Search results for "violet"

showing 10 items of 883 documents

Cytochrome c is released in a single step during apoptosis

2005

Release of cytochrome c from mitochondria is a central event in apoptotic signaling. In this study, we utilized a cytochrome c fusion that binds fluorescent biarsenical ligands (cytochrome c-4CYS (cyt. c-4CYS)) as well as cytochrome c-green fluorescent protein (cyt. c-GFP) to measure its release from mitochondria in different cell types during apoptosis. In single cells, the kinetics of cyt. c-4CYS release was indistinguishable from that of cyt. c-GFP in apoptotic cells expressing both molecules. Lowering the temperature by 7 degrees C did not affect this corelease, but further separated cytochrome c release from the subsequent decrease in mitochondrial membrane potential (DeltaPsi(m)). Cyt…

CytochromeUltraviolet RaysGreen Fluorescent ProteinsApoptosisLigandsMembrane PotentialsJurkat CellsCytochrome C1HumansCytochrome c oxidaseEnzyme InhibitorsMolecular BiologyProtein Synthesis InhibitorsMicroscopy VideobiologyTumor Necrosis Factor-alphaCytochrome bCytochrome cTemperatureCytochromes cCytochrome P450 reductaseCell BiologyStaurosporineMitochondriaCell biologyKineticsenzymes and coenzymes (carbohydrates)Coenzyme Q – cytochrome c reductaseDactinomycinbiology.proteinApoptosomeBiomarkersHeLa CellsCell Death & Differentiation
researchProduct

Effects of Short- and Long-term Ultraviolet B Irradiation on the Immune System of the Common Carp (Cyprinus carpio)¶

2005

Carp (Cyprinus carpio) were repeatedly exposed to 0, 60, 120 and 240 mJ/cm2 ultraviolet B (UVB) radiation three times in 1 week (short-term exposure) or 12 times in 4 weeks (long-term exposure). The effect of UVB on the functioning of the carp immune system was studied on day 2 after the final irradiation. After short-term UVB exposure, the whole-blood respiratory burst and cytotoxic activity were markedly enhanced, with parallel responses in both the number of circulating granulocytes and in the plasma cortisol concentration of the fish. These changes were not detectable after long-term exposure. The respiratory burst by head kidney granulocytes was suppressed dose dependently after both e…

Cytotoxicity Immunologicmedicine.medical_specialtyCarpsHydrocortisoneUltraviolet RaysLymphocyteKidneyBiochemistryCyprinusCommon carpImmune systemInternal medicinemedicineAnimalsCytotoxic T cellLymphocytesPhytohemagglutininsPhysical and Theoretical ChemistryCarpHead Kidneyintegumentary systembiologyChemistryGeneral Medicinebiology.organism_classificationRespiratory burstmedicine.anatomical_structureEndocrinologyImmunoglobulin MImmune SystemImmunologyGranulocytesPhotochemistry and Photobiology
researchProduct

Distinct Photophysics of the Isomers of B18H22 Explained

2012

The photophysics of the two isomers of octadecaborane(22), anti- and syn-B 18H 22, have been studied by UV-vis spectroscopic techniques and theoretical computational methods. In air-saturated hexane, anti-B 18H 22 shows fluorescence with a high quantum yield, Φ F = 0.97, and singlet oxygen O 2( 1Δ g) production (Φ Δ ∼ 0.008). Conversely, isomer syn-B 18H 22 shows no measurable fluorescence, instead displaying much faster, picosecond nonradiative decay of excited singlet states. Computed potential energy hypersurfaces (PEHs) for both isomers rationalize these data, pointing to a deep S 1 minimum for anti-B 18H 22 and a conical intersection (CI) between its S 0 and S 1 states that lies 0.51 e…

CzechPhotochemistryChemistryFoundation (engineering)Library science02 engineering and technology010402 general chemistry021001 nanoscience & nanotechnology01 natural sciencesFluorescencelanguage.human_language0104 chemical sciencesInorganic ChemistryIsomerismlanguagemedia_common.cataloged_instanceSpectrophotometry UltravioletChristian ministryPhysical and Theoretical ChemistryEuropean unionBoranes0210 nano-technologymedia_commonInorganic Chemistry
researchProduct

Photometric and Spectroscopic Properties of Type Ia Supernova 2018oh with Early Excess Emission from the $Kepler$ 2 Observations

2019

Supernova (SN) 2018oh (ASASSN-18bt) is the first spectroscopically-confirmed type Ia supernova (SN Ia) observed in the $Kepler$ field. The $Kepler$ data revealed an excess emission in its early light curve, allowing to place interesting constraints on its progenitor system (Dimitriadis et al. 2018, Shappee et al. 2018b). Here, we present extensive optical, ultraviolet, and near-infrared photometry, as well as dense sampling of optical spectra, for this object. SN 2018oh is relatively normal in its photometric evolution, with a rise time of 18.3$\pm$0.3 days and $\Delta$m$_{15}(B)=0.96\pm$0.03 mag, but it seems to have bluer $B - V$ colors. We construct the "uvoir" bolometric light curve hav…

DATA RELEASEULTRAVIOLETFACTORY OBSERVATIONSFOS: Physical sciencesAstrophysicsType (model theory)medicine.disease_causeSN 2011FE01 natural sciencesLuminosityPhotometry (optics)individual (SN 2018oh) [supernovae]supernovae: generalCIRCUMSTELLAR MATERIAL0103 physical sciencesmedicineSPECTRAAbsorption (logic)Ejecta010303 astronomy & astrophysicssupernovae: individualQCSolar and Stellar Astrophysics (astro-ph.SR)QBLIGHT CURVESHigh Energy Astrophysical Phenomena (astro-ph.HE)PhysicsHIGH-VELOCITY FEATURES010308 nuclear & particles physicsAstronomy and AstrophysicsLight curveSupernovaAstrophysics - Solar and Stellar Astrophysics13. Climate actionSpace and Planetary ScienceIMPROVED DISTANCESWHITE-DWARF MODELSAstrophysics - High Energy Astrophysical Phenomena[PHYS.ASTR]Physics [physics]/Astrophysics [astro-ph]general [supernovae]Ultraviolet
researchProduct

Molecular and functional analysis of the (6-4) photolyase from the hexactinellid Aphrocallistes vastus.

2003

The hexactinellid sponges (phylum Porifera) represent the phylogenetically oldest metazoans that evolved 570-750 million years ago. At this period exposure to ultraviolet (UV) light exceeded that of today and it may be assumed that this old taxon has developed a specific protection system against UV-caused DNA damage. A cDNA was isolated from the hexactinellid Aphrocallistes vastus which comprises high sequence similarity to genes encoding the protostomian and deuterostomian (6-4) photolyases. Subsequently functional studies were performed. It could be shown that the sponge gene, after transfection into mutated Escherichia coli, causes resistance of the bacteria against UV light. Recombinan…

DNA RepairDNA repairUltraviolet RaysMolecular Sequence DataBiophysicsPyrimidine dimerBiochemistryAnalytical Chemistrychemistry.chemical_compoundComplementary DNAAnimalsAmino Acid SequencePhotolyaseMolecular BiologyGenePhylogenyGeneticsbiologyHexactinellidbiology.organism_classificationRecombinant ProteinsPoriferaSpongechemistryBiochemistryDeoxyribodipyrimidine Photo-LyaseSequence AlignmentDNADNA DamageBiochimica et biophysica acta
researchProduct

Oxidative stress impairs the repair of oxidative DNA base modifications in human skin fibroblasts and melanoma cells.

2008

Irradiation of mammalian cells with solar light is associated with the generation of reactive oxygen species (ROS) and oxidative stress, which is mediated in part by endogenous photosensitizers absorbing in the visible range of the solar spectrum. Accordingly, oxidative DNA base modifications such as 7,8-dihydro-8-oxoguanine (8-oxoG) are the predominant types of DNA damage in cells irradiated at wavelengths >400 nm. We have analysed the repair of oxidative purine modifications in human skin fibroblasts and melanoma cells using an alkaline elution technique, both under normal conditions and after depletion of glutathione. Similar repair rates were observed in fibroblasts and melanoma cells f…

DNA RepairLightDNA damageUltraviolet RaysPyrimidine dimerOxidative phosphorylationBiologymedicine.disease_causeBiochemistrychemistry.chemical_compoundmedicineHumansMolecular BiologyMelanomaAgedSkinchemistry.chemical_classificationReactive oxygen speciesGuanosineCell BiologyBase excision repairGlutathioneMolecular biologyGlutathioneOxidative StresschemistryBiochemistryFemaleOxidative stressNucleotide excision repairDNA repair
researchProduct

Cell proliferation and DNA breaks are involved in ultraviolet light-induced apoptosis in nucleotide excision repair-deficient Chinese hamster cells.

2002

UV light targets both membrane receptors and nuclear DNA, thus evoking signals triggering apoptosis. Although receptor-mediated apoptosis has been extensively investigated, the role of DNA damage in apoptosis is less clear. To analyze the importance of DNA damage induced by UV-C light in apoptosis, we compared nucleotide excision repair (NER)-deficient Chinese hamster ovary cells (lines 27-1 and 43-3B mutated for the repair genes ERCC3 and ERCC1, respectively) with the corresponding DNA repair-proficient fibroblasts (CHO-9 and ERCC1 complemented 43-3B cells). NER-deficient cells were hypersensitive as to the induction of apoptosis, indicating that apoptosis induced by UV-C light is due to u…

DNA RepairTranscription GeneticDNA repairDNA damageCell SurvivalUltraviolet RaysApoptosisCHO CellsBiologyCysteine Proteinase InhibitorsRadiation ToleranceArticleMiceCricetinaeUltraviolet lightAnimalsMolecular BiologyChromosome AberrationsIntrinsic apoptosisCell CycleDNA replicationCell BiologyFibroblastsMolecular biologyCaspase InhibitorsChromatinCell biologyKineticsUVB-induced apoptosisProto-Oncogene Proteins c-bcl-2ApoptosisMutationTumor Suppressor Protein p53Cell DivisionNucleotide excision repairDNA DamageMolecular biology of the cell
researchProduct

UVA irradiation induces relocalisation of the DNA repair protein hOGG1 to nuclear speckles

2006

The DNA glycosylase hOGG1 initiates base excision repair (BER) of oxidised purines in cellular DNA. Using confocal microscopy and biochemical cell fractionation experiments we show that, upon UVA irradiation of human cells, hOGG1 is recruited from a soluble nucleoplasmic localisation to the nuclear matrix. More specifically, after irradiation, hOGG1 forms foci colocalising with the nuclear speckles, organelles that are interspersed between chromatin domains and that have been associated with transcription and RNA-splicing processes. The use of mutant forms of hOGG1 unable to bind the substrate showed that relocalisation of hOGG1 does not depend on the recognition of the DNA lesion by the en…

DNA RepairTranscription GeneticUltraviolet RaysDNA repairRecombinant Fusion ProteinsGreen Fluorescent ProteinsFluorescent Antibody TechniqueBiologyDNA GlycosylasesSubstrate Specificitychemistry.chemical_compoundDNA Repair ProteinDNA-(Apurinic or Apyrimidinic Site) LyaseHumansCell NucleusGuanosineBiological TransportCell BiologyBase excision repairNuclear matrixMolecular biologyChromatinCell biologychemistryDNA glycosylaseCell fractionationReactive Oxygen SpeciesDNAHeLa CellsJournal of Cell Science
researchProduct

Influences of histone deacetylase inhibitors and resveratrol on DNA repair and chromatin compaction

2013

Accessibility of DNA is a prerequisite for both DNA damage and repair. Therefore, the chromatin structure is expected to have major impact on both processes, with opposite consequences for the stability of the genome. To analyse the influence of chromatin compaction on the generation and repair of various types of DNA modifications, we modulated the global chromatin structure of AS52 Chinese hamster ovary cells and HeLa cells by treatment with either histone deacetylase inhibitors or resveratrol and measured the repair kinetics of (i) pyrimidine dimers induced by ultraviolet B, (ii) oxidised purines generated by photosensitisation and (iii) single-strand breaks induced by H2O2, using an alk…

DNA RepairUltraviolet RaysDNA damageDNA repairHealth Toxicology and MutagenesisCarbazolesCHO CellsHydroxamic AcidsToxicologyChromatin remodelingCricetulusStilbenesHistone H2AGeneticsmedicineAnimalsDeoxyribonuclease IHumansDNA Breaks Single-StrandedGenetics (clinical)EpigenomicsbiologyChemistryMolecular biologyChromatinCell biologyProliferating cell nuclear antigenChromatinHistone Deacetylase InhibitorsButyratesTrichostatin APyrimidine DimersResveratrolbiology.proteinHeLa Cellsmedicine.drugMutagenesis
researchProduct

Alterations of DNA Repair in Melanoma Cell Lines Resistant to Cisplatin, Fotemustine, or Etoposide

2000

Resistance to chemotherapy is a common phenomenon in malignant melanoma. In order to assess the role of altered DNA repair in chemoresistant melanoma, we investigated different DNA repair pathways in one parental human melanoma line (MeWo) and in sublines of MeWo selected in vitro for drug resistance against four commonly used drugs (cisplatin, fotemustine, etoposide, and vindesine). Host cell reactivation assays with the plasmid pRSVcat were used to assess processing of different DNA lesions. With ultraviolet-irradiated plasmids, no significant differences were found, indicating a normal (nucleotide excision) repair of DNA photoproducts. With singlet oxygen-treated plasmid, the fotemustine…

DNA RepairUltraviolet RaysDNA repairDNA damageDrug ResistanceAntineoplastic AgentsDermatologyBiologyHost-Cell Reactivationbase excision repairBiochemistryNitrosourea Compounds03 medical and health sciencesOrganophosphorus Compounds0302 clinical medicineTumor Cells CulturedmedicineHumansMelanomaMolecular BiologyEtoposide030304 developmental biology0303 health scienceschemoresistanceMicrosatellite instabilityDNA NeoplasmBase excision repairCell BiologyDNA repair protein XRCC4nucleotide excision repairmedicine.diseaseAntineoplastic Agents PhytogenicMolecular biology3. Good healthOxygenmismatch repair030220 oncology & carcinogenesisDNA mismatch repairCisplatinMicrosatellite RepeatsNucleotide excision repairJournal of Investigative Dermatology
researchProduct