Search results for "tau proteins"

showing 10 items of 37 documents

Limited agreement between biomarkers of neuronal injury at different stages of Alzheimer's disease

2013

Abstract New diagnostic criteria for Alzheimer's disease (AD) treat different biomarkers of neuronal injury as equivalent. Here, we quantified the degree of agreement between hippocampal volume on structural magnetic resonance imaging, regional glucose metabolism on positron emission tomography, and levels of phosphorylated tau in cerebrospinal fluid (CSF) in 585 subjects from all phases of the AD Neuroimaging Initiative. The overall chance-corrected agreement was poor (Cohen κ, 0.24–0.34), in accord with a high rate of conflicting findings (26%–41%). Neither diagnosis nor APOE e4 status significantly influenced the distribution of agreement between the biomarkers. The degree of agreement t…

MalePathologymedicine.medical_specialtyEpidemiologytau ProteinsHippocampus03 medical and health sciencesCellular and Molecular NeuroscienceApolipoproteins E0302 clinical medicineAtrophyCerebrospinal fluidDevelopmental NeuroscienceNeuroimagingAlzheimer DiseaseFluorodeoxyglucose F18medicineHumansDementiaCognitive DysfunctionAged030304 developmental biology0303 health sciencesChi-Square Distributionmedicine.diagnostic_testHealth PolicyMiddle Agedmedicine.diseasePsychiatry and Mental healthPositron emission tomographyPositron-Emission TomographyBiomarker (medicine)FemaleNeurology (clinical)AtrophyGeriatrics and GerontologyAlzheimer's diseaseMental Status SchedulePsychologyChi-squared distributionBiomarkers030217 neurology & neurosurgeryAlzheimer's & Dementia
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Neocortical Variation of Abeta Load in Fully Expressed, Pure Alzheimer's Disease

2010

The relationship between amyloid-beta (A beta) deposition and tau-related neurofibrillary changes is a key issue in the pathogenesis of Alzheimer's disease (AD). The aim of this study was to investigate the extent and cortical distribution of A beta and tau pathology, their mutual links and their correlation with the duration of the disease in thirty-nine patients with fully expressed AD. By tau immunohistochemistry, we identified different patterns of distribution of neurofibrillary changes that were ascribed to Braak stage V and VI. The disease duration was longer in patients at Braak stage VI than in those at V. Morphometric analysis carried out in several neocortical areas demonstrated …

MalePathologymedicine.medical_specialtyTau proteinNeocortextau ProteinsPathogenesisSuperior temporal gyrusAlzheimer Diseasemental disordersmedicineHumansSenile plaquesAgedAged 80 and overNeocortexAmyloid beta-PeptidesbiologyGeneral NeuroscienceGeneral MedicinePsychiatry and Mental healthClinical Psychologymedicine.anatomical_structureGene Expression RegulationCerebral cortexbiology.proteinDisease ProgressionFemaleGeriatrics and GerontologyPrimary motor cortexPsychologyNeuroscienceBraak staging
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Association of elevated phospho-tau levels with alzheimer-typical 18F-Fluoro-2-Deoxy-D-Glucose positron emission tomography findings in patients with…

2003

Abstract Background Mild cognitive impairment is considered to be a transitional stage between normal aging and dementia. Phosphorylated tau protein in cerebrospinal fluid and even more decrements of cerebral glucose metabolism in parietal, temporal, or cingulate regions have shown favorable specificity for the diagnosis of Alzheimer dementia and could be useful supplementary tools to determine Alzheimer pathology in early stages. Methods We measured cerebrospinal fluid tau phosphorylated at threonine 181 protein, cerebrospinal fluid total tau, and cerebral glucose metabolism using 18F-fluoro-2-deoxy-D-glucose positron emission tomography in 16 patients with mild cognitive impairment and ag…

MalePathologymedicine.medical_specialtyTau proteintau ProteinsNeuropsychological TestsStatistics NonparametricCentral nervous system diseasechemistry.chemical_compoundCerebrospinal fluidAlzheimer DiseaseFluorodeoxyglucose F18Predictive Value of Testsmental disordersmedicineHumansDementiaPhosphorylationBiological PsychiatryAgedAged 80 and overBrain ChemistryBrain Mappingmedicine.diagnostic_testbiologybusiness.industryMiddle Agedmedicine.diseaseGlucosechemistryPositron emission tomographyCase-Control Studiesbiology.proteinBiomarker (medicine)FemaleAlzheimer's diseaseCognition Disorders2-Deoxy-D-glucosebusinessTomography Emission-ComputedBiological Psychiatry
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Assessment of cerebral microbleeds by susceptibility-weighted imaging in Alzheimer's disease patients: A neuroimaging biomarker of the disease.

2017

Purpose The objective of this study was to correlate the presence and distribution of cerebral microbleeds in Alzheimer’s disease patients with cerebrospinal fluid biomarkers (amyloid-beta and phosphorylated tau 181 protein levels) and cognitive decline by using susceptibility-weighted imaging magnetic resonance sequences at 1.5 T. Material and methods Fifty-four consecutive Alzheimer’s disease patients underwent brain magnetic resonance imaging at 1.5 T to assess the presence and distribution of cerebral microbleeds on susceptibility-weighted imaging images. The images were analyzed in consensus by two neuroradiologists, each with at least 10 years’ experience. Dementia severity was assess…

MalePathologymedicine.medical_specialtytau ProteinsDisease030218 nuclear medicine & medical imaging03 medical and health sciences0302 clinical medicineCerebrospinal fluidNeuroimagingAlzheimer DiseasemedicineHumansRadiology Nuclear Medicine and imagingAlzheimer's disease; Cerebral microbleeds; magnetic resonance imaging; susceptibility-weighted imaging; Radiology Nuclear Medicine and Imaging; Neurology (clinical)AgedCerebral HemorrhageAmyloid beta-Peptidesmedicine.diagnostic_testbusiness.industryCerebral microbleedBrainMagnetic resonance imagingGeneral MedicineAlzheimer's diseaseMagnetic Resonance Imagingsusceptibility-weighted imagingSusceptibility weighted imagingBiomarker (medicine)FemaleNeurology (clinical)business030217 neurology & neurosurgeryBiomarkersThe neuroradiology journal
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Cerebrospinal fluid tau protein is not a biological marker in amyotrophic lateral sclerosis.

2009

Background:  Amyotrophic lateral sclerosis (ALS) is a neurodegenerative disorder leading to progressive motor neuron cell death. Etiopathogenesis is still imperfectly known and much effort have been undertaken to find a biological marker that could help in the early diagnosis and in the monitoring of disease progression. Cerebrospinal fluid (CSF) concentrations of tau, an axonal microtubule-associated protein, have been measured in ALS with levels found increased in some studies and unchanged in others. Methods:  Total CSF tau level was assayed in a population of ALS patients (n = 57) and controls (n = 110) using a specific ELISA method. Results:  No significant differences in the median CS…

MaleProgrammed cell deathPathologymedicine.medical_specialtyTau proteinPopulationEnzyme-Linked Immunosorbent Assaytau Proteinscerebrospinal fluidtau proteinCerebrospinal fluiddisease progressionHumansMedicineamyotrophic lateral sclerosiAmyotrophic lateral sclerosisElisa methodeducationAgededucation.field_of_studybiologybusiness.industryAmyotrophic Lateral SclerosisDisease progressionMiddle AgedMotor neuronmedicine.diseasemedicine.anatomical_structureNeurologybiology.proteinamyotrophic lateral sclerosis cerebrospinal fluid disease progression tau proteinFemaleSettore MED/26 - NeurologiaNeurology (clinical)businessBiomarkers
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Stage-dependent agreement between cerebrospinal fluid proteins and FDG-PET findings in Alzheimer's disease.

2011

Cerebral hypometabolism and abnormal levels of amyloid beta (Aβ), total (t-tau) and phosphorylated tau (ptau) proteins in cerebrospinal fluid (CSF) are established biomarkers of Alzheimer's disease (AD). We examined the agreement between these biomarkers in a single center study of patients with AD of severity extending over a wide range. Forty seven patients (MMSE 21.4 ± 3.6, range 13-28 points) with incipient and probable AD underwent positron emission tomography with [18F]-fluorodeoxyglucose (FDG-PET) and lumbar puncture for CSF assays of Aβ1-42, p-tau181, and t-tau. All findings were classified as either positive or negative for AD. Statistical analyses were performed for the whole samp…

Malemedicine.medical_specialtyPathologyAmyloid betaApolipoprotein E4tau ProteinsNeuropsychological TestsGastroenterologySensitivity and SpecificityCerebrospinal fluidAlzheimer DiseaseFluorodeoxyglucose F18Internal medicinemental disordersmedicineDementiaHumansAgedRetrospective StudiesPsychiatric Status Rating ScalesAmyloid beta-Peptidesmedicine.diagnostic_testbiologyLumbar punctureNeurodegenerationCerebrospinal Fluid ProteinsMiddle Agedmedicine.diseasePeptide FragmentsNeurologyPositron emission tomographyArea Under CurvePositron-Emission Tomographybiology.proteinFemaleNeurology (clinical)Alzheimer's diseasePsychologyCognition DisordersKappaBiomarkersFollow-Up StudiesCurrent Alzheimer research
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Increased blood mercury levels in patients with Alzheimer's disease.

1998

Alzheimer's disease (AD) is a common neurodegenerative disorder that leads to dementia and death. In addition to several genetic parameters, various environmental factors may influence the risk of getting AD. In order to test whether blood levels of the heavy metal mercury are increased in AD, we measured blood mercury concentrations in AD patients (n = 33), and compared them to age-matched control patients with major depression (MD) (n = 45), as well as to an additional control group of patients with various non-psychiatric disorders (n = 65). Blood mercury levels were more than two-fold higher in AD patients as compared to both control groups (p = 0.0005, and p = 0.0000, respectively). In…

Malemedicine.medical_specialtyPathologyNeurologychemistry.chemical_elementtau ProteinsCentral nervous system diseaseDegenerative diseaseAlzheimer DiseaseInternal medicineBlood plasmamedicineDementiaHumansBiological PsychiatryAgedAged 80 and overDepressive DisorderAmyloid beta-PeptidesNeurodegenerationMercuryMiddle Agedmedicine.diseaseMercury (element)Psychiatry and Mental healthEndocrinologyNeurologychemistryLinear ModelsFemaleNeurology (clinical)Alzheimer's diseasePsychologyBiomarkersJournal of neural transmission (Vienna, Austria : 1996)
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CSF APPsα and Phosphorylated Tau Protein Levels in Mild Cognitive Impairment and Dementia of Alzheimer's Type

2008

We exploratively measured APPs alpha, a secreted fragment of the non-amyloidogenic cleavage of amyloid precursor protein via a-secretase, and tau protein phosphorylated at threonine 181 (p tau) in the cerebrospinal fluid of 10 patients with mild cognitive impairment, 20 patients with dementia of Alzheimer's type, and 10 controls. Cerebrospinal fluid APPs alpha and p tau levels were correlated with cognitive performance. P tau levels were significantly elevated in mild cognitive impairment and in patients with dementia of Alzheimer's type, APPs alpha levels were significantly reduced in patients with dementia of Alzheimer's type compared to the controls. APPs alpha levels were associated wit…

Malemedicine.medical_specialtyPathologyTau proteintau ProteinsNeuropsychological TestsSeverity of Illness IndexCerebrospinal fluidDegenerative diseaseAlzheimer DiseaseInternal medicineTask Performance and AnalysismedicineAmyloid precursor proteinHumansDementiaSex DistributionThreonineAgedAnalysis of VariancebiologyChemistrymedicine.diseasePsychiatry and Mental healthMemory Short-TermEndocrinologyMental Recallbiology.proteinPhosphorylationFemaleNeurology (clinical)Amyloid Precursor Protein SecretasesGeriatrics and GerontologyAlzheimer's diseaseCognition DisordersBiomarkersJournal of Geriatric Psychiatry and Neurology
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FDG-PET and CSF phospho-tau for prediction of cognitive decline in mild cognitive impairment

2006

Specific patterns of cortical glucose metabolism disturbances and increased CSF phospho-tau (p-tau(181)) concentrations could be demonstrated to predict cognitive decline and shift to dementia in amnestic mild cognitive impairment (MCI). But comparisons of both diagnostic tools have not been undertaken so far. The aim of the study was to compare (18)F-fluoro-2-deoxy-d-glucose positron emission tomography (FDG-PET) findings and CSF phospho-tau (p-tau(181)) measurements in the prediction of cognitive deterioration and conversion to dementia in MCI. During follow-up (mean 19 months) eight of 16 patients (50%) showed progressive cognitive decline, and four patients shifted to dementia. Patholog…

Malemedicine.medical_specialtyTau proteinNeuroscience (miscellaneous)tau ProteinsKaplan-Meier EstimateSeverity of Illness IndexStereotaxic TechniquesCentral nervous system diseaseImaging Three-DimensionalDegenerative diseaseFluorodeoxyglucose F18Internal medicinemental disordersSeverity of illnessImage Processing Computer-AssistedmedicineHumansDementiaRadiology Nuclear Medicine and imagingLongitudinal StudiesCognitive declineAgedPsychiatric Status Rating ScalesbiologyCognitive disorderPrognosismedicine.diseasePsychiatry and Mental healthPositron-Emission TomographyStereotaxic techniquebiology.proteinCardiologyDementiaFemaleCognition DisordersMental Status SchedulePsychologyNeuroscienceBiomarkersFollow-Up StudiesPsychiatry Research: Neuroimaging
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Glycogen synthase kinase 3β links neuroprotection by 17β-estradiol to key Alzheimer processes

2004

Estrogen exerts many of its receptor-mediated neuroprotective functions through the activation of various intracellular signal transduction pathways including the mitogen activating protein kinase (MAPK), phospho inositol-3 kinase and protein kinase C pathways. Here we have used a hippocampal slice culture model of kainic acid-induced neurotoxic cell death to show that estrogen can protect against oxidative cell death. We have previously shown that MAPK and glycogen synthase kinase-3beta (GSK-3beta) are involved in the cell death/cell survival induced by kainic acid. In this model and other cellular and in vivo models we have shown that estrogen can also cause the phosphorylation and hence …

Malemedicine.medical_specialtymedicine.drug_classBlotting WesternTetrazolium SaltsEstrogen receptorCell Counttau Proteinsmacromolecular substancesBiologyHippocampusRats Sprague-DawleyGlycogen Synthase Kinase 3MiceOrgan Culture TechniquesPregnancyGSK-3Internal medicineExcitatory Amino Acid AgonistsSerinemedicineAnimalsDrug InteractionsPhosphorylationProtein kinase AGSK3BCells CulturedProtein kinase CEstrogen receptor betaGlycogen Synthase Kinase 3 betaKainic AcidCell DeathEstradiolKinaseGeneral NeuroscienceAntibodies MonoclonalEmbryo MammalianImmunohistochemistryRatsCell biologyMice Inbred C57BLThiazolesEndocrinologyAnimals NewbornEstrogenTyrosineFemalePropidiumNeuroscience
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