Search results for "viruses"

showing 10 items of 1182 documents

Classification of prokaryotic genetic replicators: between selfishness and altruism

2015

Prokaryotes harbor a variety of genetic replicators, including plasmids, viruses, and chromosomes, each having different effects on the phenotype of the hosting cell. Here, we propose a classification for replicators of bacteria and archaea on the basis of their horizontal-transfer potential and the type of relationships (mutualistic, symbiotic, commensal, or parasitic) that they have with the host cell vehicle. Horizontal movement of replicators can be either active or passive, reflecting whether or not the replicator encodes the means to mediate its own transfer from one cell to another. Some replicators also have an infectious extracellular state, thus separating viruses from other mobil…

GeneticsbiologyGeneral Neurosciencemedia_common.quotation_subjectArchaeal Virusesbiology.organism_classificationPhenotypeGeneral Biochemistry Genetics and Molecular BiologyPlasmidHistory and Philosophy of ScienceEvolutionary biologyDNA Transposable ElementsSelfishnessMobile genetic elementsmedia_commonArchaeaAnnals of the New York Academy of Sciences
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Evolutionary plasticity of SH3 domain binding by Nef proteins of the HIV-1/SIVcpz lentiviral lineage

2021

ABSTRACTThe accessory protein Nef of human and simian immunodeficiency viruses (HIV and SIV) is an important pathogenicity factor known to interact with cellular protein kinases and other signaling proteins. A canonical SH3 domain binding motif in Nef is required for most of these interactions. For example, HIV-1 Nef activates the tyrosine kinase Hck by tightly binding to its SH3 domain. An archetypal contact between a negatively charged SH3 residue and a highly conserved arginine in Nef (Arg77) plays a key role here. Combining structural analyses with functional assays, we here show that Nef proteins have also developed a distinct structural strategy - termed the “R-clamp” - that favors th…

Geneticschemistry.chemical_classification0303 health sciencesLineage (genetic)Kinaseviruses030302 biochemistry & molecular biologyHuman immunodeficiency virus (HIV)virus diseasesBiologymedicine.disease_causeSH3 domainAmino acid03 medical and health scienceschemistrymedicineSalt bridgeBinding siteTyrosine kinase030304 developmental biology
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Gene encoding capsid protein VP1 of foot-and-mouth disease virus A quasispecies model of molecular evolution

1988

A phylogenetic tree relating the VP1 gene of 15 isolates of foot-and-mouth disease virus (FMDV) of serotypes A, C, and O has been constructed. The most parsimonious tree shows that FMDV subtypes and isolates within subtypes constitute sets of related, nonidentical genomes, in agreement with a quasispecies mode of evolution of this virus. The average number of nucleotide replacements per site for all possible pairs of VP1 coding segments is higher among representatives of serotype A than serotype C or O. In comparing amino acid sequences, the values of dispersion index (variance/mean value) are greater than 1, with the highest values scored when all sequences are considered. This indicates a…

Geneticseducation.field_of_studyMultidisciplinaryPhylogenetic treebiologyNucleotidesvirusesPopulationQuasispecies modelViral quasispeciesbiology.organism_classificationViral ProteinsAphthovirusCapsidPhylogeneticsMolecular evolutionMutationAmino AcidsFoot-and-mouth disease viruseducationGenePhylogenyResearch Article
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Viral mutation and substitution: units and levels.

2011

Viruses evolve within a hierarchy of organisational levels, from cells to host species. We discuss how these nested population structures complicate the meaning and interpretation of two apparently simple evolutionary concepts: mutation rate and substitution rate. We discuss the units in which these fundamental processes should be measured, and explore why, even for the same virus, mutation and substitution can occur at very different tempos at different biological levels. In addition, we explore the ability of whole genome evolutionary analyses to distinguish between natural selection and other population genetic processes. A better understanding of the complexities underlying the molecula…

Geneticseducation.field_of_studyMutation rateNatural selectionPopulationSubstitution (logic)BiologyGenomeEvolution MolecularAmino Acid SubstitutionMolecular evolutionEvolutionary biologyVirologyViral evolutionMutation (genetic algorithm)MutationVirusesSelection GeneticeducationCurrent opinion in virology
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Transgenic Expression of a Toxin-Coding Killer Virus of the Yeast Zygosaccharomyces bailii in Saccharomyces cerevisiae: Genetic Evidence for a Possib…

1996

The killer toxin-secreting yeast Zygosaccharomyces bailii 412 contains two cytoplasmically inherited double-stranded RNA (dsRNA) viruses (ZbV-L, ZbV-M) responsible for the expression of a killer phenotype in its infected host. While ZbV-L functions as a classical helpervirus by providing capsid (cap) and RNA polymerase functions (cap/pol) necessary for packaging and replication of both viruses, M-dsRNA-containing killer viruses (ZbV-M) are satellites of ZbV-L that contain the genetic information for toxin production only. Both viruses were shown to be sufficient to confer the Z. bailii killer phenotype upon transfected spheroplasts of a S. cerevisiae non-killer strain, resulting in toxin-se…

GeneticsvirusesZygosaccharomyces bailiiMutantSaccharomyces cerevisiaeBiologybiology.organism_classificationPhenotypechemistry.chemical_compoundchemistryRNA polymeraseMycovirusHeterologous expressionGene
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Parvovirus B19 Genotype Specific Amino Acid Substitution in NS1 Reduces the Protein's Cytotoxicity in Culture

2010

A clinical association between idiopathic liver disease and parvovirus B19 infection has been observed. Fulminant liver failure, not associated with other liver-tropic viruses, has been attributed to B19 in numerous reports, suggesting a possible role for B19 components in the extensive hepatocyte cytotoxicity observed in this condition. A recent report by Abe and colleagues (Int J Med Sci. 2007;4:105-9) demonstrated a link between persistent parvovirus B19 genotype I and III infection and fulminant liver failure. The genetic analysis of isolates obtained from these patients demonstrated a conservation of key amino acids in the nonstructural protein 1 (NS1) of the disease-associated genotyp…

GenotypevirusesCytotoxicityApoptosisViral Nonstructural ProteinsProtein SParvoviruschemistry.chemical_compoundLiver diseaseStructure-Activity Relationshiphemic and lymphatic diseasesGenotypemedicineParvovirus B19 HumanHumansCytotoxicitychemistry.chemical_classificationMethioninebiologyParvovirusB19Fulminant Liver Failurevirus diseasesGeneral MedicineHep G2 Cellsmedicine.diseasebiology.organism_classificationFlow CytometryVirologyAmino acidmedicine.anatomical_structurechemistryAmino Acid SubstitutionHepatocytebiology.proteinResearch PaperInternational Journal of Medical Sciences
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The sf32 unique gene of Spodoptera frugiperda multiple nucleopolyhedrovirus (SfMNPV) is a non-essential gene that could be involved in nucleocapsid o…

2013

A recombinant virus lacking the sf32 gene (Sf32null), unique to the Spodoptera frugiperda multiple nucleopolyhedrovirus (SfMNPV), was generated by homologous recombination from a bacmid comprising the complete viral genome (Sfbac). Transcriptional analysis revealed that sf32 is an early gene. Occlusion bodies (OBs) of Sf32null contained 62% more genomic DNA than viruses containing the sf32 gene, Sfbac and Sf32null-repair, although Sf32null DNA was three-fold less infective when injected in vivo. Sf32null OBs were 18% larger in diameter and contained 17% more nucleocapsids within ODVs than those of Sfbac. No significant differences were detected in OB pathogenicity (50% lethal concentration)…

GenotypevirusesScienceGenome ViralSpodopteraSpodopteraVirus ReplicationOcclusion-derived virionsRecombinant virusHomology (biology)VirusViral Proteins03 medical and health sciencesAnimalsNucleocapsidSpodoptera frugiperda multiple nucleopolyhedrovirus (SfMNPV)Gene030304 developmental biology0303 health sciencesGenes Essential[SDV.BA.MVSA]Life Sciences [q-bio]/Animal biology/Veterinary medicine and animal HealthMultidisciplinaryNucleocapsid organizationbiology030306 microbiologyfungiQVirionRbiology.organism_classificationVirologyNucleopolyhedroviruses3. Good healthViral replicationEssential geneLarvaDNA Viral[SDV.MP.VIR]Life Sciences [q-bio]/Microbiology and Parasitology/VirologyMedicinesf32Homologous recombinationResearch ArticlePLoS ONE
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Social Interactions Among Bacteriophages

2020

Although viruses lack many of the social adaptations shown by more complex organisms, different types of social interactions have been unraveled in viruses. Phage research has contributed significantly to the development of this field, called sociovirology, with the discovery of processes such as intracellular and extracellular public good production, prudent host exploitation, cheating, and inter-phage communication. We here review and discuss these processes from a social evolution approach. Similar to other organisms, the origin and maintenance of phage-phage interactions can be explained using kin selection, group selection and game theory approaches. Key determinants of phage social ev…

Group selectionEvolutionary biologyvirusesField (Bourdieu)CheatingKin selectionPublic goodSocial evolutionBiologyGame theorySelection (genetic algorithm)
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Killing of p53-deficient hepatoma cells by parvovirus H-1 and chemotherapeutics requires promyelocytic leukemia protein

2008

To evaluate the synergistic targeting and killing of human hepatocellular carcinoma (HCC) cells lacking p53 by the oncolytic autonomous parvovirus (PV) H-1 and chemotherapeutic agents and its dependence on functional promyelocytic leukemia protein (PML).The role of p53 and PML in regulating cytotoxicity and gene transfer mediated by wild-type (wt) PV H-1 were explored in two pairs of isogenic human hepatoma cell lines with different p53 status. Furthermore, H-1 PV infection was combined with cytostatic drug treatment.While the HCC cells with different p53 status studied were all susceptible to H-1 PV-induced apoptosis, the cytotoxicity of H-1 PV was more pronounced in p53-negative than in p…

H-1 parvovirusLiver CancerH-1 parvovirusCarcinoma HepatocellularParvovirus H-1virusesAntineoplastic AgentsApoptosisPromyelocytic Leukemia ProteinPromyelocytic leukemia proteinDrug TherapyCell Line TumorHumansNuclear proteinCytotoxicityMembrane Potential MitochondrialbiologyParvovirusTumor Suppressor ProteinsLiver NeoplasmsGastroenterologyvirus diseasesNuclear ProteinsGeneral Medicinebiology.organism_classificationCombined Modality Therapydigestive system diseasesOncolytic virusApoptosisCancer researchbiology.proteinFluorouracilCisplatinTumor Suppressor Protein p53Transcription FactorsWorld Journal of Gastroenterology
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Translation of hepatitis B virus (HBV) surface proteins from the HBV pregenome and precore RNAs in Semliki Forest virus-driven expression.

2004

Hepatitis B virus (HBV) pregenome RNA (pgRNA) serves as a translation template for the HBV core (HBc) protein and viral polymerase (Pol). HBV precore RNA (pcRNA) directs the synthesis of the precore (preC) protein, a precursor of the hepatitis B e antigen (HBeAg). pgRNA and pcRNA were expressed in the Semliki Forest virus (SFV) expression system. Besides the HBc and preC proteins, there was revealed the synthesis of all three forms of HBV surface (HBs) proteins: long (LHBs), middle (MHBs) and short (SHBs), the start codons of which are located more than 1000 nt downstream of the HBc and preC start codons. Moreover, other HBV templates, such as 3′-truncated pgRNA lacking 3′ direct repeat and…

HBV RNA encapsidation signal epsilonHepatitis B virusvirusesGene ExpressionLeaky scanningDNA-Directed DNA Polymerasemedicine.disease_causeSemliki Forest virusVirus ReplicationCell LineViral Envelope ProteinsVirologymedicineAnimalsHepatitis B e AntigensRNA MessengerCloning MolecularProtein PrecursorsHepatitis B virusHepatitis B Surface Antigensbiologyvirus diseasesRNA virusTemplates Geneticbiology.organism_classificationVirologyMolecular biologyHepatitis B Core AntigensImmunohistochemistrySemliki forest virusdigestive system diseasesGenetic translationHBeAgHepadnaviridaeProtein BiosynthesisRNA ViralThe Journal of general virology
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