Search results for "type 1"

showing 10 items of 540 documents

AT1-receptor blockade with irbesartan improves peripheral but not coronary endothelial dysfunction in patients with stable coronary artery disease

2007

Activation of the renin-angiotensin-aldosterone system plays an important role in the pathogenesis of endothelial dysfunction and atherosclerosis. Studies evaluating the effect of AT1-receptor blockers on endothelial dysfunction in patients with coronary artery disease (CAD) revealed mixed results. Studies addressing the effects of AT1-receptor blockers on the coronary and peripheral function in the same study population, are still lacking. We therefore aimed to test the effects of long-term therapy with the AT1-receptor blocker irbesartan (IRB) on both, the coronary and peripheral endothelial function in patients with CAD. Seventy-two patients with CAD were randomly assigned to double-blin…

Malemedicine.medical_specialtyBrachial ArteryEndotheliumTetrazolesCoronary Artery DiseaseCoronary artery diseaseIrbesartanDouble-Blind MethodInternal medicinemedicine.arterymedicineHumanscardiovascular diseasesEndothelial dysfunctionBrachial arteryUltrasonographybusiness.industryVascular diseaseBiphenyl CompoundsIrbesartanMiddle Agedmedicine.diseaseCoronary VesselsAngiotensin IIPeripheralVasodilationmedicine.anatomical_structureEndocrinologyRegional Blood FlowCardiologyFemaleEndothelium VascularCardiology and Cardiovascular MedicinebusinessAngiotensin II Type 1 Receptor Blockersmedicine.drugAtherosclerosis
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Antidepressant-like behavioral effects of impaired cannabinoid receptor type 1 signaling coincide with exaggerated corticosterone secretion in mice.

2007

Hypothalamic-pituitary-adrenocortical (HPA) axis hyperactivity is associated with major depressive disorders, and treatment with classical antidepressants ameliorates not only psychopathological symptoms, but also the dysregulation of the HPA axis. Here, we further elucidated the role of impaired cannabinoid type 1 receptor (CB1) signaling for neuroendocrine and behavioral stress coping in the mouse forced swim test (FST). We demonstrate that the genetic inactivation of CB1 is accompanied by increased plasma corticosterone levels both under basal conditions and at different time points following exposure to the FST. The latter effect could be mimicked in C57BL/6N mice by acute, subchronic, …

Malemedicine.medical_specialtyCannabinoid receptorTime FactorsEndocrinology Diabetes and Metabolismmedicine.medical_treatmentAntidepressive Agents TricyclicStatistics NonparametricArticlechemistry.chemical_compoundMiceEndocrinologyRimonabantPiperidinesReceptor Cannabinoid CB1CorticosteroneDesipramineInternal medicineCannabinoid receptor type 1Adaptation PsychologicalmedicineAnimalsBiological PsychiatrySwimmingMice KnockoutAnalysis of VarianceEndocrine and Autonomic SystemsDepressionDesipramineMice Inbred C57BLPsychiatry and Mental healthDisease Models AnimalEndocrinologychemistrynervous systemPyrazoleslipids (amino acids peptides and proteins)FemaleCannabinoidRimonabantPsychologyCorticosteronehuman activitiesGlucocorticoidStress Psychologicalmedicine.drugBehavioural despair testSignal TransductionPsychoneuroendocrinology
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Impaired cannabinoid receptor type 1 signaling interferes with stress-coping behavior in mice.

2007

Dysregulation of the endocannabinoid system is known to interfere with emotional processing of stressful events. Here, we studied the role of cannabinoid receptor type 1 (CB1) signaling in stress-coping behaviors using the forced swim test (FST) with repeated exposures. We compared effects of genetic inactivation with pharmacological blockade of CB1 receptors both in male and female mice. In addition, we investigated potential interactions of the endocannabinoid system with monoaminergic and neurotrophin systems of the brain. Naive CB1 receptor-deficient mice (CB1-/-) showed increased passive stress-coping behaviors as compared to wild-type littermates (CB1+/+) in the FST, independent of se…

Malemedicine.medical_specialtyCannabinoid receptormedicine.medical_treatmentBiologyPharmacologyHippocampusMicePiperidinesReceptor Cannabinoid CB1Internal medicineCannabinoid receptor type 1MonoaminergicAdaptation PsychologicalGeneticsmedicineAnimalsBiogenic MonoaminesRNA MessengerReceptorMonoamine OxidaseSwimmingPharmacologyBrain-derived neurotrophic factormusculoskeletal neural and ocular physiologyBrain-Derived Neurotrophic FactorDesipraminefood and beveragesEndocannabinoid systemMice Inbred C57BLMonoamine neurotransmitterEndocrinologynervous systemVesicular Glutamate Transport Protein 1Molecular MedicinePyrazoleslipids (amino acids peptides and proteins)FemaleCannabinoidRimonabantpsychological phenomena and processesStress PsychologicalSignal TransductionThe pharmacogenomics journal
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CXCR2 blockade impairs angiotensin II-induced CC chemokine synthesis and mononuclear leukocyte infiltration.

2007

Objective—Angiotensin II (Ang-II) and mononuclear leukocytes are involved in atherosclerosis. This study reports the inhibition of Ang-II–induced mononuclear cell recruitment by CXCR2 antagonism and the mechanisms involved.Methods and Results—Ang-II (1 nmol/L, i.p. in rats) induced CXC and CC chemokines, followed by neutrophil and mononuclear cell recruitment. Administration of the CXCR2 antagonist, SB-517785-M, inhibited the infiltration of both neutrophils (98%) and mononuclear cells (60%). SB-517785-M had no effect on the increase in CXC chemokine levels but reduced MCP-1, RANTES, and MIP-1α release by 66%, 63%, and 80%, respectively. Intravital microscopy showed that pretreatment with S…

Malemedicine.medical_specialtyChemokineCXCR3Peripheral blood mononuclear cellLosartanReceptors Interleukin-8BRats Sprague-DawleyChemokine receptorInternal medicinemedicineCell AdhesionCCL17AnimalsHumansCXC chemokine receptorsSplanchnic CirculationChemokine CCL7Chemokine CCL4Chemokine CCL5Cells CulturedChemokine CCL2Chemokine CCL3InflammationbiologyAngiotensin IIMicrocirculationEndothelial CellsMacrophage Inflammatory ProteinsAtherosclerosisAngiotensin IIMonocyte Chemoattractant ProteinsRatsMononuclear cell infiltrationChemotaxis LeukocyteEndocrinologyNeutrophil Infiltrationbiology.proteinLeukocytes MononuclearCardiology and Cardiovascular MedicineAngiotensin II Type 1 Receptor BlockersArteriosclerosis, thrombosis, and vascular biology
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Angiotensin II contractile effects in mouse colon: role for pre- and post-junctional AT1A receptors

2013

Aim This study investigates whether a local renin–angiotensin system (RAS) exists in mouse colon and whether angiotensin II (Ang II) may play a role in the regulation of the contractile activity. Methods Isometric recordings were performed in vitro on the longitudinal muscle of mouse proximal and distal colon. Transcripts encoding for RAS components were investigated by RT-PCR. Results Ang II caused, in both preparations, a concentration-dependent contractile effect, antagonized by losartan, AT1 receptor antagonist, but not by PD123319, AT2 receptor antagonist. The combination of losartan plus PD123319 caused no change on the Ang II-induced contraction than losartan alone. Tetrodotoxin, neu…

Malemedicine.medical_specialtyColonPhysiologymedicine.drug_classMuscarinic AntagonistsBiologyReceptor Angiotensin Type 1Renin-Angiotensin SystemMicechemistry.chemical_compoundOrgan Culture TechniquesInternal medicineMuscarinic acetylcholine receptormedicineAnimalsReceptorAngiotensin II receptor type 1Reverse Transcriptase Polymerase Chain ReactionAngiotensin IIAntagonistMuscle Smoothangiotensin II AT1 receptors AT2 receptors enteric neurones mouse colon muscle contraction.Receptor antagonistAngiotensin IIElectrophysiologyMice Inbred C57BLEndocrinologyLosartanchemistryHexamethoniumhormones hormone substitutes and hormone antagonistsMuscle Contractionmedicine.drug
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Nerve conduction velocity and circulating immunocomplexes in type 1 diabetic children.

1991

There is evidence from several laboratories of an increased prevalence of circulating immuno-complexes (CIC) in diabetic patients. It has also been suggested that CIC are pathogenetically related to chronic diabetic complications. The aim of this study was to assess peripheral nerve function in children with Type 1 diabetes and to evaluate the relationship between the neurophysiological abnormalities and the possible presence of CIC. The investigation was carried out in 25 Type 1 diabetic patients ranging in age from 7-19 years and in 20 normal controls. Neurophysiological assessment was performed to evaluate motor and sensory conduction velocity on median and tibial nerves. IgG-CIC were de…

Malemedicine.medical_specialtyDiabetic neuropathyAdolescentSensory Receptor CellsNeural ConductionSensory systemAntigen-Antibody ComplexGastroenterologyNerve conduction velocityPathogenesisDiabetic NeuropathiesInternal medicineElectroneuronographymedicineReaction TimeHumansPeripheral NervesChildMotor NeuronsType 1 diabetesbusiness.industryGeneral Medicinemedicine.diseaseEndocrinologyDiabetes Mellitus Type 1NeurologyFemaleNeurology (clinical)ComplicationbusinessPolyneuropathyActa neurologica Scandinavica
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Oxidant/antioxidant status in obese children compared to pediatric patients with type 1 diabetes mellitus.

2009

Codoner-Franch P, Pons-Morales S, Boix-Garcia L, Valls-Belles V. Oxidant/antioxidant status in obese children compared to pediatric patients with type 1 diabetes mellitus. Background: Type 1 diabetes (T1D) mellitus and obesity are recognized risk factors for cardiovascular disease (CVD). A common mechanism underlying an increased risk for endothelial dysfunction in these two metabolic diseases is oxidative stress. Objective: To evaluate and compare the oxidant/antioxidant defense systems in children affected with T1D or obesity in order to determine the importance of oxidative stress before the emergence of complications. Subjects: Children with T1D (n = 20) or obesity (n = 22), without com…

Malemedicine.medical_specialtyLipid PeroxidesAntioxidantErythrocytesAdolescentEndocrinology Diabetes and Metabolismmedicine.medical_treatmentalpha-TocopherolProtein oxidationmedicine.disease_causeLipid peroxidationProtein Carbonylationchemistry.chemical_compoundInternal medicineMalondialdehydeInternal MedicinemedicineHumansObesityProspective StudiesChildRetrospective Studieschemistry.chemical_classificationType 1 diabetesGlutathione Peroxidasebusiness.industryGlutathione peroxidaseMalondialdehydemedicine.diseasebeta CaroteneObesityGlutathioneOxidative StressEndocrinologyDiabetes Mellitus Type 1chemistryPediatrics Perinatology and Child HealthFemaleLipid PeroxidationbusinessOxidative stressPediatric diabetes
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Altered REDD1, myostatin, and Akt/mTOR/FoxO/MAPK signaling in streptozotocin-induced diabetic muscle atrophy

2011

Type 1 diabetes, if poorly controlled, leads to skeletal muscle atrophy, decreasing the quality of life. We aimed to search highly responsive genes in diabetic muscle atrophy in a common diabetes model and to further characterize associated signaling pathways. Mice were killed 1, 3, or 5 wk after streptozotocin or control. Gene expression of calf muscles was analyzed using microarray and protein signaling with Western blotting. We identified translational repressor protein REDD1 (regulated in development and DNA damage responses) that increased seven- to eightfold and was associated with muscle atrophy in diabetes. The diabetes-induced increase in REDD1 was confirmed at the protein level. …

Malemedicine.medical_specialtyMAP Kinase Signaling SystemPhysiologyEndocrinology Diabetes and MetabolismFOXO1P70-S6 Kinase 1MyostatinBiologyMiceRandom Allocation03 medical and health sciences0302 clinical medicinePhysiology (medical)Internal medicinemedicineAnimalsRNA MessengerPhosphorylationMuscle SkeletalProtein kinase BPI3K/AKT/mTOR pathwayOligonucleotide Array Sequence Analysis030304 developmental biology0303 health sciencesForkhead Box Protein O1Gene Expression ProfilingTOR Serine-Threonine KinasesUbiquitinationForkhead Transcription FactorsOrgan SizeMyostatinProtein ubiquitinationMuscle atrophyMuscular AtrophyDNA Repair EnzymesDiabetes Mellitus Type 1EndocrinologyGene Expression Regulationbiology.proteinPhosphorylationmedicine.symptomProto-Oncogene Proteins c-akt030217 neurology & neurosurgeryTranscription FactorsAmerican Journal of Physiology-Endocrinology and Metabolism
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Psychological distress of children with early-onset type 1 diabetes and their mothers' well-being

2015

Aim Few studies have focused on the psychological adjustment of pre-adolescent children with type 1 diabetes. This study examined psychosocial functioning in nine- and 10-year-old children with early-onset type 1 diabetes, and their mothers, and associations between psychosocial functioning and diabetes management. Methods The mothers of 63 children with early-onset diabetes and 86 healthy children evaluated their own psychosocial functioning, and their child's, with standardised rating scales. We used general linear models to analyse the children's behaviour problems and the mothers' well-being. Associations between the children's behaviour problems, diabetes-related measures and the mothe…

Malemedicine.medical_specialtyMothersChild Behavior DisordersRating scaleDiabetes managementDiabetes mellitusmedicineHumansChildPsychiatryta515childhoodEarly onsetType 1 diabetesbusiness.industrymotherPsychological distressGeneral Medicinemedicine.diseaseta3123Cross-Sectional StudiesDiabetes Mellitus Type 1Type 1 diabetesPediatrics Perinatology and Child HealthWell-beingFemalepsychosocial functioningbehavioural problemsbusinessPsychosocialStress PsychologicalActa Paediatrica
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Remission phase in children diagnosed with type 1 diabetes in years 2012 to 2013 in Silesia, Poland: An observational study

2018

Background/objective The study aimed to analyze the frequency of partial remission (PR) and its association with chosen clinical and laboratory factors among pediatric patients with newly diagnosed type 1 diabetes (T1D). The long-term effect of PR on chosen parameters was also investigated. Methods In 194 patients (95 girls) aged 8.1 ± 4.3 years, we analyzed data at T1D onset: glycemia, pH, C-peptide, antibodies, weight, and concomitant autoimmune diseases. Anthropometric parameters, daily insulin requirement (DIR), and HbA1c 2 and 4 years after T1D diagnosis were also analyzed. We determined PR based on HbA1c and DIR measurements at least every 3 months. Results PR occurred in 59% of patie…

Malemedicine.medical_specialtyMultivariate analysisAdolescentEndocrinology Diabetes and Metabolismmedicine.medical_treatment030209 endocrinology & metabolismGastroenterologyDiabetic Ketoacidosis03 medical and health sciences0302 clinical medicineInternal medicineRemission phaseInternal MedicinemedicineHumans030212 general & internal medicineAge of OnsetChildGlycated HemoglobinType 1 diabetesbusiness.industryInsulinRemission InductionAge Factorsmedicine.diseaseKetoacidosisDiabetes Mellitus Type 1Child PreschoolConcomitantPediatrics Perinatology and Child HealthFemaleObservational studyPolandbusinessBody mass indexPediatric Diabetes
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