Search results for "types"

showing 10 items of 956 documents

The Factors Affecting Expansion of Reactive Tumor Infiltrating Lymphocytes (TIL) From Bladder Cancer and Potential Therapeutic Applications

2021

Tumor infiltrating lymphocytes (TIL) therapy was shown to provide durable objective response in patients with metastatic melanoma. As a fundamental first step to bring TIL therapy to clinical use, identification of patients whose tumors yield optimal numbers of reactive TIL is indispensable. We have previously shown that expansion of tumor reactive TIL from primary bladder tumors and lymph node metastases is feasible. Here, we performed TIL harvesting from additional surgical specimens (additional 31 primary tumors and 10 lymph nodes) to generate a heterogenous cohort of 53 patients with bladder cancer (BC) to evaluate the tumor characteristics that lead to tumor-reactive TIL expansion. Amo…

Malelcsh:Immunologic diseases. AllergyCD3Immunologychemical and pharmacologic phenomenaBacillus Calmette–GuerinLymphocyte ActivationCancer VaccinesImmunotherapy AdoptiveCohort StudiesBasal (phylogenetics)Tumor Necrosis Factor Receptor Superfamily Member 9Lymphocytes Tumor-InfiltratingmedicineImmunology and AllergyHumansadoptive cellular immunotherapyLymph nodeCells CulturedAgedCell ProliferationOriginal Researchmolecular subtypesBladder cancerbiologyTumor-infiltrating lymphocytesbusiness.industryhemic and immune systemsMiddle Agedmedicine.diseaseMycobacterium bovismedicine.anatomical_structureUrinary Bladder NeoplasmsLymphatic Metastasistumor-infiltrating lymphocytesCancer researchbiology.proteinInterleukin-2bladder cancerFemaleLymphAntibodyUrotheliumbusinesslcsh:RC581-607CD8Frontiers in Immunology
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Progastrin Represses the Alternative Activation of Human Macrophages and Modulates Their Influence on Colon Cancer Epithelial Cells

2014

Macrophage infiltration is a negative prognostic factor for most cancers but gastrointestinal tumors seem to be an exception. The effect of macrophages on cancer progression depends on their phenotype, which may vary between M1 (pro-inflammatory, defensive) to M2 (tolerogenic, pro-tumoral). Gastrointestinal cancers often become an ectopic source of gastrins and macrophages present receptors for these peptides. The aim of the present study is to analyze whether gastrins can affect the pattern of macrophage infiltration in colorectal tumors. We have evaluated the relationship between gastrin expression and the pattern of macrophage infiltration in samples from colorectal cancer and the influe…

Malelcsh:MedicineCell CountLigandsMonocytesWhite Blood CellsCell SignalingAnimal CellsMolecular Cell BiologyGastrointestinal CancersBasic Cancer ResearchMedicine and Health SciencesIntestinal Mucosalcsh:ScienceImmune ResponseWNT Signaling CascadeGastrinAged 80 and overMultidisciplinaryCD68Middle AgedImmunohistochemistrySignaling CascadesInterleukin 10PhenotypeOncologyColonic NeoplasmsInterleukin 12FemaleCellular TypesResearch ArticleSignal Transductionmedicine.medical_specialtyDrug Research and DevelopmentImmune CellsAdipose tissue macrophagesImmunologyAntigen-Presenting CellsGastroenterology and HepatologyBiologyCell Line TumorInternal medicineGastrinsGastrointestinal TumorsmedicineHumansProtein PrecursorsInterleukin 4AgedNeoplasm StagingInflammationPharmacologyCD86Blood CellsMacrophageslcsh:RImmunityBiology and Life SciencesCancers and NeoplasmsCancerCell BiologyMacrophage Activationmedicine.diseaseWnt ProteinsEndocrinologyCancer researchClinical Immunologylcsh:QNeoplasm GradingClinical MedicinePLoS ONE
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Identification of somatostatin receptor type 5 gene polymorphisms associated with acromegaly.

2011

ObjectiveThe aim of this study was to characterize the genetic variance of somatostatin receptor 5 (SSTR5) and investigate the possible correlation of such variants with acromegaly risk and different disease characteristics.Design and methodsThe SSTR5 gene coding region and 2000 bp upstream region was sequenced in 48 patients with acromegaly and 96 control subjects. Further, three single nucleotide polymorphisms (SNPs) were analyzed in the same group of acromegaly patients and in an additional group of 475 age- and sex-matched controls.ResultsIn total, 19 SNPs were identified in the SSTR5 gene locus by direct sequencing. Three SNPs (rs34037914, rs169068, and rs642249) were significantly ass…

Malemedicine.medical_specialty:MEDICINE [Research Subject Categories]Endocrinology Diabetes and MetabolismSingle-nucleotide polymorphismBiologyPolymorphism Single NucleotideSomatostatin receptorEndocrinologyInternal medicineAcromegalyDatabases GeneticmedicineHumansReceptors SomatostatinAlleleeducationAllele frequencyAllelesAgededucation.field_of_studySomatostatin receptor-5Polymorphism GeneticSomatostatin receptorReverse Transcriptase Polymerase Chain ReactionHaplotypeGeneral MedicineOdds ratioDNAMiddle Agedmedicine.diseaseLatviaHormonesEndocrinologyTreatment OutcomeHaplotypesAcromegalyClinical StudyFemaleEuropean journal of endocrinology
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Genetic and environmental influences on the fibrinolytic system: a twin study.

2004

SummaryThe determination of heritability is a key issue to assess the predictive power of polymorphisms for disease in clinical studies. The aim of this study was to determine the heritability of proteins and activation markers of the fibrinolytic system in a large cohort of healthy twins. Heritability was calculated as 0.76 for thrombin activatable fibrinolysis inhibitor (TAFI), 0.44 for plasminogen activator inhibitor-1 (PAI-1), and 0.43 for tissue plasminogen activator. No significant genetic influence was observed for α2-antiplasmin-plasmin-complex and D-dimer. Heritability explained by single gene polymorphisms was 25.2% for TAFI 505G>A, 31.5% for 1542C>G, and 50.0% for combinati…

Malemedicine.medical_specialtyCarboxypeptidase B2GenotypeArteriosclerosismedicine.medical_treatmentBiologyEnvironmentTissue plasminogen activatorCohort StudiesFibrin Fibrinogen Degradation ProductsInternal medicineFibrinolysisPlasminogen Activator Inhibitor 1medicineDiseases in TwinsHumansFibrinolysinGeneGeneticsalpha-2-AntiplasminPolymorphism GeneticActivator (genetics)FibrinolysisHematologyTwins MonozygoticHeritabilityTwin studyEndocrinologyPhenotypeHaplotypesHemostasisTwin Studies as TopicFemalePlasminogen activatormedicine.drugThrombosis and haemostasis
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Association between AT C573T polymorphism and cardiovascular risk factors in myocardial infarction.

2011

Abstract Background Polymorphisms in the AT1 gene have been associated with various parameters related to the pathogenesis of cardiovascular diseases and to myocardial infarction. This study analyzed the relationship between two polymorphisms of the angiotensin II AT-1 receptor gene (AT1_1166 and AT1_573) and the risk of ischemic heart disease by studying their association with several cardiovascular risk factors. Methods The sample population comprised 356 subjects: 174 patients who had survived myocardial infarction (61.01±8.15 years), and 182 age- and gender-matched controls (mean age of 60.25±9.43). The polymorphisms of the angiotensin II AT1-receptor gene (C573T and A1166C) were studie…

Malemedicine.medical_specialtyMyocardial InfarctionDiseaseGastroenterologyPolymorphism Single NucleotideRisk AssessmentReceptor Angiotensin Type 1Pathology and Forensic Medicinelaw.inventionPathogenesisGene FrequencylawRisk FactorsInternal medicineGenotypemedicineOdds RatioHumansGenetic Predisposition to DiseaseMyocardial infarctionPolymerase chain reactionAgedRetrospective StudiesAnalysis of VarianceAngiotensin II receptor type 1Chi-Square Distributionbusiness.industryGeneral MedicineAnthropometryMiddle Agedmedicine.diseaseAngiotensin IILogistic ModelsPhenotypeHaplotypesSpainCase-Control StudiesCardiologyFemaleCardiology and Cardiovascular MedicinebusinessCardiovascular pathology : the official journal of the Society for Cardiovascular Pathology
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Involvement of -308 TNF-alpha and 1267 Hsp70-2 polymorphisms and zinc status in the susceptibility of coronary artery disease (CAD) in old patients.

2006

Coronary artery disease (CAD) is characterized by an inflammatory status and it represents the major cause of death in elderly. Zinc deficiency and inflammatory genes within major histocompatibility complex (MHC) region are implicated in ischaemic heart diseases. TNF-alpha is present in coronary artery plaques and may provoke plaque instability. Hsp70 plays instead a pro-atherogenic role, via proinflammatory cytokine production, in atherosclerotic lesions contributing to plaque rupture. Contradictory data report the association between -308 TNF-alpha polymorphism and CAD, while no investigations exist on Hsp70-2 gene in CAD. In the current study, we analysed -308 TNF-alpha and 1267 Hsp70-2 …

Malemedicine.medical_specialtyPathologyAgingCoronary Artery DiseaseMajor histocompatibility complexGastroenterologyRisk AssessmentProinflammatory cytokineCoronary artery diseaseGene FrequencyRisk FactorsInternal medicineGenotypemedicineOdds RatioHumansCarotid StenosisGenetic Predisposition to DiseaseHSP70 Heat-Shock ProteinsAlleleCause of deathAgedPolymorphism Geneticbiologybusiness.industryInterleukin-6Tumor Necrosis Factor-alphaMiddle Agedmedicine.diseaseZincmedicine.anatomical_structureHaplotypesItalyCase-Control Studiesbiology.proteinTumor necrosis factor alphaFemaleGeriatrics and GerontologybusinessGerontologyBiomarkersArteryBiogerontology
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Association of a Mineralocorticoid Receptor Gene Polymorphism With Hypertension in a Spanish Population

2009

BACKGROUND To assess the association of polymorphisms and haplotypes of the mineralocorticoid receptor (MR) (NR3C2) gene to the risk of essential hypertension (HTN) in a Spanish population. METHODS This is a population-based study which included 1,502 subjects (748 women) >18 years old. Twenty-four polymorphisms of NR3C2 gene were analyzed by using SNPlex (Genotyping System based on OLA/PCR technology). RESULTS Alleles of the single-nucleotide polymorphism (SNP) rs5522 were significantly associated with the risk of HTN, both in the recessive and codominant models adjusted by age, gender, and body mass index (BMI). Genotype GG of the rs5522 showed to be protective against HTN odds ratio (OR)…

Malemedicine.medical_specialtyPopulationSingle-nucleotide polymorphismEssential hypertensionPolymerase Chain ReactionPolymorphism (computer science)Internal medicineGenotypePrevalenceInternal MedicineHumansMedicineGenetic Predisposition to Diseasecardiovascular diseaseseducationAllelesAgededucation.field_of_studyPolymorphism Geneticbusiness.industryHaplotypeDNAOdds ratioMiddle Agedmedicine.diseaseReceptors MineralocorticoidEndocrinologyHaplotypesSpainHypertensionFemaleGene polymorphismbusinessAmerican Journal of Hypertension
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Detection of Human Papillomavirus in Nonmelanoma Skin Cancer Lesions and Healthy Perilesional Skin in Kidney Transplant Recipients and Immunocompeten…

2013

Abstract Background The influence of human papillomavirus (HPV) on the development of nonmelanoma skin cancer (NMSC) is a topic of debate. HPV types from the beta genus (HPV-β) have been most frequently associated with the development of skin cancer. Objectives To analyze the prevalence and range of HPV types in NMSC lesions and healthy perilesional skin in immunodepressed and immunocompetent patients and to evaluate the influence of various clinical factors on the prevalence of HPV in skin cancer. Methods Nested polymerase chain reaction and sequencing were used to detect HPV in 120 NMSC samples obtained by biopsy from 30 kidney transplant recipients and 30 immunocompetent patients. In all…

Malemedicine.medical_specialtySkin NeoplasmsHistologyDermatologyKidney transplantPathology and Forensic MedicineBiopsymedicineHumansHuman papillomavirusPapillomaviridaeAgedSkinintegumentary systemmedicine.diagnostic_testHpv typesbusiness.industryConfoundingvirus diseasesOdds ratiomedicine.diseaseKidney TransplantationDermatologyFemaleSkin cancerbusinessImmunocompetenceNested polymerase chain reactionActas Dermo-Sifiliográficas (English Edition)
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Association of cathepsin B gene polymorphisms with tropical calcific pancreatitis

2006

Background and aims: Tropical calcific pancreatitis (TCP) is a type of chronic pancreatitis unique to countries in the tropics. Mutations in pancreatic secretory trypsin inhibitor (SPINK1) rather than cationic trypsinogen (PRSS1) explain the disease in only 50% of TCP patients. As cathepsin B (CTSB) is known to activate cationic trypsinogen, we attempted to understand the role of CTSB mutations in TCP. Evidence of epistatic interaction was investigated with the previously associated N34S SPINK1 allele, a variant considered to be a modifier rather than a true susceptibility allele. Subjects and methods: We sequenced the coding region of CTSB gene in 51 TCP patients and 25 controls and furthe…

Malemedicine.medical_specialtyTrypsinogenSingle-nucleotide polymorphismBiologyPolymerase Chain ReactionGastroenterologyCathepsin BCathepsin Bchemistry.chemical_compoundGene FrequencyPancreatitis ChronicInternal medicinemedicineHumansGenetic Predisposition to DiseaseTrypsinogen activationAlleleAllele frequencyGeneticsPolymorphism GeneticHaplotypeGastroenterologyCase-control studyCalcinosisHaplotypesPancreatitischemistryCase-Control StudiesMutationAcute DiseaseFemaleGut
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Four new cases of congenital secondary hypothyroidism due to a splice site mutation in the thyrotropin-beta gene: Phenotypic variability and founder …

2004

WOS: 000223072400081 PubMed ID: 15292359 Isolated TSH deficiency is a rare cause of congenital hypothyroidism. We here report four children from two consanguineous Turkish families with isolated TSH deficiency. Affected children who were screened at newborn age had an unremarkable TSH result and a low serum TSH level at diagnosis. Age at diagnosis and clinical phenotype were variable. All affected children carried an identical homozygous splice site mutation (IVS2 + 5 Gdouble right arrowA) in the TSHbeta gene. This mutation leads to skipping of exon 2 and a loss of the translational start codon without ability to produce a TSH-like protein. However, using specific monoclonal antibodies, we …

Malemedicine.medical_specialtyendocrine systemGuanineendocrine system diseasesEndocrinology Diabetes and MetabolismClinical BiochemistryThyrotropinLocus (genetics)Thyrotropin beta SubunitBiologyBiochemistryExonEndocrinologyHypothyroidismInternal medicinemedicineCongenital HypothyroidismHumansChildGeneGenotypingGeneticsSplice site mutationAdenineBiochemistry (medical)HaplotypeHomozygoteInfant NewbornInfantmedicine.diseaseFounder EffectIntronsCongenital hypothyroidismPedigreeEndocrinologyPhenotypeHaplotypesChild PreschoolMutationFemalehormones hormone substitutes and hormone antagonistsFounder effect
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