Search results for "toxin"

showing 10 items of 1434 documents

Altered functional connectivity in blepharospasm/orofacial dystonia

2017

Abstract Introduction Blepharospasm is characterized by involuntary eyelid spasms. It can be associated with perioral dystonia (Meige's syndrome or orofacial dystonia). We aimed at studying resting‐state functional brain connectivity in these patients and its potential modulation by therapeutic botulinum toxin injections. Methods We performed resting‐state functional MRI and a region of interest‐based analysis of functional connectivity in 13 patients with blepharospasm/Meige's syndrome in comparison to 13 healthy controls. Patients were studied before and 4 weeks after botulinum toxin treatment. Simultaneous facial electromyography was applied to control for involuntary facial movements. R…

Male0301 basic medicineCingulate cortexBotulinum ToxinsBlepharospasmNeurotoxinsBlepharospasmSomatosensory systemMeige's syndrome03 medical and health sciencesBehavioral Neuroscience0302 clinical medicineCortex (anatomy)medicineHumansOriginal ResearchAgedDystoniaBrain MappingElectromyographyPostcentral gyrusbusiness.industryfunctional connectivityBrainMeige's syndromeMiddle Agedmedicine.diseaseMagnetic Resonance ImagingBotulinum toxineye diseasesddc:DystoniaTreatment Outcome030104 developmental biologymedicine.anatomical_structureorofacial dystoniaDystonic DisordersFemaleresting‐state functional MRImedicine.symptombusinessNeuroscience030217 neurology & neurosurgerymedicine.drugBrain and Behavior
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Potassium channels contribute to the increased sensitivity of the rabbit carotid artery to hydrogen sulfide in diabetes

2019

Abstract Hydrogen sulfide (H2S) is a potential endothelium-derived hyperpolarizing factor (EDHF) and adventitium- or adipocyte-derived relaxing factor (ADRF) which vasorelaxant action is mediated by potassium channels. H2S could also play an important role in the pathophysiology of diabetic cardiovascular complications. The present study has investigated the influence of alloxan-induced diabetes on the role of potassium channels mediating the relaxant response of the rabbit carotid artery to NaHS, a donor of H2S. NaHS (10−8-3 × 10−5 M) relaxed phenylephrine-precontracted carotid arteries, with higher potency in diabetic than in control rabbits. The selective blockers of potassium channels c…

Male0301 basic medicinePotassium ChannelsCharybdotoxinCarotid arteriesHydrogen sulfidePharmacologyPotassium channelsDiabetes Mellitus ExperimentalGlibenclamide03 medical and health scienceschemistry.chemical_compound0302 clinical medicineDiabetes mellitusmedicineAnimalsHydrogen SulfidePharmacologyHydrogen sulfideDose-Response Relationship DrugChemistryDiabetesmedicine.diseasePathophysiologyPotassium channelVasodilationCarotid Arteries030104 developmental biologyRabbitsCarotid artery030217 neurology & neurosurgerymedicine.drugEuropean Journal of Pharmacology
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Effect of some indole derivatives on xenobiotic metabolism and xenobiotic-induced toxicity in cultured rat liver slices.

1999

In this study the effect of some indole derivatives on xenobiotic metabolizing enzymes and xenobiotic-induced toxicity has been examined in cultured precision-cut liver slices from male Sprague-Dawley rats. While treatment of rat liver slices for 72 hours with 2-200 microM of either indole-3-carbinol (I3C) or indole-3-acetonitrile (3-ICN) had little effect on cytochrome P-450 (CYP)-dependent enzyme activities, enzyme induction was observed after in vivo administration of I3C. The treatment of rat liver slices with 50 microM 3,3'-diindolylmethane (DIM; a dimer derived from I3C under acidic conditions) for 72 hours resulted in a marked induction of CYP-dependent enzyme activities. DIM appears…

Male33'-DiindolylmethaneAflatoxin B1IndolesCarcinogenicity TestsDiindolylmethaneIn Vitro TechniquesToxicologyXenobioticsRats Sprague-Dawleychemistry.chemical_compoundCytochrome P-450 Enzyme SystemAnimalsAnticarcinogenic AgentsDrug InteractionsEnzyme inducerMonocrotalinebiologyCytochrome P450General MedicineGlutathioneRatschemistryBiochemistryLiverToxicitybiology.proteinCarcinogensXenobioticDrug metabolismFood ScienceFood and chemical toxicology : an international journal published for the British Industrial Biological Research Association
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Vascular Leakage in Severe Dengue Virus Infections: A Potential Role for the Nonstructural Viral Protein NS1 and Complement

2006

Background Vascular leakage and shock are the major causes of death in patients with dengue hemorrhagic fever (DHF) and dengue shock syndrome (DSS). Thirty years ago, complement activation was proposed to be a key underlying event, but the cause of complement activation has remained unknown. Methods The major nonstructural dengue virus (DV) protein NS1 was tested for its capacity to activate human complement in its membrane-associated and soluble forms. Plasma samples from 163 patients with DV infection and from 19 patients with other febrile illnesses were prospectively analyzed for viral load and for levels of NS1 and complement-activation products. Blood and pleural fluids from 9 patient…

MaleAdolescentvirusesComplement C5aComplement Membrane Attack ComplexViral Nonstructural ProteinsDengue virusBiologyAntibodies Viralmedicine.disease_causeVirusCell LineDengue feverDenguemedicineHumansImmunology and AllergyAnaphylatoxinVascular DiseasesChildGlycoproteinsPleural Cavityvirus diseasesComplement System ProteinsDengue VirusViral Loadmedicine.diseaseVirologyComplement systemInfectious DiseasesCase-Control StudiesChild PreschoolImmunologybiology.proteinRNA ViralFemaleAntibodyComplement membrane attack complexViral loadThe Journal of Infectious Diseases
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Endothelial and neural factors functionally involved in the modulation of noradrenergic vasoconstriction in healthy pig internal mammary artery

2011

The role of endothelial and neural factors as modulators of neurogenic- and noradrenaline-induced vasoconstriction was examined in healthy pig internal mammary artery (IMA). Tetrodotoxin-, guanethidine-sensitive electrical field stimulation (EFS)-, and noradrenaline-elicited contractions were significantly diminished by prazosin (n=8, P0.001) and less so by rauwolscine, indicating functional α₁- and α₂-adrenoceptor-mediated noradrenergic innervation of the IMA. Endothelium removal reduced neurogenic (n=8, P0.01) but augmented noradrenaline responses (n=8, P0.01), suggesting the release of two endothelium-dependent factors with opposite effects. In the presence of endothelium, neurogenic and…

MaleAdrenergic Antagonistsmedicine.medical_specialtySympathetic Nervous SystemContraction (grammar)EndotheliumArginineSwineBlotting WesternMuscarinic AntagonistsIn Vitro TechniquesNitric OxideBiochemistryNitric oxideNorepinephrinePotassium Channels Calcium-Activatedchemistry.chemical_compoundNerve FibersKATP ChannelsInternal medicinePotassium Channel BlockersPrazosinmedicineAnimalsMammary ArteriesEndothelium-Dependent Relaxing FactorsPharmacologybusiness.industryImmunohistochemistryElectric StimulationPotassium channelmedicine.anatomical_structureEndocrinologychemistryPotassium Channels Voltage-GatedVasoconstrictionProstaglandinsTetrodotoxinEndothelium Vascularmedicine.symptombusinessVasoconstrictionmedicine.drugBiochemical Pharmacology
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Rat liver endothelial and Kupffer cell-mediated mutagenicity of polycyclic aromatic hydrocarbons and aflatoxin B1.

1990

The ability of isolated rat liver endothelial and Kupffer cells to activate benzo(a)pyrene (BP), trans-7,8-dihydroxy-7,8-dihydrobenzo(a)pyrene (DDBP), trans-1,2-dihydroxy-1,2-dihydrochrysene (DDCH), and aflatoxin B1 (AFB1) to mutagenic metabolites was assessed by means of a cell-mediated bacterial mutagenicity assay and compared with the ability of parenchymal cells to activate these compounds. Endothelial and Kupffer cells from untreated rats were able to activate AFB1 and DDBP; DDBP was activated even in the absence of an NADPH-generating system. Pretreating the animals with Aroclor 1254 strongly enhanced the mutagenicity of the dihydrodiol, whereas the mutagenicity of AFB1 showed a sligh…

MaleAflatoxin B1EndotheliumKupffer CellsLiver cytologyHealth Toxicology and MutagenesisIn Vitro TechniquesBiologychemistry.chemical_compoundAflatoxinsmedicineOrganoidAnimalsPolycyclic CompoundsTestosteroneBiotransformationCarcinogenKupffer cellPublic Health Environmental and Occupational Healthfood and beveragesRats Inbred StrainsRatsmedicine.anatomical_structureLiverBiochemistrychemistryBenzopyreneToxicityMicrosomeEndothelium VascularResearch ArticleMutagensEnvironmental Health Perspectives
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Interspecies differences in cancer susceptibility and toxicity.

1999

One of the most complex challenges to the toxicologist represents extrapolation from laboratory animals to humans. In this article, we review interspecies differences in metabolism and toxicity of heterocyclic amines, aflatoxin B1, 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD), and related compounds, endocrine disrupters, polycyclic aromatic hydrocarbons, tamoxifen, and digitoxin. As far as possible, extrapolations to human toxicity and carcinogenicity are performed. Humans may be more susceptible to the carcinogenic effect of heterocyclic amines than monkeys, rats, and mice. Especially, individuals with high CYP1A2 and 3A4 activities and the rapid acetylator phenotype may be expected to have …

MaleAflatoxinAflatoxin B1Cardiotonic AgentsPolychlorinated DibenzodioxinsAntineoplastic Agents HormonalHamsterEndocrine SystemPharmacologyToxicologychemistry.chemical_compoundMiceDigitoxinSpecies SpecificityHeterocyclic CompoundsCricetinaeNeoplasmsBenzo(a)pyreneAnimalsHumansPharmacology (medical)General Pharmacology Toxicology and PharmaceuticsCarcinogenCYP1A2EstrogensGlutathioneAntiestrogenRatsTamoxifenBenzo(a)pyrenechemistryToxicityMicrosomes LiverFemaleDisease SusceptibilityRabbitsDrug metabolism reviews
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Mechanisms involved in the chemoprevention of flavonoids

2001

Flavonoids, widespread in edible plants, have been studied extensively for their anticarcinogenic properties. However, only few studies have been done with these constituents being administered by the dietary route. In our research, the effects of feeding rats with flavone, flavanone, tangeretin, and quercetin were investigated on two steps of aflatoxin B1 (AFB1)-induced hepatocarcinogenesis (initiation and promotion). Nonpolar flavonoids such as flavone, flavanone and tangeretin administered through the initiation period, decreased the number of -gamma-glutamyl transpeptidase-preneoplastic foci. In the same conditions of administration, quercetin, a polyhydroxylated flavonoid, showed no pr…

MaleAflatoxinAflatoxin B1[SDV]Life Sciences [q-bio]Clinical BiochemistryFlavonoidChemopreventionBiochemistryFlavones03 medical and health scienceschemistry.chemical_compoundTangeretinCytosolLiver Neoplasms Experimental0302 clinical medicineAnimalsAnticarcinogenic Agentsheterocyclic compoundsRats WistarComputingMilieux_MISCELLANEOUS030304 developmental biologyFlavonoidschemistry.chemical_classification0303 health sciencesDNAGeneral MedicineGlutathioneFlavonesGlutathioneCANCERDietRats3. Good health[SDV] Life Sciences [q-bio]LiverchemistryBiochemistryPhenobarbital030220 oncology & carcinogenesisFlavanonesCarcinogensChemoprotectiveMolecular MedicineQuercetinQuercetinFlavanoneBioFactors
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DNA binding, adduct characterisation and metabolic activation of aflatoxin B1 catalysed by isolated rat liver parenchymal, Kupffer and endothelial ce…

1991

In vitro studies with rat liver parenchymal, Kupffer and endothelial cells isolated from male Sprague-Dawley rats were undertaken to investigate cell-specific bioactivation of aflatoxin B1, DNA binding and adduct formation. In the mutagenicity studies, using homogenates of all three separated liver cell populations (co-incubated with NADP+ and glucose-6-phosphate as cofactors for the cytochrome P-450 monooxygenase system) parenchymal, Kupffer and endothelial cells were able to activate aflatoxin B1 to a metabolite mutagenic to Salmonella typhimurium TA 98. In the case of nonparenchymal cells (i.e. Kupffer and endothelial cells) 10-fold higher concentrations of aflatoxin B1 had to be used to…

MaleAflatoxinAroclorsAflatoxin B1Kupffer CellsHealth Toxicology and MutagenesisMetaboliteBiologyIn Vitro TechniquesToxicologychemistry.chemical_compoundmedicineAnimalsTestosteroneEndotheliumBiotransformationMutagenicity TestsLiver cellKupffer cellfood and beveragesRats Inbred StrainsGeneral MedicineDNAMonooxygenaseChlorodiphenyl (54% Chlorine)In vitroRatsEndothelial stem cellmedicine.anatomical_structurechemistryBiochemistryLiverMicrosomeArchives of toxicology
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In vitro antimutagenic and in vivo anticlastogenic effects of carotenoids and solvent extracts from fruits and vegetables rich in carotenoids.

1998

The water insoluble residues of some carotenoid-rich fruits and vegetables, such as apricots, oranges, brussels sprouts, carrots, yellow-red peppers, and tomatoes, were sequentially extracted with n-hexane, dichloromethane, acetone, and 2-propanol, and solvent extracted materials were tested for inhibition of mutagenicities induced by aflatoxin B1 (AFB1), benzo[a]pyrene (BaP), 2-amino-3-methylimidazo[4,5-f]quinoline (IQ), and cyclophosphamide (CP) in histidine-deficient strains of Salmonella typhimurium. Antimutagenic activities were found in many extracts, but especially in the n-hexane extracts. For example, in the case of oranges, 100 microg of this extract reduced the bacterial mutageni…

MaleAflatoxinLuteinHealth Toxicology and MutagenesisRats Sprague-Dawleychemistry.chemical_compoundMiceStructure-Activity RelationshipAstaxanthinVegetablesGeneticsAnimalsCanthaxanthinFood scienceCarotenoidchemistry.chemical_classificationDose-Response Relationship DrugPlant Extractsfood and beveragesAntimutagenic AgentsCarotenoidsLycopeneRatsZeaxanthinchemistryBiochemistryFruitAntimutagenMutation research
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