0000000001217044

AUTHOR

J. Chapman

showing 3 related works from this author

Contributory presentations/posters

1999

0303 health sciencesbiologyGeneral Medicine010402 general chemistry01 natural sciencesHorseradish peroxidaseGeneral Biochemistry Genetics and Molecular Biology0104 chemical sciences03 medical and health sciencesBiochemistryManganese porphyrinbiology.proteinEnzyme reconstitutionGeneral Agricultural and Biological Sciences030304 developmental biologyJournal of Biosciences
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Direct measurement of the W boson width

2009

We present a direct measurement of the width of the W boson using the shape of the transverse mass distribution of W->enu candidates selected in 1 fb-1 of data collected with the D0 detector at the Fermilab Tevatron collider in ppbar collisions at sqrt{s}=1.96 TeV. We use the same methods and data sample that were used for our recently published W boson mass measurement, except for the modeling of the recoil, which is done with a new method based on a recoil library. Our result, 2.028 +- 0.072 GeV, is in agreement with the predictions of the standard model.

Particle physicsTevatronGeneral Physics and AstronomyFOS: Physical sciences= 1.8 TEVElementary particle01 natural sciencesHigh Energy Physics - ExperimentStandard Modellaw.inventionNuclear physicsCOLLIDERParticle decayHigh Energy Physics - Experiment (hep-ex)Physics and Astronomy (all)RecoilRATIOPBARP COLLISIONSlaw0103 physical sciences[PHYS.HEXP]Physics [physics]/High Energy Physics - Experiment [hep-ex]RADIATIVE-CORRECTIONSFermilabCollider010306 general physicsNuclear ExperimentBosonPhysics010308 nuclear & particles physicsComputer Science::Information Retrieval14.70.Fm 13.38.Be 13.85.QkTransverse mass= 1.8 TEV; PBARP COLLISIONS; RADIATIVE-CORRECTIONS; RATIO; COLLIDER; DECAYHigh Energy Physics::ExperimentCollider Detector at FermilabDECAY
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Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer's disease

2013

Eleven susceptibility loci for late-onset Alzheimer's disease (LOAD) were identified by previous studies; however, a large portion of the genetic risk for this disease remains unexplained. We conducted a large, two-stage meta-analysis of genome-wide association studies (GWAS) in individuals of European ancestry. In stage 1, we used genotyped and imputed data (7,055,881 SNPs) to perform meta-analysis on 4 previously published GWAS data sets consisting of 17,008 Alzheimer's disease cases and 37,154 controls. In stage 2, 11,632 SNPs were genotyped and tested for association in an independent set of 8,572 Alzheimer's disease cases and 11,312 controls. In addition to the APOE locu…

MaleApolipoprotein Eepidemiology [Alzheimer Disease]SORL1Medizingenetics [Alzheimer Disease]Genome-wide association studySingle-nucleotide polymorphismBiologyPolymorphism Single NucleotideArticlePICALMCohort StudiesAlzheimer Diseaseddc:570PSEN2GeneticsmedicineHumansGenetic Predisposition to DiseaseAge of OnsetBiologyAgedGenetic associationAged 80 and overGeneticsMiddle Agedmedicine.diseaseGenetic LociCase-Control StudiesFemaleHuman medicineAlzheimer's diseasestatistics & numerical data [Genome-Wide Association Study]Genome-Wide Association StudyNature Genetics
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