Search results for "tritium"

showing 10 items of 165 documents

Proliferative response of cells of the dentogingival junction to mechanical stimulation

2001

SUMMARY The aim of this research was to study the proliferative response of junctional epithelium (JE) and gingival connective tissue (GCT) to mechanical stimulation in vivo with regard to the potential occurrence of apical migration of JE and loss of GCT attachment during orthodontic tooth movement. Elastic bands were inserted between the maxillary first and second molars of male rats aged 8 weeks, which were pulse-labelled with 3 H-thymidine and subsequently killed in groups, together with labelled control animals (a total of 98 rats) after periods of 1‐168 hours. Autoradiographs were prepared from plastic mesiodistal sections, and parameters of cell proliferation for JE and GCT of the pa…

MaleMolarPathologymedicine.medical_specialtyTooth Movement TechniquesPeriodontal LigamentEpithelial AttachmentGingivaJunctional epitheliumConnective tissueOrthodonticsStimulationTritiumStatistics NonparametricOrthodontic AppliancesCell MovementPressuremedicineAnimalsPeriodontal fiberConnective Tissue CellsAnalysis of VarianceHyperplasiaChemistryAnatomyHyperplasiaEpithelial Attachmentmedicine.diseaseMolarRatsMajor duodenal papillamedicine.anatomical_structureAutoradiographyStress MechanicalRadiopharmaceuticalsCell DivisionThymidineThe European Journal of Orthodontics
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A nuclear juvenile hormone-binding protein from larvae of Manduca sexta: a putative receptor for the metamorphic action of juvenile hormone

1994

0027-8424 (Print) Journal Article Research Support, Non-U.S. Gov't Research Support, U.S. Gov't, Non-P.H.S. Research Support, U.S. Gov't, P.H.S.; A 29-kDa nuclear juvenile hormone (JH)-binding protein from the epidermis of Manduca sexta larvae was purified by using the photoaffinity analog for JH II ([3H]epoxyhomofarnesyldiazoacetate) and partially sequenced. A 1.1-kb cDNA was isolated by using degenerate oligonucleotide primers for PCR based on these sequences. The cDNA encoded a 262-amino acid protein that showed no similarity with other known proteins, except for short stretches of the interphotoreceptor retinoid-binding protein, rhodopsin, and human nuclear protein p68. Recombinant bacu…

MaleMoths/growth & development/*metabolism/physiologyBase SequenceMetamorphosisPolymerase Chain Reaction/methodsSesquiterpenes/metabolismMolecular Sequence DataDNABiological/*physiologyTritiumJuvenile Hormones/metabolismMolecular WeightKineticsIsomerismOligodeoxyribonucleotidesLarvaAnimalsComplementary/isolation & purificationInsect ProteinsAmino Acid SequenceCarrier Proteins/genetics/isolation & purification/*metabolism
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Regulation of α4β2α5 nicotinic acetylcholinergic receptors in rat cerebral cortex in early and late adolescence: Sex differences in response to chron…

2013

Chronic nicotine administration in animals, and smoking in humans, causes up-regulation of α4β2* neuronal nicotinic receptors (nAChRs), which has been hypothesized to contribute to the addictive actions of nicotine. We used a rat model to test whether such up-regulatory effects differ in adolescents versus adults, and in males versus females. Following chronic treatment with nicotine or saline via subcutaneous osmotic minipumps, we measured α4β2 and α4β2α5 nAChRs in cerebral cortex using [3H]epibatidine to label assembled nAChRs, and selective antibodies to measure the individual subunits via immunoprecipitation. For the first time, we provide a detailed characterization of the response of …

MaleNicotinemedicine.medical_specialtyPyridinesmedia_common.quotation_subjectReceptors NicotinicTritiumArticleRats Sprague-DawleyNicotineCellular and Molecular NeuroscienceDownregulation and upregulationInternal medicinemedicineAnimalsImmunoprecipitationNicotinic AgonistsReceptormedia_commonCerebral CortexPharmacologySex CharacteristicsBinding SitesAddictionBridged Bicyclo Compounds HeterocyclicUp-RegulationNicotinic agonistEndocrinologymedicine.anatomical_structureCerebral cortexEpibatidineFemalePsychologymedicine.drugSex characteristicsNeuropharmacology
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PURINE AND PYRIMIDINE NUCLEOTIDES IN THE BRAIN OF NORMAL AND CONVULSANT RATS

1969

— Purine and pyrimidine nucleotides were measured in the brain of normal and electroshocked rats after chromatographic separation on ion-exchange resin of mono-, di- and tri-phosphorylated derivatives. CMP, IMP and NAD did not show any significant quantitative change. Adenine nucleotides showed an abrupt change followed by a rapid return to the control value. GTP was the only purine nucleotide exhibiting a relatively slow return to its starting concentration. The greatest percentage increase after electroshock was observed in UMP, which returned to its control value only after 5 min; UDPCoenzymes (i.e. UDPA plus UDPG) showed a relatively small drop during the development of the seizure and …

MalePurineGTP'Uracil NucleotidesCytosine NucleotidesTritiumBiochemistryCellular and Molecular Neurosciencechemistry.chemical_compoundAdenosine TriphosphateSeizuresAdenine nucleotideAnimalsNucleotidechemistry.chemical_classificationChromatographyElectroshockAdenine NucleotidesNucleotidesChemistryBrainNADGuanine NucleotidesUridineRatsPyrimidinesBiochemistryPurinesConvulsantIon Exchange ResinsNAD+ kinasePyrimidine NucleotidesJournal of Neurochemistry
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Effects of 5-HT4 receptor stimulation on basal and electrically evoked release of acetylcholine from guinea-pig myenteric plexus

1992

The effects of 5-methoxytryptamine and 5-hydroxytryptamine (5-HT) on both basal and electrically evoked outflow of tritium were studied in guinea-pig myenteric plexus preparations preincubated with [3H]-choline. Basal outflow. 5-Methoxytryptamine caused a transient and calcium-dependent increase in basal outflow of [3H]acetylcholine that was abolished by tetrodotoxin. Ondansetron (1 μmol/1) did not affect the stimulatory response of 5-methoxytryptamine but ICS 205-930 (1 and 3 μmol/1) produced parallel rightward displacements of the concentration-response curve to 5-methoxytryptamine. The PKB value for ICS 205-930 was 6.6 suggesting an involvement of 5-HT4 receptors. 5-HT caused an increase…

MaleSerotoninmedicine.medical_specialtyGuinea PigsNeuromuscular JunctionMyenteric Plexus5-HT4 receptorStimulationIn Vitro TechniquesBiologyTritium5-HT3 receptorCholine5-Methoxytryptaminechemistry.chemical_compoundIleumInternal medicinemedicineAnimalsReceptorMyenteric plexusPharmacologyMuscle SmoothGeneral MedicineSmooth muscle contractionAcetylcholineElectric StimulationStimulation ChemicalEndocrinologychemistryReceptors SerotoninMetitepinebiology.proteinFemaleCholinesterase InhibitorsAcetylcholinemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Thimerosal induces calcium mobilization, fructose 2,6-bisphosphate synthesis and cytoplasmic alkalinization in rat thymus lymphocytes

1991

The effect of thimerosal on intracellular calcium ([Ca2+]i), pH (pHi) and fructose 2,6-bisphosphate (Fru 2,6-P2) in thymus lymphocytes was investigated. The effect of thimerosal on cell growth was also examined. Thimerosal produced a dose-dependent increase in [Ca2+]i, pHi and in the level of fructose 2,6-bisphosphate. Thimerosal was, however, unable to produce cell proliferation and inhibited [3H]thymidine incorporation when cells were challenged with PHA and costimulator. In the absence of external calcium, thimerosal produced only a slight increase in [Ca2+]i. In Na(+)-containing buffer, thimerosal induced an initial acidification (0.05 +/- 0.01 pH units), followed by an alkalinization o…

Malemedicine.medical_specialtyCytoplasmT-LymphocytesFluorescence spectrometrychemistry.chemical_elementBiologyCalciumLymphocyte ActivationTritiumCalcium in biologychemistry.chemical_compoundInternal medicinemedicineFructosediphosphatesAnimalsLactic AcidMolecular BiologyIon transporterProtein kinase CThimerosalSodiumThimerosalFructoseRats Inbred StrainsCell BiologyHydrogen-Ion ConcentrationRatsEndocrinologyFructose 26-bisphosphatechemistryLactatesCalciumGlycolysisHydrogenThymidine
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Nicotine receptors do not modulate the 3H-noradrenaline release from the isolated rat heart evoked by sympathetic nerve stimulation.

1982

Isolated rat hearts with the right sympathetic nerves attached were perfused at a constant flow rate of 7 ml/min with Tyrode's solution. (-)-3H-Noradrenaline (final concentration 10–13.9 nM) was infused for 10 min to label the noradrenaline stores. After wash-out the sympathetic nerves were stimulated electrically (3 Hz, 180 impulses, 1 ms, 20–30 mA) three times (S1–S3) at intervals of 15 min. 3H-Noradrenaline and its metabolites were determined by liquid scintillation counting according to Graefe et al. (1973). Both, nicotine 50 μM and p-aminophenethyltrimethylammonium (PAPETA) 30 μM, enhanced the 3H-noradrenaline overflow in the absence of nerve stimulation. The effect of PAPETA was bipha…

Malemedicine.medical_specialtyNicotineSympathetic Nervous SystemSympathetic nerveStimulationIn Vitro TechniquesReceptors NicotinicTritiumReuptakeMethoxyhydroxyphenylglycol3h noradrenalineNicotinechemistry.chemical_compoundNorepinephrineInternal medicinemedicineAnimalsReceptors CholinergicReceptorPharmacologyNeuronsMyocardiumHeartRats Inbred StrainsGeneral MedicineRat heartElectric StimulationRatsQuaternary Ammonium CompoundsEndocrinologychemistryMandelic AcidsHexamethoniumFemalemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Suppression by cholinesterase inhibition of a Ca(2+)-independent efflux of [3H]acetylcholine from the neuromuscular junction of the isolated rat diap…

1992

Abstract Endplate preparations of the left rat hemidiaphragm were incubated with [ 3 H]choline to label neuronal acetylcholine stores. Elevation of the concentration (13.5–135 mmol/l) of extracellular potassium chloride (KC1) stimulated the release of [ 3 H]acetylcholine in a concentration-dependent manner. KC1 (27 mmol/l) still caused a significant efflux of [ 3 H]acetylcholine in a Ca 2+ -free medium. Inhibitors of cholinesterase (physostigmine, diisopropylfluorophosphate) suppressed by 80% this Ca 2+ -independent efflux of [ 3 H]acetylcholine. Vesamicol (10 μmol/l), the blocker of the vesicular acetylcholine carrier, also suppressed the stimulated, Ca 2+ -independent efflux of [ 3 H]acet…

Malemedicine.medical_specialtyPhysostigmineVesamicolPhysostigmineDiaphragmNeuromuscular JunctionIn Vitro TechniquesTritiumNeuromuscular junctionCholinePotassium ChlorideRats Sprague-Dawleychemistry.chemical_compoundPiperidinesInternal medicinemedicineCholineAnimalsCholinesterasePharmacologyMuscarinebiologyAcetylcholineRatsEndocrinologymedicine.anatomical_structurechemistrybiology.proteinCalciumFemaleCholinesterase Inhibitorsmedicine.symptomAcetylcholinemedicine.drugMuscle contractionEuropean journal of pharmacology
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Transient oligemia is associated with long-term changes in binding densities of cortical inhibitory GABAA receptors in the rat brain

2009

Recently, we could demonstrate in rats that a short transient oligemic period of only 20-minute duration, induced by systemic hypotension, resulted in a transient decline of spatial memory capacities without any histological damage over a subsequent period of 6 months. In our present study, we checked for more subtle alterations within the highly vulnerable hippocampal CA1 subfield using quantification of neuronal cell density and semi-quantitative analysis of the ischemia-sensitive protein MAP2. Since hippocampal excitatory and inhibitory neurotransmitter receptors are crucially involved in spatial memory processes, quantitative in vitro receptor autoradiography was performed using [(3)H]M…

Malemedicine.medical_specialtyTime FactorsDown-RegulationAMPA receptorHippocampal formationTritiumInhibitory postsynaptic potentialBinding CompetitiveHippocampusReceptors N-Methyl-D-AspartateTimeRadioligand Assaychemistry.chemical_compoundParietal LobeInternal medicinemedicineAnimalsReceptors AMPARats WistarReceptorGABA AgonistsMolecular Biologygamma-Aminobutyric AcidCerebral CortexMemory DisordersMuscimolChemistryGABAA receptorGeneral NeuroscienceReceptors GABA-ARatsDisease Models AnimalEndocrinologynervous systemMuscimolHypoxia-Ischemia BrainExcitatory postsynaptic potentialNMDA receptorNeurology (clinical)Microtubule-Associated ProteinsDevelopmental BiologyBrain Research
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Effect of previous exercise on fracture healing: a biochemical study with mice.

1974

(1974). Effect of Previous Exercise on Fracture Healing: A Biochemical Study with Mice. Acta Orthopaedica Scandinavica: Vol. 45, No. 1-4, pp. 481-489.

Malemedicine.medical_specialtyTime FactorsNitrogenPhysical Exertionmacromolecular substancesBone healingTritiummedicineAnimalsOrthopedics and Sports MedicineBony CallusWound HealingPhysical Education and Trainingbusiness.industryHexosaminesDNAAdaptation PhysiologicalSurgeryRatsTibial FracturesHydroxyprolineRNA RibosomalSurgeryCalciumbusinessActa orthopaedica Scandinavica
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