Search results for " ACTIVATION"

showing 10 items of 1535 documents

Herpes virus saimiri-transformed human T lymphocytes: normal functional phenotype and preserved T cell receptor signalling

1993

Herpes virus saimiri (HVS), a primate herpes virus, transforms human CD4+ and CD8+ T lymphocytes to continuous growth in vitro. We have previously shown that HVS-transformed human T cells (HVS-T cells) respond to stimulation via CD2 with autocrine growth. In the present study we have investigated the functional characteristics of HVS-T cells. We describe that these cells can perform all the functions of normal T cells, i.e. cytokine secretion, cytotoxicity, and exocytosis of granule esterases. All these activities can be triggered via CD2 by binding to its natural ligand or via the TCR, e.g. by anti-TCR antibodies, by recognition of a bacterial superantigen and by MHC-restricted recognition…

Antigens Differentiation T-LymphocyteT-Lymphocytesmedicine.medical_treatmentImmunologyCD2 AntigensReceptors Antigen T-Cellchemical and pharmacologic phenomenaBiologyLymphocyte ActivationHerpesvirus 2 SaimiriineTCIRG1AntigenmedicineHumansImmunology and AllergyCytotoxic T cellAntigensReceptors ImmunologicCell Line TransformedT-cell receptorGeneral MedicineT lymphocyteCell Transformation ViralVirologyCell biologyPhenotypeCytokineInterleukin-2Cytokine secretionCD8International Immunology
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Defective T cell receptor/CD3 complex signaling in human type I diabetes

1994

The autoimmune process leading to the destruction of pancreatic β-cells is mediated by T lymphocytes. Peripheral T cells from subjects with preclinical and clinical type I diabetes respond weakly in vitro to lectin stimulation. We, therefore, investigated in a group of newly diagnosed diabetic patients the presence of a defect in the signal transduction pathway of the T cell receptor (TcR)/CD3 complex. Following stimulation with anti-CD3-coupled beads, the proliferative response in diabetic T cells was significantly decreased in comparison with that from normal T cells. Interestingly, addition of either recombinant interleukin (IL)-2 or phorbol 12-myristate 13-acetate to the cell culture wa…

Antigens Differentiation T-Lymphocytemedicine.medical_specialtyT-LymphocytesCD3ImmunologyBiologyLymphocyte ActivationInterleukin 21Antigens CDInternal medicinemedicineHumansImmunology and AllergyCytotoxic T cellLectins C-TypeIL-2 receptorProtein Kinase CInterleukin 3ZAP70T-cell receptorCD28Molecular biologyDiabetes Mellitus Type 1EndocrinologyReceptor-CD3 Complex Antigen T-Cellbiology.proteinCalciumEuropean Journal of Immunology
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Importance of the inducible costimulator molecule for the induction of allergic immune responses and its decreased expression on T helper cells after…

2004

The inducible costimulator (ICOS), a newly identified member of the CD28 receptor family that is induced after T-cell activation, and its ligand (ICOSL), being expressed on activated monocytes and dendritic cells play a key role in T-cell-mediated immune responses. As ICOS costimulation also seems to regulate T helper 2 effector cells, the aim of this study was to analyse the function of this molecule in allergic immune responses and their specific therapy, mainly venom immunotherapy (VIT). CD4+ T cells from grass pollen-, or bee or wasp venom-allergic donors were stimulated in the presence of autologous mature dendritic cells, which were pulsed with different allergen doses. In this system…

Antigens Differentiation T-Lymphocytemedicine.medical_treatmentImmunologyReceptors Antigen T-CellBiologyLymphocyte ActivationInducible T-Cell Co-Stimulator ProteinTh2 CellsImmune systemAntigenHypersensitivitymedicineHumansImmunology and AllergyInducible T-Cell Co-Stimulator ProteinReceptorArthropod VenomsHistocompatibility Antigens Class IICD28InterleukinOriginal ArticlesDendritic CellsImmunotherapyAllergensTh1 CellsCoculture TechniquesInterleukin-10CytokineDesensitization ImmunologicImmunologyImmunotherapyImmunology
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Resistance to Gemcitabine in a Lymphoma Cell Line Resistant to Fas-mediated Apoptosis

2004

BACKGROUND: The T-cell lymphoma cell line HuT78B1, selected for resistance to Fas-mediated apoptosis, resulted unexpectedly resistant to the apoptotic and cytotoxic effects of gemcitabine (dFdC). We investigated whether this resistance was due to the impairment of the Fas/Fas-ligand (FasL) system. MATERIALS AND METHODS: dFdC effects were studied in HuT78B1 and in the parental Fas-sensitive HuT78 cells exposed to inhibitors of the Fas/FasL system. RESULTS: FasL- and Fas-blocking antibodies did not interfere with dFdC-induced apoptosis in HuT78 cells, whereas inhibitors of caspase-8, -9, -1 or -3 had partial inhibitory effects. Notably, in HuT78B1 cells there was a markedly reduced dFdC accum…

Antimetabolites AntineoplasticFas Ligand ProteinMembrane GlycoproteinsApoptosisLymphoma T-CellCaspase InhibitorsDeoxycytidineGemcitabineEnzyme ActivationLymphoma T-Cell Cell Line Tumor gemcitabineDrug Resistance NeoplasmCaspasesCell Line TumorDeoxycytidine KinaseHumansfas Receptor
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PTEN Mediates the Antioxidant Effect of Resveratrol at Nutritionally Relevant Concentrations

2014

Introduction.Antioxidant properties of resveratrol have been intensively studied for the last years, bothin vivoandin vitro. Its bioavailability after an oral dose is very low and therefore it is very important to make sure that plasma concentrations of free resveratrol are sufficient enough to be active as antioxidant.Aims.In the present study, using nutritionally relevant concentrations of resveratrol, we aim to confirm its antioxidant capacity on reducing peroxide levels and look for the molecular pathway involved in this antioxidant effect.Methods.We used mammary gland tumor cells (MCF-7), which were pretreated with different concentrations of resveratrol for 48 h, and/or a PTEN inhibit…

Antioxidantendocrine system diseasesArticle Subjectmedicine.medical_treatmentlcsh:MedicineResveratrolGeneral Biochemistry Genetics and Molecular BiologyAntioxidantschemistry.chemical_compoundDownregulation and upregulationStilbenesmedicinePTENHumansPhosphorylationskin and connective tissue diseasesHydrogen peroxidePI3K/AKT/mTOR pathwayGeneral Immunology and MicrobiologybiologyAkt/PKB signaling pathwaySuperoxide Dismutaseorganic chemicalslcsh:RPTEN Phosphohydrolasefood and beveragesGeneral MedicineHydrogen PeroxideCatalaseUp-RegulationEnzyme ActivationBiochemistrychemistryCatalaseResveratrolbiology.proteinMCF-7 CellsProto-Oncogene Proteins c-akthormones hormone substitutes and hormone antagonistsSignal TransductionResearch Article
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DNA replication arrest in response to genotoxic stress provokes early activation of stress-activated protein kinases (SAPK/JNK).

2009

Abstract The impact of DNA damage-induced replication blockage for early activation of stress kinases [stress-activated protein kinase (SAPK)/c-Jun N-terminal kinase (JNK)] is largely unknown. Here, we show that induction of dual phosphorylation of SAPK/JNK by the DNA polymerase inhibitor aphidicolin was not ameliorated by additional exposure to ultraviolet (UV) light, indicating that overlapping mechanisms participate in signaling to SAPK/JNK triggered by both agents. UV-induced DNA replication blockage, cyclobutane pyrimidine dimer formation and DNA strand break induction coincided with SAPK/JNK phosphorylation at early (≤ 30 min) but not late (≥ 2 h) time points after exposure. Genotoxin…

AphidicolinDNA ReplicationDNA damageUltraviolet RaysPoly ADP ribose polymeraseCell Linechemistry.chemical_compoundMiceAphidicolinStructural BiologyCricetinaeAnimalsHumansLymphocytesPhosphorylationProtein kinase AMolecular BiologyNucleic Acid Synthesis InhibitorsBRCA2 ProteinMice KnockoutbiologyKinaseCell CycleDNA replicationJNK Mitogen-Activated Protein KinasesFibroblastsMolecular biologyProliferating cell nuclear antigenDNA-Binding ProteinsEnzyme ActivationchemistryPyrimidine Dimersbiology.proteinPhosphorylationApoptosis Regulatory ProteinsDNA DamageJournal of molecular biology
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The apolipoprotein(a) moiety of lipoprotein(a) interacts with the complement activation fragment iC3b but does not functionally affect C3 activation …

1992

A previous study has shown that complement component C3 binds to recombinant apolipoprotein(a) (r-apo(a)). In the present report we have investigated the interactions between lipoprotein(a) (Lp(a)), r-apo(a) and C3 in relation to complement activation and degradation. Neither Lp(a) nor r-apo(a) affected complement activation as indicated by sheep and rabbit red blood cell hemolytic assays, and by assessment of the amount of C3a generated in zymosan-activated human serum in the presence or absence of Lp(a). Crossed immunoelectrophoretic analyses indicated that Lp(a) retarded the migration of iC3b in complement-activated serum but had no effects on C3, C3b, C3c or C3dg. Recombinant apo(a) exh…

Apolipoprotein BLipoproteinsApoprotein(a)chemistry.chemical_compoundHumansComplement ActivationbiologyComplement C3Lipoprotein(a)N-Acetylneuraminic AcidComplement systemSialic acidApolipoproteinsBiochemistrychemistryLow-density lipoproteinComplement C3bSialic Acidsbiology.proteiniC3bElectrophoresis Polyacrylamide Gellipids (amino acids peptides and proteins)Cardiology and Cardiovascular MedicineImmunoelectrophoresis Two-DimensionalN-Acetylneuraminic acidLipoprotein(a)LipoproteinAtherosclerosis
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Determination of organic fluorine in aqueous samples with neutron activation analysis in comparison with the DIN method

1996

Neutron activation analysis (NAA) has been used as detection technique for adsorbable organic fluorine in aqueous samples. The results have been compared with the standardized DIN method which uses a fluoride-ion-selective electrode (ISE).

Aqueous solutionChemistryRadiochemistryElectrodeFluorinechemistry.chemical_elementNeutron activation analysisBiochemistryAnalytical ChemistryNeutron activationAnalytical and Bioanalytical Chemistry
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Isolation and characterization of a complement-activating lipid extracted from human atherosclerotic lesions.

1990

The major characteristics of human atherosclerotic lesions are similar to those of a chronic inflammatory reaction, namely fibrosis, mesenchymal cell proliferation, the presence of resident macrophages, and cell necrosis. Atherosclerosis exhibits in addition the feature of lipid (mainly cholesterol) accumulation. The results of the present report demonstrate that a specific cholesterol-containing lipid particle present in human atherosclerotic lesions activates the complement system to completion. Thus, lipid could represent a stimulatory factor for the inflammatory reaction, whose underlying mechanistic basis may be, at least in part, complement activation. The complement-activating lipid …

ArteriosclerosisComplement Pathway AlternativeImmunologyInflammationMuscle Smooth VascularC5-convertasechemistry.chemical_compoundMesenchymal cell proliferationmedicineHumansImmunology and AllergyComplement ActivationImmunoelectrophoresisAortaTriglyceridesCholesterolFatty AcidsComplement System ProteinsArticlesLipidsComplement systemCarotid ArteriesCholesterolchemistryBiochemistryLow-density lipoproteinChromatography GelAlternative complement pathwaylipids (amino acids peptides and proteins)Lipid particlemedicine.symptomJournal of Experimental Medicine
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Complement and atherogenesis: The unknown connection

1999

The question why low-density lipoprotein (LDL) stranded in the subendothelium of arteries should acquire the proinflammatory properties that initiate and sustain atherogenesis has puzzled researchers for decades. The most popular concept contends that oxidative processes are crucial because oxidized LDL (ox-LDL) produced in vitro has atherogenic properties and small amounts of it are found in atherosclerotic lesions. Recently, a possible role for vascular infections has also been considered because infectious agents, in particular Chlamydia pneumoniae, are sometimes present in the lesions. Here, evidence is summarized for a different concept of atherogenesis, which evolves from the fact tha…

ArteriosclerosisVascular diseaseInflammationGeneral MedicineChlamydia InfectionsChlamydophila pneumoniaeMacrophage ActivationBiologymedicine.diseaseProinflammatory cytokineLipoproteins LDLPathogenesischemistry.chemical_compoundImmune systemchemistryLow-density lipoproteinImmunologymedicineHumansMacrophagelipids (amino acids peptides and proteins)medicine.symptomComplement ActivationLipoproteinAnnals of Medicine
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