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RESEARCH PRODUCT

Depressive symptoms and career-related goal appraisals: genetic and environmental correlations and interactions.

Eero VuoksimaaTellervo KorhonenSanna ReadRichard J. RoseJaakko KaprioDanielle M. DickKatariina Salmela-aro

subject

AdultMaleEmotionsTwinsmedicine.disease_causeStructural equation modelingArticleDevelopmental psychologyYoung Adultdepressive symptomsSurveys and QuestionnairesmedicineDiseases in TwinsPsychological stressHumansYoung adultGene–environment interactionta515Genetics (clinical)Depression (differential diagnoses)Depressive symptomsFinlandnuoret aikuisetcareer-related goal appraisalsDepressionObstetrics and GynecologyTargeted interventionsTwins Monozygoticgenetic correlationgene-environment interactionPediatrics Perinatology and Child HealthtwinFemaleGene-Environment InteractionPsychologyGoalsStress Psychological

description

Abstract: In order to further understand why depressive symptoms are associated with negative goal appraisals, the present study examined the genetic and environmental correlations and interactions between depressive symptoms and career-related goal appraisals. A total of 1,240 Finnish twins aged 21–26 years completed a questionnaire containing items on the appraisal of their career goals along five dimensions: importance, progress, effort, strain, and self-efficacy. In the same questionnaire, the 10-item General Behavior Inven- tory assessed depressive symptoms. Structural equation modeling was used to evaluate the genetic and environmental correlations and gene–environment interactions between the career-goal appraisals and depressive symptoms. Associations were identified, and were attributed to environmental factors. Of the career-related goal appraisals, the shared environmental component was of a higher magnitude for the dimension of strain among the depressed compared with non-depressed subjects. The results indicate that the interplay between depressive symptoms and negative career-related goal appraisals is significantly affected by environmental factors, and thus possibly susceptible to targeted interventions. peerReviewed

10.1017/thg.2014.33https://pubmed.ncbi.nlm.nih.gov/24932581