Search results for "tumor necrosis factor alpha"

showing 10 items of 479 documents

TNFalpha, IFNgamma and IL-10 gene polymorphisms in a sample of Sicilian patients with coeliac disease.

2005

Coeliac disease is associated with DQ2 and DQ8 alleles, but other genes also confer an additional genetic risk.Defining whether the genetic profiles of interleukin-10, tumour necrosis factor alpha and interferon gamma are associated with an increased coeliac disease risk.The functionally gene polymorphisms of tumour necrosis factor alpha (-308G/A), interferon gamma (+874T/A) and interleukin-10 (-1082G/A) were typed using sequence specific primer-polymerase chain reaction in 110 Sicilian coeliac disease patients and in 220 Sicilian healthy controls.No differences in genotype frequencies of interleukin-10 polymorphisms were found between coeliac disease patients and healthy controls. A signif…

AdultMaleNecrosisAdolescentGenotypeCoeliac diseaseInterferon-gammaGene FrequencymedicineHumansGenetic Predisposition to DiseaseAlleleChildGeneSicilyPolymorphism GeneticHepatologybusiness.industryTumor Necrosis Factor-alphaGastroenterologynutritional and metabolic diseasesInfantGluten intoleranceMiddle Agedmedicine.diseaseGenotype frequencyInterleukin-10Interleukin 10Celiac DiseaseCase-Control StudiesChild PreschoolImmunologyTumor necrosis factor alphaFemalemedicine.symptombusinessDigestive and liver disease : official journal of the Italian Society of Gastroenterology and the Italian Association for the Study of the Liver
researchProduct

Expression of the tumor necrosis factor receptor—associated factors 1 and 2 and regulation of the nuclear factor—kB antiapoptotic activity in human g…

2005

Object. Tumor necrosis factor receptor (TNFR)—associated factors (TRAFs) are a recently established group of proteins involved in the intracellular signaling of the TNFR superfamily members. The TRAFs have been implicated in promoting cell survival through the activation of transcription factor nuclear factor (NF)—κB. The authors investigated the expression of NF-κB, caspase 3, TRAF1, TRAF2, and TRAF-associated NF-κB activator/TRAF—interacting protein (TANK/I-TRAF), a regulator of TRAF activity, in human gliomas. Methods. Tumor samples were obtained in 27 adult patients harboring seven low-grade gliomas, nine anaplastic astrocytomas, and 11 glioblastomas multiforme. The NF-κB activation was…

AdultMalePathologymedicine.medical_specialtyBlotting WesternTRAF1Apoptosis Blotting Brain Neoplasms/metabolism Brain Neoplasms/pathology Caspase GliomaApoptosisCaspase 3BiologyGliomamedicineHumansTranscription factorAgedAged 80 and overTankyrasesBrain NeoplasmsCaspase 3NF-kappa BGliomaMiddle AgedTNF Receptor-Associated Factor 2medicine.diseaseTNF Receptor-Associated Factor 1Up-RegulationIntracellular signal transductionBlotTumor Necrosis Factor Receptor-Associated FactorsCaspasesCancer researchFemaleTumor necrosis factor alphaSignal TransductionJournal of Neurosurgery
researchProduct

Molecular interactions between human cartilaginous endplates and nucleus pulposus cells: a preliminary investigation.

2014

Study Design. Conditioned media (CM) of cartilaginous endplates (CEPs) of intervertebral discs were analyzed in a bioassay with regard to their influence on matrix turnover and inflammatory factors on nucleus pulposus (NP) cells of the same patient. CEP tissue underwent further histological and ultrastructural analysis. Objective. To identify possible interactions between the CEP and the disc via molecular factors that may influence disc matrix degradation and to determine degenerative changes of CEP tissue. Summary of Background Data. Impaired endplate perme-ability due to degeneration and calcification is considered to be a key contributor to disc degeneration. An upregulation of metallop…

AdultMalePathologymedicine.medical_specialtyCellIntervertebral Disc DegenerationMatrix metalloproteinaseMatrix (biology)Proinflammatory cytokineDownregulation and upregulationMatrix Metalloproteinase 13MedicineHumansOrthopedics and Sports MedicineAggrecansIntervertebral DiscAggrecanCells CulturedAgedbusiness.industryInterleukin-6Interleukin-8Middle AgedCell biologyTissue Degenerationmedicine.anatomical_structureCartilageTumor necrosis factor alphaFemaleMatrix Metalloproteinase 3Neurology (clinical)businessSpine
researchProduct

Immune-mediated necrotizing myopathy is characterized by a specific Th1-M1 polarized immune profile.

2012

Immune-mediated necrotizing myopathy (IMNM) is considered one of the idiopathic inflammatory myopathies, comprising dermatomyositis, polymyositis, and inclusion body myositis. The heterogeneous group of necrotizing myopathies shows a varying amount of necrotic muscle fibers, myophagocytosis, and a sparse inflammatory infiltrate. The underlying immune response in necrotizing myopathy has not yet been addressed in detail. Affected muscle tissue, obtained from 16 patients with IMNM, was analyzed compared with eight non-IMNM (nIMNM) tissues. Inflammatory cells were characterized by IHC, and immune mediators were assessed by quantitative real-time PCR. We demonstrate that immune- and non–immune-…

AdultMalePathologymedicine.medical_specialtyT cellBiopsyCell CountBiologyCD8-Positive T-LymphocytesMajor histocompatibility complexReal-Time Polymerase Chain ReactionPolymyositisPathology and Forensic MedicineYoung AdultImmune systemSarcolemmamedicineHumansAgedAged 80 and overB-LymphocytesMyositisMacrophagesMusclesHistocompatibility Antigens Class IAutoantibodyImmunityComplement System ProteinsDermatomyositisMiddle AgedTh1 Cellsmedicine.diseaseCapillariesmedicine.anatomical_structureChild PreschoolImmunologybiology.proteinTumor necrosis factor alphaFemaleInclusion body myositisThe American journal of pathology
researchProduct

Inflammatory capacity of exosomes released in the early stages of acute pancreatitis predicts the severity of the disease

2021

As acute pancreatitis progresses to the severe form, a life-threatening systemic inflammation is triggered. Although the mechanisms involved in this process are not yet well understood, it has been proposed that circulating exosomes may be involved in the progression of inflammation from the pancreas to distant organs. Here, the inflammatory capacity and protein profile of plasma exosomes obtained during the first 24 h of hospitalization of patients diagnosed with acute pancreatitis were characterized and compared with the final severity of the disease. We found that the final severity of the disease strongly correlates with the inflammatory capacity of exosomes in the early stages of acute…

AdultMaleProteomicsInflammationSystemic inflammationExosomesS100A9Pathology and Forensic MedicineS100A8medicineHumansS100A8PancreasS100A9AgedAged 80 and overInflammationbusiness.industryMiddle Agedmedicine.diseaseMicrovesiclesAcute pancreatitisPancreatitisAcute DiseaseImmunologyDisease ProgressionPancreatitisAcute pancreatitisFemaleTumor necrosis factor alphamedicine.symptombusinessSignal TransductionJournal of Pathology
researchProduct

Cytokine Gene Transcription By NF-kappaB Family Members in Patients with Inflammatory Bowel Disease

1998

We examined the expression of the transcription factor NF-kappa B, a nuclear trans-acting factor known to play a key role in cytokine gene regulation, in patients with inflammatory bowel disease (IBD). It was found that LP macrophages in Crohn's disease (CD) and ulcerative colitis (UC) display high levels of NF-kappa B DNA-binding activity accompanied by an increased production of interleukin (IL)-1, IL-6, and tumor necrosis factor (TNF) alpha. Western blot studies showed an increased expression of the p50 and c-rel subunits of NF-kappa B; however, the most striking finding was an increased expression level of NF-kappa B p65 in patients with CD and UC. Selective downregulation of p65 in IBD…

AdultMaleShort Bowel SyndromeAdolescentTranscription GeneticColonBiologyInflammatory bowel diseaseGeneral Biochemistry Genetics and Molecular BiologyProinflammatory cytokinechemistry.chemical_compoundCrohn DiseaseHistory and Philosophy of ScienceDownregulation and upregulationmedicineHumansIntestinal MucosaTranscription factorCells CulturedRegulation of gene expressionMacrophagesGeneral NeuroscienceNF-kappa BInterleukinNF-κBMiddle Agedmedicine.diseaseGene Expression RegulationchemistryImmunologyCancer researchCytokinesColitis UlcerativeFemaleTumor necrosis factor alphaAnnals of the New York Academy of Sciences
researchProduct

Tumour necrosis factor (TNF) production by T cell receptor-primed T lymphocytes is a target for low dose methotrexate in rheumatoid arthritis

1999

SUMMARYMethotrexate (MTX) is an effective immunosuppressive agent in various chronic inflammatory diseases such as rheumatoid arthritis (RA). However, its mechanisms of action are only partially understood. In this study, we assessed the effects of MTX on the differentiation of peripheral blood (PB) CD4+CD45RA ‘naive’ and CD4+CD45RO ‘memory’ T cells from healthy controls and patients with RA. Accordingly, purified T cells were primed and restimulated in vitro via the T cell receptor (TCR) in the presence of IL-2 to generate effector T cells secreting large amounts of Th1 and Th2 cytokines. We observed that low doses of MTX strongly suppress TNF and to a lesser extent interferon-gamma (IFN-γ…

AdultMaleTime FactorsT-LymphocytesT cellImmunologyReceptors Antigen T-CellPriming (immunology)Enzyme-Linked Immunosorbent AssayMonocytesArthritis RheumatoidInterferon-gammaAntigens CDimmune system diseasesmedicineHumansImmunology and AllergyCytotoxic T cellCells CulturedB-LymphocytesDose-Response Relationship DrugTumor Necrosis Factor-alphabusiness.industryMonocyteSynovial MembraneT-cell receptorCell DifferentiationOriginal ArticlesT lymphocyteMiddle Agedmedicine.diseaseMethotrexatemedicine.anatomical_structureAntirheumatic AgentsRheumatoid arthritisImmunologyCytokinesFemaleTumor necrosis factor alphabusinessClinical and Experimental Immunology
researchProduct

Different Transcriptional Activity and In Vitro TNF-α Production in Psoriasis Patients Carrying the TNF-α 238A Promoter Polymorphism

2000

Genes encoded on chromosome 6 within the major histocompatibility complex region are thought to play an important role in the pathogenesis of psoriasis. A potential candidate gene is tumor necrosis factor alpha. The tumor necrosis factor alpha promoter contains several polymorphisms including two G--A transitions at position -308 and -238, which are the most common in Caucasian populations. The TNF238.2 (-238A) allele has been strongly associated with psoriasis. We have investigated the effect of the -238 and -308 variants on transcription of the tumor necrosis factor alpha gene in luciferase reporter gene assays. In addition, peripheral blood mononuclear cells of 47 patients with psoriasis…

AdultMaleTranscription Geneticmedicine.medical_treatmentT cellDermatologyBiologyBiochemistryPeripheral blood mononuclear cellAntigenPsoriasisTNFαmedicineSuperantigenHumansPsoriasisPromoter Regions GeneticMolecular Biologytranscriptional activityAgedAged 80 and overPBMGPolymorphism GeneticTumor Necrosis Factor-alphaPromoterCell BiologyMiddle Agedmedicine.diseaseMolecular biologypromoter polymorphismCytokinemedicine.anatomical_structureImmunologyLeukocytes MononuclearFemaleTumor necrosis factor alphaJournal of Investigative Dermatology
researchProduct

Continuous therapy with transdermal nitroglycerin does not affect biomarkers of vascular inflammation and injury in healthy volunteers.

2009

Continuous exposure to nitroglycerin (GTN) results in development of tolerance and is associated with increased free radical production and abnormal endothelial function. Elevated plasma biomarkers of inflammation have been shown to be associated with endothelial dysfunction in most cardiovascular conditions. It remains unclear whether exposure to GTN is also associated with increased biomarkers of endothelial and vascular injury or vascular inflammation. In an investigator-blind study, a total of 28 healthy volunteers were randomized to continuous therapy with GTN (0.6 mg/h 24 h/day for 7 days) or no therapy. Venous blood was collected on day 0 and day 7. Plasma levels of markers such as …

AdultMaleVasculitisTime FactorsEndotheliumAdolescentPhysiologyVasodilator AgentsInflammationPharmacologyAdministration CutaneousArginineLesionNitroglycerinYoung AdultPhysiology (medical)MedicineHumansEndothelial dysfunctionTransdermalPharmacologyDose-Response Relationship Drugbusiness.industryInterleukin-6Tumor Necrosis Factor-alphaGeneral MedicineVenous bloodmedicine.diseaseLipoproteins LDLDose–response relationshipP-Selectinmedicine.anatomical_structureAnesthesiacardiovascular systemTumor necrosis factor alphaEndothelium Vascularmedicine.symptombusinessCell Adhesion MoleculesBiomarkerscirculatory and respiratory physiologyCanadian journal of physiology and pharmacology
researchProduct

Recombinant human granulocyte-macrophage colony-stimulating factor induces secretion of autoinhibitory monokines by U-937 cells

1988

Colony-stimulating factors are required for survival proliferation, differentiation and functional activation of granulocytes, macrophages and their precursor cells. In the present report, however, we demonstrate antiproliferative activity of recombinant human (rh) granulocyte-macrophage colony-stimulating factor (GM-CSF) on monoblast cell line U-937 and provide evidence for the involvement of tumor necrosis factor alpha TNF-alpha and interleukin 1 beta (IL 1 beta) in its growth inhibitory action. GM-CSF (but not granulocyte CSF, G-CSF or macrophage CSF, M-CSF) suppressed DNA synthesis and self renewal of U-937 cells. Similarly, medium conditioned by U-937 cells in response to GM-CSF (GM-CS…

AdultMalemedicine.drug_classImmunologyMonoblastBiologyGranulocyteMonoclonal antibodyMonocytesColony-Stimulating FactorsTumor Cells CulturedmedicineHumansImmunology and AllergyMacrophageTumor Necrosis Factor-alphaMolecular biologyRecombinant ProteinsStimulation ChemicalGranulocyte macrophage colony-stimulating factormedicine.anatomical_structureCell culturebiology.proteinTumor necrosis factor alphaLymphoma Large B-Cell DiffuseAntibodyCell DivisionInterleukin-1medicine.drugEuropean Journal of Immunology
researchProduct