Search results for "von Willebrand factor"

showing 10 items of 81 documents

Cancers in Patients with von Willebrand Disease: A Survey from the Italian Association of Haemophilia Centres

2015

Besides its essential role in hemostasis, there is growing evidence that von Willebrand factor (VWF) has an additional antitumor effect. To elucidate the clinical significance of this biological activity we conducted a retrospective study on cancers among Italian patients with von Willebrand disease (VWD) on behalf of the Italian Association of Haemophilia Centres (AICE). A questionnaire to collect demographic, clinical, and treatment data of VWD patients with cancer was sent to all the 54 Italian Haemophilia Treatment Centres (HTCs) members of AICE. Overall, 18 HTCs (33%) provided information on 92 VWD patients (61 alive and 31 deceased) with 106 cancers collected during the period 1981 to…

Adultcongenital hereditary and neonatal diseases and abnormalitiesPediatricsmedicine.medical_specialtySettore MED/09 - Medicina InternaPopulationbleeding; cancer; mortality; von Willebrand disease; Hematology; Cardiology and Cardiovascular MedicineHaemophiliaHospitals Specialbleeding; cancer; mortality; von Willebrand disease; Adult; Aged; Aged 80 and over; Humans; Italy; Middle Aged; Prospective Studies; Hospitals Special; Neoplasms; Surveys and Questionnaires; von Willebrand Diseases; Hematology; Cardiology and Cardiovascular MedicineVon Willebrand factorNeoplasmsSurveys and Questionnaireshemic and lymphatic diseases80 and overmedicineVon Willebrand diseaseHumanscancerProspective StudiesDesmopressinProspective cohort studyeducationAgedAged 80 and overeducation.field_of_studySpecialbiologybusiness.industryRetrospective cohort studyHematologyMiddle Agedmedicine.diseasebleedingmortalityHospitalsbleeding; cancer; mortality; von Willebrand disease; Adult; Aged; Aged 80 and over; Humans; Italy; Middle Aged; Prospective Studies; Hospitals Special; Neoplasms; Surveys and Questionnaires; von Willebrand Diseasesvon Willebrand DiseasesItalyHemostasisbiology.proteinbusinessvon Willebrand diseaseCardiology and Cardiovascular Medicinemedicine.drug
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Clinical benefits of a Bayesian model for plasma-derived factor VIII/VWF after one year of pharmacokinetic-guided prophylaxis in severe/moderate hemo…

2021

Abstract Introduction Individual pharmacokinetic (PK) profiling in hemophilia A (HA) helps to individualize prophylaxis using population PK models (popPK). A specific popPK model for plasma-derived factor VIII containing von-Willebrand Factor (pdFVIII/VWF) was developed. Aim To compare standard versus PK-driven prophylaxis, using a generic or a specific popPK model for pdFVIII/VWF. Materials and methods A prospective study conducted in HA patients in prophylaxis with pdFVIII/VWF (Fanhdi®) comparing three one-year study periods: (1) standard prophylaxis, (2) PK-guided prophylaxis using a generic pdFVIII popPK model which described FVIII activity irrespective of FVIII concentrate, and (3) PK-…

Adultmedicine.medical_specialtyPopulationHemophilia ABayesian methodPharmacokineticsInternal medicinehemic and lymphatic diseasesvon Willebrand FactorHemarthrosisMedicineHumansPharmacokineticsProspective StudieseducationProspective cohort studyeducation.field_of_studyFactor VIIIbusiness.industryPlasma derivedProphylaxisBayes TheoremHematologyHemarthrosismedicine.diseaseSevere moderateCohortbusinessFactor VIII vWF
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Immune Tolerance Induction in Hemophilia A: A Review

2003

In this article, a comparative analysis of the data stemming from the studies conducted in the field of immune tolerance treatment (ITT) of hemophilia A was attempted. Comparisons are difficult because previous studies differ in many respects, including the dosage of factor (F) VIII, the number of FVIII administrations per day, the association with immunosuppressive drugs (prednisone, cyclophosphamide), and, most importantly, the definition of success in terms of the reacquisition of tolerance. However, a number of variables consistently influenced outcome: the inhibitor titer, either the maximum one or the one assayed before immune tolerance (IT) start and age. As to the FVIII dose, result…

Adultmedicine.medical_specialtyTime FactorsAdolescentDoseCyclophosphamideHemophilia AImmune toleranceVon Willebrand factorPrednisoneInternal medicineImmune ToleranceCoagulopathyHumansMedicineChildFactor VIIIbiologybusiness.industryInfantHematologymedicine.diseaseRecombinant ProteinsKineticsTreatment OutcomeChild PreschoolImmunologyChemoprophylaxisbiology.proteinCardiology and Cardiovascular MedicinebusinessBypassing agentmedicine.drugSeminars in Thrombosis and Hemostasis
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The gut microbiota: An emerging risk factor for cardiovascular and cerebrovascular disease

2018

Commensal gut microbiota have recently been implicated in cardiovascular disease (CVD) and cerebrovascular disease. Atherosclerotic plaque formation depends on the colonization status of the host. In addition to host nutrition and the related microbiota-dependent metabolic changes, activation of innate immune pathways triggers the development of atherosclerosis and supports arterial thrombosis. Gnotobiotic mouse models have uncovered that activation of Toll-like receptor-2 by gut microbial ligands supports von Willebrand factor-integrin mediated platelet deposition to the site of vascular injury. Depending on nutritional factors, the microbiota-derived choline-metabolite trimethylamine N-ox…

Blood Platelets0301 basic medicineIntegrinsEmerging riskImmunologyDiseaseBiologyGut floradigestive systemMethylaminesMice03 medical and health sciencesVon willebrandRisk Factorsvon Willebrand FactormedicineAnimalsHumansImmunology and AllergyPlateletSymbiosisInnate immune systemInfant NewbornInfantThrombosisAtherosclerosisbiology.organism_classificationmedicine.diseaseThrombosisImmunity InnatePlaque AtheroscleroticToll-Like Receptor 2Gastrointestinal MicrobiomeCerebrovascular Disorders030104 developmental biologyCardiovascular DiseasesArterial thrombusImmunologyEuropean Journal of Immunology
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The Gut Microbiota as an Influencing Factor of Arterial Thrombosis

2018

The mutualistic gut microbiota does not only impact the development and function of various immune cell types, but it also influences the function of the hepatic vascular endothelium and prothrombotic platelet function. With germ-free mouse models, we have demonstrated that gut-derived microbial-associated molecular patterns could stimulate hepatic von Willebrand factor (VWF) synthesis and plasmatic VWF levels through Toll-like receptor-2 (TLR2), thus defining the extent of platelet deposition to the subendothelial matrix of the ligation-injured common carotid artery. In addition to the microbiota-derived choline metabolite trimethylamine N-oxide and the microbiota's regulatory role on the …

Blood Platelets030204 cardiovascular system & hematologyGut floradigestive system03 medical and health sciences0302 clinical medicineImmune systemVon Willebrand factorAnimalsHumansPlateletInnate immune systembiologyEndothelial CellsThrombosisArteriesHematologybiology.organism_classificationGastrointestinal MicrobiomeTLR2CoagulationImmunologybiology.proteinSignal transductionSignal Transduction030215 immunologyHämostaseologie
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Targeted SERPIN (TaSER): A dual‐action antithrombotic agent that targets platelets for SERPIN delivery

2021

BACKGROUND Occlusive thrombi are not homogeneous in composition. The core of a thrombus is rich in activated platelets and fibrin while the outer shell contains resting platelets. This core is inaccessible to plasma proteins. We produced a fusion protein (targeted SERPIN-TaSER), consisting of a function-blocking VH H against glycoprotein Ibα (GPIbα) and a thrombin-inhibiting serine protease inhibitor (SERPIN; α1-antitrypsin 355 AIAR358 ) to interfere with platelet-driven thrombin formation. AIM To evaluate the antithrombotic properties of TaSER. METHODS Besides TaSER, we generated three analogous control variants with either a wild-type antitrypsin subunit, a non-targeting control VH H, or …

Blood PlateletsbiologyChemistryHematologySerpinFibrinCell biologyTissue factorPlatelet AdhesivenessThrombinFibrinolytic AgentsVon Willebrand factorvon Willebrand FactorAntithromboticmedicinebiology.proteinHumansPlateletPlatelet activationSerpinscirculatory and respiratory physiologymedicine.drugJournal of Thrombosis and Haemostasis
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Tumorigenic conversion of endothelial cells.

2003

Tumors of endothelial origin develop rarely. Until now, only two angiosarcoma (AS)-derived endothelial cell lines have been be isolated, ISO-HAS and AS-M. Both AS-derived endothelial cell lines presented the typical endothelial characteristics, such as the expression of CD31 and von Willebrand factor, but differed from normal endothelial cells in a nuclear expression of p53, in a delayed angiogenic reaction, and a reduced expression of caveolin. In addition, differences in the expression of cytokines and cell adhesion molecules responsive to proinflammatory stimuli were observed. While AS-M showed an expression pattern similar to that of human umbilical vein endothelial cells (HUVEC), ISO-H…

CD31AdultLipopolysaccharidesTelomerasePathologymedicine.medical_specialtyClinical BiochemistryCaveolin 1Vascular Cell Adhesion Molecule-1BiologyCaveolinsPathology and Forensic Medicinevon Willebrand FactormedicineCell AdhesionHumansMolecular BiologyTelomeraseCells CulturedCell NucleusCell adhesion moleculeReverse Transcriptase Polymerase Chain ReactionGranulocyte-Macrophage Colony-Stimulating FactorTelomereIntercellular Adhesion Molecule-1Cell biologyVascular endothelial growth factor BEndothelial stem cellDNA-Binding ProteinsPlatelet Endothelial Cell Adhesion Molecule-1Vascular endothelial growth factor ACell Transformation NeoplasticVascular endothelial growth factor CCell cultureEndothelium VascularTumor Suppressor Protein p53Experimental and molecular pathology
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Primary angiosarcoma of the alveolar mucosa in a haemodialysis patient: case report and discussion

1994

A case of a haemodialysis patient with a primitive angiosarcoma of the alveolar mucosa is reported. The vascular origin of the tumor was confirmed by the immunohistochemical data which showed strong positivity for Factor VIII-related antigen and for vimentin, whereas stains for desmin and cytokeratins were negative.

Cancer ResearchPathologymedicine.medical_specialtyHemangiosarcomaVimentinMandiblemacromolecular substancesPathology and Forensic MedicineImmunoenzyme TechniquesAntigenRenal Dialysisvon Willebrand FactorHumansVimentinMedicineAngiosarcomaDental alveolusAlveolar mucosaUremiaGingival Neoplasmsbiologybusiness.industryMouth MucosaMiddle AgedPrimary AngiosarcomaOtorhinolaryngologybiology.proteinPeriodonticsImmunohistochemistryFemaleDesminOral SurgerybusinessJournal of Oral Pathology and Medicine
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Procoagulant activity during viral infections.

2017

The abundance of evidence suggest that inflammation of immune and non-immune cells may lead to an imbalance of the pro- and anti-coagulant state during viral infections. During systemic infections, the endothelium plays a critical role in regulating hemostasis, and severe imbalances of endothelial function and activation can contribute to organ failure. Viral infections may elevate plasma levels of procoagulant markers such as TAT and D-dimer TF-positive MPs as well as von Willebrand factor (vWF). Although multiple clinical studies are showing the association of viral infection and increased prothrombotic risk, the pathological mechanisms have not been fully identified for most viral infect…

Cell typeEndotheliumInflammation030204 cardiovascular system & hematologyThromboplastin03 medical and health sciences0302 clinical medicineImmune systemVon Willebrand factorvon Willebrand FactormedicineAnimalsHumansReceptorBlood CoagulationHemostasisbiologybusiness.industryToll-Like ReceptorsBlood Coagulation Factorsmedicine.anatomical_structureCoagulationVirus DiseasesHemostasisImmunologybiology.proteinmedicine.symptombusiness030217 neurology & neurosurgeryBiomarkersFrontiers in bioscience (Landmark edition)
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Evaluation of Clotting Factor Concentrates for Treatment of Thrombotic Thrombocytopenic Purpura.

2010

Abstract Abstract 3678 Introduction: Thrombotic thrombocytopenic purpura (TTP) is characterized by microthrombi, hemolytic anemia as well as thrombocytopenia. These symptoms are caused by a decreased activity of the protease ADAMTS13 which cleaves the von Willebrand Factor (VWF), due to mutation of the ADAMTS13-gene or autoantibodies. At the moment, the only available immediate therapy is plasmapheresis with Fresh Frozen Plasma (FFP) which may induce side effects. Therefore an alternative therapy might be the treatment with clotting factor concentrates. Methods: 40 plasma samples were tested, consisting of FFP and solvent/detergent treated plasma, four batches of each blood group; VWF/VIII …

Clotting factorHemolytic anemiamedicine.medical_specialtybiologyChemistrymedicine.medical_treatmentImmunologyAutoantibodyThrombotic thrombocytopenic purpuraCell BiologyHematologymedicine.diseaseBiochemistryADAMTS13EndocrinologyVon Willebrand factorhemic and lymphatic diseasesInternal medicineImmunologymedicinebiology.proteinPlasmapheresisFresh frozen plasmaBlood
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