Search results for " ACTIVATION"

showing 10 items of 1535 documents

Association of cathepsin B gene polymorphisms with tropical calcific pancreatitis

2006

Background and aims: Tropical calcific pancreatitis (TCP) is a type of chronic pancreatitis unique to countries in the tropics. Mutations in pancreatic secretory trypsin inhibitor (SPINK1) rather than cationic trypsinogen (PRSS1) explain the disease in only 50% of TCP patients. As cathepsin B (CTSB) is known to activate cationic trypsinogen, we attempted to understand the role of CTSB mutations in TCP. Evidence of epistatic interaction was investigated with the previously associated N34S SPINK1 allele, a variant considered to be a modifier rather than a true susceptibility allele. Subjects and methods: We sequenced the coding region of CTSB gene in 51 TCP patients and 25 controls and furthe…

Malemedicine.medical_specialtyTrypsinogenSingle-nucleotide polymorphismBiologyPolymerase Chain ReactionGastroenterologyCathepsin BCathepsin Bchemistry.chemical_compoundGene FrequencyPancreatitis ChronicInternal medicinemedicineHumansGenetic Predisposition to DiseaseTrypsinogen activationAlleleAllele frequencyGeneticsPolymorphism GeneticHaplotypeGastroenterologyCase-control studyCalcinosisHaplotypesPancreatitischemistryCase-Control StudiesMutationAcute DiseaseFemaleGut
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Enzymatically Degraded, Nonoxidized LDL Induces Human Vascular Smooth Muscle Cell Activation, Foam Cell Transformation, and Proliferation

2000

Background —Enzymatic, nonoxidative modification transforms LDL to an atherogenic molecule (E-LDL) that activates complement and macrophages and is present in early atherosclerotic lesions. Methods and Results —We report on the atherogenic effects of E-LDL on human vascular smooth muscle cells (SMC). E-LDL accumulated in these cells, and this was accompanied by selective induction of monocyte chemotactic protein-1 in the absence of effects on the expression of interleukin (IL)-8, RANTES, or monocyte inflammatory proteins-1α and -β). Furthermore, E-LDL stimulated the expression of gp130, the signal-transducing chain of the IL-6 receptor (IL-6R) family, and the secretion of IL-6. E-LDL invok…

Malemedicine.medical_specialtyVascular smooth muscleArteriosclerosismedicine.medical_treatmentBiologyFibroblast growth factorMuscle Smooth VascularStatistics NonparametricPhysiology (medical)Internal medicinemedicineHomeostasisHumansRNA MessengerAutocrine signallingAortaCells CulturedChemokine CCL2AgedFoam cellInterleukin-6Cell growthGrowth factorMonocyteCholesterol LDLReceptors Interleukin-6EnzymesCell biologymedicine.anatomical_structureEndocrinologyFemaleCardiology and Cardiovascular MedicineCell activationOxidation-ReductionCell DivisionFoam CellsCirculation
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CAPD + hemoperfusion once a week in the management of children with end-stage renal disease

1983

With hemoperfusion there is a sharp decline in the solute concentration of the blood compartment, but immediately after HP the concentration of these substances rises to the original level (post-Hp rebound). It may be inferred that the result would be better and the rebound phenomenon less marked if hemoperfusion could be combined with a continuous dialysis system such as CAPD. The study was performed in 3 children of our CAPD programm. All patients were dialysed with 5 daily exchange each of one liter. The patients underwent at 2-hours hemoperfusion with a charcoal column (Gambro Adsorba 150 c) once a week. The data obtained with this form of treatment are indicative for a: significant in…

Malemedicine.medical_specialtymedicine.medical_treatment030232 urology & nephrologyBiomedical EngineeringUrologyMedicine (miscellaneous)Bioengineering030204 cardiovascular system & hematologyEnd stage renal diseasePeritoneal dialysisBiomaterials03 medical and health scienceschemistry.chemical_compound0302 clinical medicinePeritoneal Dialysis Continuous AmbulatoryHumansMedicinePlatelet activationChildIntensive care medicineDialysisCreatininebusiness.industryLiterGeneral MedicineHemoperfusionUric AcidHemoperfusionchemistryCreatinineKidney Failure ChronicUric acidFemalebusinessPeritoneal Dialysis
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Cell-free DNA and Microvascular Damage in ST-segment Elevation Myocardial Infarction Treated With Primary Percutaneous Coronary Intervention.

2019

INTRODUCTION AND OBJECTIVES Cell-free DNA (cfDNA) in ST-segment elevation myocardial infarction might originate from hyperactivated leukocytes at the coronary lesion. Our aim was to investigate the relationship between cfDNA and coronary reperfusion. METHODS We studied 116 patients treated with primary angioplasty using thrombus aspiration. Coronary (during aspiration) and peripheral (at the end of the procedure) blood samples were drawn for cfDNA, as well as high-sensitivity troponin T and myeloperoxidase quantification. The primary endpoint was no ST-segment resolution (STR) (≥ 70%) and the secondary endpoint was lack of final Thrombolysis In Myocardial Infarction flow 3 (TIMI 3). RESULTS…

Malemedicine.medical_specialtymedicine.medical_treatmentMyocardial ReperfusionMyocardial Reperfusion Injury030204 cardiovascular system & hematologyLymphocyte ActivationLesion03 medical and health sciences0302 clinical medicinePercutaneous Coronary InterventionTroponin TInternal medicinemedicineClinical endpointLeukocytesST segmentHumanscardiovascular diseasesMyocardial infarctionProspective StudiesPeroxidaseTroponin Tbusiness.industryPercutaneous coronary interventionGeneral MedicineThrombolysisMiddle Agedmedicine.diseaseTreatment OutcomeMicrovesselsCardiologyST Elevation Myocardial InfarctionFemalemedicine.symptombusinessCell-Free Nucleic AcidsTIMIRevista espanola de cardiologia (English ed.)
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Neuromuscular Adaptations to Low-Frequency Stimulation Training in a Patient with Chronic Heart Failure

2008

Electrical myostimulation (EMS) has recently been proposed as an interesting rehabilitation tool in patients with chronic heart failure (CHF). Although its beneficial effects on exercise capacity and muscle strength have been demonstrated, the neuromuscular adaptations responsible for improved performance are not well understood. This preliminary case report was therefore planned to investigate the neural and muscular adaptations to a 5-wk EMS program in one patient. An increase in maximal strength (10.5%) was accompanied by increased twitch torque (13.9%) and slowing of muscle contractile properties (half-relaxation time, time to peak torque, and maximal rate of relaxation increased by 7.1…

Malemedicine.medical_specialtymedicine.medical_treatmentPhysical Therapy Sports Therapy and RehabilitationPhysical medicine and rehabilitationHumansMedicineIn patientMuscle SkeletalLow frequency stimulationHeart FailureLegRehabilitationRelaxation (psychology)Electromyographybusiness.industryRehabilitationHeartStroke VolumeMuscle activationMiddle Agedmedicine.diseaseAdaptation PhysiologicalMyocardial ContractionElectric StimulationImproved performanceTorqueHeart failureFeasibility StudiesTime to peakCardiomyopathiesbusinessAmerican Journal of Physical Medicine & Rehabilitation
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Transcranial direct current stimulation improves isometric time to exhaustion of the knee extensors

2016

International audience; Transcranial direct current stimulation (tDCS) can increase cortical excitability of a targeted brain area, which may affect endurance exercise performance. However, optimal electrode placement for tDCS remains unclear. We tested the effect of two different tDCS electrode montages for improving exercise performance. Nine subjects underwent a control (CON), placebo (SHAM) and two different tDCS montage sessions in a randomized design. In one tDCS session, the anodal electrode was placed over the left motor cortex and the cathodal on contralateral forehead (HEAD), while for the other montage the anodal electrode was placed over the left motor cortex and cathodal electr…

Malemedicine.medical_treatmentIsometric exerciseFunctional LateralitytDCSRandom Allocation0302 clinical medicineHeart RateSingle-Blind Methodprefrontal cortexprimary motor cortexTranscranial direct-current stimulationexerciseGeneral NeuroscienceMotor Cortexvoluntary activationTranscranial Magnetic StimulationPeripheralmedicine.anatomical_structureMuscle FatiguePsychologyFemoral Nerveperformancemedicine.medical_specialtyShoulderintracortical inhibitionNeuroscience(all)B100brain stimulationPlacebo03 medical and health sciencesYoung AdultPhysical medicine and rehabilitationEndurance trainingIsometric ContractionHeart rateexcitabilitymedicineHumansneuromuscular functionMuscle Skeletalmagnetic stimulationhuman muscle fatigueLeg030229 sport sciencesEvoked Potentials MotorC600QPElectric Stimulationbody regionsBrain stimulation[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]ForeheadPhysical therapyPerceptionsupraspinal factors030217 neurology & neurosurgery
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The role of accessory cells in polyclonal T cell activation. I. Both induction of interleukin 2 production and of interleukin 2 responsiveness by con…

1983

Recent studies from other laboratories have shown that concanavalin A (Con A) acts at two separate steps in polyclonal T cell activation: interleukin 2 (IL2) production, and induction of responsiveness to IL2. Using a combination of techniques for the depletion of accessory cells from lymph node T cells, we have investigated which of these steps, if not both, is responsible for the known requirement for accessory cells in the Con A response. It was found that with increasing T cell purification, first the ability is lost to produce sufficient levels of endogenous IL2, whereas induction of IL2 responsiveness can still take place. Further removal of accessory cells however yields a population…

Malemusculoskeletal diseasesInterleukin 2medicine.medical_specialtyComplement Activating EnzymesT-LymphocytesT cellLymphocyte CooperationImmunologyPopulationchemical and pharmacologic phenomenaLymphocyte ActivationMiceInterleukin 21immune system diseasesInternal medicineConcanavalin AmedicineAnimalsImmunology and AllergyAntigen-presenting celleducationInterleukin 3LymphokinesMice Inbred BALB Ceducation.field_of_studybiologyComplement C1qImmune SeraHistocompatibility Antigens Class IIhemic and immune systemsCell biologyKineticsstomatognathic diseasesEndocrinologymedicine.anatomical_structurePolyclonal antibodiesConcanavalin Abiology.proteinInterleukin-2FemaleLymph NodesSpleenmedicine.drugEuropean Journal of Immunology
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Monocytes from patients with rheumatoid arthritis and type 2 diabetes mellitus display an increased production of interleukin (IL)-1β via the nucleot…

2015

Summary A better understanding about the mechanisms involved in the pathogenesis of type 2 diabetes mellitus (T2D) showed that inflammatory cytokines such as tumour necrosis factor (TNF) and interleukin (IL)-1β play a pivotal role, mirroring data largely reported in rheumatoid arthritis (RA). IL-1β is produced mainly by monocytes (MO), and hyperglycaemia may be able to modulate, in the cytoplasm of these cells, the assembly of a nucleotide-binding domain and leucine-rich repeat containing family pyrin (NLRP3)-inflammosome, a cytosolic multi-protein platform where the inactive pro-IL-1β is cleaved into active form, via caspase-1 activity. In this paper, we evaluated the production of IL-1 β …

Maletype 2 diabetes mellituInflammasomesMessengerIL-1β; NLRP3-inflammasome; rheumatoid arthritis; type 2 diabetes mellitus; Adult; Arthritis Rheumatoid; Carrier Proteins; Caspase 1; Cells Cultured; Diabetes Mellitus Type 2; Enzyme Activation; Female; Glucose; Humans; Hyperglycemia; Inflammasomes; Inflammation; Interleukin-1beta; Leukocytes Mononuclear; Male; Middle Aged; RNA Messenger; Tumor Necrosis Factor-alphaInterleukin-1betaArthritisPyrin domainInflammasomeArthritis RheumatoidRheumatoidImmunology and AllergyCells CulturedCulturedCaspase 1InterleukinDiabetes MellituMiddle AgedIL-1βTumor necrosis factor alphaNLRP3-inflammasomeFemalemedicine.symptomType 2ArthritiHumanAdultmedicine.medical_specialtyMononuclearImmunologyCaspase 1InflammationProinflammatory cytokineInternal medicineNLR Family Pyrin Domain-Containing 3 ProteinmedicineHumansRNA MessengerInflammationbusiness.industryTumor Necrosis Factor-alphaType 2 Diabetes MellitusOriginal Articlesrheumatoid arthritiLeukocytemedicine.diseaseEnzyme ActivationEndocrinologyGlucoseDiabetes Mellitus Type 2HyperglycemiaImmunologyLeukocytes MononuclearRNACellbusinessCarrier ProteinsCarrier Protein
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Alkali activated materials using pumice from the Aeolian Islands (Sicily, Italy) and their potentiality for cultural heritage applications: Prelimina…

2020

Abstract In this paper, the potentialities of pumice-based geopolymers have been explored with the aim to evaluate their use as restoration materials. In particular, the behaviour of Aeolian pumice during alkali activation process, either alone or in binary mixtures with metakaolin, has been assessed using sodium hydroxide and sodium silicate activators. Hardened products were characterized by: X-ray diffraction; infrared spectroscopy; thermo gravimetry and electron microscopy. Furthermore, Hg-intrusion porosimetry, water absorption, compressive strength and colorimetric measurements were carried out in order to define the technical features of the produced geopolymers. The results reveal t…

Materials scienceAbsorption of waterMetallurgySodium silicateAlkali activation; Cultural Heritage; Geopolymer binders; PumiceBuilding and ConstructionPorosimetryCultural HeritageGeopolymer binderschemistry.chemical_compoundSettore ING-IND/22 - Scienza E Tecnologia Dei MaterialiCompressive strengthAlkali activationPumicechemistrySodium hydroxidePumiceGeneral Materials SciencePorosityMetakaolinCivil and Structural Engineering
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Modification of human platelet adhesion on biomaterial surfaces by protein preadsorption under static and flow conditions.

2004

Biomaterial-induced thrombosis remains one of the main complications of vascular implant devices. Preadsorbed proteins on the biomaterial/blood interface will modify the adhesion and activation of platelets (PTLs) during the initial contact-phase. Our results clearly show that PTL-adherence on biomaterials is influenced not only by protein preadsorption, but also by flow conditions. The covalent coating of TCPS and glass by phosphorylcholine (PC) induces a significant decrease of PTL adhesion but leads to a slight, but nevertheless significant activation of PTL, which was detected by the induction of P-selectin expression using FACS analysis. Methodologically, the visualization of PTL adhes…

Materials scienceBiomedical EngineeringBiophysicschemistry.chemical_elementBioengineeringBiocompatible MaterialsCalciumIn Vitro TechniquesBiomaterialsBlood cellPlatelet AdhesivenessIn vivoMaterials TestingmedicineHumansPlateletPhosphorylcholineBiomaterialThrombosisAdhesionBlood ProteinsFlow CytometryPlatelet ActivationBiomechanical PhenomenaBlood Vessel ProsthesisP-Selectinmedicine.anatomical_structurechemistryembryonic structuresImmunologyHemorheologyBiophysicsAdsorptionProtein adsorptionJournal of materials science. Materials in medicine
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