Search results for "tumor necrosis factor alpha"

showing 10 items of 479 documents

Both IL-12p70 and IL-23 are synthesized during active Crohnʼs disease and are down-regulated by treatment with anti-IL-12 p40 monoclonal antibody

2005

Background: Interleukin (IL)-12p70 and IL-23 are key T helper-1 (TH1) cytokines that drive the inflammation seen in numerous models of intestinal inflammation. These molecules contain an identical p40 chain that is bound to a p35 chain in IL-12 and a p19 chain in IL-23, making both potentially susceptible to modulation by an anti-IL-12p40 monoclonal antibody (mAb). Methods: In the present study, we sought to determine whether active inflammation in Crohn's disease (CD) is associated with the increased synthesis of both of these cytokines and whether patients treated with an anti-IL-12p40 mAb down-regulate IL-23 as well as IL-12p70 as previous reported. Results: To this end we initially dete…

AdultMaleAdolescentBiopsyT-Lymphocytesmedicine.medical_treatmentT cellDown-RegulationInterleukin-23Crohn DiseaseInterleukin 23medicineHumansImmunology and AllergyCells CulturedAgedLamina propriaMucous MembraneCD40biologyInterleukin-12 Subunit p40Interleukin-6InterleukinsMacrophagesGastroenterologyAntibodies MonoclonalInterleukinMiddle AgedInterleukin-12Protein Subunitsmedicine.anatomical_structureCytokineImmunologyInterleukin-23 Subunit p19biology.proteinInterleukin 12FemaleTumor necrosis factor alphaInterleukin-1Inflammatory Bowel Diseases
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Functional patterns of cytomegalovirus (CMV) pp65 and immediate early-1-specific CD8+T cells that are associated with protection from and control of …

2015

Background The functional profile of cytomegalovirus (CMV)-specific CD8+ T cells that associate with protection from and control of CMV DNAemia in allogeneic stem cell transplant (allo-SCT) recipients remains incompletely characterized. Methods We enumerated pp65 and immediate early (IE)-1-specific CD8+ T cells expressing interferon-gamma, tumor necrosis factor-alpha, and CD107a, by flow cytometry in 94 patients at days +30 and +60 after allo-SCT. Results Fifty of 94 patients had CMV DNAemia within the first 100 days after transplant. CMV-specific CD8+ T-cell responses (of any functional type) were more likely to be detected in patients who did not display CMV DNAemia than in those who did …

AdultMaleAdolescentCongenital cytomegalovirus infectionCytomegalovirusCD8-Positive T-LymphocytesLower riskFlow cytometryCohort StudiesViral Matrix ProteinsInterferon-gammaYoung AdultmedicineHumansTransplantation HomologousCytotoxic T cellAgedTransplantationmedicine.diagnostic_testTumor Necrosis Factor-alphabusiness.industryvirus diseasesMiddle AgedPhosphoproteinsmedicine.diseaseVirologyTransplantationInfectious DiseasesCytomegalovirus InfectionsDNA ViralImmunologyFemaleTumor necrosis factor alphaStem cellbusinessCD8Stem Cell TransplantationTransplant Infectious Disease
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T cells involved in psoriasis vulgaris belong to the Th1 subset

1994

Although the pathogenesis of psoriasis vulgaris is still unknown, several characteristics point to an immunologically mediated process. Epidermal psoriatic lesions are characterized by a hyperproliferation of keratinocytes and an infiltration of T lymphocytes and granulocytes. Because the former may be mediated in part by lymphokines secreted by T cells, we have focused our interest on the in vivo and in vitro cytokine secretion patterns of T lymphocytes from psoriatic lesions. In five patients T lymphocytes were obtained from epidermal specimens. The cells were propagated with lectin and irradiated feeder cells and subsequently cloned by limiting dilution. The resulting T-cell clones were …

AdultMaleBiopsyCD8 AntigensT-Lymphocytesmedicine.medical_treatmentMolecular Sequence DataDermatologyBiologyPolymerase Chain ReactionBiochemistryInterferon-gammaT-Lymphocyte SubsetsPsoriasisSynovial FluidmedicineHumansPsoriasisRNA MessengerMolecular BiologyCells CulturedAgedSkinAged 80 and overBase SequenceTumor Necrosis Factor-alphaArthritis PsoriaticLymphokineInterleukinT-Lymphocytes Helper-InducerCell BiologyT lymphocyteMiddle Agedmedicine.diseaseInterleukin-10PhenotypeCytokineepidermal T cellsCD4 AntigensImmunologyCytokinesInterleukin-2cytokine productionFemaleCytokine secretionTumor necrosis factor alphaInterleukin-4CD8
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Clinical staging and serum cytokines in bipolar patients during euthymia

2017

Aims: Changes in serum cytokines and altered neutrophin concentration have been associated with bipolar disorder (BD). Our aim here was to analyze peripheral blood biomarkers according to the clinical stages of BD. Method: Euthymic BD-I patients were grouped according to their level of functioning in early-stage (n = 25) and late-stage (n = 23), and compared to healthy siblings (n = 23) and genetically unrelated healthy controls (n = 21). Neurotrophin (neurotrophin-3 and BDNF) concentration and biomarkers of inflammation, including cytokines (IL-6, IL-10 and TNF-alpha), leukocytes count and acute phase proteins, were measured. Results: IL-10 concentration was significantly increased in earl…

AdultMaleBipolar DisorderBipolar disorderInflammationNeurotrophinsYoung Adult03 medical and health sciences0302 clinical medicineNeurotrophin 3medicineLeukocytesHumansNerve Growth FactorsBipolar disorderBiological PsychiatryPharmacologyInflammationbiologyInterleukin-6Tumor Necrosis Factor-alphabusiness.industryBrain-Derived Neurotrophic FactorSiblingsAcute-phase proteinMiddle Agedmedicine.diseaseBlood Cell CountInterleukin-10030227 psychiatryPeripheralAffectSerum cytokineClinical stagingCase-Control StudiesImmunologybiology.proteinBiomarker (medicine)CytokinesFemaleTumor necrosis factor alphamedicine.symptombusinessBiomarkers030217 neurology & neurosurgeryAcute-Phase ProteinsNeurotrophin
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TNF-α in CRPS and 'normal' trauma--significant differences between tissue and serum.

2011

Posttraumatic TNF-alpha signaling may be one of the factors responsible for pain and hyperalgesia in complex regional pain syndromes (CRPS). In order to further specify the role of TNF-alpha we investigated tissue (skin) and serum concentrations in three different patient groups: patients with osteoarthritis and planned surgery, with acute traumatic upper limb bone fracture waiting for surgery, and with CRPS I. Thirty patients (10 in each group) were recruited. Mean CRPS duration was 36.1 ± 8.1 weeks (range 8- 90 weeks). Skin punch biopsies were taken at the beginning of the surgery in osteoarthritis and fracture patients and from the affected side in CRPS patients. Blood samples were taken…

AdultMaleBone pathologyEnzyme-Linked Immunosorbent AssayPilot ProjectsOsteoarthritisFractures BoneOsteoarthritismedicineHumansAgedSkinAged 80 and overmedicine.diagnostic_testbusiness.industryTumor Necrosis Factor-alphaBone fractureMiddle Agedmedicine.diseaseUp-RegulationAnesthesiology and Pain Medicinemedicine.anatomical_structureComplex regional pain syndromeNeurologyAnesthesiaSkin biopsyHyperalgesiaAcute DiseaseUpper limbWounds and InjuriesTumor necrosis factor alphaFemaleNeurology (clinical)medicine.symptombusinessComplex Regional Pain SyndromesPainReferences
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Serum levels of cytokines in chronic liver diseases

1992

Serum levels of interleukin-1 (IL-1 beta), interleukin-6 (IL-6), tumor necrosis factor alpha (TNF-alpha), interferon gamma (IFN-gamma), and C-reactive protein (CRP) were investigated in patients with chronic liver diseases (CLD) and correlated with the type of underlying disease and various clinical and laboratory parameters. Two hundred sixty-four patients suffering from various CLD were studied; 136 cases presented with liver cirrhosis, and 128 patients were in the noncirrhotic stage of their underlying liver diseases. Serum levels of IL-1 beta, IL-6, TNF-alpha, IFN-gamma, and CRP were elevated in patients with CLD. Endogenous cytokine patterns in CLD were stage dependent and only margina…

AdultMaleCirrhosisAdolescentmedicine.medical_treatmentDiseaseReference ValuesHumansMedicineInterferon gammaChildBeta (finance)Hepatologybiologybusiness.industryLiver DiseasesC-reactive proteinGastroenterologyMiddle Agedmedicine.diseasePathophysiologyC-Reactive ProteinCytokineChronic DiseaseImmunologybiology.proteinCytokinesFemaleTumor necrosis factor alphabusinessmedicine.drugGastroenterology
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IL-10 and TNF-α polymorphisms and the recovery from HCV infection

2003

Hepatitis C virus (HCV) infection becomes chronic in about 85% of infected individuals, whereas only 15% of infected people clear spontaneously the virus. It is conceivable that the host immunogenetic background influences the course of infection in term of recovery. Thus, in this study we have evaluated the effect of functionally relevant polymorphisms at tumor necrosis factor-alpha (TNF-alpha, i.e., 2 biallelic polymorphisms at nt -863 and nt-308 of the promoter) and interleukin-10 (IL-10) loci (i.e., 1 biallelic polymorphism at nt -1082 of the promoter), on the clearance of HCV infection. To this purpose, we compared 18 Sicilian patients who had spontaneously recovered from previous HCV …

AdultMaleGenotypeHepatitis C virusmedicine.medical_treatmentImmunologyInfection groupBiologymedicine.disease_causePolymorphism Single NucleotideViruspolymorphismImmunoenzyme TechniquesGenotypemedicinecytokineImmunology and AllergyHumansIn patientTumor Necrosis Factor-alphaGeneral MedicineHepatitis C ChronicVirologyHepatitis CInterleukin-10Interleukin 10CytokineTNF-αImmunologyHCVIL-10Tumor necrosis factor alphaFemale
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The -308G/A polymorphism of TNF-alpha influences immunological parameters in old subjects affected by infectious diseases.

2005

Abnormal increments of pro-inflammatory cytokines (IL-6 and TNF-alpha) characterize the outbreak of infectious diseases, which are the major cause of death in the elderly. A counterbalance to the inflammation is exerted by IL-10 with an inhibitory role on TNF-alpha production. As is well known, some cytokine gene polymorphisms influence the cytokine production, playing a role as susceptibility or resistance factors against immune-mediated and infectious disease. Genetic variations in the -308A/G locus for TNF-alpha seems to affect the clinical outcome of some infectious diseases. In fact, the -308A allele is associated with severe septic shock and death. On this basis, we have screened heal…

AdultMaleGenotypemedicine.medical_treatmentImmunologyCommunicable DiseasesGene FrequencyGenotypeGeneticsmedicineBronchopneumoniaHumansAlleleInterleukin 6Molecular BiologyAllele frequencyGenetics (clinical)AgedPolymorphism GeneticbiologyInterleukin-6Tumor Necrosis Factor-alphaOutbreakGeneral MedicineMiddle AgedBronchitis ChronicKiller Cells NaturalZincCytokineInfectious disease (medical specialty)Immunologybiology.proteinTumor necrosis factor alphaFemaleMetallothioneinInternational journal of immunogenetics
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Relevance of gamma interferon, tumor necrosis factor alpha, and interleukin-10 gene polymorphisms to susceptibility to Mediterranean spotted fever.

2009

Several studies have demonstrated that cellular immunity plays a critical role in the protective immune response against Rickettsia conorii. Immune CD4+ and CD8+ T cells are both involved in the control of rickettsial infection (38). Perivascular infiltrated CD4+ and CD8+ T lymphocytes, macrophages, and natural killer cells produce chemokines and cytokines that activate endothelial rickettsicidal activities. Infected human cells, including endothelial cells, hepatocytes, and macrophages, activated by gamma interferon (IFN-γ), tumor necrosis factor alpha (TNF-α), and interleukin-1β (IL-1β), kill intracellular rickettsiae by one or a combination of three mechanisms, involving nitric oxide syn…

AdultMaleMicrobiology (medical)Meningitidesgene polymorphismgene polymorphisms; mediterranean spotted fevermedicine.medical_treatmentClinical BiochemistryImmunologyBiologyBoutonneuse FeverPolymorphism Single NucleotideInterferon-gammaImmune systemInterferonmedicineHumansImmunology and AllergyCytotoxic T cellSicilyTumor Necrosis Factor-alphaMiddle AgedInterleukin-10mediterranean spotted feverInterleukin 10CytokineImmunologyFemaleTumor necrosis factor alphaDisease SusceptibilityMicrobial ImmunologyCD8medicine.drug
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The Acute Phase Response in Sicilian Patients with Boutonneuse Fever Admitted to Hospitals in Palermo, 1992–1997

2001

Abstract Objectives : To study the modifications of some components of the acute phase response (APR) in Sicilian patients with boutonneuse fever (BF) caused by Rickettsia conorii . Methods : Sera from 500 Sicilian patients with confirmed BF were studied at the time of diagnosis and every week after treatment, and after recovery for the presence of various inflammatory mediators. Tumour necrosis factor α (TNFα), interleukin(IL)-6, IL-1α, IL-8, soluble TNF receptors (sTNF-R) and sIL-6R were assayed by commercially ELISA kits. C3, C4, factor B, C-reactive protein (CRP), fibrinogen, ceruloplasmin (Cp) and α 1 -antitrypsin (AAT) were assayed by a rate nephelometry. Results : Interferon gamma (I…

AdultMaleMicrobiology (medical)Time Factorsmedicine.medical_treatmentBoutonneuse FeverFibrinogenmedicineHumansInterferon gammaAcute-Phase ReactionAgedbiologybusiness.industryAcute-phase proteinInterleukinMiddle Agedmedicine.diseasebiology.organism_classificationAntibodies BacterialBoutonneuse feverRickettsia conoriiInfectious DiseasesCytokineItalyImmunologyCytokinesFemaleTumor necrosis factor alphaRickettsia conoriibusinessAcute-Phase Proteinsmedicine.drugJournal of Infection
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