Search results for "stre"

showing 10 items of 10887 documents

Oxidative stress in the oral cavity is driven by individualspecific bacterial communities

2018

The term “bacterial dysbiosis” is being used quite extensively in metagenomic studies, however, the identification of harmful bacteria often fails due to large overlap between the bacterial species found in healthy volunteers and patients. We hypothesized that the pathogenic oral bacteria are individual-specific and they correlate with oxidative stress markers in saliva which reflect the inflammatory processes in the oral cavity. Temporally direct and lagged correlations between the markers and bacterial taxa were computed individually for 26 volunteers who provided saliva samples during one month (21.2 ± 2.7 samples/volunteer, 551 samples in total). The volunteers’ microbiomes differed sig…

0301 basic medicineSalivaACID REACTING SUBSTANCES030106 microbiologyPhysiologyDiseasemedicine.disease_causeApplied Microbiology and BiotechnologyMicrobiologylcsh:Microbial ecologyArticle03 medical and health sciencesmedicineMicrobiomeGENE-EXPRESSIONTOTAL ANTIOXIDANT CAPACITYScience & TechnologyDENTAL-CARIESPLASMASTABILITYbiologybiology.organism_classificationmedicine.diseaseSALIVARY MARKERSSTREPTOCOCCUS-MUTANSStreptococcus mutansMICROBIOME030104 developmental biologyBiotechnology & Applied MicrobiologyMetagenomicslcsh:QR100-130Life Sciences & BiomedicineDysbiosisRESISTANCEBacteriaOxidative stressBiotechnology
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Salivary Stress/Immunological Markers in Crohn’s Disease and Ulcerative Colitis

2020

There is continuous and growing interest in research into new alternatives to standard biomarkers to detect and follow-up disease, reducing physical and psychological stress in patients needing regular and invasive medical examinations for the evaluation of pathologies, including inflammatory bowel diseases (IBD). Saliva is one of the most promising body fluids in the research of new biomarkers, thanks to the large number of molecules it contains. Many molecules present in saliva are often directly correlated to their concentration in the blood but may be affected by the condition of the oral cavity. This means that a careful selection of a specific biomarker is required for each pathology,…

0301 basic medicineSalivaReviewDiseaseInflammatory bowel diseaseCatalysislcsh:ChemistryInorganic Chemistrystress03 medical and health sciences0302 clinical medicineImmune systemCrohn Diseaseinflammatory bowel diseasemedicineHumansoxidative stressSalivary Proteins and PeptidesPhysical and Theoretical Chemistrylcsh:QH301-705.5Molecular BiologySpectroscopysalivaCrohn's diseasebusiness.industryOrganic Chemistry030206 dentistryGeneral Medicinemedicine.diseaseUlcerative colitisdigestive system diseasescytokinesComputer Science Applicationsimmune system030104 developmental biologylcsh:Biology (General)lcsh:QD1-999ImmunologyBiomarker (medicine)Colitis UlcerativeCalprotectinbusinessBiomarkersInternational Journal of Molecular Sciences
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Heat- and pH-induced BSA conformational changes, hydrogel formation and application as 3D cell scaffold

2016

Aggregation and gelation of globular proteins can be an advantage to generate new forms of nanoscale biomaterials based on the fibrillar architecture. Here, we report results obtained by exploiting the proteins' natural tendency to self-organize in 3D network, for the production of new material based on BSA for medical application. In particular, at five different pH values the conformational and structural changes of the BSA during all the steps of the thermal aggregation and gelation have been analyzed by FTIR spectroscopy. The macroscopic mechanical properties of these hydrogels have been obtained by rheological measurements. The microscopic structure of the gels have been studied by AFM…

0301 basic medicineScaffoldHot TemperatureNanostructureBSACell SurvivalProtein ConformationGlobular proteinBiophysics?-aggregatesBiocompatible Materials02 engineering and technologymacromolecular substancesMicroscopy Atomic ForceBiochemistryMice03 medical and health sciencesProtein structureRheologySpectroscopy Fourier Transform Infraredβ-aggregateAnimalsCell-scaffoldFourier transform infrared spectroscopyMolecular BiologyNanoscopic scalechemistry.chemical_classificationTissue ScaffoldsChemistrySerum Albumin BovineHydrogelsHydrogen-Ion Concentration021001 nanoscience & nanotechnologySettore FIS/07 - Fisica Applicata(Beni Culturali Ambientali Biol.e Medicin)NanostructuresHydrogelCrystallography030104 developmental biologyMechanical spectraBiophysicChemical engineeringFTIRSelf-healing hydrogelsMicroscopy Electron ScanningCattleStress MechanicalRheology0210 nano-technology
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Large strain stimulation promotes extracellular matrix production and stiffness in an elastomeric scaffold model

2016

Mechanical conditioning of engineered tissue constructs is widely recognized as one of the most relevant methods to enhance tissue accretion and microstructure, leading to improved mechanical behaviors. The understanding of the underlying mechanisms remains rather limited, restricting the development of in silico models of these phenomena, and the translation of engineered tissues into clinical application. In the present study, we examined the role of large strip-biaxial strains (up to 50%) on ECM synthesis by vascular smooth muscle cells (VSMCs) micro-integrated into electrospun polyester urethane urea (PEUU) constructs over the course of 3 weeks. Experimental results indicated that VSMC …

0301 basic medicineScaffoldVascular smooth muscleMaterials scienceIn silico0206 medical engineeringMyocytes Smooth MuscleBiomedical Engineering02 engineering and technologyECM (extracellular matrix)ArticleMuscle Smooth VascularBiomaterialsExtracellular matrix03 medical and health sciencesTissue engineeringmedicineAnimalsMechanical conditioningCells CulturedTissue EngineeringTissue ScaffoldsRational designStiffnessModels Theoretical020601 biomedical engineeringBiomaterialElasticityExtracellular MatrixPolyesterElastomeric scaffold030104 developmental biologyElastomersRats Inbred LewMechanics of MaterialsBiophysicsCollagenStress Mechanicalmedicine.symptomMechanical propertieBiomedical engineering
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The SCO1731 methyltransferase modulates actinorhodin production and morphological differentiation of Streptomyces coelicolor A3(2)

2018

AbstractStreptomyces coelicolor is a Gram-positive microorganism often used as a model of physiological and morphological differentiation in streptomycetes, prolific producers of secondary metabolites with important biological activities. In the present study, we analysed Streptomyces coelicolor growth and differentiation in the presence of the hypo-methylating agent 5′-aza-2′-deoxycytidine (5-aza-dC) in order to investigate whether cytosine methylation has a role in differentiation. We found that cytosine demethylation caused a delay in spore germination, aerial mycelium development, sporulation, as well as a massive impairment of actinorhodin production. Thus, we searched for putative DNA…

0301 basic medicineScienceMutantAnthraquinonesStreptomyces coelicolorDecitabineSettore BIO/19 - Microbiologia GeneraleDNA methyltransferaseArticleActinorhodin03 medical and health scienceschemistry.chemical_compoundBacterial ProteinsSpore germinationSpores BacterialRegulation of gene expressionMultidisciplinaryMyceliumbiologyStreptomyces coelicolorfungiQRActinorhodin ProductionCell DifferentiationGene Expression Regulation BacterialMethyltransferasesbiology.organism_classificationTn5 Mutant Strains030104 developmental biologychemistryBiochemistryHypomethylating AgentsStreptomyces coelicolor bacterial differentiation epigenetic cytosine methylationDNA methylationMedicineCytosineCytosine Methylation
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Vascular ageing and endothelial cell senescence: Molecular mechanisms of physiology and diseases

2016

Ageing leads to a progressive deterioration of structure and function of all organs over the time. During this process endothelial cells undergo senescence and manifest significant changes in their properties, resulting in impairment of the vascular functionality and neo-angiogenic capability. This ageing-dependent impairment of endothelial functions is considered a key factor contributing to vascular dysfunctions, which is responsible of several age-related diseases of the vascular system and other organs. Several mechanisms have been described to control ageing-related endothelial cell senescence including microRNAs, mitochondrial dysfunction and micro environmental stressors, such as hyp…

0301 basic medicineSenescenceAgingEndotheliump73Biologymedicine.disease_cause03 medical and health sciencesEndotheliocyte; Endothelium; Hypoxia; MicroRNAs; Mitochondrial dysfunction; Oxidative stress; P53 family; P73; Transglutaminase 2; VEGF; Aging; Developmental BiologymicroRNAmedicineAnimalsHumansSettore MED/05 - Patologia ClinicaEndotheliocyte; Endothelium; Hypoxia; Mitochondrial dysfunction; Oxidative stress; Transglutaminase 2; VEGF; microRNAs; p53 family; p73Vascular DiseasesEndotheliumHypoxiaCellular SenescenceEndothelial CellsMicroRNASettore MED/23 - Chirurgia CardiacaBECN1Hypoxia (medical)VEGFMitochondriamicroRNAsEndothelial stem cellTransglutaminase 2030104 developmental biologymedicine.anatomical_structureOxidative stressAgeingImmunologyOxidative stremedicine.symptomMitochondrial dysfunctionp53 familyEndotheliocyteNeuroscienceOxidative stressDevelopmental BiologyMechanisms of Ageing and Development
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Paracrine effects of human adipose-derived mesenchymal stem cells in inflammatory stress-induced senescence features of osteoarthritic chondrocytes

2016

Aging and exposure to stress would determine the chondrocyte phenotype in osteoarthritis (OA). In particular, chronic inflammation may contribute to stress-induced senescence of chondrocytes and cartilage degeneration during OA progression. Recent studies have shown that adipose-derived mesenchymal stem cells exert paracrine effects protecting against degenerative changes in chondrocytes. We have investigated whether the conditioned medium (CM) from adipose-derived mesenchymal stem cells may regulate senescence features induced by inflammatory stress in OA chondrocytes. Our results indicate that CM down-regulated senescence markers induced by interleukin-1β including senescence-associated β…

0301 basic medicineSenescenceAgingPathologymedicine.medical_specialtyadipose-derived mesenchymal stem cells conditioned mediumsenescenceCaveolin 1chondrocytesAdipose tissueInflammationmedicine.disease_cause03 medical and health sciencesParacrine signalling0302 clinical medicineOsteoarthritisParacrine CommunicationmedicineHumansCellular SenescenceInflammation030203 arthritis & rheumatologybiologySirtuin 1KinaseMesenchymal stem cellMesenchymal Stem CellsCell Biologybeta-GalactosidaseCell biologyOxidative Stress030104 developmental biologyAdipose Tissuebiology.proteinmedicine.symptomOxidative stressResearch PaperAging
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Oxidative stress, autophagy, epigenetic changes and regulation by miRNAs as potential therapeutic targets in osteoarthritis

2015

Aging is a natural process characterized by the declining ability of the different organs and tissues to respond to stress, increasing homeostatic imbalance and risk of disease. Osteoarthritis (OA) is a multifactorial disease in which cartilage degradation is a central feature. Aging is the main risk factor for OA. In OA cartilage, a decrease in the number of chondrocytes and in their ability to regenerate the extracellular matrix and adequately respond to stress has been described. OA chondrocytes show a senescence secretory phenotype (SSP) consisting on the overproduction of cytokines (interleukins 1 and 6), growth factors (e.g., epidermal growth factor) and matrix metalloproteinases (MMP…

0301 basic medicineSenescenceMAPK/ERK pathwayAgingProgrammed cell deathDNA damageBiologymedicine.disease_causeBiochemistryChondrocyteEpigenesis Genetic03 medical and health sciencesChondrocytesOsteoarthritisAutophagymedicineAnimalsHumansMolecular Targeted TherapyEpigeneticsCellular SenescencePharmacologyAutophagyDNA MethylationCell biologyMicroRNAsOxidative Stress030104 developmental biologymedicine.anatomical_structureImmunologyReactive Oxygen SpeciesOxidative stressDNA DamageBiochemical Pharmacology
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Differential proteomics reveals age-dependent liver oxidative costs of innate immune activation in mice

2016

Abstract Individual response to an immune challenge results from the optimization of a trade-off between benefits and costs of immune cell activation. Age-related immune disorders may have several mechanistic bases, from immune cell defects to chronic pro-inflammatory status and oxidative imbalance, but we are still lacking experimental data showing the relative importance of each of these mechanisms. Using a proteomic approach and subsequent biochemical validations of proteomics-derived hypotheses, we found age-dependent regulations in the liver of 3-months and 1-year old-mice in response to an acute innate immune activation. Old mice presented a chronic up-regulation of several proteins i…

0301 basic medicineSenescenceMaleProteomicsAgingImmunosenescenceBiophysicsBiologyProteomicsmedicine.disease_causeBiochemistry[ SDE ] Environmental Sciences03 medical and health sciencesMice0302 clinical medicineImmune systemmedicineAnimalsOrganismInnate immune systemImmunosenescenceImmunity Innate3. Good healthAgeing030104 developmental biologyLiverAgeingOxidative stressImmunology[SDE]Environmental SciencesOxidation-Reduction030217 neurology & neurosurgeryOxidative stress
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Mediterranean nutraceutical foods: Strategy to improve vascular ageing.

2015

Ageing is characterized by a decline in all systemic functions. A greater susceptibility to apoptosis and senescence may contribute to proliferative and functional impairment of endothelial progenitor cells. They play an important role in neo-angiogenesis and endothelial repair. Vascular ageing is associated with changes in the structure and functions of vessels' wall. There are many possible causes of this damage. For sure, inflammation and oxidative stress play a fundamental role in the pathogenesis of endothelial dysfunction, commonly attributed to a reduced availability of nitric oxide. Inflammageing, the chronic low-grade inflammation that characterizes elderly people, aggravates vascu…

0301 basic medicineSenescencePathologymedicine.medical_specialtyAgingInflammationDisease030204 cardiovascular system & hematologyBioinformaticsmedicine.disease_causeDiet MediterraneanEndothelial progenitor cellPathogenesis03 medical and health sciences0302 clinical medicineNutraceuticalmedicineHumansVascular DiseasesEndothelial dysfunctionEndothelial Progenitor CellsSettore MED/04 - Patologia Generalebusiness.industryVascular ageingmedicine.diseaseInflammageing030104 developmental biologyAgeingNutraceuticalEndothelium Vascularmedicine.symptombusinessOxidative stressDevelopmental BiologyMechanisms of ageing and development
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