Search results for "techniques"

showing 10 items of 4426 documents

Short- and long-latency muscarinic inhibition of noradrenaline release from rabbit atria induced by vagal stimulation.

1988

1. The influence of the time interval between vagal and sympathetic nerve stimuli on the magnitude of muscarinic inhibition of noradrenaline release was studied in the isolated perfused rabbit atria preparation. The transmitter stores were labelled with [14C]choline and [3H]noradrenaline. 2. The right cardiac postganglionic sympathetic nerves were stimulated at 3 Hz for 3 min three times at intervals of 10 min. The [3H]noradrenaline outflow evoked by the second stimulation equalled the averaged means of the log values of amine outflows evoked by the first and third stimulations. 3. During the second sympathetic stimulation the right vagus nerve was stimulated (3 Hz, 3 min) in such a way tha…

AtropineMalemedicine.medical_specialtySympathetic Nervous SystemTime FactorsPhysiologyAdrenergicTubocurarineStimulationIn Vitro TechniquesInhibitory postsynaptic potentialCholineNorepinephrineInternal medicineMuscarinic acetylcholine receptormedicineAnimalsChemistryMyocardiumVagus NerveReceptors MuscarinicAcetylcholineAtropineEndocrinologyCholinergicSilent periodFemaleRabbitsAcetylcholinemedicine.drugResearch Article
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A muscarinic inhibition of the noradrenaline release evoked by postganglionic sympathetic nerve stimulation

1969

1. The noradrenaline output from isolated rabbit hearts perfused with Tyrode solution was estimated fluorimetrically. The postganglionic sympathetic nerves of the heart were stimulated (10 shocks/sec; 1 msec) for three 1 min periods with intervals of 10 min. 2. The noradrenaline output evoked by 3 consecutive stimulation periods decreased exponentially. 3. Acetylcholine (10−9–10−6 g/ml) administered continuously one min before to one min after the second stimulation caused a dose-dependent reduction of the noradrenaline output evoked by the second stimulation to as low as 19% of the normal value. Acetylcholine in the concentrations applied did not cause a noradrenaline output by itself. 4. …

AtropineMalemedicine.medical_specialtySympathetic Nervous SystemTyramineStimulationHexamethonium CompoundsIn Vitro TechniquesPiperazinesNorepinephrinechemistry.chemical_compoundInternal medicineMuscarinic acetylcholine receptormedicineAnimalsMethacholine CompoundsFluorometryReceptors CholinergicPharmacologyHeartAdrenergic nervous systemGeneral MedicineCoronary VesselsAcetylcholineElectric StimulationReceptors AdrenergicPerfusionQuaternary Ammonium CompoundsAtropineEndocrinologyParasympathomimeticschemistryDepression ChemicalAutonomic Fibers PostganglionicCholinergicFemaleHexamethoniumMethacholineRabbitsAcetylcholinemedicine.drugNaunyn-Schmiedebergs Archiv f�r Pharmakologie
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Electrical stimulation of glossopharyngeal nerve and oesophageal EMG response in the pigeon

1985

The effects of the efferent glossopharyngeal nerve stimulation, on EMG activity of the pigeon cervical oesophagus, were studied. In control animals, stimulation caused a biphasic response characterized by an intra-stimulus excitatory component followed by a post-stimulus inhibitory one. The EMG response to glossopharyngeal stimulation appeared simultaneously throughout the cervical oesophagus. A bell-shaped mechanical wave was detected relating to the electrical excitatory component. Atropine administration antagonized the excitatory component, while the inhibitory one persisted. It occurs intra-stimulus, and its duration is increased, compared to control ones. A reduction in the oesophagea…

AtropinePhysiologyEfferentStimulationHexamethonium CompoundsElectromyographyIn Vitro TechniquesInhibitory postsynaptic potentialBiochemistrychemistry.chemical_compoundEsophagusmedicineAnimalsColumbidaeGlossopharyngeal Nervemedicine.diagnostic_testElectromyographybusiness.industryElectric Stimulationmedicine.anatomical_structurechemistryPeripheral nervous systemAnesthesiaGlossopharyngeal nerveExcitatory postsynaptic potentialHexamethoniumbusinessArchives Internationales de Physiologie et de Biochimie
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Cholinesterase activity and exposure time to acetylcholine as factors influencing the muscarinic inhibition of [3H]-noradrenaline overflow from guine…

1985

Guinea-pig isolated atria were incubated and loaded with [3H]-noradrenaline. The release of 3H and of [3H]-noradrenaline was induced by field stimulation (6-9 trains of 150 pulses at 5 Hz). The stimulation-evoked overflows of 3H and of [3H]-noradrenaline were determined. In the absence of an inhibitor of acetylcholinesterase, acetylcholine (12 min preincubation before nerve stimulation, up to 10 microM) failed to inhibit the evoked [3H]-noradrenaline overflow. In the presence of atropine, an increase by acetylcholine of evoked release was observed in the same atria. In contrast, the selective muscarinic agonist methacholine significantly decreased the evoked overflow. The inhibition was ant…

AtropinePhysostigminemedicine.medical_specialtyTime FactorsPhysostigmineGuinea PigsHexamethonium CompoundsIn Vitro TechniquesHexamethoniumMuscarinic agonistNorepinephrinechemistry.chemical_compoundCocaineInternal medicineMuscarinic acetylcholine receptormedicineAnimalsMethacholine CompoundsDrug InteractionsHeart AtriaPhentolamineMethacholine ChlorideCholinesterasePharmacologybiologyHeartPropranololReceptors MuscarinicAcetylcholineAtropineEndocrinologychemistryAcetylcholinesterasebiology.proteinMethacholineHexamethoniumCorticosteroneAcetylcholineResearch Articlemedicine.drugBritish Journal of Pharmacology
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Acetylcholine via Muscarinic Receptors Inhibits Histamine Release from Human Isolated Bronchi

1997

Human bronchi were incubated in organ baths to measure histamine release. The calcium ionophore A23187 (10 mumol/L; 1 min) stimulated histamine release by 148 +/- 28% (n = 11) above the prestimulation level but was ineffective in epithelium-denuded bronchi. Neither bradykinin (0.1 mumol/L) nor compound 48/80 (10 micrograms/ml) triggered the release of histamine from epithelium-intact bronchi. Acetylcholine did not affect spontaneous histamine release (about 2 nmol/g x 5 min) but inhibited A23187-evoked histamine release in an atropine-sensitive manner. Already a concentration as low as 0.1 nmol/L acetylcholine was effective, the maximal inhibition (by 89%) occurred at 100 nmol/L, whereas a …

AtropinePulmonary and Respiratory MedicineAgonistPhysostigminemedicine.medical_specialtyTime Factorsmedicine.drug_classPhysostigmineBradykininBronchiMuscarinic AntagonistsMuscarinic AgonistsCritical Care and Intensive Care MedicineHistamine Releasechemistry.chemical_compoundCulture TechniquesInternal medicineMuscarinic acetylcholine receptormedicineOxotremorineHumansDrug InteractionsCalcimycinDose-Response Relationship DrugIonophoresbusiness.industryOxotremorineImmunoglobulin EReceptors MuscarinicAcetylcholineEndocrinologychemistryAcetylcholinesterase inhibitorDepression ChemicalCholinesterase InhibitorsbusinessAcetylcholineHistaminemedicine.drugAmerican Journal of Respiratory and Critical Care Medicine
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Release of [3H]Acetylcholine in Human Isolated Bronchi: Effect of Indomethacin on Muscarinic Autoinhibition

1995

Receptor-mediated regulation of acetylcholine release in the airways, particularly in humans, remains unclear. In the present study, the tissue content of acetylcholine and release of [3H]acetylcholine were measured in freshly dissected human bronchi obtained at thoracotomy. Large (main and lobar bronchi) and small (segmental and subsegmental bronchi) airways contained considerable amounts of endogenous acetylcholine (300 +/- 50 pmol/100 mg wet weight), whereas significantly less was found in lung parenchyma (60 +/- 30 pmol/100 mg). Isolated small bronchi incubated in an organ bath with the precursor [3H]choline synthesized significant amounts of [3H]acetylcholine (26,000 +/- 4,000 dpm/100 …

AtropinePulmonary and Respiratory Medicinemedicine.medical_specialtyIndomethacinStimulationBronchiIn Vitro TechniquesCritical Care and Intensive Care Medicinechemistry.chemical_compoundInternal medicineMuscarinic acetylcholine receptormedicineOxotremorineCholineHumansbusiness.industryOxotremorinerespiratory systemReceptors MuscarinicAcetylcholineElectric StimulationAtropineEndocrinologychemistryTetrodotoxinCholinergicbusinessAcetylcholinemedicine.drugAmerican Journal of Respiratory and Critical Care Medicine
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Delivering Inorganic and Organic Reagents and Enzymes from Zein and Developing Optical Sensors.

2018

Nowadays, interest in using environmentally friendly materials is increasing in many fields. However, the rational design of sensors with biodegradable materials is a challenge. The main aim of this work is to show the possibility of using zein, a protein from corn, as a biodegradable and low-cost material for immobilizing, stabilizing, and delivering different kind of reagents for developing optical sensors. Enzymes, metallic salts, and aromatic and small organic compounds were tested. In addition, different techniques of immobilization, entrapment and adsorption, were used, and different formats, such as solid devices and also multiwell platforms, were proposed. The capacity of zein for i…

AtropineZein02 engineering and technologyBiosensing Techniques01 natural sciencesZea maysAnalytical ChemistryPhosphatesAdsorptionHumanschemistry.chemical_classificationFerric hydroxamateChemistry010401 analytical chemistryRational designA proteinSubstrate (chemistry)021001 nanoscience & nanotechnologyEnzymes ImmobilizedCombinatorial chemistryEnvironmentally friendly0104 chemical sciencesEnzymeSpectrometry FluorescencePharmaceutical PreparationsReagentColorimetryIndicators and Reagents0210 nano-technologyAnalytical chemistry
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On the opioid receptor subtype inhibiting the evoked release of 3H-noradrenaline from guinea-pig atria in vitro

1986

1. Guinea-pig isolated atria were incubated and loaded with 3H-(−)-noradrenaline. The intrinsic nerves were stimulated with trains of 5 or 35 field pulses (4 Hz), and the evoked efflux of 3H-noradrenaline and of total tritium was determined in the presence of atropine, corticosterone, desipramine, and phentolamine by liquid scintillation spectrometry. 2. Ethylketocyclazocine (1.4 nmol/l, IC50), MR 2033 (9.1 nmol/l), dynorphin A (1–13) (25 nmol/l, peptidase inhibitors present), etorphine (71 nmol/l), and [d-Ala2, d-Leu5]-enkephalin (>10 μmol/l, peptidase inhibitors present) inhibited the stimulation-evoked efflux of 3H-noradrenaline in a concentration-dependent manner, but not morphine up to…

Atropinemedicine.medical_specialtyEthylketocyclazocinemedicine.drug_classGuinea PigsPopulationEthylketocyclazocine(+)-NaloxoneIn Vitro TechniquesPharmacologyBinding CompetitiveDynorphinsNorepinephrinechemistry.chemical_compoundOpioid receptorInternal medicinemedicineAnimalsCyclazocineHeart AtriaPhentolamineeducationEndogenous opioidPharmacologyeducation.field_of_studyMorphineNaloxoneMyocardiumReceptors Opioid kappaDesipramineEtorphineDynorphin AGeneral MedicineEnkephalin Leucine-2-AlaninePeptide FragmentsBenzomorphansEndocrinologyEtorphineOpioidchemistryReceptors OpioidSynapsesCorticosteroneEnkephalin Leucinemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Subtypes of muscarinic receptor on cholinergic nerves and atrial cells of chicken and guinea-pig hearts

1988

1. Electrically driven chicken and guinea-pig atria were used to investigate the negative inotropic effects of the muscarinic agonists methacholine and acetylcholine (ACh). The release of ACh from isolated hearts into the perfusate in response to (preganglionic) vagal or (pre- and postganglionic) field stimulation was bioassayed on the guinea-pig ileum or determined by labelling with [3H]-choline. 2. Concentration-response curves for the negative inotropic effect of methacholine were shifted to the right by pirenzepine in various concentrations (0.03 to 10 mumol l-1). The pA2 values were 7.76 in chicken atria and 6.53 in guinea-pig atria. Pirenzepine and atropine antagonized the negative in…

Atropinemedicine.medical_specialtyGuinea PigsTubocurarineStimulationIn Vitro TechniquesBiologyNeuroeffector junctionParasympathetic Nervous SystemInternal medicineMuscarinic acetylcholine receptormedicineAnimalsMethacholine CompoundsPharmacologyHeartVagus NervePirenzepineMyocardial ContractionReceptors MuscarinicPirenzepineAcetylcholineElectric StimulationVagus nerveAtropineEndocrinologyMethacholineChickensAcetylcholineResearch Articlemedicine.drugBritish Journal of Pharmacology
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Mode and mechanism of neurotensin action in rat proximal colon

1997

Abstract This study examined the mechanism of action of neurotensin on intraluminal pressure in rat proximal colon. The direct and indirect contractile response to neurotensin (100 nM) was abolished in Ca 2+ -free solution, and was antagonized by nifedipine (1–5–10 nM) and potentiated by Bay K 8644 (methyl 1,4-dihydro-2,6-dimethyl-3-nitro-4-(2-trifluoromethylphenyl)-pyridine-5-carboxylate) (10–100–1000 nM). Neurotensin, in the presence of nifedipine (10 nM) and atropine (1 μM), induced a tetrodotoxin-insensitive inhibitory effect, which was antagonized by SR 48692 (2[(1-(7-chloro-4-quinolinyl)-5-(2,6-dimethoxy-phenyl)pyrazol-3-yl) carbonyl amino]tricyclo (3.3.1.1. 3.7 ) decan-2-carboxylic a…

Atropinemedicine.medical_specialtyNifedipineColonchemistry.chemical_elementCholinergic AgonistsIn Vitro TechniquesCalciumInhibitory postsynaptic potentialApaminCholinergic Antagonistschemistry.chemical_compoundNifedipineInternal medicinemedicineAnimalsReceptors NeurotensinRats WistarNeurotensinPharmacologyChemistryMuscle Smooth3-Pyridinecarboxylic acid 14-dihydro-26-dimethyl-5-nitro-4-(2-(trifluoromethyl)phenyl)- Methyl esterBethanecholCalcium Channel BlockersRatsCalcium Channel AgonistsEndocrinologyApaminMechanism of actionQuinolinesExcitatory postsynaptic potentialBiophysicsPyrazolesCalciummedicine.symptomMuscle Contractionmedicine.drugMuscle contractionNeurotensinEuropean Journal of Pharmacology
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