Search results for "spines"

showing 10 items of 28 documents

Neocortical Layer 6B as a Remnant of the Subplate - A Morphological Comparison.

2015

The fate of the subplate (SP) is still a matter of debate. The SP and layer 6 (which is ontogenetically the oldest and innermost neocortical lamina) develop coincidentally. Yet, the function of sublamina 6B is largely unknown. It has been suggested that it consists partly of neurons from the transient SP, however, experimental evidence for this hypothesis is still missing. To obtain first insights into the neuronal complement of layer 6B in the somatosensory rat barrel cortex, we used biocytin stainings of SP neurons (aged 0-4 postnatal days, PND) and layer 6B neurons (PND 11-35) obtained during in vitro whole-cell patch-clamp recordings. Neurons were reconstructed for a quantitative charac…

Cell typeDendritic spinePatch-Clamp TechniquesCognitive NeuroscienceDendritic SpinesNeocortexBiologySomatosensory systemCellular and Molecular Neurosciencechemistry.chemical_compoundBiocytinSubplatemedicineImage Processing Computer-AssistedAnimalsPatch clampRats WistarNeuronsNeocortexPyramidal CellsCell PolarityDendritesSomatosensory CortexBarrel cortexAxonsRatsmedicine.anatomical_structurenervous systemchemistryAnimals NewbornNeuroscienceCerebral cortex (New York, N.Y. : 1991)
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Efectos de la exposición crónica al etanol sobre el tráfico intracelular y citoesqueleto como factores implicados en la migración neuronal

2013

El consumo de etanol durante la gestación puede inducir una serie de alteraciones graves en el desarrollo del feto, la manifestación más extrema da lugar al Síndrome Alcohólico Fetal (SAF). La exposición prenatal al alcohol es la causa conocida y, además evitable, más importante de retraso mental en el mundo occidental. Además de déficits cognitivos, los niños con SAF presentan múltiples anomalías estructurales en el sistema nervioso central, como reducción de la masa cerebral, y a nivel celular, daños en la migración neuronal, en el proceso de formación de espinas dendríticas y establecimiento de sinapsis. En la actualidad, los mecanismos moleculares implicados en la teratogénesis inducida…

Central Nervous Systemaparato de Golgietanolespinas dendríticasneuronasneurons:CIENCIAS DE LA VIDA::Neurociencias [UNESCO]migración neuronalMAP2Fetal Alcoholic SyndromeRho GTPasasactinaRho GTPasesendocitosis:CIENCIAS MÉDICAS::Toxicología [UNESCO]endocytosismicrotúbuloneuronal migrationUNESCO::CIENCIAS MÉDICAS::Toxicologíatráfico intracelularUNESCO::CIENCIAS DE LA VIDA::Biología celular::Cultivo celularcytoskeletondendritic spinesSAFSíndrome Alcohólico Fetalcitoesqueleto:CIENCIAS DE LA VIDA::Biología celular::Cultivo celular [UNESCO]Golgi apparatusUNESCO::CIENCIAS DE LA VIDA::Neurocienciasethanolintracellular trafficactinSistema Nervioso Centralmicrotubule
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Presence of Genital Spines in a Male Corynosoma cetaceum Johnston and Best, 1942 (Acanthocephala)

2002

We collected 83 females and 80 males of Corynosoma cetaceum from 2 common dolphins, Delphinus delphis, collected in northern Patagonia (Argentina). Worms were most similar to specimens collected in other South American localities. However, 1 male had 2 spines adjacent to the genital pore and isolated from the rest of body spines. This finding confirms the recent reassignment of C. cetaceum to Corynosoma. Absence of genital spines is suggested to be avoided as the sole criterion to exclude specimens from Corynosoma or Andracantha. Aznar Avendaño, Francisco Javier, Francisco.Aznar@uv.es ; Raga Esteve, Juan Antonio, Toni.Raga@uv.es

Male CorynosomaGenital spines ; Male Corynosoma ; Acanthocephela:CIENCIAS DE LA VIDA::Biología animal (Zoología) [UNESCO]UNESCO::CIENCIAS DE LA VIDAAcanthocephelaUNESCO::CIENCIAS DE LA VIDA::Biología animal (Zoología)Genital spines:CIENCIAS DE LA VIDA [UNESCO]
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Dopamine restores limbic memory loss, dendritic spine structure, and NMDAR-dependent LTD in the nucleus accumbens of alcohol-withdrawn rats

2018

Alcohol abuse leads to aberrant forms of emotionally salient memory, i.e., limbic memory, that promote escalated alcohol consumption and relapse. Accordingly, activity-dependent structural abnormalities are likely to contribute to synaptic dysfunctions that occur from suddenly ceasing chronic alcohol consumption. Here we show that alcohol-dependent male rats fail to perform an emotional-learning task during abstinence but recover their functioning byl-3,4-dihydroxyphenylalanin (l-DOPA) administration during early withdrawal.l-DOPA also reverses the selective loss of dendritic “long thin” spines observed in medium spiny neurons of the nucleus accumbens (NAc) shell of alcohol-dependent rats d…

Male0301 basic medicineDendritic spineDendritic SpinesAlcohol abuseDopamineDopamine AgentsAMPA receptorMotor ActivityNucleus accumbensMedium spiny neuronReceptors N-Methyl-D-AspartateNucleus AccumbensLevodopaRats Sprague-Dawley03 medical and health sciences0302 clinical medicineDopamineMemoryLimbic SystemmedicineAnimalsReceptors AMPAResearch ArticlesMemory DisordersAlcohol Abstinencebusiness.industryLong-Term Synaptic DepressionGeneral NeuroscienceDopaminergicRatsConfocal microscopyAlcoholism030104 developmental biologySynaptic plasticityLTDSettore BIO/14 - FarmacologiaNMDA receptorGlutamatebusinessNeuroscience030217 neurology & neurosurgerymedicine.drug
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Dysregulated Expression of Neuregulin-1 by Cortical Pyramidal Neurons Disrupts Synaptic Plasticity

2014

Summary Neuregulin-1 ( NRG1 ) gene variants are associated with increased genetic risk for schizophrenia. It is unclear whether risk haplotypes cause elevated or decreased expression of NRG1 in the brains of schizophrenia patients, given that both findings have been reported from autopsy studies. To study NRG1 functions in vivo, we generated mouse mutants with reduced and elevated NRG1 levels and analyzed the impact on cortical functions. Loss of NRG1 from cortical projection neurons resulted in increased inhibitory neurotransmission, reduced synaptic plasticity, and hypoactivity. Neuronal overexpression of cysteine-rich domain (CRD)-NRG1, the major brain isoform, caused unbalanced excitato…

MaleDendritic SpinesNeuregulin-1Nonsynaptic plasticityGene ExpressionMice TransgenicNeurotransmissionInhibitory postsynaptic potentialSynaptic TransmissionGeneral Biochemistry Genetics and Molecular BiologyCell MovementInterneuronsConditioning Psychologicalmental disordersAnimalsNeuregulin 1lcsh:QH301-705.5CA1 Region HippocampalNeuronal PlasticitybiologyPyramidal CellsAnatomyFearCortex (botany)Synaptic fatiguelcsh:Biology (General)Synaptic plasticitybiology.proteinExcitatory postsynaptic potentialFemaleNerve NetNeuroscience
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Hampered long-term depression and thin spine loss in the nucleus accumbens of ethanol-dependent rats.

2014

Alcoholism involves long-term cognitive deficits, including memory impairment, resulting in substantial cost to society. Neuronal refinement and stabilization are hypothesized to confer resilience to poor decision making and addictive-like behaviors, such as excessive ethanol drinking and dependence. Accordingly, structural abnormalities are likely to contribute to synaptic dysfunctions that occur from suddenly ceasing the use of alcohol after chronic ingestion. Here we show that ethanol-dependent rats display a loss of dendritic spines in medium spiny neurons of the nucleus accumbens (Nacc) shell, accompanied by a reduction of tyrosine hydroxylase immunostaining and postsynaptic density 95…

MaleDendritic spineDendritic SpinesGlutamic AcidNucleus accumbensNeurotransmissionMedium spiny neuronSynaptic TransmissionNucleus AccumbensOrgan Culture TechniquesAnimalsRats WistarLong-term depressionLong-Term Synaptic Depressiondopamine synaptic plasticity Golgi glutamateMultidisciplinaryNeuronal PlasticityEthanolDopaminergic NeuronsLong-Term Synaptic DepressionCentral Nervous System DepressantsRatsAlcoholismPNAS PlusSynaptic plasticitySettore BIO/14 - FarmacologiaPsychologyNeurosciencePostsynaptic densityProceedings of the National Academy of Sciences of the United States of America
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Chronic stress induces changes in the structure of interneurons and in the expression of molecules related to neuronal structural plasticity and inhi…

2011

Chronic stress in experimental animals, one of the most accepted models of chronic anxiety and depression, induces structural remodeling of principal neurons in the amygdala and increases its excitation by reducing inhibitory tone. These changes may be mediated by the polysialylated form of the neural cell adhesion molecule (PSA-NCAM), a molecule related to neuronal structural plasticity and expressed by interneurons in the adult CNS, which is downregulated in the amygdala after chronic stress. We have analyzed the amygdala of adult mice after 21 days of restraint stress, studying with qRT-PCR the expression of genes related to general and inhibitory neurotransmission, and of PSA synthesizi…

MaleDendritic spineInterneuronDendritic SpinesSynaptophysinNeural Cell Adhesion Molecule L1BiologyNeurotransmissionSynaptic TransmissionAmygdalaImmobilizationMiceDevelopmental NeuroscienceInterneuronsmedicineAnimalsChronic stressNeuronal PlasticityGlutamate DecarboxylaseDendritesAmygdalaImmunohistochemistrySialyltransferasesDisease Models Animalmedicine.anatomical_structurenervous systemNeurologySialic AcidsSynaptophysinbiology.proteinNeural cell adhesion moleculeNeuroscienceStress PsychologicalBasolateral amygdalaExperimental Neurology
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The dendritic spines of interneurons are dynamic structures influenced by PSA-NCAM expression.

2013

Excitatory neurons undergo dendritic spine remodeling in response to different stimuli. However, there is scarce information about this type of plasticity in interneurons. The polysialylated form of the neural cell adhesion molecule (PSA-NCAM) is a good candidate to mediate this plasticity as it participates in neuronal remodeling and is expressed by some mature cortical interneurons, which have reduced dendritic arborization, spine density, and synaptic input. To study the connectivity of the dendritic spines of interneurons and the influence of PSA-NCAM on their dynamics, we have analyzed these structures in a subpopulation of fluorescent spiny interneurons in the hippocampus of glutamic …

MaleDendritic spineTime FactorsInterneuronCognitive NeuroscienceDendritic SpinesGreen Fluorescent ProteinsHippocampusNeuraminidaseMice TransgenicNerve Tissue ProteinsNeural Cell Adhesion Molecule L1BiologyHippocampal formationIn Vitro TechniquesHippocampus03 medical and health sciencesCellular and Molecular NeuroscienceMice0302 clinical medicineOrgan Culture TechniquesInterneuronsmedicineAnimals030304 developmental biology0303 health sciencesPolysialic acidGlutamate DecarboxylaseDendritic filopodiamedicine.anatomical_structurenervous systemAnimals NewbornGene Expression RegulationCalbindin 2Excitatory postsynaptic potentialSialic AcidsNeural cell adhesion moleculeCholecystokininSomatostatinNeuroscience030217 neurology & neurosurgeryVasoactive Intestinal PeptideCerebral cortex (New York, N.Y. : 1991)
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Chronic fluoxetine treatment alters the structure, connectivity and plasticity of cortical interneurons

2014

Novel hypotheses suggest that antidepressants, such as the selective serotonin reuptake inhibitor fluoxetine, induce neuronal structural plasticity, resembling that of the juvenile brain, although the underlying mechanisms of this reopening of the critical periods still remain unclear. However, recent studies suggest that inhibitory networks play an important role in this structural plasticity induced by fluoxetine. For this reason we have analysed the effects of a chronic fluoxetine treatment in the hippocampus and medial prefrontal cortex (mPFC) of transgenic mice displaying eGFP labelled interneurons. We have found an increase in the expression of molecules related to critical period pla…

MalePERINEURONAL NET EXPRESSIONTime FactorsDendritic spinePSA-NCAMCritical period plasticityHippocampusCell CountADULT BRAIN PLASTICITYTREATMENT INCREASESHippocampusMice0302 clinical medicinePharmacology (medical)Prefrontal cortexCerebral Cortex0303 health sciencesNeuronal PlasticitybiologyGlutamate DecarboxylaseMEDIAL PREFRONTAL CORTEXPOLYSIALIC ACIDmusculoskeletal neural and ocular physiologyPerineuronal net3. Good healthPsychiatry and Mental healthParvalbuminsmedicine.anatomical_structureCerebral cortexCELL-ADHESION MOLECULEAntidepressive Agents Second-GenerationDendritic SpinesGreen Fluorescent ProteinseducationMice TransgenicNerve Tissue ProteinsNeural Cell Adhesion Molecule L1Inhibitory postsynaptic potentialRAT HIPPOCAMPUS03 medical and health sciencesmedicineAnimalsPSA-NCAM EXPRESSION030304 developmental biologyPharmacologyperineuronal netsinterneuronsCENTRAL-NERVOUS-SYSTEMfluoxetine3112 NeurosciencesGene Expression Regulationnervous systemVesicular Glutamate Transport Protein 1Sialic Acidsbiology.proteinNeural cell adhesion moleculeNerve NetNeuroscience030217 neurology & neurosurgeryParvalbuminThe International Journal of Neuropsychopharmacology
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The Cannabinoid Receptor CB1 Interacts with the WAVE1 Complex and Plays a Role in Actin Dynamics and Structural Plasticity in Neurons.

2015

The molecular composition of the cannabinoid type 1 (CB1) receptor complex beyond the classical G-protein signaling components is not known. Using proteomics on mouse cortex in vivo, we pulled down proteins interacting with CB1 in neurons and show that the CB1 receptor assembles with multiple members of the WAVE1 complex and the RhoGTPase Rac1 and modulates their activity. Activation levels of CB1 receptor directly impacted on actin polymerization and stability via WAVE1 in growth cones of developing neurons, leading to their collapse, as well as in synaptic spines of mature neurons, leading to their retraction. In adult mice, CB1 receptor agonists attenuated activity-dependent remodeling o…

MaleReceptor complexCannabinoid receptorDendritic spineQH301-705.5medicine.medical_treatmentDendritic SpinesNeurogenesisRecombinant Fusion ProteinsGrowth ConesWiskott-Aldrich Syndrome Protein NeuronalNerve Tissue ProteinsBiologyCannabinoidergicGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesActin remodeling of neurons0302 clinical medicineReceptor Cannabinoid CB1Parietal LobeChlorocebus aethiopsmedicineAnimalsBiology (General)Cells Cultured030304 developmental biologyMice KnockoutNeurons0303 health sciencesNeuronal PlasticityGeneral Immunology and MicrobiologyCannabinoidsGeneral NeuroscienceNeurogenesisActin cytoskeletonEmbryo MammalianCell biologyFrontal LobeMice Inbred C57BLActin CytoskeletonLuminescent Proteinsnervous systemCOS Cellslipids (amino acids peptides and proteins)CannabinoidGeneral Agricultural and Biological Sciences030217 neurology & neurosurgeryResearch ArticlePLoS Biology
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