Search results for "vascular cell adhesion molecule-1"

showing 10 items of 65 documents

Antibodies to proteinase 3 mediate expression of vascular cell adhesion molecule-1 (VCAM-1).

1996

SUMMARY VCAM-1 was first identified as an adhesion molecule induced on human endothelial cells (HEC) by inflammatory cytokines such as IL-1, tumour necrosis factor (TNF), and lipopolysaccharide (LPS). The molecule binds to a variety of leucocytes, including B cells, T cells, basophils, eosinophils and monocytes. Vascular expression of VCAM-1 has been associated with a number of disease states, including rheumatoid arthritis and vasculitis. The detection of antineutrophil cytoplasmic antibodies (ANCA), especially to proteinase 3 (PR3), has become important in the diagnosis of Wegener’s granulomatosis (WG) and related vasculitides. Recently we were able to demonstrate a direct effect of anti-…

MyeloblastinT-LymphocytesImmunologyMolecular Sequence DataVascular Cell Adhesion Molecule-1BiologyAntibodiesProinflammatory cytokinechemistry.chemical_compoundAntigenProteinase 3Cell AdhesionImmunology and AllergyHumanscardiovascular diseasesRNA MessengerVCAM-1Cell adhesionCells CulturedBase SequenceCell adhesion moleculeSerine EndopeptidasesOriginal ArticleschemistryImmunologybiology.proteinTumor necrosis factor alphaEndothelium VascularAntibodyClinical and experimental immunology
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Effects of transdermal hormone replacement therapy on levels of soluble P- and E-selectin in postmenopausal healthy women

2002

Abstract Objective: To study the adhesion molecule pattern in postmenopausal women who were not receiving hormone replacement therapy (HRT), HRT users, and fertile women. Design: Case-control study. Setting: Second University of Naples, Naples, Italy. Patient(s): Fifty healthy naturally postmenopausal women and 20 fertile women. Intervention(s): Twenty-six women received no HRT and 24 received continuous transdermal 17β−estradiol, 0.05 mg/d, plus oral acetate nomegestrol, 5 mg/d. Main Outcome Measure(s): Levels of the soluble forms of intercellular adhesion molecule-1 (ICAM-1), vascular cell adhesion molecule-1 (VCAM-1), E-selectin, and P-selectin. Result(s): Women who did not received HRT …

Nomegestrolmedicine.medical_specialtyAdministration Cutaneougenetic structuresP-selectinmedicine.drug_classHormone Replacement Therapymedicine.medical_treatmentHRTVascular Cell Adhesion Molecule-1Administration Cutaneouschemistry.chemical_compoundInternal medicineE-selectinmedicineHumansICAM-1biologyEstradiolbusiness.industryCase-control studyObstetrics and GynecologyHormone replacement therapy (menopause)Megestrolmedicine.diseaseIntercellular Adhesion Molecule-1AtherosclerosisMenopausePostmenopauseP-SelectinEndocrinologyReproductive MedicinechemistryEstrogenCase-Control Studiesbiology.proteinAdhesion molecules; Atherosclerosis; HRTFemalebusinessCase-Control StudieE-SelectinAdhesion moleculesHuman
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Transfusion-associated graft-versus-host disease-report of two further cases with an immunohistochemical analysis

1994

Summary Transfusion-associated graft-vs.-host disease (tGVHD) is a severe disease usually affecting immunocompromised hosts with haematological neoplasia. Two patients with acute leukaemia are reported, who developed fatal tGVHD after blood transfusions. Intercellular adhesion molecule 1 (ICAM-1), vascular cell adhesion molecule 1 (VCAM-1) and endothelial leucocyte adhesion molecule 1 (ELAM-1) expression and the CD4/CD8 ratio were assessed in lesional skin. ICAM-1 was strongly expressed on epidermal keratinocytes and endothelial cells (EC) and correlated with HLA-DR staining. VCAM-1 was strongly expressed on EC in the superficial dermal vessels. ELAM-1 stained weakly on EC in some of the su…

Pathologymedicine.medical_specialtyBlood transfusionCD3medicine.medical_treatmentIntercellular Adhesion Molecule-1CD4-CD8 RatioGraft vs Host DiseaseVascular Cell Adhesion Molecule-1CD18DermatologyTransfusion-associated graft versus host diseaseFatal OutcomeHumansLeukemia-Lymphoma Adult T-CellMedicineSkinbiologybusiness.industryCell adhesion moleculeTransfusion ReactionMiddle AgedIntercellular Adhesion Molecule-1medicine.diseaseLeukemia Myeloid AcuteLeukemiaImmunologybiology.proteinFemalebusinessCell Adhesion MoleculesCD8Clinical and Experimental Dermatology
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Comparative Study of Adhesion Molecule Expression in Cultured Human Macro- and Microvascular Endothelial Cells

2002

Culture systems as models for disease are only valid as long as they are comparable to in vivo conditions. The phenotype of cultured endothelial cells (ECs) has only been sporadically compared to the corresponding phenotype in vivo. Thus, we compared by immunolocalization the endothelial expression of ICAM-1, VCAM, and E-selectin in vivo in stimulated/unstimulated human umbilical vein endothelial cells (HUVEC) as a model for macrovascular ECs and stimulated/unstimulated HPMEC (human pulmonary microvessel endothelial cells) as a model for pulmonary microvascular ECs with that in human lungs in vivo (normal and ARDS). Proinflammatory stimuli in vitro were used to stimulate conditions relevant…

Pathologymedicine.medical_specialtyEndotheliumClinical BiochemistryVascular Cell Adhesion Molecule-1Umbilical veinPathology and Forensic MedicineIn vivoE-selectinCell AdhesionmedicineHumansMolecular BiologyMicrovesselCells CulturedRespiratory Distress SyndromebiologyCell adhesion moleculeIntercellular Adhesion Molecule-1ImmunohistochemistryMolecular biologyEndothelial stem cellPhenotypemedicine.anatomical_structureCell culturecardiovascular systembiology.proteinEndothelium VascularE-SelectinExperimental and Molecular Pathology
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Interleukin 6 (IL-6) deficiency delays lupus nephritis in MRL-Faslpr mice: the IL-6 pathway as a new therapeutic target in treatment of autoimmune ki…

2009

Objective.To investigate the pathophysiological effect of interleukin 6 (IL-6) on lupus nephritis in MRL-Faslprmice.Methods.We generated IL-6-deficient MRL-Faslprmice using a backcross/intercross breeding scheme. Renal pathology was evaluated using immunohistochemistry detection for macrophages, lymphocytes, vascular cell adhesion molecule-1 (VCAM-1), and TUNEL (terminal deoxynucleotide transferase-mediated dUTP nick end-labeling) for apoptotic cells, and renal IgG and C3 deposition by immunofluorescence staining. Expression of inflammatory markers in the spleen was analyzed by quantitative real-time reverse transcription-polymerase chain reaction. Serum cytokine concentrations were detecte…

Pathologymedicine.medical_specialtyMice Inbred MRL lprImmunologyLupus nephritisVascular Cell Adhesion Molecule-1SpleenKidneyInterferon-gammaMiceLife ExpectancyRheumatologyImmunology and AllergyMedicineAnimalsHumansLupus Erythematosus Systemicfas ReceptorInterleukin 6InflammationMice KnockoutKidneyMice Inbred BALB CLupus erythematosusbiologybusiness.industryInterleukin-6Kidney metabolismmedicine.diseaseLupus NephritisInterleukin-10Proteinuriamedicine.anatomical_structureRenal pathologyImmunologybiology.proteinFemalebusinessBiomarkersSpleenKidney diseaseSignal TransductionThe Journal of rheumatology
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trans -Lycopene from tomato juice attenuates inflammatory biomarkers in human plasma samples: An intervention trial

2017

cope : To evaluate the effects of carotenoids from tomato juice (TJ) on inflammatory biomarkers, by performing a 4-week dose-response nutritional trial in a population at high cardiovascular risk. Methods and results : An open, prospective, randomized, cross-over, and controlledclinical trial was carried out with 28 volunteers (mean age 69.7 ± 3.1 years; mean BMI 31.5 ± 3.6 kg/m2) at high cardiovascular risk, which were assigned to consume daily for 4 weeks in random order: 200 mL (LD) or 400 mL (HD) of TJ, or water as a control (C), with a 21-day wash-out period between each intervention. Blood samples were collected at baseline (B) and after each intervention. Endpoints included significa…

Risk0301 basic medicinemedicine.medical_specialtyChemokinePopulationVascular Cell Adhesion Molecule-1Inflammation030204 cardiovascular system & hematologyGastroenterologyBody Mass Index03 medical and health scienceschemistry.chemical_compoundLycopene0302 clinical medicineSolanum lycopersicumFunctional FoodInternal medicinemedicineHumansObesityIntervention trialeducationCarotenoidAgedchemistry.chemical_classificationeducation.field_of_studyCross-Over Studies030109 nutrition & dieteticsbiologyAnti-Inflammatory Agents Non-SteroidalStereoisomerismMiddle AgedAtherosclerosisIntercellular Adhesion Molecule-1CarotenoidsInflammatory biomarkersLycopeneSurgeryFruit and Vegetable JuiceschemistrySpainHuman plasmabiology.proteinInflammation Mediatorsmedicine.symptomBiomarkersFollow-Up StudiesFood ScienceBiotechnologyMolecular Nutrition & Food Research
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eNOS Activation by HDL Is Impaired in Genetic CETP Deficiency.

2014

Mutations in the CETP gene resulting in defective CETP activity have been shown to cause remarkable elevations of plasma HDL-C levels, with the accumulation in plasma of large, buoyant HDL particles enriched in apolipoprotein E. Genetic CETP deficiency thus represents a unique tool to evaluate how structural alterations of HDL impact on HDL atheroprotective functions. Aim of the present study was to assess the ability of HDL obtained from CETP-deficient subjects to protect endothelial cells from the development of endothelial dysfunction. HDL isolated from one homozygous and seven heterozygous carriers of CETP null mutations were evaluated for their ability to down-regulate cytokine-induced…

Settore MED/09 - Medicina InternaCHOLESTEROL EFFLUXApolipoprotein BEpidemiologylcsh:MedicineANTIINFLAMMATORY PROPERTIESmedicine.disease_causeBiochemistryVascular Medicinechemistry.chemical_compoundHigh-density lipoproteinEnosMedicine and Health SciencesEndothelial dysfunctionlcsh:ScienceMutationMultidisciplinarybiologyHomozygoteCETP; eNOS; HDL;NeurochemistryLipidsGenetic EpidemiologyeNOSlipids (amino acids peptides and proteins)AnatomyNeurochemicalsLipoproteins HDLResearch Articlemedicine.medical_specialtyDrug Research and DevelopmentHDLNitric Oxide Synthase Type IIILipoproteinsENDOTHELIAL FUNCTIONINHIBITIONCardiologyDown-RegulationVascular Cell Adhesion Molecule-1Nitric OxideCELL-ADHESION MOLECULE-1Lipid Metabolism Inborn ErrorsESTER TRANSFER PROTEINInternal medicineCETPCholesterylester transfer proteinHuman Umbilical Vein Endothelial CellsmedicineHumansNITRIC-OXIDE SYNTHASEInflammationClinical GeneticsPharmacologyCholesterollcsh:RTorcetrapibEndothelial CellsBiology and Life SciencesProteinsnutritional and metabolic diseasesLipid MetabolismAtherosclerosismedicine.diseasebiology.organism_classificationCholesterol Ester Transfer Proteinscarbohydrates (lipids)MetabolismEndocrinologychemistryOther Clinical MedicineMutationImmunologyCardiovascular Anatomybiology.proteinlcsh:QTORCETRAPIBClinical MedicineHIGH-DENSITY-LIPOPROTEINSCAVENGER RECEPTOR BI
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Cutaneous Exposure to the Superantigen Staphylococcal Enterotoxin B Elicits a T-Cell-Dependent Inflammatory Response

1996

We analyzed the impact of superantigens secreted by skin-colonizing Staphylococci on the skin and the associated lymphoid tissue following epicutaneous application and intracutaneous injection of small amounts of staphylococcal enterotoxin B (SEB). A single intracutaneous injection of 50 ng of SEB elicited a strong inflammatory response in the skin of BALB/c mice. Three to 6 h later, we observed langerhans cell activation, mast cell degranulation, vasodilation, upregulation of ICAM-1, and induction of VCAM-1 on dermal blood vessels, with vascular adhesion of granulocytes. by 12 to 24 h, cell infiltration of the dermis increased, reaching the epidermis. Among the infiltrating leukocytes, a s…

Staphylococcus aureusLangerhans cellT cellVascular Cell Adhesion Molecule-1InflammationDermatitischemical and pharmacologic phenomenaDermatologyCD8-Positive T-LymphocytesPeripheral blood mononuclear cellBiochemistryEnterotoxinsMicemedicineSuperantigenAnimalsIntradermal injectionMolecular BiologyMice Inbred BALB CSuperantigensbusiness.industryDegranulationhemic and immune systemsCell Biologybiological factorsmedicine.anatomical_structureImmunologyTumor necrosis factor alphaFemalemedicine.symptombusinessJournal of Investigative Dermatology
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Prostacyclin inhibits adhesion of polymorphonuclear leukocytes to human vascular endothelial cells due to adhesion molecule independent regulatory me…

2002

Prostacyclin is an important endothelial mediator involved in the interaction of neutrophils (PMN) with the vessel wall. Many studies have shown the beneficial effects of prostacyclin in ischemia and reperfusion. However, no previous study has investigated the direct effects of the prostacyclin analogs iloprost (ILO) and alprostadil (PGE(1)) on the endothelial part of the adhesion process. Human umbilical vein endothelial cells (HUVECs) were grown to confluence, stimulated with 300 U/ml TNF-alpha and treated with increasing concentrations of ILO and PGE(1). The cells were washed to remove TNF and the inhibitors and adhesion of fluorescence-green labeled PMN was determined microscopically. I…

Umbilical VeinsEndotheliumNeutrophilsPhysiologyVascular Cell Adhesion Molecule-1ProstacyclinPharmacologyUmbilical veinPhysiology (medical)Cell AdhesionmedicineHumansIloprostAlprostadilChemoattractant activityCells CulturedCell NucleusTumor Necrosis Factor-alphaChemistryChemotaxisAdhesionrespiratory systemEpoprostenolrespiratory tract diseasesEndothelial stem cellmedicine.anatomical_structureBiochemistrycardiovascular systemlipids (amino acids peptides and proteins)Tumor necrosis factor alphaEndothelium VascularCardiology and Cardiovascular MedicineCell Adhesion MoleculesIloprostmedicine.drugBasic Research in Cardiology
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Isolation and molecular characterization of brain microvascular endothelial cells from human brain tumors.

2002

Brain tumor formation and growth is accompanied by the proliferation and infiltration of blood capillaries. The phenotypes of endothelial cells that make up capillaries are known to differ not only in the tissues in which endothelial cells are located but also as a result of the microenvironment to which they are exposed. For this reason, primary cultures of brain endothelial cells were isolated from human brain tumors removed by surgery and compared with cells from normal tissue. The primary confluent monolayers that grew out of isolated capillary fragments consisted of closely associated, elongated, fusiform-shaped cells. But brain tumor-derived endothelial cells in culture exhibited sign…

Vascular Cell Adhesion Molecule-1Cell SeparationBiologyBlood–brain barrierAntigenvon Willebrand FactormedicineTumor Cells CulturedHumansCell adhesionCell SizeFluorescent DyesTight junctionBrain NeoplasmsBrainMembrane ProteinsCell BiologyGeneral MedicineHuman brainCarbocyaninesmedicine.diseaseIntercellular Adhesion Molecule-1PhosphoproteinsCell biologyMicroscopy Electronmedicine.anatomical_structureCell cultureBlood-Brain BarrierZonula Occludens-1 ProteinEndothelium VascularStem cellPlant LectinsE-SelectinInfiltration (medical)Developmental BiologyIn vitro cellulardevelopmental biology. Animal
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