Search results for "trace"

showing 10 items of 3218 documents

Mechanism of vascular relaxation by thaligrisine

2000

Abstract In the present study we examine the mechanism by which thaligrisine, a bisbenzyltetrahydroisoquinoline alkaloid, inhibits the contractile response of vascular smooth muscle. The work includes functional studies on rat isolated aorta and tail artery precontracted with noradrenaline or KCl. In other experiments rat aorta was precontracted by caffeine in the presence or absence of extracellular Ca 2 +. In order to assess whether thaligrisine interacts directly with calcium channel binding sites or with α-adrenoceptors we examined the effect of the alkaloid on [ 3 H]-(+)- cis diltiazem, [ 3 H]-nitrendipine and [ 3 H]-prazosin binding to cerebral cortical membranes. The functional studi…

AortaVascular smooth muscleStereochemistrychemistry.chemical_elementGeneral MedicineCalciumGeneral Biochemistry Genetics and Molecular BiologyTetrandrinechemistry.chemical_compoundchemistrymedicine.arteryExtracellularBiophysicsmedicineChannel blockerCalcium Channel BindingGeneral Pharmacology Toxicology and PharmaceuticsBinding siteLife Sciences
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Heart valve tissue engineering: how far is the bedside from the bench?

2015

Heart disease, including valve pathologies, is the leading cause of death worldwide. Despite the progress made thanks to improving transplantation techniques, a perfect valve substitute has not yet been developed: once a diseased valve is replaced with current technologies, the newly implanted valve still needs to be changed some time in the future. This situation is particularly dramatic in the case of children and young adults, because of the necessity of valve growth during the patient's life. Our review focuses on the current status of heart valve (HV) therapy and the challenges that must be solved in the development of new approaches based on tissue engineering. Scientists and physicia…

Aortic valveHeart diseaseSwine030204 cardiovascular system & hematology0302 clinical medicineHeart valve tissue engineeringHyaluronic AcidChildProsthetic valve0303 health sciencesMARROW-DERIVED CELLSTissue ScaffoldsFetal BloodHeart Valves3. Good healthmedicine.anatomical_structureHeart Valve ProsthesisCardiologyMolecular MedicineCollagenmedicine.medical_specialtyPULMONARY VALVEBONE-MARROWInduced Pluripotent Stem CellsVENTRICULAR OUTFLOW TRACTMESENCHYMAL STEM-CELLS03 medical and health sciencesTissue scaffoldsInternal medicineEXTRACELLULAR-MATRIXmedicineAnimalsHumansHeart valveIntensive care medicineENDOTHELIAL PROGENITOR CELLSMolecular Biology030304 developmental biologyBioprosthesisAORTIC-VALVEFibrinSheepTissue Engineeringbusiness.industryEndothelial Cellsmedicine.diseaseTransplantationPulmonary valveUMBILICAL-CORD BLOOD1182 Biochemistry cell and molecular biologybusinessHUMAN AMNIOTIC-FLUIDExpert Reviews in Molecular Medicine
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Constitutive modeling of ascending thoracic aortic aneurysms using microstructural parameters.

2016

Ascending thoracic aortic aneurysm (ATAA) has been associated with diminished biomechanical strength and disruption in the collagen fiber microarchitecture. Additionally, the congenital bicuspid aortic valve (BAV) leads to a distinct extracellular matrix structure that may be related to ATAA development at an earlier age than degenerative aneurysms arising in patients with the morphological normal tricuspid aortic valve (TAV). The purpose of this study was to model the fiber-reinforced mechanical response of ATAA specimens from patients with either BAV or TAV. This was achieved by combining image-analysis derived parameters of collagen fiber dispersion and alignment with tensile testing dat…

Aortic valvemedicine.medical_specialtyMaterials scienceBicuspid aortic valve0206 medical engineeringFinite Element AnalysisBiomedical EngineeringBiophysics02 engineering and technology030204 cardiovascular system & hematologyThoracic aortic aneurysmModels BiologicalArticleExtracellular matrixSettore ING-IND/14 - Progettazione Meccanica E Costruzione Di Macchine03 medical and health sciencesAortic aneurysmFinite Element Analysi0302 clinical medicineBicuspid aortic valveFinite elementmedicine.arteryInternal medicineTensile StrengthAscending aortamedicineHumansAortaMechanical PhenomenaAortaAortic aneurysmAortic Aneurysm ThoracicSettore ING-IND/34 - Bioingegneria IndustrialeBiomechanical strengthExtracellular matrixAnatomymedicine.disease020601 biomedical engineeringBiomechanical Phenomenamedicine.anatomical_structureBiophysicCardiologycardiovascular systemCollagenStress MechanicalAortic failureHumanMedical engineeringphysics
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Amyloid-β toxicity and tau hyperphosphorylation are linked via RCAN1 in Alzheimer's disease.

2011

Amyloid-β peptide (Aβ) toxicity and tau hyperphosphorylation are hallmarks of Alzheimer’s disease (AD). How their molecular relationships may affect the etiology, progression, and severity of the disease, however, has not been elucidated. We now report that incubation of foetal rat cortical neurons with Aβ up-regulates expression of the Regulator of Calcineurin gene RCAN1, and this is mediated by Aβ-induced oxidative stress. Calcineurin (PPP3CA) is a serine-threonine phosphatase that dephosphorylates tau. RCAN1 proteins inhibit this phosphatase activity of calcineurin. Increased expression of RCAN1 also causes up-regulation of glycogen synthase kinase-3beta (GSK3β), a tau kinase. Thus, incr…

Apolipoprotein EAdultMuscle Proteinstau ProteinsBiologymedicine.disease_causeTransfectionArticleDephosphorylationGlycogen Synthase Kinase 3GSK-3Alzheimer DiseasemedicineAnimalsHumansLymphocytesPhosphorylationRNA Small InterferingGSK3BCells CulturedChromatography High Pressure LiquidRegulation of gene expressionCerebral CortexNeuronsAmyloid beta-PeptidesGlycogen Synthase Kinase 3 betaGeneral NeuroscienceCalcineurinIntracellular Signaling Peptides and ProteinsGeneral MedicineMiddle Agedmedicine.diseaseEmbryo MammalianMolecular biologyGlutathionePeptide FragmentsCell biologyRatsCalcineurinDNA-Binding ProteinsPsychiatry and Mental healthClinical PsychologyOxidative StressGene Expression RegulationFemaleGeriatrics and GerontologyAlzheimer's diseaseOxidative stressJournal of Alzheimer's disease : JAD
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Functional role of the low-density lipoprotein receptor-related protein in Alzheimer's disease.

2006

Alzheimer’s disease (AD) is the most common age-related neurodegenerative disorder, characterized by neuronal loss, neurofibrillary tangle formation and the extracellular deposition of amyloid-β (Aβ) plaques. The amyloid precursor protein (APP) and the enzymes responsible for Aβ generation seem to be the base elements triggering the destructive processes. Initially, the low-density lipoprotein receptor-related protein (LRP) was genetically linked to AD and later it emerged to impact on many fundamental events related to this disease. LRP is not only involved in Aβ clearance but is also the major receptor of several AD-associated ligands, e.g. apolipoprotein E and α<sub>2</sub>-m…

Apolipoprotein EFunctional rolemedicine.medical_specialtyPathologybiologyChemistryDiseaseLRP1Amyloid beta-Protein PrecursorEndocrinologyNeurologyAlzheimer DiseaseInternal medicineLDL receptormedicineExtracellularAmyloid precursor proteinbiology.proteinNeurofibrillary tangle formationAnimalsHumansNeurology (clinical)LDL-Receptor Related ProteinsNeuro-degenerative diseases
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Reduced VLDL clearance in ApoeNpc1 mice is associated with increased Pcsk9 and Idol expression and decreased hepatic LDL-receptor levels

2010

Niemann-Pick type C1 (NPC1) promotes the transport of LDL receptor (LDL-R)-derived cholesterol from late endosomes/lysosomes to other cellular compartments. NPC1-deficient cells showed impaired regulation of liver_X receptor (LXR) and sterol regulatory element-binding protein (SREBP) target genes. We observed that Apoe(-/-)Npc1(-/-) mice displayed a marked increase in total plasma cholesterol mainly due to increased VLDL, reflecting decreased clearance. Although nuclear SREBP-2 and Ldlr mRNA levels were increased in Apoe(-/-)Npc1(-/-) liver, LDL-R protein levels were decreased in association with marked induction of proprotein convertase subtilisin/kexin type 9 (Pcsk9) and inducible degrade…

Apolipoprotein EreceptorCholesterol VLDLLDL/metabolismMacrophages Peritoneal/cytologyBiochemistryMiceEndocrinologyhemic and lymphatic diseasesReceptorsOrphan Nuclear Receptors/geneticspolycyclic compoundsnuclear receptorCells CulturedResearch ArticlesLiver X ReceptorsMice KnockoutCulturedSterol Regulatory Element Binding Protein 2/geneticslipoproteinSerine EndopeptidasesIntracellular Signaling Peptides and ProteinsLamin Type AOrphan Nuclear ReceptorsTriglycerides/bloodCholesterolLiverProteins/geneticsKexinlipids (amino acids peptides and proteins)Proprotein ConvertasesProprotein Convertase 9Sterol Regulatory Element Binding Protein 1Niemann-Pick diseaseSterol Regulatory Element Binding Protein 2medicine.medical_specialtyCellsKnockoutUbiquitin-Protein LigasesReceptors LDL/metabolismSerine Endopeptidases/geneticsQD415-436BiologyCholesterol/blooddigestive systemApolipoproteins ELiver/physiologySterol Regulatory Element Binding Protein 1/geneticsNiemann-Pick C1 ProteinInternal medicinemedicineAnimalsPeritoneal/cytologyCholesterol VLDL/metabolismUbiquitin-Protein Ligases/geneticsLiver X receptorTriglyceridesMacrophagesPCSK9Proteinsnutritional and metabolic diseasesVLDL/metabolismLamin Type A/metabolismCell BiologySterol regulatory element-binding proteinEndocrinologyReceptors LDLLDL receptorMacrophages PeritonealSterol regulatory element-binding protein 2atherosclerosisApolipoproteins E/geneticsLipoproteinJournal of Lipid Research
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Superoxide Flux in Endothelial Cells via the Chloride Channel-3 Mediates Intracellular Signaling

2007

Reactive oxygen species (ROS) have been implicated in both cell signaling and pathology. A major source of ROS in endothelial cells is NADPH oxidase, which generates superoxide (O2.−) on the extracellular side of the plasma membrane but can result in intracellular signaling. To study possible transmembrane flux of O2.−, pulmonary microvascular endothelial cells were preloaded with the O2.−-sensitive fluorophore hydroethidine (HE). Application of an extracellular bolus of O2.−resulted in rapid and concentration-dependent transient HE oxidation that was followed by a progressive and nonreversible increase in nuclear HE fluorescence. These fluorescence changes were inhibited by superoxide dism…

ApoptosisMembrane PotentialsSuperoxide dismutasechemistry.chemical_compoundChloride ChannelsSuperoxidesExtracellularAnimalsHumansEnzyme InhibitorsRNA Small InterferingMolecular BiologyLungCells CulturedFluorescent Dyeschemistry.chemical_classificationReactive oxygen speciesNADPH oxidasebiologySuperoxideAngiotensin IIThrombinAcetophenonesEndothelial CellsNADPH OxidasesCell BiologyArticlesCell biologyMitochondriaPhenanthridinesOxygenchemistryDIDSbiology.proteinCalciumSignal transductionOxidation-ReductionIntracellularSignal Transduction
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Polycyclic aromatic hydrocarbons and trace metal contamination of coastal sediment and biota from Togo

2011

The state of contamination of tropical environments, particularly in Africa, remains a relatively under explored subject. Here, we determined polycyclic aromatic hydrocarbon (PAH) and trace metal concentrations in coastal sediment and biota samples (fish and mussels) from Togo (West Africa). In the sediments, the ∑21 PAH concentrations ranged from4 ng g(-1) to 257 ng g(-1), averaging 92 ng g(-1). Concentration ratios of low molecular weight PAHs (2-3 rings) versus high molecular weight PAHs (≥4 rings) were always lower than 1 (ranging from 0.08 to 0.46) indicating that high molecular weight PAHs were dominant in all sediment samples, and that PAHs originated mainly from anthropogenic combus…

Aquatic OrganismsGeologic SedimentsPolycyclic aromatic hydrocarbonManagement Monitoring Policy and LawAnimalsSeawaterTrace metalPolycyclic Aromatic Hydrocarbonschemistry.chemical_classificationChemistryTerrigenous sedimentFishesPublic Health Environmental and Occupational HealthSedimentBiotaGeneral MedicineBiotaBivalviaPhosphoriteMetalsTogoEnvironmental chemistryBioaccumulationEnrichment factorWater Pollutants ChemicalEnvironmental MonitoringJournal of Environmental Monitoring
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Morphogenetic (Mucin Expression) as Well as Potential Anti-Corona Viral Activity of the Marine Secondary Metabolite Polyphosphate on A549 Cells

2020

The mucus layer of the nasopharynx and bronchial epithelium has a barrier function against inhaled pathogens such as the coronavirus SARS-CoV-2. We recently found that inorganic polyphosphate (polyP), a physiological, metabolic energy (ATP)-providing polymer released from blood platelets, blocks the binding of the receptor binding domain (RBD) to the cellular ACE2 receptor in vitro. PolyP is a marine natural product and is abundantly present in marine bacteria. Now, we have approached the in vivo situation by studying the effect of polyP on the human alveolar basal epithelial A549 cells in a mucus-like mucin environment. These cells express mucins as well as the ectoenzymes alkaline phospha…

Aquatic OrganismsSecondary MetabolismVirus AttachmentPharmaceutical ScienceAdenylate kinaseRespiratory MucosaMucin 5ACdigestive systemArticle03 medical and health sciences0302 clinical medicinemucinPolyphosphatesDrug Discoveryotorhinolaryngologic diseasesExtracellularHumansReceptorlcsh:QH301-705.5neoplasmsinnate immunityPharmacology Toxicology and Pharmaceutics (miscellaneous)MUC1030304 developmental biologyA549 cellBiological Products0303 health sciencesBacteriaSARS-CoV-2ChemistryCell growthMucin-1MucinCOVID-19polyphosphaterespiratory systemImmunity Innatedigestive system diseasesSARS-CoV-2 spike proteinADKCell biologyATPlcsh:Biology (General)A549 Cells030220 oncology & carcinogenesishydrogelhuman alveolar cellsMarine Drugs
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Public Reason and the Limits of Liberal Anti-Racism in Latvia

2011

My paper is a critical analysis of anti-racist and tolerance promotion initiatives in Latvia. First, I trace the historical and geopolitical conditions that enable the emergence of two discursive positions that are central to arguments about racism - that of liberally inclined tolerance activists and that of Latvians with politically objectionable nationalist sensibilities. Subsequently, I argue that, plagued by developmentalist thinking, anti-racist and tolerance promotion initiatives fail in their analysis of contemporary racism. They posit backward attitudes as the main hindrance to the eradication of racism and displace racism as a constitutive feature of modern political forms onto ind…

ArcheologyAnti-racismmedia_common.quotation_subjectGeopoliticsRacismNationalismTrace (semiology)PoliticsPromotion (rank)Arts and Humanities (miscellaneous)AnthropologyPolitical economyLawSociologyPublic reasonmedia_commonEthnos
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