Search results for "vasodilator"

showing 10 items of 128 documents

The lack of the effect of DA-1 and DA-2 dopamine agonists on the isolated guinea-pig atria.

1987

1 The effects of dopamine and both DA-1 and DA-2 dopamine receptor agonists have been studied on the contractile force of electrically driven guinea-pig left atria and frequency of spontaneously beating right atria. 2 Pretreatment of animals with reserpine caused a parallel rightward shift of the concentration-response curve to dopamine of either preparation. 3 Propranolol, but not domperidone, shifted to the right the dose-response curve for the positive inotropic and chronotropic effects of dopamine. 4 Neither apomorphine, fenoldopam, bromocriptine nor piribedil had effects on the frequency and contractile force of the isolated guinea-pig atria. 5 These results suggest that DA-1 and DA-2 …

ChronotropicMalemedicine.medical_specialtyFenoldopamApomorphineVasodilator AgentsGuinea PigsPharmacologyFenoldopamIn Vitro TechniquesReceptors DopaminePiribedilDopamineInternal medicinemedicineAnimalsBromocriptinePharmacologyChemistryGeneral NeurosciencePiribedilHeartReserpineBenzazepinesPropranololBromocriptineDomperidoneElectric StimulationApomorphineEndocrinologyDopamine receptorcardiovascular systemFemalemedicine.drugJournal of autonomic pharmacology
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In vitro and in vivo characterization of a new organic nitrate hybrid drug covalently bound to pioglitazone.

2014

<b><i>Background/Aims:</i></b> Organic nitrates represent a group of nitrovasodilators that are clinically used for the treatment of ischemic heart disease. The new compound CLC-3000 is an aminoethyl nitrate (AEN) derivative of pioglitazone, a thiazolidinedione antidiabetic agent combining the peroxisome proliferator-activated receptor γ agonist activity of pioglitazone with the NO-donating activity of the nitrate moiety. <b><i>Methods:</i></b> In vitro and in vivo characterization was performed by isometric tension recording, platelet function, bleeding time and detection of oxidative stress. <b><i>Results:</i></…

DrugBlood PlateletsMaleBleeding TimePlatelet Aggregationmedia_common.quotation_subjectVasodilator Agentsmedicine.disease_causeMitochondria Heartchemistry.chemical_compoundNitrateFibrinolytic AgentsIn vivomedicineAnimalsHumansHypoglycemic AgentsRats WistarAortamedia_commonPharmacologyNitratesPioglitazoneChemistryGeneral MedicineReactive Nitrogen SpeciesIn vitroOrganic nitratesMice Inbred C57BLVasodilationBiochemistryCovalent bondVasoconstrictionThiazolidinedionesReactive Oxygen SpeciesPioglitazoneOxidative stressmedicine.drugPharmacology
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Mechanisms underlying diabetes enhancement of endothelin-1-induced contraction in rabbit basilar artery

2004

The influence of alloxan-induced diabetes on the reactivity of rabbit basilar artery to endothelin-1 was examined. Endothelin-1 induced concentration-dependent contraction of basilar arteries that was higher in diabetic than in control rabbits. Endothelium removal produced a higher enhancement of the endothelin-1-induced contraction in control than in diabetic rabbits. N(G)-nitro-L-arginine (L-NOArg) enhanced the maximal contraction induced by endothelin-1 in control rabbits and potentiated this response in diabetic rabbits. Endothelin ETA receptor antagonist, cyclo(D-Asp-Pro-D-Val-Leu-D-Trp) (BQ-123), inhibited endothelin-1-induced contraction in both rabbit groups. Endothelin ETB receptor…

Endothelin Receptor AntagonistsMaleNitroprussidemedicine.medical_specialtyContraction (grammar)Vascular smooth muscleEndotheliumEndothelin A Receptor AntagonistsVasodilator AgentsEndothelin B Receptor AntagonistsNitroargininePeptides CyclicMuscle Smooth VascularDiabetes Mellitus ExperimentalPiperidinesIsometric Contractionmedicine.arteryInternal medicinemedicineBasilar arteryAnimalsEnzyme InhibitorsAntihypertensive AgentsPharmacologyDiabetisEndothelin-1Artèriesbusiness.industryEndoteli vascularReceptor Endothelin AReceptor Endothelin BEndothelin 1Òxid nítricEndothelin A Receptor AntagonistsEndothelin B Receptor AntagonistsEndocrinologymedicine.anatomical_structureBasilar Arterycardiovascular systemRabbitsbusinessEndothelin receptorOligopeptidesEuropean Journal of Pharmacology
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Myocardial contrast echocardiography in biopsy-proven primary cardiac amyloidosis.

2008

Abstract Cardiac vasculature is affected in 88-90% of patients with primary cardiac amyloidosis (CA). Myocardial contrast echocardiography (MCE) relies on the ultrasound detection of microbubble contrast agents that are solely confined to the intravascular space, and are therefore useful in the evaluation of flow in the microvasculature. This is the first case report describing the use of MCE during vasodilator stress to evaluate coronary flow reserve in a patient with biopsy-proven primary CA and angiographically normal coronaries. Qualitative MCE demonstrated delayed replenishment of microbubbles during peak stress; quantitative analysis was consistent with a reduction in total myocardial…

Gadolinium DTPAMalemedicine.medical_specialtyHeart DiseasesBiopsyVasodilator AgentsContrast MediaInternal medicinemedicineHumansechocardiography cardiac amyloidosis.Radiology Nuclear Medicine and imagingFluorocarbonsmedicine.diagnostic_testbusiness.industryAmyloidosisUltrasoundCoronary flow reserveMagnetic resonance imagingAmyloidosisGeneral MedicineBlood flowMiddle Agedmedicine.diseaseMagnetic Resonance ImagingSettore MED/11 - Malattie Dell'Apparato CardiovascolareCardiac amyloidosisEchocardiographyStrain rate imagingCardiologyMicrobubblesRadiologyCardiology and Cardiovascular MedicinebusinessEchocardiography Stress
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In situ kinetic modelling of intestinal efflux in rats: functional characterization of segmental differences and correlation with in vitro results.

2007

The objective was to devise and apply a novel modelling approach to combine segmental in situ rat perfusion data and in vitro cell culture data, in order to elucidate the contribution of efflux in drug absorption kinetics. The fluoroquinolone CNV97100 was used as a model P-gp substrate. In situ intestinal perfusion was performed in rat duodenum, jejunum, ileum and colon to measure the influence of P-gp expression on efflux. Inhibition studies of CNV97100 were performed in the presence of verapamil, quinidine, cyclosporin A and p-aminohippuric acid. Absorption/efflux parameters were modelled simultaneously, using data from both in situ studies as well as in vitro studies. The maximal efflux …

In situAbsorption (pharmacology)MaleColonVasodilator AgentsPharmaceutical ScienceIleumMuscarinic AntagonistsModels BiologicalIntestinal absorptionPermeabilityJejunumCiprofloxacinCyclosporin aIntestine SmallmedicineAnimalsPharmacology (medical)ATP Binding Cassette Transporter Subfamily B Member 1Intestinal MucosaRats WistarP-glycoproteinPharmacologybiologyDose-Response Relationship DrugMolecular StructureChemistryGeneral MedicineQuinidineRatsKineticsmedicine.anatomical_structureBiochemistryIntestinal AbsorptionVerapamilbiology.proteinBiophysicsCyclosporinep-Aminohippuric AcidEffluxAlgorithmsImmunosuppressive AgentsFluoroquinolonesBiopharmaceuticsdrug disposition
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Uncoupling of Endothelial Nitric Oxide Synthase in Perivascular Adipose Tissue of Diet-Induced Obese Mice

2015

Objective— The present study was conducted to investigate the contribution of perivascular adipose tissue (PVAT) to vascular dysfunction in a mouse model of diet-induced obesity. Approach and Results— Obesity was induced in male C57BL/6J mice with a high-fat diet for 20 weeks, and vascular function was studied with myograph. In PVAT-free aortas isolated from obese mice, the endothelium-dependent, nitric oxide–mediated vasodilator response to acetylcholine remained normal. In contrast, a clear reduction in the vasodilator response to acetylcholine was observed in aortas from obese mice when PVAT was left in place. Adipocytes in PVAT were clearly positive in endothelial nitric oxide synthase…

Male0301 basic medicinemedicine.medical_specialtyNitric Oxide Synthase Type IIIVasodilator AgentsAdipose tissueAorta ThoracicVasodilation030204 cardiovascular system & hematologyArginineDiet High-FatNitric OxideNitric oxide03 medical and health scienceschemistry.chemical_compound0302 clinical medicineAdipokinesSuperoxidesEnosInternal medicineParacrine CommunicationAdipocytesmedicineAnimalsObesityEnzyme InhibitorsPhosphorylationAdiposityArginaseDose-Response Relationship DrugbiologyNitric Oxide Synthase Type IIIbiology.organism_classificationMice Inbred C57BLVasodilationArginaseDisease Models Animal030104 developmental biologyEndocrinologyAdipose TissuechemistryCytokinesInflammation MediatorsCardiology and Cardiovascular MedicineDiet-induced obeseSignal TransductionMyographArteriosclerosis, Thrombosis, and Vascular Biology
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Chronic Administration of Quercetin Induces Biomechanical and Pharmacological Remodeling in the Rat Coronary Arteries

2017

Acute dilation brought about by the dietary flavonoid quercetin in coronary arterioles has been described earlier, but no information is available on its chronic effects. Male Wistar rats (body weight about 190 g) were divided to two groups: the quercetin-treated group (n=22) had quercetin supplementation of approximately 30 mg/kg/day, whereas the control group (n=20) had none. After eight weeks of treatment, intramural coronary arterioles with identical passive diameters (178+/-14 microm and 171+/-9 microm) were prepared and their biomechanics and pharmacological reactivities were tested using pressure arteriography ex vivo. The spontaneous tone of quercetin-treated arteries was higher (16…

Male0301 basic medicinemedicine.medical_specialtyPhysiologyVasodilator AgentsLumen (anatomy)Vascular Remodeling030204 cardiovascular system & hematologyBody weightDrug Administration ScheduleNitric oxide03 medical and health scienceschemistry.chemical_compoundCoronary circulation0302 clinical medicineInternal medicinemedicineAnimalsRats WistarNo releaseGeneral MedicineCoronary VesselsRatsVasodilationCoronary arteries030104 developmental biologyEndocrinologymedicine.anatomical_structurechemistryQuercetinQuercetinEx vivoPhysiological Research
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Prevalence and prognostic impact of nonischemic late gadolinium enhancement in stress cardiac magnetic resonance

2020

Aim To assess the prevalence and prognostic significance of NI-LGE in patients undergoing stress-CMR. Methods Stress-CMR with either dipyridamole or adenosine was performed in 283 patients (228 men, 81%) including perfusion imaging, wall motion evaluation and LGE. Follow-up was completed in all enrolled patients (median time: 1850 days; interquartile range: 1225-2705 days). Composite endpoint included cardiac death, ventricular tachycardia, myocardial infarction, stroke, hospitalization for cardiac cause and coronary revascularization performed beyond 90 days from stress-CMR scans. Results One hundred and twelve patients (40%) had negative LGE (no-LGE), 140 patients (49%) I-LGE and 31 patie…

MaleAdenosineTime FactorsVasodilator AgentsContrast MediaPerfusion scanning030204 cardiovascular system & hematologyVentricular tachycardia0302 clinical medicineRisk FactorsInterquartile rangePrevalence030212 general & internal medicineMyocardial infarctionStrokenonischemic fibrosislate gadolinium enhancement; nonischemic cardiac findings; nonischemic fibrosis; prognosis; stress perfusion cardiac magnetic resonanceDipyridamoleGeneral MedicineMiddle AgedMagnetic Resonance ImagingDipyridamolelate gadolinium enhancementstress perfusion cardiac magnetic resonanceembryonic structurescardiovascular systemCardiologyFemaleCardiology and Cardiovascular MedicinePerfusionmedicine.drugnonischemic cardiac findingsmedicine.medical_specialtyHeart DiseasesPerfusion ImagingRisk Assessment03 medical and health sciencesPredictive Value of TestsInternal medicinemedicineHumanscardiovascular diseasesSurvival analysisAgedRetrospective Studiesbusiness.industryMyocardiummedicine.diseaseFibrosisLate gadolinium enhancement Nonischemic cardiac findings Nonischemic fibrosis Prognosis Stress perfusion cardiac magnetic resonanceprognosisbusinessJournal of Cardiovascular Medicine
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Riociguat versus sildenafil on hypoxic pulmonary vasoconstriction and ventilation/perfusion matching

2017

Introduction Current treatment with vasodilators for pulmonary hypertension associated with respiratory diseases is limited by their inhibitory effect on hypoxic pulmonary vasoconstriction (HPV) and uncoupling effects on ventilation-perfusion (V'/Q'). Hypoxia is also a well-known modulator of the nitric oxide (NO) pathway, and may therefore differentially affect the responses to phosphodiesterase 5 (PDE5) inhibitors and soluble guanylyl cyclase (sGC) stimulators. So far, the effects of the sGC stimulator riociguat on HPV have been poorly characterized. Materials and methods Contraction was recorded in pulmonary arteries (PA) in a wire myograph. Anesthetized rats were catheterized to record …

MaleAnoxemiaPulmonologyPulmonary FibrosisVasodilator Agentslcsh:MedicineVasodilation030204 cardiovascular system & hematologyPharmacologyVascular Medicinechemistry.chemical_compound0302 clinical medicineSoluble Guanylyl CyclaseHypoxic pulmonary vasoconstrictionPulmonary fibrosisMedicine and Health SciencesPulmonary Arterieslcsh:ScienceHypoxiaMultidisciplinaryVasodilatorsDrugsRespiratory organs diseasesArteriesMiddle AgedChemistryPhysical Sciencescardiovascular systemFemalemedicine.symptomAnatomyMedicamentsmedicine.drugResearch ArticleChemical Elementsinorganic chemicalsSildenafilHypertension PulmonaryChronic Obstructive Pulmonary DiseaseEnzyme ActivatorsIn Vitro TechniquesPulmonary ArteryRiociguatSildenafil CitrateMalalties de l'aparell respiratori03 medical and health sciencesMedical HypoxiamedicineVentilation-Perfusion RatioAnimalsHumansRats WistarAgedPharmacologybusiness.industrylcsh:RAnoxèmiaBiology and Life SciencesHypoxia (medical)Phosphodiesterase 5 Inhibitorsmedicine.diseasePulmonary hypertensionFibrosisRatsOxygenDisease Models AnimalPyrimidines030228 respiratory systemchemistryVasoconstrictionCardiovascular AnatomyPyrazolesBlood Vesselslcsh:QSoluble guanylyl cyclasebusinessDevelopmental Biology
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Nitric oxide modulates cerebral blood flow stimulation by acetazolamide in the rat cortex: a laser Doppler scanning study

2001

Abstract The involvement of nitric oxide (NO) in cerebral blood flow (CBF) stimulation by acetazolamide was studied in anaesthetised, mechanically ventilated Wistar rats. CBF was monitored by laser Doppler scanning. Acetazolamide induced a long-lasting significant rCBF-increase. Application of N G -Nitro- l -arginine (L-NNA), an inhibitor of all NO synthetases (NOS), prevented CBF stimulation by acetazolamide. Continuous infusion of the exogenous NO donor SIN-1 (3-morpholinosydnonimine) suppressed L-NNA induced increases of mean arterial blood pressure without effect on rCBF in comparison to baseline. Additional acetazolamide injection then again caused a significant increase of rCBF in spi…

MaleArginineVasodilator AgentsHemodynamicsBlood PressureStimulationPharmacologyNitric OxideNitroarginineNitric oxidechemistry.chemical_compoundLaser-Doppler FlowmetrymedicineAnimalsNitric Oxide DonorsRats WistarCarbonic Anhydrase InhibitorsCerebral CortexChemistryGeneral NeuroscienceLaser Doppler velocimetryRatsAcetazolamidemedicine.anatomical_structurenervous systemCerebral blood flowCerebral cortexCerebrovascular CirculationMolsidomineAnesthesiaNitric Oxide SynthaseAcetazolamidecirculatory and respiratory physiologymedicine.drugNeuroscience Letters
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