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showing 10 items of 10618 documents

Genome-wide analysis for micro-aberrations in familial exstrophy of the bladder using array-based comparative genomic hybridization

2007

OBJECTIVE: Exstrophy of the bladder (EB) is part of the bladder-exstrophy-epispadias complex (BEEC). Because familial occurrence of BEEC is rare, exogenous factors are thought to play a major role in the etiology of most BEEC cases. We aimed to investigate a possible genetic basis of BEEC in a consanguineous kindred of Moroccan origin with three members showing the same phenotypic expression of BEEC. PATIENTS AND METHODS: The three affected males (two cousins and their maternal uncle) all presenting with nonsyndromic classic EB, were born in Morocco or The Netherlands. One Moroccan patient had an open bladder surface for 22 years due to late surgical reconstruction, avoided upright posture …

AdultMaleEpispadiasAdolescentUrologyClone (cell biology)GenomeMedicineHumansAbnormalities MultipleGenetic Predisposition to DiseaseGeneIn Situ Hybridization FluorescenceOligonucleotide Array Sequence AnalysisGeneticsChromosome AberrationsGenomebusiness.industryBladder ExstrophyNucleic Acid HybridizationKaryotypeDNAmedicine.diseasePhenotypePedigreeBladder exstrophyMoroccoEtiologybusinessComparative genomic hybridizationBJU International
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A novel mutation in the coagulation factor 12 gene in subjects with hereditary angioedema and normal C1-inhibitor.

2011

In hereditary angioedema with normal C1-inhibitor two different missense mutations of codon p.Thr328* in the coagulation factor 12 gene have been reported in some families. In this study a novel factor 12 gene mutation, the deletion of 72 base pairs (bp) (c.971_1018+24del72*), was identified in a family of Turkish origin, in two sisters with recurrent skin swellings and abdominal pain attacks and in their symptom-free father. This deletion caused a loss of 48 bp of exon 9 (coding amino acids 324* to 340*) in addition to 24 bp of intron 9, including the authentic donor splice site of exon 9. The large deletion of 72 bp was located in the same F12 gene region as the missense mutations p.Thr32…

AdultMaleFactor XII DeficiencyTurkeyImmunologyDNA Mutational AnalysisMutation MissenseGene mutationmedicine.disease_causeC1-inhibitorExonImmunology and AllergyMedicineMissense mutationHumansHereditary Angioedema Type IIISequence DeletionGeneticsMutationAngioedemabiologybusiness.industryAngioedemas HereditaryExonsmedicine.diseaseMolecular biologyIntronsPedigreeHereditary angioedemaFactor XIIMutationbiology.proteinFemalemedicine.symptombusinessComplement C1 Inhibitor ProteinClinical immunology (Orlando, Fla.)
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Sensory-specific satiety for a food is unaffected by the ad libitum intake of other foods during a meal. Is SSS subject to dishabituation?

2012

Sensory-specific satiety (SSS) is defined as a decrease in the pleasantness of a specific food that has just been eaten to satiation, while other non-eaten foods remain pleasant. The objectives of this study were the following: (1) to investigate whether SSS for a food is affected by the ad libitum intake of other foods presented sequentially during a meal, (2) to compare the development of SSS when foods are presented simultaneously or sequentially during a meal, and (3) to examine whether SSS is modified when foods are presented in an unusual order within a meal. Twelve participants participated in three tasting sessions. In session A, SSS for protein-, fat- and carbohydrate-rich sandwich…

AdultMaleFood intakeSensory-specific satiety[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionSatiationBody Mass IndexFood PreferencesYoung AdultAnimal scienceDishabituationHumansHabituation PsychophysiologicMealsGeneral PsychologyMealCommunicationNutrition and Dieteticsbusiness.industrySingle typedigestive oral and skin physiologyFeeding BehaviorOlfactory PerceptionPostprandial PeriodSSS*TasteFemaleWine tastingbusinessPsychology[SDV.AEN]Life Sciences [q-bio]/Food and NutritionAppetite
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Functional and Postural Lateral Preferences in Humans: Interrelations and Life-Span Age Differences

2002

This study aimed to provide data on lateral preferences among older subjects, to analyze age differences, and to determine interrelations between lateral preferences. Four functional preferences (handedness, footedness, eyedness, earedness) and three postural lateral preferences (hand-clasping, arm-folding, leg-crossing) were assessed in 628 Germans (252 men, 376 women) aged between 19 and 90 years. Sex differences, age differences, and associations between lateralities were analyzed applying chi-square tests. Logistic regression analyses considering age, sex, and interactions between variables were applied to analyze combined effects on laterality measures. Right-sided preference for hande…

AdultMaleFootednessPostureLogistic regressionFunctional LateralityOcular dominanceGermanyGeneticsHumansOcular Physiological PhenomenaGenetics (clinical)Ecology Evolution Behavior and SystematicsAgedAged 80 and overLegChi-Square DistributionLife spanAge differencesRightward shiftAge FactorsEarMiddle AgedLogistic ModelsPhenotypeLateralityArmFemalePsychologyChi-squared distributionDemographyHuman Biology
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Microsatellite allele A5.1 of MHC class I chain-related gene A is associated with latent autoimmune diabetes in adults in Latvia.

2006

NIDDM is one of the most common forms of diabetes. The diagnosis is based on WHO classification, which is a clinical classification and misses the autoimmune diabetes in adults. Therefore, among the clinically diagnosed NIDDM cases, there can be a certain number of patients with latent autoimmune diabetes in adults (LADA). The MICA gene is located in the MHC class I region and is expressed by monocytes, keratinocytes, and endothelial cells. Sequence determination of the MICA gene identifies trinucleotide repeat (GCT) microsatellite polymorphism, which identifies 5 alleles with 4, 5, 6, and 9 repetitions of GCT (A4, A5, A6, and A9) or 5 repetitions of GCT with 1 additional G insertion for al…

AdultMaleGeneral Biochemistry Genetics and Molecular Biologylaw.inventionHistory and Philosophy of ScienceGene FrequencylawDiabetes mellitusMHC class ImedicineHumansGenetic Predisposition to DiseaseAlleleAge of OnsetPolymerase chain reactionAllelesbiologyGeneral NeuroscienceHistocompatibility Antigens Class Imedicine.diseaseLatviastomatognathic diseasesDiabetes Mellitus Type 2HaplotypesImmunologybiology.proteinMicrosatelliteFemaleAge of onsetAntibodyTrinucleotide repeat expansionMicrosatellite RepeatsAnnals of the New York Academy of Sciences
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Further evidence of genetic heterogeneity in familial essential tremor.

2007

Familial essential tremor (FET) is a common hereditary movement disorder with phenotypic variability and genetic heterogeneity. To date, linkage analyses revealed three loci associated to essential tremor (ET) (ETM1 on 3q13, ETM2 on 2p22-25, and a locus on 6p23). We performed a genetic analysis of these candidate chromosomal regions in a fifth-generation Italian kindred with autosomal-dominant ET. Of the 22 clinically evaluated family members, nine were affected by ET. The genetic study indicates that the ET in this family is not associated to any of the known ET loci. These findings support evidence of further genetic heterogeneity for such disease. (C) 2007 Elsevier Ltd. All rights reserv…

AdultMaleGenetic LinkageLocus (genetics)DiseaseBiologyGenetic analysisGenetic HeterogeneityGenetic linkagemedicineHumansAge of OnsetAgedGeneticsEssential tremorGenetic heterogeneityMiddle Agedmedicine.diseasePhenotypePedigreeNeurologySettore MED/03 - Genetica MedicaDisease ProgressionEssential tremorFemaleSettore MED/26 - NeurologiaNeurology (clinical)Geriatrics and GerontologyAge of onsetLinkage analysiNeurological disease
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Familial ring (18) mosaicism in a 23-year-old young adult with 46,XY,r(18) (::p11→q21::)/46,XY karyotype, intellectual disability, motor retardation …

2010

We report on a 23-year-old man with craniofacial findings of the holoprosencephaly spectrum disorder (microcephaly, hypotelorism, depressed nasal bridge, single median maxillary central incisor), fusion of C2-C3 vertebrae, intellectual disability, and severe sleep apnea. Chromosome analysis of blood lymphocytes showed 75% ring (18) cells and 25% normal cells, karyotype mos 46,XY,r(18)(::p11→q21::)[75]/46,XY[25]. His mother was phenotypically normal except for a double ureter and bifid renal pelvis as in his son. She had a supernumerary ring (18) in 10% of blood lymphocytes, karyotype mos 47,XX,+r(18)(::p11→q21::)[10]/46,XX[90]. Familial ring (18) is a rare cytogenetic abnormality. This is t…

AdultMaleGeneticsMonosomyMicrocephalyMosaicismRing chromosomeMothersAneuploidyKaryotypeAnatomyMotor ActivityBiologymedicine.diseasePhenotypeChromosome 18Intellectual DisabilityKaryotypingGeneticsRing 18medicineHumansFemaleSupernumeraryGenetics (clinical)American Journal of Medical Genetics Part A
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Temporal lobe grey matter volume in schizophrenia is associated with a genetic polymorphism influencing glycogen synthase kinase 3-beta activity

2010

At the crossroad of multiple pathways regulating trophism and metabolism, glycogen synthase kinase (GSK)3 is considered a key factor in influencing the susceptibility of neurons to harmful stimuli (neuronal resilience) and is a target for several psychiatric drugs that directly inhibit it or increase its inhibitory phosphorylation. Inhibition of GSK3 prevents apoptosis and could protect against the neuropathological processes associated with psychiatric disorders. A GSK3-beta promoter single-nucleotide polymorphism (rs334558) influences transcriptional strength, and the less active form was associated with less detrimental clinical features of mood disorders. Here we studied the effect of r…

AdultMaleGenotypeApoptosisNeuropathologyBiologyGrey matterGene Expression Regulation EnzymologicTemporal lobe03 medical and health sciencesBehavioral NeuroscienceSuperior temporal gyrusGlycogen Synthase Kinase 30302 clinical medicineGSK-3GeneticsmedicineHumansGenetic Predisposition to DiseasePromoter Regions GeneticGSK3B030304 developmental biology0303 health sciencesGlycogen Synthase Kinase 3 betaPolymorphism GeneticGenetic VariationBrodmann area 21medicine.diseaseTemporal LobeEnzyme Activationmedicine.anatomical_structureNeurologySchizophreniaChronic DiseaseNerve DegenerationSchizophreniaFemaleAtrophyNeuroscience030217 neurology & neurosurgeryGenes, Brain and Behavior
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Associations of classic Kaposi sarcoma with common variants in genes that modulate host immunity

2006

AbstractClassic Kaposi sarcoma (CKS) is an inflammatory-mediated neoplasm primarily caused by Kaposi sarcoma–associated herpesvirus (KSHV). Kaposi sarcoma lesions are characterized, in part, by the presence of proinflammatory cytokines and growth factors thought to regulate KSHV replication and CKS pathogenesis. Using genomic DNA extracted from 133 CKS cases and 172 KSHV-latent nuclear antigen-positive, population-based controls in Italy without HIV infection, we examined the risk of CKS associated with 28 common genetic variants in 14 immune-modulating genes. Haplotypes were estimated for IL1A, IL1B, IL4, IL8, IL8RB, IL10, IL12A, IL13, and TNF. Compared with controls, CKS risk was decrease…

AdultMaleGenotypeEpidemiologyPopulationSingle-nucleotide polymorphismBiologySettore MED/42 - Igiene Generale E ApplicataIL12AmedicineHumansGenetic Predisposition to DiseaseRisk factoreducationSarcoma KaposiAgedAged 80 and overeducation.field_of_studyClassic Kaposi SarcomaPolymorphism GeneticCase-control studyCancerHerpesvirus InfectionOdds ratioMiddle Agedmedicine.diseaseOncologyHaplotypesItalyGenetic VariantCase-Control StudiesImmunologyHerpesvirus 8 HumanCytokinesFemaleClassic Kaposi Sarcoma
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Cholinesterase variants: rapid characterisation by PCR/SSCP and evidence for molecular homogeneity.

1995

We have applied the technique of PCR-SSCP (polymerase chain reaction-single stranded conformation polymorphism) to characterise the molecular basis of cholinesterase deficiency and variants in a Jordanian family. PCR-SSCP proved to be a quick and sensitive method of screening cholinesterase variants in a clinical setting. An AG insertion at position 351 was found to cause a silent allele, for which the parents were heterozygous and three children homozygous. In addition, the father and two sons were heterozygous for an A to G transition at position 209, known to cause the dibucaine resistant variant. No linkage to the K variant was found, which has been reported previously in white populati…

AdultMaleGenotypeGenetic LinkageMolecular Sequence DataDibucainePolymerase Chain ReactionFrameshift mutationlaw.inventionlawGenetic linkageGenotypeGeneticsCholinesterasesHumansPoint MutationGenetic TestingAlleleFrameshift MutationGenetics (clinical)PolymerasePolymerase chain reactionAllelesPolymorphism Single-Stranded ConformationalCholinesteraseGeneticsJordanbiologyBase SequencePoint mutationSequence Analysis DNAMolecular biologyPedigreebiology.proteinFemaleMetabolism Inborn ErrorsResearch ArticleJournal of medical genetics
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