0000000000117641

AUTHOR

Yadong Wang

showing 6 related works from this author

Fatal Tumour Lysis Syndrome Induced by Brigatinib in a Lung Adenocarcinoma Patient Treated With Sequential ALK Inhibitors: A Case Report

2021

Tumour lysis syndrome (TLS) represents a group of fatal metabolic derangements resulting from the rapid breakdown of tumour cells. TLS typically occurs soon after the administration of chemotherapy in haematologic malignancies but is rarely observed in solid tumours. Here, we report a case of brigatinib-induced TLS after treatment with sequential anaplastic lymphoma kinase (ALK) inhibitors in a patient with advanced ALK-rearranged lung adenocarcinoma. The patient was treated sequentially with crizotinib, alectinib, and ensartinib. High-throughput molecular profiling after disease progression indicated that brigatinib may overcome ALK resistance mutations, so the patient was administered bri…

Pharmacologybrigatinibacute kidney injurycase reportPharmacology (medical)Therapeutics. PharmacologyRM1-950targeted therapynon-small cell lung cancertumour lysis syndromeFrontiers in Pharmacology
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Existence and dynamics of pairs of temporal cavity solitons weakly-bound through kelly sidebands in a passive optical fiber resonator

2015

We report experimental observations of weaklybound states of temporal cavity solitons, with separations of 10 soliton widths and beyond, in a 100-m single mode fiber cavity. Bound state dynamics has been studied using the real time dispersive Fourier transform technique for roundtrip-byroundtrip measurements. Temporal oscillations in the cavity soliton background, associated with the presence of Kelly sidebands, have been identified as the binding mechanism.

PhysicsOptical fiberSingle-mode optical fiberPhysics::Opticslaw.inventionAmplitude modulationsymbols.namesakeResonatorFourier transformlawQuantum mechanicsBound stateDispersion (optics)symbolsPhysics::Accelerator PhysicsSolitonAtomic physicsNonlinear Sciences::Pattern Formation and Solitons2015 Spatiotemporal Complexity in Nonlinear Optics (SCNO)
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Dissipative polarization domain walls as persisting topological defects

2018

We experimentally demonstrate the existence of dissipative polarization domain walls in a normally dispersive Kerr resonator. We excite and trap them with appropriate external signals thus realizing an all-optical buffer for topological data.

PhysicsCondensed matter physicsCross-phase modulationPhysics::OpticsNonlinear opticsOptical polarizationPolarization (waves)01 natural sciencesTopological defect010309 opticsResonator0103 physical sciencesDissipative system010306 general physicsSelf-phase modulationCLEO Pacific Rim Conference
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Buffering optical topological data using passive Kerr resonators

2018

We experimentally demonstrate the existence of dissipative polarization domain walls, in a normally dispersive Kerr resonator. Through deterministic manipulation of the laser driving the resonator, we achieve systematic excitation and locking of the domain walls.

Physicsbusiness.industryCross-phase modulationPhysics::OpticsOptical ring resonatorsNonlinear opticsOptical polarization02 engineering and technology021001 nanoscience & nanotechnologyPolarization (waves)Laser01 natural scienceslaw.invention010309 opticsResonatorOpticslaw0103 physical sciencesDissipative system0210 nano-technologybusinessConference on Lasers and Electro-Optics
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Imperfect symmetry breaking

2018

By considering a nonlinear resonator driven by an elliptically polarized beam, we study experimentally the robustness of spontaneous symmetry breaking to controlled asymmetries. In particular, we reveal that different asymmetries can balance each other.

PhysicslawCross-phase modulationSpontaneous symmetry breakingQuantum electrodynamicsLight beamHigh Energy Physics::ExperimentSymmetry breakingElliptical polarizationPolarization (waves)Bose–Einstein condensateBeam (structure)law.inventionAdvanced Photonics 2018 (BGPP, IPR, NP, NOMA, Sensors, Networks, SPPCom, SOF)
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Potential treatment strategy for the rare osimertinib resistant mutation EGFR L718Q

2020

Epidermal growth factor receptor (EGFR) L718Q is a rare resistant mutation which independently leads to third-generation tyrosine kinase inhibitor (TKI) resistance. Although a few studies have examined its resistance mechanisms, no effective treatment strategy has yet been proposed for patients with this mutation. Here, we report an effective treatment strategy for the rare EGFR L718Q mutation for the first time. A 44-year-old Chinese male patient initially presented with the sensitizing EGFR L858R mutation, and the progression-free survival (PFS) time after initial icotinib treatment was 9 months. When the progression of the disease (PD) and the EGFR T790M mutation were identified, he did …

0301 basic medicinePulmonary and Respiratory MedicineOncologyiMDT Cornermedicine.medical_specialtymedicine.drug_classmedicine.medical_treatmentnon-small cell lung cancer (NSCLC)Tyrosine-kinase inhibitorTargeted therapy03 medical and health sciencesT790M0302 clinical medicineInternal medicinemedicineOsimertinibEpidermal growth factor receptorbiologybusiness.industrymedicine.disease030104 developmental biology030220 oncology & carcinogenesisMutation (genetic algorithm)Icotinibbiology.proteinbusiness
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