Effect of noradrenaline and isoproterenol on lipopolysaccharide-induced tumor necrosis factor-alpha production in whole blood from patients with chronic heart failure and the role of beta-adrenergic receptors

6533b860fe1ef96bd12c390d

RESEARCH PRODUCT

Effect of noradrenaline and isoproterenol on lipopolysaccharide-induced tumor necrosis factor-alpha production in whole blood from patients with chronic heart failure and the role of beta-adrenergic receptors

Stefan D. Anker Stefan D. Anker Sabine Genth-zotz Stephan Von Haehling Aidan P. Bolger Philip A. Poole-wilson Ian M. Adcock Rainer Dietz Paul R. Kalra Michael Kemp

subject

Adult Lipopolysaccharides Male medicine.medical_specialty Lipopolysaccharide Adrenergic receptor Adrenergic beta-Antagonists Norepinephrine chemistry.chemical_compound Internal medicine Receptors Adrenergic beta medicine Bisoprolol Humans Receptor Whole blood Heart Failure Tumor Necrosis Factor-alpha business.industry Isoproterenol Cardiovascular Agents medicine.disease Endocrinology chemistry Bisoprolol Heart failure cardiovascular system Cardiology Female Tumor necrosis factor alpha Cardiology and Cardiovascular Medicine business Ex vivo medicine.drug

description

Increased levels of tumor necrosis factor-alpha (TNF-alpha) correlate with poor prognoses in chronic heart failure (CHF). This study demonstrated that noradrenaline and isoproterenol inhibit TNF-alpha production in patients with CHF in ex vivo whole blood in a dose-dependent fashion. The beta-blocker bisoprolol abolishes this effect.

year	journal	country	edition	language
2005-04-01	The American Journal of Cardiology

https://doi.org/10.1016/j.amjcard.2004.12.022

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