Search results for "script"

showing 10 items of 5143 documents

Long-term therapeutic levels of human alpha-1 antitrypsin in plasma after hydrodynamic injection of nonviral DNA

2003

The transfection efficacy of several vectors containing the full genomic hAAT gene with its natural promoter (pTG7101) and others containing the cDNA of hAAT gene driven by cytomegalovirus immediate-early promoter or the 0.5 kb upstream of hAAT gene sequence has been studied by hydrodynamic tail-vein injection (20 microg/mouse). pTG7101 (but not the other plasmids) results in therapeutic and stable concentration of hAAT in plasma. A dose-response study with this plasmid (0.3-320 microg/mouse) confirms that hAAT remains long-term stable in plasma, with therapeutic concentrations of hAAT (>0.9 mg/ml). The parameters of the dose-response curve were: R: 0.98, E(max) 3449.0+/- 279.7 microg/ml an…

MaleTime FactorsTransgeneGenetic enhancementMolecular Sequence DataGene ExpressionBiologyGene deliveryTransfectionInjectionsMicePlasmidComplementary DNAGene expressionGeneticsAnimalsHumansTransgenesMolecular BiologyGeneBase SequenceReverse Transcriptase Polymerase Chain ReactionDNAGenetic TherapyTransfectionImmunohistochemistryMolecular biologyMice Inbred C57BLLiveralpha 1-AntitrypsinMolecular MedicineGene Therapy
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Olfactory discrimination ability and brain expression of c-fos, Gir and Glut1 mRNA are altered in n-3 fatty acid-depleted rats

2007

Abstract The long-chain polyunsaturated n  − 3 fatty acids ( n  − 3 PUFA), particularly docosahexaenoic acid (DHA), are abundantly present in the central nervous system and play an important role in cognitive functions such as learning and memory. We, therefore, investigated the effects of n  − 3 PUFA-depletion in rats (F2 generation) on the learning of an olfactory discrimination task, progressively acquired within a four-arm maze, and on the mRNA expression of some candidate genes, i.e., c-fos, Gir and glucose transporter (Glut1), which could reflect the level of cerebral activity. We observed that DHA contents were dramatically decreased in the olfactory bulb, the piriform cortex and the…

MaleTime Factors[SDV]Life Sciences [q-bio]OLFACTORY LEARNINGReceptors G-Protein-CoupledDiscrimination LearningBehavioral Neuroscience0302 clinical medicineDiscrimination PsychologicalPiriform cortexBRAINDiet Fat-Restricted0303 health sciencesGlucose Transporter Type 1NeocortexBehavior AnimalGIRReverse Transcriptase Polymerase Chain ReactionSmellmedicine.anatomical_structureBiochemistryDocosahexaenoic acidN−3 DEFICIENCYOlfactory LearningProto-Oncogene Proteins c-fosmedicine.medical_specialtyN-3 PUFAC-FOSCentral nervous systemOlfactionBiologyAPPRENTISSAGE03 medical and health sciencesInternal medicineFatty Acids Omega-3medicineAnimalsRNA MessengerRats WistarUnsaturated fatty acid030304 developmental biologyAnalysis of VarianceBody WeightDOCOSAHEXAENOIC ACIDOlfactory bulbRatsEndocrinologyGene Expression RegulationRAT030217 neurology & neurosurgeryGLUT1
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Controlled intermittent shortening contractions of a muscle-tendon complex: muscle fibre damage and effects on force transmission from a single head …

2005

This study was performed to examine effects of prolonged (3 h) intermittent shortening (amplitude 2 mm) contractions (muscles were excited maximally) of head III of rat extensor digitorum longus muscle (EDL III) on indices of muscle damage and on force transmission within the intact anterior crural compartment. Three hours after the EDL III exercise, muscle fibre damage, as assessed by immunohistochemical staining of structural proteins (i.e. dystrophin, desmin, titin, laminin-2), was found in EDL, tibialis anterior (TA) and extensor hallucis longus (EHL) muscles. The damaged muscle fibres were not uniformly distributed throughout the muscle cross-sections, but were located predominantly ne…

MaleTime Factorsanimal structuresPhysiologyMolecular Sequence DataMuscle Fibers SkeletalVesicular Transport ProteinsMuscle ProteinsBiochemistryDesminExtensor digitorum longus muscleTendonsSDG 3 - Good Health and Well-beingmedicineAnimalsRNA MessengerMuscle fibreRats WistarMuscle SkeletalbiologyChemistryReverse Transcriptase Polymerase Chain ReactionMETIS-229512Compartment (ship)Gene Expression Profilingmusculoskeletal neural and ocular physiologyMembrane ProteinsCell BiologyAnatomyOrgan Sizemusculoskeletal systemTendonRatsSlack lengthmedicine.anatomical_structureIR-104003Models Animalbiology.protein/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingTitinDesminStress MechanicalDystrophintissuesMuscle Contraction
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Autoimmune polyendocrine syndrome type 1: an Italian survey on 158 patients

2021

Abstract Background Autoimmune Polyglandular Syndrome type 1 (APS-1) is a rare recessive inherited disease, caused by AutoImmune Regulator (AIRE) gene mutations and characterized by three major manifestations: chronic mucocutaneous candidiasis (CMC), chronic hypoparathyroidism (CH) and Addison’s disease (AD). Methods Autoimmune conditions and associated autoantibodies (Abs) were analyzed in 158 Italian patients (103 females and 55 males; F/M 1.9/1) at the onset and during a follow-up of 23.7 ± 15.1 years. AIRE mutations were determined. Results The prevalence of APS-1 was 2.6 cases/million (range 0.5–17 in different regions). At the onset 93% of patients presented with one or more component…

MaleTranscription FactorEndocrinology Diabetes and MetabolismAutoimmune hepatitisGene mutationGastroenterologyChronic mucocutaneous candidiasisEndocrinologyAddison DiseaseAutoimmune Polyglandular Syndrome type 1 (APS-1)PrevalenceMedicineChronic mucocutaneous candidiasisPolyendocrinopathies AutoimmuneCandidiasis Chronic MucocutaneouAddison’s disease AIRE gene mutations Autoimmune Polyglandular Syndrome type 1 (APS-1) Autoimmune-poly-endocrine-candidiasis-ectodermal-dystrophy (APECED) Chronic hypoparathyroidism Chronic mucocutaneous candidiasis Interferon autoantibodiesCandidiasis Chronic MucocutaneousAIRE gene mutations; Addison’s disease; autoimmune polyglandular syndrome type 1 (APS-1); autoimmune-poly-endocrine-candidiasis-ectodermal-dystrophy (APECED); chronic hypoparathyroidism; chronic mucocutaneous candidiasis; interferon autoantibodiesAutoimmune regulatorAutoantibodieItalyInterferon autoantibodieAddison's diseaseInterferon Type IOriginal ArticleFemaleChronic hypoparathyroidismHumanAdultmedicine.medical_specialtyAutoimmune GastritisHypoparathyroidismAddison’s diseaseAIRE gene mutationsInternal medicineInterferon autoantibodiesHumansMortalityAutoantibodiesAddison’s disease; AIRE gene mutations; Autoimmune Polyglandular Syndrome type 1 (APS-1); Autoimmune-poly-endocrine-candidiasis-ectodermal-dystrophy (APECED); Chronic hypoparathyroidism; Chronic mucocutaneous candidiasis; Interferon autoantibodiesbusiness.industryChronic mucocutaneous candidiasiAIRE gene mutationAutoantibodymedicine.diseaseAutoimmune-poly-endocrine-candidiasis-ectodermal-dystrophy (APECED)Interferon autoantibodies.Autoimmune polyendocrine syndrome type 1MutationbusinessTranscription Factors
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Further Delineation of Duplications of ARX Locus Detected in Male Patients with Varying Degrees of Intellectual Disability

2022

The X-linked gene encoding aristaless-related homeobox (ARX) is a bi-functional transcription factor capable of activating or repressing gene transcription, whose mutations have been found in a wide spectrum of neurodevelopmental disorders (NDDs); these include cortical malformations, paediatric epilepsy, intellectual disability (ID) and autism. In addition to point mutations, duplications of the ARX locus have been detected in male patients with ID. These rearrangements include telencephalon ultraconserved enhancers, whose structural alterations can interfere with the control of ARX expression in the developing brain. Here, we review the structural features of 15 gain copy-number variants …

MaleTranscription FactorUltraconserved enhancersIntellectual disability3D structureCatalysisInorganic ChemistryMiceAnimalsHumansPhysical and Theoretical ChemistryChildMolecular BiologySpectroscopyHomeodomain ProteinsAnimalKDM5C-SYN1 axiOrganic ChemistryKDM5C-SYN1 axisGenes HomeoboxHomeodomain ProteinGeneral MedicineXp21.3 duplicationComputer Science ApplicationsUltraconserved enhancerSettore MED/03 - Genetica MedicaMutationARXHumanTranscription Factors
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Different micrococcal nuclease cleavage patterns characterize transcriptionally active and inactive sea-urchin histone genes.

1986

Micrococcal nuclease (MNase) and DNaseI have made a great contribution to our present understanding of the structural organization of the eucaryotic genome [l - 31. The enhanced sensitivity of active portions of the genome to DNaseI gave, in fact, the first indication of differences in the nucleoproteic arrangement of the transcribed as compared to the silent DNA regions [4]. The use of MNase, as a probe of the chromatin organization of specific genes in the active and inactive state, provided additional evidence for this and further showed that the packing of several coding segments of DNA in a regular array of nucleosomal particles is severely but reversibly affected by the transcriptiona…

MaleTranscription GeneticBiologyBiochemistryGenomeHistoneschemistry.chemical_compoundTranscriptional regulationAnimalsMicrococcal NucleaseGeneGeneticsNucleaseDNA Restriction EnzymesSpermatozoaChromatinChromatinBlastocystHistoneGeneschemistrySea Urchinsbiology.proteinDNADensitometryMicrococcal nuclease
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Glyphosate-based herbicides are toxic and endocrine disruptors in human cell lines.

2009

International audience; Glyphosate-based herbicides are the most widely used across the world; they are commercialized in different formulations. Their residues are frequent pollutants in the environment. In addition, these herbicides are spread on most eaten transgenic plants, modified to tolerate high levels of these compounds in their cells. Up to 400 ppm of their residues are accepted in some feed. We exposed human liver HepG2 cells, a well-known model to study xenobiotic toxicity, to four different formulations and to glyphosate, which is usually tested alone in chronic in vivo regulatory studies. We measured cytotoxicity with three assays (Alamar Blue (R), MTT ToxiLight (R)), plus gen…

MaleTranscription GeneticEstrogen receptor010501 environmental sciencesEndocrine DisruptorsToxicologymedicine.disease_cause01 natural scienceschemistry.chemical_compoundGenes ReporterAromataseCytotoxicityendocrine disruptor0303 health sciencesroundupsexual steroidsEndocrine disruptorBiochemistryReceptors AndrogenComet Assaymedicine.medical_specialtyHepG2AdolescentGlycine[SDV.TOX.TCA]Life Sciences [q-bio]/Toxicology/Toxicology and food chainBiology03 medical and health sciencesAromataseglyphosateInternal medicineCell Line TumorToxicity TestsmedicineEstrogen Receptor betaHumansRNA MessengerCarcinogen030304 developmental biology0105 earth and related environmental sciencesDose-Response Relationship DrugHerbicidesEstrogen Receptor alphaPesticide ResiduesComet assayEndocrinologychemistry13. Climate actionbiology.proteinXenobioticGenotoxicityDNA DamageToxicology
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Nuclear fos domains in transcriptionally activated supraoptic nucleus neurons

1993

This study has analysed by light and electron microscopy immunolocalization the nuclear pattern of distribution of Fos-related proteins in supraotic neurons. Two experimental models of transcriptional activation have been used: sustained, global transcriptional activation, at relatively near physiological conditions, by six days of chronic intermittent salt loading; and superinduction of c-fos gene by this salt loading regime plus cycloheximide treatment for 4 h. In the first condition, the ultrastructural analysis showed a distribution of Fos-like immunoreactivity on the reticular network of dispersed chromatin that extends between the nucleolar surface and the nuclear envelope, whereas th…

MaleTranscription GeneticEuchromatinNucleolusHeterochromatinCycloheximideBiologyEuchromatinRats Sprague-Dawleychemistry.chemical_compoundGenes junmedicineAnimalsCycloheximideRibonucleoproteinCell NucleusNeuronsGeneral NeuroscienceGenes fosRNAImmunohistochemistryMolecular biologyChromatinRatsChromatinCell biologyMicroscopy ElectronCell nucleusmedicine.anatomical_structurechemistryProto-Oncogene Proteins c-fosSupraoptic NucleusNeuroscience
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Lysosomal Signaling Licenses Embryonic Stem Cell Differentiation via Inactivation of Tfe3

2019

International audience; Self-renewal and differentiation of pluripotent murine embryonic stem cells (ESCs) is regulated by extrinsic signaling pathways. It is less clear whether cellular metabolism instructs developmental progression. In an unbiased genome-wide CRISPR/Cas9 screen, we identified components of a conserved amino-acid-sensing pathway as critical drivers of ESC differentiation. Functional analysis revealed that lysosome activity, the Ragulator protein complex, and the tumor-suppressor protein Folliculin enable the Rag GTPases C and D to bind and seclude the bHLH transcription factor Tfe3 in the cytoplasm. In contrast, ectopic nuclear Tfe3 represses specific developmental and met…

MaleTranscription GeneticGTPaseGTP PhosphohydrolasesPATHWAYMice0302 clinical medicineNeural Stem CellsCRISPRTUMOR-SUPPRESSORCell Self RenewalPhosphorylationSPECIFICATIONdevelopmental disorder0303 health sciencesGenomeBasic Helix-Loop-Helix Leucine Zipper Transcription FactorsCell DifferentiationMouse Embryonic Stem CellsFlcndifferentiationCell biologymedicine.anatomical_structuremTORMolecular MedicineFemaleSignal transductionProtein BindingSignal TransductionRECRUITMENTBiology03 medical and health sciencesRag GTPasesLysosomeGeneticsmedicineAnimalsHumansPoint MutationNAIVE PLURIPOTENCYAMINO-ACID LEVELSTranscription factorAllelesPI3K/AKT/mTOR pathway030304 developmental biologyCOMPLEXFOLLICULINRagulatorCell Biologypluripotencyembryonic stem cellEmbryonic stem cellTfe3[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human geneticsCytoplasmLysosomes030217 neurology & neurosurgeryCell Stem Cell
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The murine nuclear orphan receptor GCNF is expressed in the XY body of primary spermatocytes

1998

AbstractWe have studied the expression of the nuclear orphan receptor GCNF (germ cell nuclear factor) on the mRNA and protein level in pubertal and adult mouse testes. We show by Northern and Western blot analyses and by in situ hybridization that GCNF is expressed in spermatocytes and round spermatids of adult mouse testis suggesting that GCNF may be a transcriptional regulator of spermatogenesis. Since the GCNF protein is accumulated in the XY body of late pachytene spermatocytes, it may be involved in transcriptional inactivation of sex chromosomes.

MaleTranscription GeneticGerm cell nuclear factorBiophysicsFluorescent Antibody TechniqueReceptors Cytoplasmic and NuclearIn situ hybridizationBiologyIn Vitro TechniquesGerm cell nuclear factorBiochemistryEpitheliumMiceWestern blotStructural BiologySpermatocytesNuclear Receptor Subfamily 6 Group A Member 1TestisGeneticsTranscriptional regulationmedicineAnimalsRNA MessengerRetinoid receptor-related testis-associated receptorSpermatogenesisMolecular BiologyOrphan receptorMessenger RNAMice Inbred BALB Cmedicine.diagnostic_testCell BiologyMolecular biologySpermatidsNeuron-derived orphan receptor 1DNA-Binding ProteinsGene Expression RegulationNuclear orphan receptorSpermatogenesisNeuronal cell nuclear factorXY bodyFEBS Letters
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