Search results for " ACTIVATION"

showing 10 items of 1535 documents

Tonic T cell signalling and T cell tolerance as opposite effects of self-recognition on dendritic cells.

2010

Naive T cells spend most of their time scanning the surface of dendritic cells (DCs), indicating that self-MHC/T cell receptor (TCR) interactions between these immune cells occur routinely in peripheral organs during the steady state. Peripheral self-MHC recognition on DCs drives seemingly opposing effects in the absence of inflammatory stimuli such as deletion of certain self-reactive T cells as well as maintenance of the T cell responsiveness to antigen, both of which shape the T cell repertoire and regulate T cell responses. Here we review recent data on the role of self-MHC recognition on steady-state DCs in the periphery and propose that interactions between T cells and steady-state DC…

T cellT-LymphocytesImmunologyAntigen presentation610 Medicine & healthchemical and pharmacologic phenomenaBiologyLymphocyte ActivationMajor Histocompatibility ComplexmedicineImmunology and AllergyCytotoxic T cellAnimalsHumansIL-2 receptorAntigen-presenting cell2403 ImmunologyAntigen PresentationZAP70CD28Dendritic CellsNatural killer T cellCell biologymedicine.anatomical_structureSelf Tolerance10032 Clinic for Oncology and Hematology2723 Immunology and AllergySignal TransductionCurrent opinion in immunology
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Cortical neurons selectively inhibit MHC class II induction in astrocytes but not in microglial cells.

1993

Astrocytes have been shown to act as potent accessory cells for MHC class II-restricted T cell responses in vitro after treatment with interferon-gamma. In contrast, even under conditions of severe central nervous system (CNS) inflammation, they seem to express little, if any, class II molecules in vivo. Thus the role of astroglial cells as accessory cells in immune responses in the CNS remains to be determined. We have studied neuron--glia interactions with respect to induction of MHC class II molecules. Surprisingly, in a co-culture system, viable neurons inhibited the induction of class II restriction elements on astrocytes. This effect was only observed when neurons had contact to astro…

T cellT-LymphocytesImmunologyAntigen presentationAntigen-Presenting CellsDown-RegulationLymphocyte ActivationMHC class ImedicineImmunology and AllergyAnimalsCells CulturedCerebral CortexNeuronsMHC class IIbiologyMicrogliaHistocompatibility Antigens Class IIGeneral MedicineCell biologyRatsmedicine.anatomical_structurenervous systemAstrocytesImmunologybiology.proteinNeurogliaNeuronNeurogliaAstrocyteInternational immunology
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Enhanced dendritic cell maturation by TNF-alpha or cytidine-phosphate-guanosine DNA drives T cell activation in vitro and therapeutic anti-tumor immu…

2000

Abstract Dendritic cells (DC) manipulated ex vivo can induce tumor immunity in experimental murine tumor models. To improve DC-based tumor vaccination, we studied whether DC maturation affects the T cell-activating potential in vitro and the induction of tumor immunity in vivo. Maturation of murine bone marrow-derived DC was induced by GM-CSF plus IL-4 alone or by further addition of TNF-α or a cytidine-phosphate-guanosine (CpG)-containing oligonucleotide (ODN-1826), which mimics the immunostimulatory effect of bacterial DNA. Flow cytometric analysis of costimulatory molecules and MHC class II showed that DC maturation was stimulated most by ODN-1826, whereas TNF-α had an intermediate effec…

T cellT-LymphocytesImmunologyAntineoplastic AgentsCell CommunicationBiologyLymphocyte ActivationImmunotherapy AdoptiveMiceImmune systemAdjuvants ImmunologicIn vivomedicineTumor Cells CulturedImmunology and AllergyAnimalsInterleukin 4Cells CulturedMice Inbred BALB CTumor Necrosis Factor-alphaCell DifferentiationDendritic cellDendritic CellsMolecular biologyInterleukin-12Coculture TechniquesGrowth InhibitorsMice Inbred C57BLmedicine.anatomical_structureOligodeoxyribonucleotidesColonic NeoplasmsInterleukin 12Cancer researchTumor necrosis factor alphaCpG IslandsFemaleInterleukin-4Ex vivoNeoplasm TransplantationJournal of immunology (Baltimore, Md. : 1950)
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Suppressive effects of C3b on monocyte-dependent T cell proliferation.

1987

The effect of C3b treatment of human monocytes on secondary antigen-dependent T cell response was studied. When antigen-specific T cell blasts were cultivated together with C3b-treated monocytes the proliferative response was inhibited in a dose-dependent fashion. This suppressive effect was specific for C3b because heat-inactivated C3b or buffer alone had no influence on T cell proliferation. In part, this suppressive effect is mediated through a C3b-induced decreased expression of class II antigens on the surface of treated monocytes, but another suppressive mechanism exists because the C3b pretreatment of monocytes also led to an inhibition of the proliferative response in a class II ant…

T cellT-LymphocytesImmunologyIndomethacinchemical and pharmacologic phenomenaBiologyIn Vitro TechniquesInhibitory postsynaptic potentialT cell responseLymphocyte ActivationMonocytesmedicineImmune ToleranceImmunology and AllergyHumansCells CulturedMonocyteComplement C3Molecular biologyProliferative responsemedicine.anatomical_structureComplement C3dComplement C3bImmunologic MemoryClass II Antigenscirculatory and respiratory physiologyEuropean journal of immunology
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Tumors as elusive targets of T-cell-based active immunotherapy.

2003

The understanding of tumor-host interactions remains elusive despite significant progress in the identification of tumor antigens (TAs) recognized by autologous T cells. In particular, most human tumors do not regress and continue to grow in spite of spontaneous or immunization-induced immune responses demonstrated in circulating lymphocytes. Indeed, systemic immune responses might insufficiently address the complexity of tumor-host interactions because of factors, such as (1) the lack of productive T-cell receptor (TCR) engagement with epitope owing to qualitative and/or quantitative defects in the generation and maintenance of the immune response, (2) insufficient costimulation provided b…

T cellmedicine.medical_treatmentT-LymphocytesImmunologyReceptors Antigen T-CellEpitopes T-Lymphocytechemical and pharmacologic phenomenaActive immunotherapyBiologyLymphocyte ActivationCancer VaccinesEpitopeImmune systemAntigenAntigens NeoplasmNeoplasmsmedicineImmunology and AllergyAnimalsHumansTumor microenvironmentImmunity CellularT-cell receptorImmunotherapy ActiveImmunotherapybiochemical phenomena metabolism and nutritionmedicine.anatomical_structureImmunologybacteriaTrends in immunology
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NFATc1 affects mouse splenic B cell function by controlling the calcineurin–NFAT signaling network

2011

Mouse B cells lacking NFATc1 exhibit defective proliferation, survival, isotype class switching, cytokine production, and T cell help.

T-LymphocytesImmunologyNaive B cellB-cell receptorReceptors Antigen B-CellLymphocyte ActivationArticleMice03 medical and health sciences0302 clinical medicinemedicineAnimalsImmunology and AllergyB cell030304 developmental biologyB-Lymphocytes0303 health sciencesCD40NFATC Transcription Factorsintegumentary systembiologyCalcineurinCD22Germinal centerImmunoglobulin Class SwitchingMolecular biology3. Good healthB-1 cellCalcineurinmedicine.anatomical_structure030220 oncology & carcinogenesisCancer researchbiology.proteinCalciumSpleenSignal TransductionJournal of Experimental Medicine
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Different Efficiency of Heat Shock Proteins (HSP) to Activate Human Monocytes and Dendritic Cells: Superiority of HSP60

2002

Abstract One essential immunoregulatory function of heat shock protein (HSP) is activation of the innate immune system. We investigated the activation of human monocytes and monocyte-derived dendritic cells (DC) by recombinant human HSP60, human inducible HSP72, and preparations of human gp96 and HSP70 under stringent conditions, in the absence of serum and with highly purified monocytes. HSP60 induced human DC maturation and activated human DC to secrete proinflammatory cytokines. HSP72 induced DC maturation to a lesser extent, but activated human monocytes and immature DC as efficiently as HSP60 to release proinflammatory cytokines. The independence of the effects of HSP60 and HSP72 from …

T-Lymphocytesmedicine.medical_treatmentImmunologyHSP72 Heat-Shock ProteinsPeptide bindingBiologyLymphocyte ActivationMonocytesProinflammatory cytokineAntigens NeoplasmHeat shock proteinmedicineHumansImmunology and AllergyHSP70 Heat-Shock ProteinsSecretionHeat-Shock ProteinsInnate immune systemCell DifferentiationChaperonin 60Dendritic CellsMolecular biologyCoculture TechniquesRecombinant ProteinsHsp70CytokineCytokinesHSP60Inflammation MediatorsSignal TransductionThe Journal of Immunology
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Specialized subsets of innate-like T cells and dendritic cells protect from lethal pneumococcal infection in the lung

2021

SummaryInnate-like T cells, including invariant natural killer T (iNKT) cells, mucosal-associated invariant T (MAIT) cells and γδ T cells, are present in various barrier tissues, including the lung. They carry out protective responses during infections, but the mechanisms for protection are not completely understood. Here, we investigated their roles during pulmonary infection with Streptococcus pneumoniae. Following infection, innate-like T cells rapidly increased in lung tissue, in part through recruitment, but TCR activation and cytokine production occurred mostly in IL-17-producing NKT17 and γδ T cells. NKT17 cells were preferentially located outside the vasculature prior to infection, …

TCR ActivationLungChemistrymedicine.medical_treatmentT cellAntigen presentationPulmonary infectionmedicine.disease_causeMolecular biologymedicine.anatomical_structureCytokineStreptococcus pneumoniaemedicineLung tissue
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Infectious Tolerance

2002

Regulatory CD4(+)CD25(+) T cells (Treg) are mandatory for maintaining immunologic self-tolerance. We demonstrate that the cell-cell contact-mediated suppression of conventional CD4(+) T cells by human CD25(+) Treg cells is fixation resistant, independent from membrane-bound TGF-beta but requires activation and protein synthesis of CD25(+) Treg cells. Coactivation of CD25(+) Treg cells with Treg cell-depleted CD4(+) T cells results in anergized CD4(+) T cells that in turn inhibit the activation of conventional, freshly isolated CD4(+) T helper (Th) cells. This infectious suppressive activity, transferred from CD25(+) Treg cells via cell contact, is cell contact-independent and partially medi…

TGF-βCD4-Positive T-Lymphocyteshuman regulatory T cellsT-LymphocytesImmunologyCellchemical and pharmacologic phenomenaIn Vitro TechniquesLymphocyte ActivationT-Lymphocytes RegulatoryImmune toleranceInterleukin 21AntigenTransforming Growth Factor betaCD4+CD25+ T cellsCell AdhesionImmune TolerancemedicineHumansImmunology and AllergyCytotoxic T cellIL-2 receptorbiologyBrief Definitive ReportModels ImmunologicalReceptors Interleukin-2hemic and immune systemsT-Lymphocytes Helper-InducerTransforming growth factor betainfectious tolerancemedicine.anatomical_structureT cell inhibitionImmunologyCancer researchbiology.proteinTransforming growth factorJournal of Experimental Medicine
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Type-II transmembrane prolyl dipeptidases and matrix metalloproteinases in membrane vesicles of active endothelial cells.

2006

Conclusions: Endothelia cells in sparse culture are migratory and increase the production of gelatinases of serine- and metallo-classes in membrane vesicles. Collectively, proteases associated with membrane vesicles degrade extracellular matrix components including type-I and type-IV collagens, laminin and fibronectin. Inhibitor studies suggest the existence of small gelatinases that were derived from these serine- and metallo-proteases. Thus, further studies are warranted to demonstrate the cooperative action of metallo- and serine proteases on cell surfaces and in extracellular vesicles during endothelial cell migration in 3D collagenous matrices, and potential proteolytic activation mech…

TUMOR-CELLSCell MembraneBREAST-CARCINOMA CELLSEndothelial CellsUP-REGULATIONANGIOGENESISMatrix MetalloproteinasesExtracellular MatrixACTIVATIONEnzyme ActivationNEUROPEPTIDE-YCell MovementSEPRASESettore BIO/10 - BiochimicaMETASTASISPEPTIDASE-IVHumansDipeptidyl-Peptidases and Tripeptidyl-PeptidasesINTEGRINCells CulturedAdvances in experimental medicine and biology
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