Search results for " ACTIVATION"

showing 10 items of 1535 documents

Gamma interferon blocks gammaherpesvirus reactivation from latency in a cell type-specific manner

2007

Gammaherpesviruses are important pathogens whose lifelong survival in the host depends critically on their capacity to establish and reactivate from latency, processes regulated by both viral genes and the host immune response. Previous work has demonstrated that gamma interferon (IFN-gamma) is a key regulator of chronic infection with murine gammaherpesvirus 68 (gammaHV68), a virus that establishes latent infection in B lymphocytes, macrophages, and dendritic cells. In mice deficient in IFN-gamma or the IFN-gamma receptor, gammaHV68 gene expression is altered during chronic infection, and peritoneal cells explanted from these mice reactivate more efficiently ex vivo than cells derived from…

1109 Insect Sciencemedicine.medical_treatmentImmunologyCellSpleen610 Medicine & healthBiology10263 Institute of Experimental ImmunologyMicrobiologyInterferon-gammaGammaherpesvirinaeImmune systemVirologyVirus latencymedicineAnimalsHumansInterferon gammaDiphtheria toxinB-Lymphocytes2403 ImmunologyMacrophages2404 MicrobiologyHerpesviridae Infectionsmedicine.diseaseVirus LatencyCell biologyChronic infectionCytokinemedicine.anatomical_structureInsect ScienceImmunology2406 VirologyPathogenesis and Immunity570 Life sciences; biologyVirus Activationmedicine.drug
researchProduct

Endogenous peroxynitrite modulates PGHS-1–dependent thromboxane A2 formation and aggregation in human platelets

2008

Aggregation of activated platelets is considerably mediated by the autocrine action of thromboxane A2 (TxA2) which is formed in a prostaglandin endoperoxide H2 synthase-1 (PGHS-1 or COX-1)-dependent manner. The activity of PGHS-1 can be stimulated by peroxides, an effect termed "peroxide tone", that renders PGHS-1 the key regulatory enzyme in the formation of TxA2. Activated platelets release nitric oxide (*NO) and superoxide (O*2) but their interactions with the prostanoid pathway have been controversially discussed in platelet physiology and pathophysiology. The current study demonstrates that endogenously formed peroxynitrite at nanomolar concentrations, originating from the interaction …

1303 BiochemistryPlatelet AggregationSuperoxideProstanoid610 Medicine & healthBiochemistryNitric oxideCell biologychemistry.chemical_compoundPeroxynitrous acidThromboxane A2Thromboxane A22737 Physiology (medical)BiochemistrychemistryPhysiology (medical)Peroxynitrous Acid10209 Clinic for CardiologyCyclooxygenase 1Humanslipids (amino acids peptides and proteins)PlateletPlatelet activationPeroxynitrite
researchProduct

Effect of high temperature annealing (T > 1650 °C) on the morphological and electrical properties of p-type implanted 4H-SiC layers

2019

This work reports on the effect of high temperature annealing on the electrical properties of p-type implanted 4H-SiC. Ion implantations of Aluminum (Al) at different energies (30-200 keV) were carried out to achieve 300 nm thick acceptor box profiles with a concentration of about 10(20) at/cm(3). The implanted samples were annealed at high temperatures (1675-1825 degrees C). Morphological analyses of the annealed samples revealed only a slight increase of the surface roughness RMS up to 1775 degrees C, while this increase becomes more significant at 1825 degrees C (RMS = 1.2 nm). Room temperature Hall measurements resulted in a hole concentration in the range 0.65-1.34 x 10(18)/cm(3) and m…

4H-SiCMaterials scienceFabricationAnnealing (metallurgy)Analytical chemistrychemistry.chemical_element02 engineering and technologyActivation energy01 natural sciencesIonAluminium0103 physical sciencesSurface roughnessGeneral Materials ScienceElectrical measurements010302 applied physicsCondensed Matter - Materials ScienceMechanical EngineeringPhysics - Applied Physics021001 nanoscience & nanotechnologyCondensed Matter PhysicsAcceptorPost implantation annealingchemistryMechanics of MaterialsElectrical activationp-type implantation0210 nano-technologyMaterials Science in Semiconductor Processing
researchProduct

Statins and the squalene synthase inhibitor zaragozic acid stimulate the non-amyloidogenic pathway of amyloid-beta protein precursor processing by su…

2010

Cholesterol-lowering drugs such as statins influence the proteolytic processing of the amyloid-beta protein precursor (AbetaPP) and are reported to stimulate the activity of alpha-secretase, the major preventive secretase of Alzheimer's disease. Statins can increase the alpha-secretase activity by their cholesterol-lowering properties as well as by impairment of isoprenoids synthesis. In the present study, we elucidate the contribution of these pathways in alpha-secretase activation. We demonstrate that zaragozic acid, a potent inhibitor of squalene synthase which blocks cholesterol synthesis but allows synthesis of isoprenoids, also stimulates alpha-secretase activity. Treatment of human n…

ADAM10Blotting Westernchemistry.chemical_compoundSqualeneADAM10 ProteinAmyloid beta-Protein PrecursorCell Line TumormedicineHumansLovastatinRNA Small InterferingProtein precursorLuciferasesLipid raftNeuronsbiologyATP synthaseChemistryReverse Transcriptase Polymerase Chain ReactionTerpenesGeneral NeuroscienceAnticholesteremic AgentsCell MembraneMembrane ProteinsTricarboxylic AcidsZaragozic acidGeneral MedicineBridged Bicyclo Compounds HeterocyclicEnzyme ActivationPsychiatry and Mental healthClinical PsychologyADAM ProteinsCholesterolFarnesyl-Diphosphate FarnesyltransferaseBiochemistrybiology.proteinlipids (amino acids peptides and proteins)LovastatinGeriatrics and GerontologyAmyloid Precursor Protein SecretasesHydroxymethylglutaryl-CoA Reductase InhibitorsAmyloid precursor protein secretasemedicine.drugJournal of Alzheimer's disease : JAD
researchProduct

Alpha-secretase as a therapeutic target.

2007

In the non-amyloidogenic pathway the alpha-secretase cleaves the amyloid precursor protein (APP) within the sequence of Abeta-peptides and precludes their formation. In addition, alpha-secretase cleavage releases an N-terminal extracellular domain with neurotrophic and neuroprotective properties. The disintegrin metalloproteinase ADAM10 has been shown to act as alpha-secretase in vivo, to prevent amyloid plaque formation and hippocampal defects in an Alzheimer disease mouse model. An increase in alpha-secretase activity therefore is an attractive strategy for treatment of AD and may be achieved by modulating selective signalling pathways. Functional characterization of the human ADAM10 prom…

ADAM10Retinoic acidModels BiologicalReceptors G-Protein-Coupledchemistry.chemical_compoundADAM10 ProteinDownregulation and upregulationAlzheimer DiseaseExtracellularAmyloid precursor proteinAnimalsHumansTranscription factorG protein-coupled receptorbiologyMembrane ProteinsCell biologyEnzyme ActivationADAM ProteinsDisease Models AnimalNeurologychemistryAlpha secretasebiology.proteinNeurology (clinical)Amyloid Precursor Protein SecretasesCurrent Alzheimer research
researchProduct

Determination of phosphorus by instrumental neutron activation and bremsstrahlung measurement in bone samples

1997

A non destructive method based on the31P(n,γ)32P reaction for the assay of phosphorus in bone samples is described. This method involves a thermal neutron irradiation of 2 minutes in a reactor followed by the measurement of the bremsstrahlung produced by the β− of32P in a Ge-detector surrounded by an anti-Compton shield. Accuracy and precision were tested by analysing the certified NIST 1486 Bone Meal reference material and tri-calcium phosphate (Ca3(PO4)2) samples. The value obtained for the reference material was in good agreement with the certified value and with relative standard deviation of 4.1% the precision was acceptable. The value obtained for Ca3(PO4)2 shows a deviation of −6% fr…

Accuracy and precisionMaterials scienceHealth Toxicology and MutagenesisPhosphorusRadiochemistryPublic Health Environmental and Occupational HealthAnalytical chemistryBremsstrahlungchemistry.chemical_elementPollutionNeutron temperatureBone mealAnalytical ChemistryNuclear Energy and EngineeringchemistryNon destructiveRadiology Nuclear Medicine and imagingIrradiationSpectroscopyNeutron activationJournal of Radioanalytical and Nuclear Chemistry
researchProduct

Halogenated (arylsulfanyl)pyridines syntheses and their derivatives by C-S and C-X couplings catalyzed by palladium complexes

2018

C–H activation of aryl compounds directed by 2-sulfonylpyridine and 2-sulfanylpyridine moiety for the formation of C–X (X = F, Cl, Br, I) bonds was studied. First, the syntheses of thioethers (2-(arylsulfanyl)pyridines) and sulfones (2-(arylsulfonyl)pyridines) were necessary for the progress of this project. A C–S coupling catalyzed by palladium complexe between thioaryls and halogenated heterocycles was performed for the synthesis of thioethers. The cheap ligand bis(diphenylphosphino)ferrocene (dppf) was used for this convenient reaction. Then, oxidation of thioethers was performed to synthesize sulfones. Halogenation of these compounds was studied with ortho-directed C–H activation cataly…

Activation C–H[CHIM.OTHE] Chemical Sciences/OtherC–halogen couplingOxidationCouplage C–halogène[CHIM.CATA] Chemical Sciences/CatalysisC–H activationOxydationCouplage C–S[CHIM.CATA]Chemical Sciences/Catalysis[CHIM.OTHE]Chemical Sciences/OtherPalladium
researchProduct

Synthesis of novel organic-based fluorophores – Implementation to the design of fluorogenic enzyme substrates based on the principle of in situ synth…

2020

Detection and fluorescence imaging of biologic systems requires the implementation of efficient, robust and easy-to-use tools. Conventional fluorogenic probes currently used in microbiology lack efficiency since they are based on the single chemical modification of a fluorophore bearing an optically tunable reactive group, which often leads to incomplete fluorescence quenching. The main goal of my Ph.D thesis was to develop novel fluorogenic enzymatic substrates based on the "covalent assembly" principle. This approach also named "in situ synthesis" is based on the use of domino reactions to form a fluorescent moiety starting from a "caged" non-fluorescent molecule. In our case, the bioanal…

Activation enzymatique[CHIM.ORGA]Chemical Sciences/Organic chemistryHétéro-XanthènesSynthèse in situHetero-Xanthene dyesQuinoxalinonesIn situ synthesis[CHIM.ORGA] Chemical Sciences/Organic chemistryEnzymatic activationFluorogenic probesSonde fluorogéniquesFluorescence
researchProduct

CD73-generated extracellular adenosine in chronic lymphocytic leukemia creates local conditions counteracting drug-induced cell death

2011

Abstract Extracellular adenosine (ADO), generated from ATP or ADP through the concerted action of the ectoenzymes CD39 and CD73, elicits autocrine and paracrine effects mediated by type 1 purinergic receptors. We have tested whether the expression of CD39 and CD73 by chronic lymphocytic leukemia (CLL) cells activates an adenosinergic axis affecting growth and survival. By immunohistochemistry, CD39 is widely expressed in CLL lymph nodes, whereas CD73 is restricted to proliferation centers. CD73 expression is highest on Ki-67+ CLL cells, adjacent to T lymphocytes, and is further localized to perivascular areas. CD39+/CD73+ CLL cells generate ADO from ADP in a time- and concentration-dependen…

AdenosineCellular differentiationChronic lymphocytic leukemia5'-Nucleotidase; Adenosine; Adenosine Diphosphate; Adenosine Triphosphate; Antigens CD; Antineoplastic Agents Phytogenic; Apyrase; Autocrine Communication; Cell Death; Cell Differentiation; Cell Movement; Cell Survival; Etoposide; Extracellular Space; GPI-Linked Proteins; Humans; Leukemia Lymphocytic Chronic B-Cell; Paracrine Communication; Receptor Adenosine A2A; Tumor Cells Cultured; Biochemistry; Immunology; Hematology; Cell BiologyMICROENVIRONMENTCD38BiochemistryACTIVATIONAdenosine TriphosphateCell MovementPhytogenichemic and lymphatic diseasesTumor Cells CulturedChronic5'-NucleotidaseEtoposideLeukemiaCulturedCell DeathTUMOR-GROWTHApyrasePurinergic receptorCell DifferentiationHematologyLymphocyticCDTumor CellsCell biologyAdenosine DiphosphateAutocrine CommunicationLeukemiaReceptorIMMUNE SUPPRESSIONReceptor Adenosine A2ACell SurvivalImmunologyAntineoplastic AgentsAdenosinergicBiologyGPI-Linked ProteinsDAMAGE-INDUCED APOPTOSISAdenosine A2AParacrine signallingAntigens CDParacrine CommunicationmedicineHumansAntigensAutocrine signallingImmunobiologyB-CellCell BiologyDAMAGE-INDUCED APOPTOSIS; T-CELLS; IMMUNE SUPPRESSION; ZAP-70 EXPRESSION; TUMOR-GROWTH; RECEPTOR; CD73; ACTIVATION; CD38; MICROENVIRONMENTmedicine.diseaseAntineoplastic Agents PhytogenicLeukemia Lymphocytic Chronic B-CellSettore MED/15 - MALATTIE DEL SANGUET-CELLSCD73Extracellular SpaceZAP-70 EXPRESSIONCD38Blood
researchProduct

The transcription factor NFATc2 controls IL-6–dependent T cell activation in experimental colitis

2008

The nuclear factor of activated T cells (NFAT) family of transcription factors controls calcium signaling in T lymphocytes. In this study, we have identified a crucial regulatory role of the transcription factor NFATc2 in T cell–dependent experimental colitis. Similar to ulcerative colitis in humans, the expression of NFATc2 was up-regulated in oxazolone-induced chronic intestinal inflammation. Furthermore, NFATc2 deficiency suppressed colitis induced by oxazolone administration. This finding was associated with enhanced T cell apoptosis in the lamina propria and strikingly reduced production of IL-6, -13, and -17 by mucosal T lymphocytes. Further studies using knockout mice showed that IL-…

Adjuvants Immunologic; Animals; Humans; Interleukin-13; Interleukin-17; Interleukin-6; Lymphocyte Activation; Mice; Mice Inbred BALB C; Mice Knockout; Mice SCID; Models Biological; NFATC Transcription Factors; Oxazolone; T-LymphocytesT cellT-LymphocytesImmunologyMice SCIDBiologyLymphocyte ActivationInflammatory bowel diseaseModels BiologicalArticleOxazoloneTCIRG1chemistry.chemical_compoundMiceAdjuvants ImmunologicmedicineImmunology and AllergyCytotoxic T cellAnimalsHumansColitisMice KnockoutMice Inbred BALB CInterleukin-13NFATC Transcription FactorsInterleukin-6Interleukin-17OxazoloneArticlesmedicine.diseasemedicine.anatomical_structurechemistryImmunologyInterleukin 13Cancer researchInterleukin 17The Journal of Experimental Medicine
researchProduct