0000000000806860

AUTHOR

Luisa Benussi

showing 2 related works from this author

Search forBs0→μ+μ−andB0→μ+μ−Decays with CDF II

2011

A search has been performed for B{sub s}{sup 0} {yields} {mu}{sup +}{mu}{sup -} and B{sup 0} {yields} {mu}{sup +}{mu}{sup -} decays using 7 fb{sup -1} of integrated luminosity collected by the CDF II detector at the Fermilab Tevatron collider. The observed number of B{sup 0} candidates is consistent with background-only expectations and yields an upper limit on the branching fraction of {Beta}(B{sup 0} {yields} {mu}{sup +}{mu}{sup -}) < 6.0 x 10{sup -9} at 95% confidence level. We observe an excess of B{sub s}{sup 0} candidates. The probability that the background processes alone could produce such an excess or larger is 0.27%. The probability that the combination of background and the expe…

Flight directionNuclear and High Energy PhysicsParticle physicsMesonTevatronGeneral Physics and Astronomy01 natural sciences7. Clean energyLuminosityStandard Modellaw.inventionNuclear physicsParticle decaychemistry.chemical_compoundlawTheoryofComputation_ANALYSISOFALGORITHMSANDPROBLEMCOMPLEXITY0103 physical sciencesInvariant massLimit (mathematics)FermilabCollider010306 general physicsPhysicsMuon010308 nuclear & particles physicsBranching fractionSupersymmetryD0 experimentIMesCrystallographychemistryDecay lengthHigh Energy Physics::ExperimentLeptonPhysical Review Letters
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An APOE haplotype associated with decreased ε4 expression increases the risk of late onset Alzheimer's disease.

2011

This paper addresses a tenet of the literature on APOE, i.e., the relationship between the effects of the e4, one of the established genetic risk factor for Alzheimer's disease (AD), and its expression levels as determined by APOE promoter polymorphisms. Five polymorphisms (-491 rs449647, -427 rs769446, -219 rs405509, and e rs429358-rs7412) were studied in 1308 AD patients and 1082 control individuals from the Central-Northern Italy. Major findings of the present study are the following: 1) the variants -219T and e4 increase the risk for late onset AD (LOAD) when they are both present in cis on the same chromosome (in phase); 2) the correlation between the haplotype (-219T/e4) and AD risk p…

Apolipoprotein EMaleLinkage disequilibriumGENETICSApolipoprotein E4Late onsetGenome-wide association studyBiologyPolymorphism Single NucleotideLinkage DisequilibriumAlzheimer DiseaseRisk FactorsmedicineHumansGenetic Predisposition to DiseaseLongitudinal StudiesAlleleGeneticsChi-Square DistributionGeneral NeuroscienceHaplotypeAge FactorsGeneral MedicineSingle Nucleotidemedicine.diseasePOLYMORPHISMPsychiatry and Mental healthClinical PsychologyHaplotypesItalyFemaleApolipoprotein EGeriatrics and GerontologyAlzheimer's diseaseAge of onsetGenome-Wide Association StudyJournal of Alzheimer's disease : JAD
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