Search results for "thrombus"

showing 10 items of 91 documents

Anticoagulant drugs in noncompaction: A mandatory therapy?

2008

BACKGROUND: Noncompaction of left ventricular myocardium is a rare congenital cardiomyopathy resulting from an incomplete myocardial morphogenesis that leads to the persistence of the embryonic myocardium. This condition is characterized by a thin compacted epicardial and an extremely thickened endocardial layer with prominent trabeculations and deep intertrabecular recesses. It is not clear, in noncompaction of myocardium, whether intertrabecular recesses could be responsible for thrombi formation and thromboembolic complications. METHODS: The prevalence of stroke and echocardiographic finding of thrombus was evaluated in a continuous series of 229 patients (men and women) affected by nonc…

MaleRegistrieTime FactorsEmbolismAdministration OralHeart VentricleRisk FactorsRegistriesStrokeIschemic strokeAnticoagulantCongenital cardiomyopathyGeneral MedicineMiddle AgedStrokeAnticoagulant drugsCardiologycardiovascular systemFemaleAnticoagulant drugs noncompactionCardiomyopathiesCardiology and Cardiovascular MedicineHumanAdultHeart Defects Congenitalmedicine.medical_specialtyStroke etiologyTime Factormedicine.drug_classIsolated left ventricular noncompactionHeart Ventriclesanticoagulant; embolism; ischemic stroke; Isolated left ventricular noncompactionInternal medicineThromboembolismmedicineHumanscardiovascular diseasesThrombusNoncompactionCardiomyopathiebusiness.industryRisk FactorAnticoagulantAnticoagulantsInfantmedicine.diseaseSettore MED/11 - Malattie Dell'Apparato CardiovascolareEmbolismIschemic strokeThrombuLeft ventricular myocardiumbusiness
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Acute coronary artery closure following intracoronary ultrasound examination

1995

Two patients undergoing intracoronary ultrasound examination were complicated by acute coronary artery closure. One of the complications was thought to be caused by intimal dissection and thrombus formation and the other was thought to be caused by intimal dissection and subsequent embolization. The complications were successfully managed conservatively in both cases. © 1995 Wiley-Liss, Inc.

Malemedicine.medical_specialtymedicine.medical_treatmentEmbolismCoronary DiseaseConstriction PathologicCoronary AngiographymedicineHumansEmbolizationThrombusUltrasonography InterventionalIntimal Dissectionbusiness.industryCoronary ThrombosisfungiMiddle AgedCoronary artery closuremedicine.diseaseCoronary VesselsThrombosisSurgeryAcute DiseaseCirculatory systemIntracoronary ultrasoundRadiologyCardiology and Cardiovascular MedicineComplicationbusinessCatheterization and Cardiovascular Diagnosis
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Benign and malignant mimickers of infiltrative hepatocellular carcinoma: tips and tricks for differential diagnosis on CT and MRI

2021

Hepatocellular carcinoma (HCC) may have an infiltrative appearance in about 8-20% of cases. Infiltrative HCC can be a challenging diagnosis and it is associated with the worst overall survival among HCC patients. Infiltrative HCC is characterized by the spread of multiple minute nodules throughout the liver, without a dominant one, ultimately resulting into macrovascular invasion. On CT and MRI, infiltrative HCC appears as an ill-defined, large mass, with variable degree of enhancement, and satellite neoplastic nodules in up to 52% of patients. On MRI, it may show restriction on diffusion weighted imaging, hyperintensity on T2- and hypointensity on T1-weighted images, and, if hepatobiliary …

Gadolinium DTPAmedicine.medical_specialtyCarcinoma HepatocellularHepatocellular carcinomaDifferential diagnosiContrast Media030218 nuclear medicine & medical imagingDiagnosis Differential03 medical and health sciences0302 clinical medicineMagnetic resonance imagingmedicineHumansRadiology Nuclear Medicine and imagingThrombusVeinComputed tomographyRetrospective Studiesmedicine.diagnostic_testbusiness.industryAmyloidosisLiver NeoplasmsMagnetic resonance imagingmedicine.diseaseThrombosisdigestive system diseasesHyperintensitymedicine.anatomical_structure030220 oncology & carcinogenesisHepatocellular carcinomaRadiologyDifferential diagnosisbusinessTomography X-Ray Computed
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Analysis of platelet function and dysfunction.

2014

SummaryAlthough platelets act as central players of haemostasis only their cross-talk with other blood cells, plasma factors and the vascular compartment enables the formation of a stable thrombus. Multiple activation processes and complex signalling networks are responsible for appropriate platelet function. Thus, a variety of platelet function tests are available for platelet research and diagnosis of platelet dysfunction. However, universal platelet function tests that are sensitive to all platelet function defects do not exist and therefore diagnostic algorithms for suspected platelet function disorders are still recommended in clinical practice.Based on the current knowledge of human p…

0301 basic medicineBlood PlateletsPlatelet Function TestsPlatelet dysfunctionPoint-of-care testingPoint-of-Care SystemsVascular compartment030204 cardiovascular system & hematologySensitivity and Specificity03 medical and health sciences0302 clinical medicineThrombinmedicineHumansPlateletThrombusbusiness.industryReproducibility of ResultsHematologymedicine.disease030104 developmental biologyPlatelet function testImmunologyBlood Platelet DisordersbusinessFunction (biology)medicine.drugHamostaseologie
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Crosstalk of the plasma contact system with bacteria.

2012

Activation of the plasma contact system triggers several cascade systems such as the kallikrein-kinin system, the intrinsic pathway of coagulation, the classical complement cascade and the fibrinolytic system. Recent studies have shown a critical role of the contact system for arterial and venous thrombus formation and thromboembolic disease. In contrast, the function of the contact system for host-defense reactions and its physiological functions have remained enigmatic. Experimental animal studies and clinical data have linked the contact system to bacterial infections with implications for sepsis disease. The present review summarizes the role of the contact system and its activation for…

Kallikrein-Kinin SystemVascular permeabilityBiologySepsisCapillary PermeabilitySepsismedicineAnimalsHumansComplement Pathway ClassicalThrombusBlood CoagulationFactor XIIFibrinInnate immune systemBacteriaFibrinolysisHematologyBacterial Infectionsmedicine.diseaseImmunity InnateComplement systemCrosstalk (biology)ImmunologySignal transductionSignal TransductionThrombosis research
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The gut microbiota: An emerging risk factor for cardiovascular and cerebrovascular disease

2018

Commensal gut microbiota have recently been implicated in cardiovascular disease (CVD) and cerebrovascular disease. Atherosclerotic plaque formation depends on the colonization status of the host. In addition to host nutrition and the related microbiota-dependent metabolic changes, activation of innate immune pathways triggers the development of atherosclerosis and supports arterial thrombosis. Gnotobiotic mouse models have uncovered that activation of Toll-like receptor-2 by gut microbial ligands supports von Willebrand factor-integrin mediated platelet deposition to the site of vascular injury. Depending on nutritional factors, the microbiota-derived choline-metabolite trimethylamine N-ox…

Blood Platelets0301 basic medicineIntegrinsEmerging riskImmunologyDiseaseBiologyGut floradigestive systemMethylaminesMice03 medical and health sciencesVon willebrandRisk Factorsvon Willebrand FactormedicineAnimalsHumansImmunology and AllergyPlateletSymbiosisInnate immune systemInfant NewbornInfantThrombosisAtherosclerosisbiology.organism_classificationmedicine.diseaseThrombosisImmunity InnatePlaque AtheroscleroticToll-Like Receptor 2Gastrointestinal MicrobiomeCerebrovascular Disorders030104 developmental biologyCardiovascular DiseasesArterial thrombusImmunologyEuropean Journal of Immunology
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Innate Effector-Memory T-Cell Activation Regulates Post-Thrombotic Vein Wall Inflammation and Thrombus Resolution

2016

Rationale: Immune cells play an important role during the generation and resolution of thrombosis. T cells are powerful regulators of immune and nonimmune cell function, however, their role in sterile inflammation in venous thrombosis has not been systematically examined. Objective: This study investigated the recruitment, activation, and inflammatory activity of T cells in deep vein thrombosis and its consequences for venous thrombus resolution. Methods and Results: CD4 + and CD8 + T cells infiltrate the thrombus and vein wall rapidly on deep vein thrombosis induction and remain in the tissue throughout the thrombus resolution. In the vein wall, recruited T cells largely consist of effect…

CD4-Positive T-Lymphocytes0301 basic medicineChemokineMice 129 StrainPhysiologyMice TransgenicInflammationCD8-Positive T-Lymphocytes030204 cardiovascular system & hematologyVaricose VeinsMice03 medical and health sciences0302 clinical medicineImmune systemmedicineAnimalsHumansThrombusVeinInflammationVenous ThrombosisbiologyEffector Memory T-CellThrombosismedicine.diseaseThrombosisImmunity InnateCell biologyMice Inbred C57BLVenous thrombosis030104 developmental biologymedicine.anatomical_structureImmunologybiology.proteinmedicine.symptomCardiology and Cardiovascular MedicineCirculation Research
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Plasminogen activator inhibitor‐1 augments damage by impairing fibrinolysis after traumatic brain injury

2019

Objective Plasminogen activator inhibitor-1 (PAI-1) is the key endogenous inhibitor of fibrinolysis, and enhances clot formation after injury. In traumatic brain injury, dysregulation of fibrinolysis may lead to sustained microthrombosis and accelerated lesion expansion. In the present study, we hypothesized that PAI-1 mediates post-traumatic malfunction of coagulation, with inhibition or genetic depletion of PAI-1 attenuating clot formation and lesion expansion after brain trauma. Methods We evaluated PAI-1 as a possible new target in a mouse controlled cortical impact (CCI) model of traumatic brain injury. We performed the pharmacological inhibition of PAI-1 with PAI-039 and stimulation b…

Male0301 basic medicineTraumatic brain injurymedicine.medical_treatmentBrain damagePharmacologyLesionMice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineBrain Injuries TraumaticSerpin E2FibrinolysisAnimalsMedicineThrombusResearch ArticlesIndoleacetic Acidsbusiness.industryFibrinolysisBrainmedicine.diseaseMice Inbred C57BL030104 developmental biologyNeurologychemistryPlasminogen activator inhibitor-1Neurology (clinical)medicine.symptombusinessPlasminogen activator030217 neurology & neurosurgeryIntravital microscopyResearch ArticleAnnals of Neurology
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Clot Composition Analysis as a Diagnostic Tool to Gain Insight into Ischemic Stroke Etiology: A Systematic Review

2021

Mechanical thrombectomy renders the occluding clot available for analysis. Insights into thrombus composition could help establish the stroke cause. We aimed to investigate the value of clot composition analysis as a complementary diagnostic tool in determining the etiology of large vessel occlusion (LVO) ischemic strokes (International Prospective Register of Systematic Reviews [PROSPERO] registration # CRD42020199436). Following the Preferred Reporting Items for Systematic Reviews and Meta-Analyses (PRISMA) guidelines, we ran searches on Medline (using the PubMed interface) and Web of Science for studies reporting analyses of thrombi retrieved from LVO stroke patients subjected to mechani…

medicine.medical_specialtyetiologyMEDLINEReviewComposition analysiscerebral thrombusCerebral thrombus Etiology Ischemic stroke Systematic review ThrombectomyEmbolussystematic reviewischemic strokeMedicineDiseases of the circulatory (Cardiovascular) systemcardiovascular diseasesThrombusIntensive care medicineStrokebusiness.industrymedicine.diseaseSystematic reviewthrombectomyRC666-701EtiologyNeurology (clinical)Cardiology and Cardiovascular MedicinebusinessCohort studyJournal of Stroke
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Effects of combined therapy of clopidogrel and aspirin in preventing thrombus formation on mechanical heart valves in an ex vivo rabbit model.

2002

Abstract Background: The aim of our study was to investigate the efficacy of the combination of clopidogrel and aspirin in the prevention of thrombus formation on artificial heart valves in an experimental rabbit model as compared to anticoagulation with warfarin. Methods: Studies were performed after oral administration of clopidogrel and aspirin in group I (n=9) for 5 days, after 5±2 days treatment with warfarin in group II (n=9) and without medication in group III (n=9). Leaflets from Sulzer Carbomedics bileaflet valves were placed in a flow chamber. The flow chamber was filled with blood in a continuous circulation between the carotid artery and the jugular vein. Results: In group III, …

MaleTiclopidineArtificial heart valveDrug Evaluation PreclinicalIn Vitro Techniquesmedicine.disease_causeJugular veinmedicineAnimalscardiovascular diseasesHeart valveThrombusAspirinAspirinbusiness.industryWarfarinThrombosisHematologymedicine.diseaseClopidogrelThrombosisClopidogrelMicroscopy Electronmedicine.anatomical_structureAnesthesiaHeart Valve Prosthesiscardiovascular systemDrug Therapy CombinationRabbitsWarfarinbusinessmedicine.drugThrombosis research
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