferation

2007

We have studied the effects of dihydrocucurbitacin B, a triterpene isolated from Cayaponia tayuya roots, on different models of delayed type hypersensitivity (DTH) in mice, as well as on T-lymphocyte proliferation and the mediators involved. In experiments with mice, dihydrocucurbitacin B inhibited the inflammatory reactions induced by oxazolone, dinitrofluorobenzene, and sheep red blood cells, reducing both the edema and cell infiltration. Moreover, the analysis of inflamed tissues showed that dihydrocucurbitacin B reduced the presence of the most relevant cytokines implicated in these processes, including interleukin-1 beta, interleukin-4, and tumor necrosis factor-alpha. Dihydrocucurbita…

CellLymphocyte proliferationLymphocyte ActivationResting Phase Cell CycleOxazoloneMicechemistry.chemical_compoundCyclinsmedicineAnimalsHypersensitivity DelayedCyclinInflammationPharmacologyNFATC Transcription FactorsbiologyNFATCell cyclebiology.organism_classificationMolecular biologyTriterpenesCayaponia tayuyaDisease Models Animalmedicine.anatomical_structurechemistryImmunologyCytokinesMolecular MedicineTumor necrosis factor alphaJournal of Pharmacology and Experimental Therapeutics
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