Search results for "specialist"

showing 10 items of 798 documents

10qter deletion: A new case

2008

Vertebrate telomeres consist of tandem repeats of the TTAGGG sequence that cap the ends of chromosomes, protecting them from degradation and fusion. Extensive evidence has shown that telomere shortening and erosion lead lo chromo¬some end-to-end fusions and genomic instability, causing mental retardation and/or malformation syndromes. So far, over 19,000 patients with mental retardation have been tested and reported of whom -2.5% appeared to have a subtelomeric rearrange¬ment [Ravnan et al., 2006; Ballif et al., 2007; Ledbetter and Martin, 2007]. Since the identification of sub¬microscopic subtelomeric rearrangements as a major cause of mental retardation [Flint et al., 1995], testing for s…

MaleChromosomes Human Pair 1010qter deletionDevelopmental DisabilitiesBiologyCraniofacial AbnormalitiesMonosomySettore MED/38 - Pediatria Generale E SpecialisticaChild PreschoolGeneticsHumansAbnormalities MultipleChromosome DeletionIn Situ Hybridization FluorescenceGenetics (clinical)Oligonucleotide Array Sequence Analysis
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The first case of myoclonic epilepsy in a child with a de novo 22q11.2 microduplication

2011

Chromosome 22, particularly the q11.2 sub-band, has long been recognized as responsible for multiple congenital anomaly disorders. In particular, its susceptibility to subtle microdeletions or, more rarely, microduplications has been attributed to the presence of several low-copy repeats spanning the region as mediators of nonallelic homologous recombination that result in 22q11.2 rearrangements. While recent data suggest that the frequency of 22q11.2 microduplications could be approximately half of all deletions, now only 50 unrelated cases have been reported thus far. However, it is reasonable to suppose that microduplications of 22q11.2 may be largely undetected as a result of a less-dis…

MaleChromosomes Human Pair 22Non-allelic homologous recombinationEpilepsies MyoclonicMultiple congenital anomalyBiologyRAB36 genemyoclonic epilepsySettore MED/38 - Pediatria Generale E SpecialisticaChromosome DuplicationGene duplicationClinical heterogeneityGeneticsmedicineHumansChildIn Situ Hybridization FluorescenceGenetics (clinical)GeneticsComparative Genomic HybridizationFaciesmedicine.diseaseMild learning difficultiesdevelopmental delayPhenotypeSettore MED/03 - Genetica MedicaChild PreschoolMyoclonic epilepsynonallelic homologous recombinationChromosome 2222q11.2 microduplicationComparative genomic hybridizationAmerican Journal of Medical Genetics Part A
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Laboratory investigations in genetic syndromes: examples of clinical approach in the neonatal unit.

2010

Contiguous gene deletion syndromes: the importance of an accurate genetic definition for a careful clinical monitoring. Contiguous gene deletion syndromes are so named because the deletion manifests as a distinctive cluster of otherwise unrelated single-gene disorders in the same subject. An accurate genetic definition of the deleted region is extremely important for the appropriate management of these patients and for unravelling the function of the involved genes. The microarray-based comparative genomic hybridization (CGH arrays) analysis is the actual molecular method able to accurately define the bounds of a deleted region, since it allows an evaluation of DNA copy number alterations a…

MaleComparative Genomic Hybridizationgenetic syndrome neonategenetic syndromesDisorders of Sex DevelopmentGene DosageGenetic Diseases InbornInfant NewbornLaboratories HospitalEarly DiagnosisPhenotypeSettore MED/38 - Pediatria Generale E SpecialisticaIntensive Care Units NeonatalIntensive Care NeonatalChromosomes HumanHumansFemaleGenetic TestingGene Deletion
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Kawasaki disease: Guidelines of Italian Society of Pediatrics, part II - Treatment of resistant forms and cardiovascular complications, follow-up, li…

2018

Abstract This second part of practical Guidelines related to Kawasaki disease (KD) has the goal of contributing to prompt diagnosis and most appropriate treatment of KD resistant forms and cardiovascular complications, including non-pharmacologic treatments, follow-up, lifestyle and prevention of cardiovascular risks in the long-term through a set of 17 recommendations. Guidelines, however, should not be considered a norm that limits the treatment options of pediatricians and practitioners, as treatment modalities other than those recommended may be required as a result of peculiar medical circumstances, patient’s condition, and disease severity or individual complications.

MaleDrug ResistanceReviewCoronary Artery Disease030204 cardiovascular system & hematologySeverity of Illness IndexCoronary artery diseaseEfficacy0302 clinical medicineCardiovascular Diseasecoronary artery abnormalitiesChildCoronary artery abnormalitieSocieties MedicalPediatricAnti-Inflammatory Agents Non-Steroidallcsh:RJ1-570Immunoglobulins IntravenousSettore MED/38Settore MED/38 - PEDIATRIA GENERALE E SPECIALISTICACardiovascular DiseasesInnovative biotechnologieChild PreschoolPractice Guidelines as TopicFemaleRisk assessmentmedicine.drugHumanmedicine.medical_specialtypediatricsMucocutaneous Lymph Node SyndromeRisk AssessmentFollow-Up Studie03 medical and health sciencesinnovative biotechnologies030225 pediatricsDiabetes mellitusSeverity of illnessmedicineHumansIntensive care medicineIntravenous immunoglobulinAspirinKawasaki diseasebusiness.industryWarfarinlcsh:Pediatricsmedicine.diseasePersonalized medicineInfliximabAspirin; Child; Coronary artery abnormalities; Innovative biotechnologies; Intravenous immunoglobulin; Kawasaki disease; Personalized medicine;Immunoglobulins IntravenouPediatrics Perinatology and Child Healthperinatology and child healthKawasaki diseaseaspirin; child; coronary artery abnormalities; innovative biotechnologies; intravenous immunoglobulin; Kawasaki disease; personalized medicine; pediatrics perinatology and child healthbusinessFollow-Up Studies
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Epidemiology of Toxoplasma and CMV serology and of GBS colonization in pregnancy and neonatal outcome in a Sicilian population

2013

Background: Aim of our study is to analyze the immunological status in pregnancy for two main TORCH agents, Toxoplasma and Cytomegalovirus (CMV), and the results of group B streptococcus (GBS) screening, assessing the risk for congenital infection in a population from Palermo, Italy. Methods: We retrospectively analyzed the medical records of all inborn live newborns who were born in our division during 2012, gathering information about the mother, the pregnancy and neonatal hospitalization at birth. Whenever data were available, we categorized the serologic status of the mothers for Toxoplasma and CMV. We also considered the results of rectal and vaginal swabs for GBS. We compared the resu…

MaleEpidemiologyAntibodies ProtozoanCytomegalovirusmedicine.disease_causeAntibodies ViralGroup BInfant Newborn DiseasesSerologySettore MED/38 - Pediatria Generale E SpecialisticaPregnancyRisk FactorsPrenatal DiagnosisEpidemiologyPrevalencePregnancy Complications InfectiousIntrauterine infectionSicilyeducation.field_of_studyPregnancy OutcomeCMVMiddle AgedAntibodies BacterialAntibodies Anti-IdiotypicCytomegalovirus InfectionsFemaleToxoplasmaToxoplasmosisAdultmedicine.medical_specialtyAdolescentPopulationCongenital cytomegalovirus infectionGBSRisk AssessmentStreptococcus agalactiaeYoung AdultStreptococcal InfectionsmedicineAnimalsHumanseducationRetrospective StudiesPregnancybusiness.industryResearchInfant Newbornmedicine.diseaseToxoplasmosisStreptococcus agalactiaeImmunoglobulin GImmunologybusinessItalian Journal of Pediatrics
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17β-Hydroxysteroid dehydrogenase-3 deficiency: From pregnancy to adolescence

2009

OBJECTIVE: Aim of this study is to report on basal clinical phenotype and follow up after diagnosis, of patients with 17beta-hydroxysteroid-dehydrogenase type 3 (17beta-HSD3) deficiency in Italy. SETTING: Pediatric Endocrine Departments, University Hospitals. PATIENTS: The cases of 5 Italian subjects affected by 17beta-HSD3 deficiency are presented in this study. INTERVENTIONS: Laboratory and genetic assessment. Gonadectomy and female sex assignment (4 patients) or GnRH analog therapy to regress puberty and gender identity disorder (1 patient). RESULTS: Presentation lasted from pregnancy (pre-natal diagnosis of a 46,XY fetus with female external genitalia) to infancy (inguinal hernia contai…

MaleGender Identity DisorderPediatricsmedicine.medical_specialty17-Hydroxysteroid DehydrogenasesEndocrinology Diabetes and MetabolismSex assignmentPrenatal diagnosisGene mutationBiologyClitoromegalyAdolescence pregnancy 17beta-Hydroxysteroid dehydrogenase-3 deficiencySettore MED/38 - Pediatria Generale E SpecialisticaEndocrinologyPregnancyPrenatal DiagnosismedicineHumansDisorders of sex developmentDISORDERS OF SEX DEVELOPMENTTestosterone17-BETA-HYDROXYSTEROID DEHYDROGENASE TYPE 3 GENEGynecologyPregnancyPubertymedicine.diseaseFemaleMALE/FEMALE SEX REVERSALTESTOSTERONE/D4-ANDROSTENEDIONE RATIO17-BETA-HYDROXYSTEROID DEHYDROGENASE TYPE 3 DEFICIENCYmedicine.symptomJournal of Endocrinological Investigation
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A finely tuned strategy adopted by an egg parasitoid to exploit chemical traces from host adults.

2009

SUMMARY Scelionid egg parasitoids can obtain reliable information on the presence of host eggs by discriminating host gender on the basis of chemical footprints of their co-evolved hosts, with a strong preference for the footprint left by host females. Based on the concept of dietary specialization and infochemical use in natural enemies, it could be predicted that host gender discrimination in specialist species belonging to the genus Trissolcus is further tuned to specific cues from distinctive chemical traces left by host females as a consequence of copulation and/or oviposition. To test this hypothesis we used the system Murgantia histrionica – Trissolcus brochymenae. Our results showed…

MaleGender discriminationPhysiologyOvipositionTrissolcus brochymenae; Murgantia histrionica; egg parasitoid; indirect host-related cues; arrestment responseWaspsZoologyAquatic ScienceGeneralist and specialist speciesParasitoidHost-Parasite InteractionsHeteropteraCopulationAnimalsNatural enemiesMolecular BiologyEcology Evolution Behavior and SystematicsMurgantia histrionicaSex CharacteristicsbiologyHost (biology)Ecologyindirect host-related cuesExtremitiesINDIRECT HOST RELATED CUES EGG PARASITOIDbiology.organism_classificationarrestment responseTrissolcus brochymenaeInsect Scienceegg parasitoidAnimal Science and ZoologyFemaleCuesThe Journal of experimental biology
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A novel mutation in KCNQ3-related benign familial neonatal epilepsy: electroclinical features and neurodevelopmental outcome.

2019

Benign familial neonatal epilepsy (BFNE) is caused, in about 5% of families, by mutations in the KCNQ3 gene encoding voltage-gated potassium channel subunits. Usually, newborns with BFNE show a normal neurological outcome, but recently, refractory seizures and/or developmental disability have been reported suggesting phenotype variability associated with KCNQ3-related BFNE. Here, we describe a proband from a BFNE family carrying a novel variant in the KCNQ3 gene. Regarding the paucity of data in the literature, we describe the presented case with a view to further establishing: (1) a genotype/phenotype correlation in order to define a BFNE phenotype associated with favourable outcome; (2) a…

MaleGenotypeelectroclinical featureInfantElectroencephalographygenotype-phenotype correlationSettore MED/39 - Neuropsichiatria InfantileEpilepsy Benign NeonatalKCNQ3 Potassium ChannelKCNQSettore MED/38 - Pediatria Generale E SpecialisticaPhenotypevoltage-gated potassium channelsSettore M-PSI/08 - Psicologia ClinicaHumansbenign familial neonatal epilepsyEpileptic SyndromesEpileptic disorders : international epilepsy journal with videotape
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Hormonal, auxological and clinical follow-up in children with connatal HIV infection: personal records

2006

Auxological and endocrinological complications frequently occur in children with connatal HIV infection. These complications seem to be related both to the infection itself and the antiretroviral therapy. Many children consequently show height-weight and pubertal retardation without any evidence of hormonal deficit. We studied 10 children with connatal HIV infection who were enrolled in this analysis and followed up for 7 years in order to evaluate their height-weight growth, pubertal maturation, bone age progression and hormonal pattern [basal Growth hormone (GH) and GH after Clonidine or Insulin stimulation, Insulin-like Growth Factor 1 (IGF-1), Insulin-like Growth Factor Binding Protein …

MaleHIV hormonal follow-up auxological follow-up clinical follow-upSettore MED/17 - Malattie InfettiveBody WeightInfant NewbornInfantHIV InfectionsChild DevelopmentSettore MED/38 - Pediatria Generale E SpecialisticaChild PreschoolBody SizeHumansFemaleLongitudinal StudiesChildFollow-Up Studies
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DIET QUALITY AFFECTS WARNING COLORATION INDIRECTLY: EXCRETION COSTS IN A GENERALIST HERBIVORE

2009

Aposematic herbivores are under selection pressure from their host plants and predators. Although many aposematic herbivores exploit plant toxins in their own secondary defense, dealing with these harmful compounds might underlay costs. We studied whether the allocation of energy to detoxification and/or sequestration of host plant defense chemicals trades off with warning signal expression. We used a generalist aposematic herbivore Parasemia plantaginis (Arctiidae), whose adults and larvae show extensive phenotypic and genetic variation in coloration. We reared larvae from selection lines for small and large larval warning signals on Plantago lanceolata with either low or high concentratio…

MaleHerbivoreLarvaPlantagobiologyfungiColorGenetic VariationZoologyAposematismMothsbiology.organism_classificationGeneralist and specialist speciesDietPredationParasemia plantaginisBotanyGeneticsAnimalsFemaleChemical defenseGeneral Agricultural and Biological SciencesEcology Evolution Behavior and SystematicsEvolution
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