Search results for "toxin"

showing 10 items of 1434 documents

Pore formation by Vibrio cholerae cytolysin requires cholesterol in both monolayers of the target membrane

2007

Vibrio cholerae cytolysin (VCC) forms oligomeric transmembrane pores in cholesterol-rich membranes. To better understand this process, we used planar bilayer membranes. In symmetric membranes, the rate of the channel formation by VCC has a superlinear dependency on the cholesterol membrane fraction. Thus, more than one cholesterol molecule can facilitate VCC-pore formation. In asymmetric membranes, the rate of pore formation is limited by the leaflet with the lower cholesterol content. Methyl-beta-cyclodextrin, which removes cholesterol from membranes, rapidly inhibits VCC pore formation, even when it is added to the side opposite that of VCC addition. The results suggest that cholesterol i…

Pore Forming Cytotoxic Proteinsgenetic structuresLipid BilayersBiologymedicine.disease_causeBiochemistrychemistry.chemical_compoundMonolayermedicineAnimalsMoleculeVibrio choleraePore-forming toxinMembrane GlycoproteinsPerforinCholesterolbeta-CyclodextrinsGeneral Medicineeye diseasesTransmembrane proteinCholesterolMembraneBiochemistrychemistryVibrio choleraeBiophysicsCattlelipids (amino acids peptides and proteins)sense organsCytolysinBiochimie
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Correct oligomerization is a prerequisite for insertion of the central molecular domain of staphylococcal α-toxin into the lipid bilayer

1995

Staphylococcal alpha-toxin is a primarily hydrophilic molecule that binds as a monomer to target membranes and then aggregates to form amphiphilic oligomers that represent water-filled transmembrane channels. Current evidence indicates that a region located in the center of the molecule inserts deeply into the bilayer. In the present study, we sought to determine whether membrane insertion was triggered by the oligomerization process, and whether insertion correlated with pore formation. Double mutants of alpha-toxin were prepared in which His-35 was replaced by Arg, and cysteine residues were introduced at positions 69, 130 and 186. Substitution of His-35 with Arg rendered the toxin molecu…

Pore formationBacterial ToxinsLipid BilayersMolecular ConformationBiophysics(Staphylococcus)Arginineα-ToxinBiochemistryHemolysin ProteinsMembrane Lipidschemistry.chemical_compound2-NaphthylamineAmphiphileOligomerizationCysteineLipid bilayerFluorescent DyesTransmembrane channelsPore-forming toxinBilayerCell BiologyMembraneMonomerchemistryBiochemistryMutationPore-forming toxinBiophysicsMembrane insertionCysteineBiochimica et Biophysica Acta (BBA) - Biomembranes
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Mode of primary binding to target membranes and pore formation induced by Vibrio cholerae cytolysin (hemolysin).

1997

Vibrio cholerae cytolysin (VCC) is produced by many non-choleratoxigenic strains of V. cholerae, and possibly represents a relevant pathogenicity determinant of these bacteria. The protein is secreted as a pro-toxin that is proteolytically cleaved to yield the active toxin with a molecular mass of approximately 63 kDa. We here describe a simple procedure for preparative isolation of mature VCC from bacterial culture supernatants, and present information on its mode of binding and pore formation in biological membranes. At low concentrations, toxin monomers interact with a high-affinity binding site on highly susceptible rabbit erythrocytes. This as yet unidentified binding site is absent on…

Pore-forming toxinBinding SitesToxinCytotoxinsErythrocyte MembraneMolecular Sequence DataAerolysinHemolysinBiologymedicine.disease_causeBiochemistryTransmembrane proteinMolecular WeightBiochemistryVibrio choleraemedicineAnimalsHumansCytolysinAmino Acid SequenceRabbitsBinding siteVibrio choleraeEuropean journal of biochemistry
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Cholesterol Specificity of Some Heptameric β-Barrel Pore-Forming Bacterial Toxins: Structural and Functional Aspects

2010

Apart from the thiol-specific/cholesterol-dependent cytolysin family of toxins (see Chapter 20) there are a number of other unrelated bacterial toxins that also have an affinity for plasma membrane cholesterol. Emphasis is given here on the Vibrio cholerae cytolysin (VCC) and the cytolysins from related Vibrio species. The inhibition of the cytolytic activity of these toxins by prior incubation with extracellular cholesterol or low density lipoprotein emerges as a unifying feature, as does plasma membrane cholesterol depletion. Incubation of VCC with cholesterol produces a heptameric oligomer, which is not equivalent to the pre-pore since it is unable to penetrate the plasma membrane. In st…

Pore-forming toxinHemolysinmedicine.disease_causeOligomerchemistry.chemical_compoundMembranechemistryBiochemistryVibrio choleraeLow-density lipoproteinExtracellularmedicinelipids (amino acids peptides and proteins)Cytolysin
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Phasic GABAA-receptor activation is required to suppress epileptiform activity in the CA3 region of the immature rat hippocampus

2012

Summary Purpose:  Despite the consistent observation that γ-aminobutyric acid A (GABAA) receptors mediate excitatory responses at perinatal stages, the role of the GABAergic system in the generation of neonatal epileptiform activity remains controversial. Therefore, we analyzed whether tonic and phasic GABAergic transmission had differential effects on neuronal excitability during early development. Methods:  We performed whole cell patch-clamp and field potential recordings in the CA3 region of hippocampal slices from immature (postnatal day 4–7) rats to analyze the effect of specific antagonists and modulators of tonic and phasic GABAergic components on neuronal excitability. Key Findings…

Postsynaptic CurrentGABAA receptormusculoskeletal neural and ocular physiologyHippocampal formationTonic (physiology)chemistry.chemical_compoundnervous systemNeurologychemistryGabazinemedicineExcitatory postsynaptic potentialGABAergicNeurology (clinical)Neurosciencemedicine.drugPicrotoxinEpilepsia
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Application of White Mustard Bran and Flour on Bread as Natural Preservative Agents.

2021

In this study, the antifungal activity of white mustard bran (MB), a by-product of mustard (Sinapis alba) milling, and white mustard seed flour (MF) was tested against mycotoxigenic fungi in the agar diffusion method. The results obtained were posteriorly confirmed in a quantitative test, determining the minimum concentration of extract that inhibits the fungal growth (MIC) and the minimum concentration with fungicidal activity (MFC). Since MF demonstrated no antifungal activity, the MB was stored under different temperature conditions and storage time to determine its antifungal stability. Finally, an in situ assay was carried out, applying the MB as a natural ingredient into the dough to …

PreservativeHealth (social science)food.ingredientPlant Sciencelcsh:Chemical technologyShelf life01 natural sciencesHealth Professions (miscellaneous)MicrobiologyArticleIngredientchemistry.chemical_compound0404 agricultural biotechnologyfoodmustard branmycotoxinsby-productmycotoxigenic fungilcsh:TP1-1185Food scienceAgar diffusion testbakery productsbiologyBran010401 analytical chemistryfood and beverages04 agricultural and veterinary sciencesMustard seedbiology.organism_classification040401 food sciencemustard flour0104 chemical sciencesshelf-lifefood safetychemistrySodium propionateSinapis albaWhite mustardantifungal<i>Sinapis alba</i>Food ScienceFoods (Basel, Switzerland)
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Novel path to apoptosis: small transmembrane pores created by staphylococcal alpha-toxin in T lymphocytes evoke internucleosomal DNA degradation.

1994

Peripheral-blood human T lymphocytes were treated with Staphylococcus aureus alpha-toxin. Membrane permeabilization was assessed by measuring efflux of K+ and Rb+ and influx of Na+, Ca2+, and propidium iodide. Cellular ATP and [3H]thymidine incorporation following lectin stimulation were measured as parameters for cell viability. Internucleosomal cleavage characteristic of programmed cell death was assessed by agarose gel electrophoresis and by quantifying low-molecular-weight, [3H]thymidine-labeled DNA fragments. Nanomolar concentrations of alpha-toxin evoked protracted, irreversible ATP depletion in both activated and resting T lymphocytes. Toxin-damaged cells also lost their ability to i…

Programmed cell deathCell Membrane PermeabilityStaphylococcusT-LymphocytesImmunologyBacterial ToxinsApoptosisBiologyMicrobiologychemistry.chemical_compoundHemolysin ProteinsAdenosine TriphosphateHumansPropidium iodideViability assaySodiumT lymphocyteDNANucleosomesInfectious DiseaseschemistryBiochemistryApoptosisAgarose gel electrophoresisBiophysicsPotassiumParasitologyCalciumThymidineAdenosine triphosphateResearch Article
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Pro-inflammatory T helper 17 directly harms oligodendrocytes in neuroinflammation.

2021

Significance Multiple sclerosis (MS) is a neuroinflammatory, demyelinating disease that represents one of the most frequent causes of irreversible disability in young adults. Treatment options to halt disability are limited. We discovered that T helper (Th)17 cells in contact with oligodendrocytes produce higher levels of glutamate and induce significantly greater oligodendrocyte damage than their Th2 counterpart. Blockade of CD29, which is linked to glutamate release pathways and expressed in high levels on Th17 cells, preserved human oligodendrocyte processes from Th17-mediated injury. Our data thus provide evidence for the direct and deleterious attack of Th17 cells on the myelin compart…

Programmed cell deathEncephalomyelitis Autoimmune ExperimentalCentral nervous systemFreund's AdjuvantoligodendrocytesMice Transgenicglutamate03 medical and health sciencesMyelinMice0302 clinical medicineImmunology and Inflammationintravital microscopymedicineAnimalsNeuroinflammation030304 developmental biologyInflammationMice Knockout0303 health sciencesMultidisciplinaryChemistryMultiple sclerosisGlutamate receptorMembrane ProteinsCD29Biological SciencesCD29 blockademedicine.disease420Oligodendrocyte3. Good healthCell biologyDNA-Binding ProteinsMice Inbred C57BLOligodendrogliamedicine.anatomical_structurePertussis ToxinTh17 CellsMyelin-Oligodendrocyte Glycoprotein030217 neurology & neurosurgeryProceedings of the National Academy of Sciences of the United States of America
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Natural and induced apoptosis during lymphocyte development in the axolotl

1999

Lymphocytes apoptosis was characterized in a urodele amphibian, the axolotl, by morphology using electron microscopy and by flow cytometry after propidium iodide staining, as well as by biochemical criteria with the detection of DNA ladders after glucocorticoid treatment. The morphological and biochemical features observed in treated axolotls are in accordance with the criteria of apoptosis found in different models of mammalian lymphocyte programmed cell death. The onset of natural apoptosis was then detected by DNA fragmentation in thymus and in spleen during lymphocyte development and ontogenesis. A typical DNA ladder characteristic of apoptosis is detectable in the thymus as early as 5 …

Programmed cell deathHydrocortisoneT-LymphocytesLymphocyteImmunologyApoptosisBiologyAmbystomaFlow cytometryEnterotoxinschemistry.chemical_compoundAxolotlmedicineSuperantigenAnimalsLymphocytesPropidium iodideSuperantigensmedicine.diagnostic_testCell Differentiationbiology.organism_classificationMolecular biologymedicine.anatomical_structurechemistryApoptosisLarvaDNA fragmentationDevelopmental BiologyDevelopmental &amp; Comparative Immunology
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Excitotoxin-induced changes in transglutaminase during differentiation of cerebellar granule cells

2002

Excitotoxicity induced by NMDA receptor stimulation is able to increase the activity of many enzymes involved in neuronal cell death. Primary cultures of rat cerebellar granule cells were used to elucidate the role of transglutaminase reaction in the excitotoxic cell response, and to evaluate the role of glutamate receptors in cell survival and degeneration. Granule neurons, maintained in vitro for two weeks, were exposed to NMDA at different stages of differentiation. Following NMDA receptor activation, increases in transglutaminase activity were observed in cell cultures. The levels of enzyme activity were higher in cells at 5 days in vitro than in those at 8-9 or 13-14 days in vitro. Mor…

Programmed cell deathN-MethylaspartateTime FactorsCell SurvivalTissue transglutaminaseNeurotoxinsClinical BiochemistryExcitotoxicityStimulationmedicine.disease_causeReceptors N-Methyl-D-AspartateBiochemistryCerebellummedicineAnimalsRats WistarNeuronsTransglutaminasesbiologyOrganic ChemistryGlutamate receptorCell DifferentiationIn vitroRatsCell biologyAnimals Newbornnervous systemApoptosisNerve Degenerationbiology.proteinNMDA receptorTransglutaminase – Excitotoxicity – Neurodegenerative diseases – Apoptosis – Glutamate – Cerebellar granule neuronsAmino Acids
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