Search results for " ADR"

showing 10 items of 285 documents

Ovarian suppression reduces clinical and endocrine expression of late-onset congenital adrenal hyperplasia due to 21-hydroxylase deficiency.

1994

Objective To determine the effectiveness of GnRH-agonist (GnRH-a) treatment in women with late onset congenital adrenal hyperplasia. Design Prospective assessment of GnRH-a treatment in six women with documented late-on-set congenital adrenal hyperplasia who were not preselected. Comparisons were made to previous responses in the same patients receiving dexamethasone. Eight age- and weight-matched ovulatory women served as controls. Setting Academic medical center. Intervention Baseline blood determinations before and after IV ACTH, before and after 6months of GnRH-a treatment. Estrogen and progestin replacement was begun in all women after the 3rd month of treatment. Main Outcome Measures …

Adultendocrine systemmedicine.medical_specialtyHirsutismAdolescentmedicine.drug_classOvaryDexamethasoneInternal medicineEndocrine GlandsmedicineHydroxyprogesteronesHumansCongenital adrenal hyperplasiaProspective StudiesAge of OnsethirsutismDexamethasoneTriptorelin PamoatebiologyAdrenal Hyperplasia Congenitalbusiness.industry17-alpha-HydroxyprogesteroneOvary21-HydroxylaseObstetrics and Gynecologymedicine.diseaseAndrogenmedicine.anatomical_structureEndocrinologyReproductive MedicineEstrogenbiology.proteinAndrogensFemalebusinessProgestinhormones hormone substitutes and hormone antagonistsGonadotropinsmedicine.drugFertility and sterility
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Refractory Acne and 21-Hydroxylase Deficiency in a Selected Group of Female Patients.

2009

<i>Background:</i> Excessive androgen production, suspected in women when acne is accompanied by hirsutism and menstrual irregularities, may be due to congenital adrenal hyperplasia. This inherited disorder of cortisol biosynthesis is caused in more than 90–95% of all cases by 21-hydroxylase deficiency (21-OHD). The steroid 21-hydroxylase gene <i>(CYP21)</i> has a high degree of variability. <i>Objective:</i> This study was conducted to evaluate <i>CYP21 </i>gene mutations in a selected group of women with papulopustular and comedonal acne refractory to treatment, irregular menses and hirsutism. <i>Methods:</i> 30 out of 61 women e…

Adultmedicine.medical_specialtyAdolescentDrug ResistancePhysiologyDermatologyAdrenocorticotropic hormoneYoung AdultSettore MED/38 - Pediatria Generale E SpecialisticaPapulopustularInternal medicineAcne VulgarismedicineHumansPoint MutationCongenital adrenal hyperplasiaGenetic TestingRefractory acne Excessive androgen production Non-classical 21-hydroxylase deficiency CYP21 gene mutations.AcnehirsutismAdrenal Hyperplasia Congenitalmedicine.diagnostic_testbiologybusiness.industry17-alpha-HydroxyprogesteroneACTH stimulation test21-Hydroxylasemedicine.diseasePolycystic ovaryEndocrinologybiology.proteinFemaleSteroid 21-HydroxylaseHyperandrogenismbusinessPolycystic Ovary Syndrome
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The ratio of androstenedione: 11β-hydroxyandrostenedione is an important marker of adrenal androgen excess in women

1992

To determine if the ratio of serum androstenedione (A):11 beta-hydroxyandrostenedione (OHA) would be helpful in differentiating adrenal from ovarian hyperandrogenism.Prospective study of outpatients being evaluated for hyperandrogenism.Normal women (n = 27), those with hyperandrogenic chronic anovulation (n = 25), and 7 with adult onset of congenital adrenal hyperplasia (CAH) because of 21-hydroxylase deficiency.Fasting serum between 8:00 A.M. and 9:00 A.M. Patients with hyperandrogenic chronic anovulation and CAH received dexamethasone (DEX) 2 mg for 7 days.Serum testosterone (T), unbound T, dehydroepiandrosterone sulfate (DHEAS), A, and 11 beta-OHA by radioimmunoassay.Serum 11 beta-OHA an…

Adultmedicine.medical_specialtyAdolescentmedicine.drug_classAdrenal Gland DiseasesRadioimmunoassayDexamethasoneAnovulationchemistry.chemical_compoundDehydroepiandrosterone sulfateInternal medicineAdrenal GlandsmedicineHumansCongenital adrenal hyperplasiaProspective StudiesAndrostenedioneDexamethasoneHyperplasiaAdrenal Hyperplasia Congenitalbusiness.industryHyperandrogenismAndrostenedioneObstetrics and GynecologyRadioimmunoassaymedicine.diseaseAndrogenEndocrinologyReproductive MedicinechemistryAndrogensFemalebusinessBiomarkersmedicine.drugFertility and Sterility
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Female pelvic congenital malformations. Part I: embryology, anatomy and surgical treatment.

2011

This review covers the most important female congenital pelvic malformations. The first part focuses on the embryological development of the urogenital and anorectal apparatus, morphological features, and the diagnostic and surgical approach to abnormalities. Comprehension of the embryological development of the urogenital and anorectal apparatus is essential to understand the morphology of congenital pelvic abnormalities and their surgical treatment. Congenital pelvic malformations are characterized by specific common features; the severity of which often subverts the pelvic morphology completely and makes it difficult to comprehend before surgery. The development of imaging, mainly magnet…

Adultmedicine.medical_specialtyReconstructive surgeryanorectal malformations46 XX Disorders of Sex DevelopmentAdolescentEmbryonic DevelopmentUrogenital SystemKidneyCongenital AbnormalitiesPelvisAnus Imperforatemedicinemayer-rokitansky-küster-hauser syndromeHumanscongenital adrenal hyperplasiaCongenital adrenal hyperplasiaMayer-Rokitansky-Kuster-Hauser SyndromeAbnormalities MultipleIntestine LargeMullerian Ductsmayer-rokitansky-kuster-hauser syndrome; mayer–rokitansky–kuster–hauser syndrome; mayer-rokitansky-küster-hauser syndrome; congenital adrenal hyperplasia; anorectal malformations; bladder exstrophymedicine.diagnostic_testAdrenal Hyperplasia Congenitalbusiness.industryGenitourinary systemmayer–rokitansky–kuster–hauser syndromeUterusObstetrics and GynecologyInfantMagnetic resonance imagingAnatomyAnusmedicine.diseaseSpineSurgeryBladder exstrophymedicine.anatomical_structureReproductive MedicineSomitesEmbryologyVaginamayer-rokitansky-kuster-hauser syndromeFemalebusinessbladder exstrophy
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Sympathetic nervous activity and the pressor effect of noradrenaline under chronic?-?-adrenoceptor blockade with labetalol in hypertension

1983

In 14 patients with essential hypertension, the influence of the alpha- and beta-adrenoceptor blocking drug labetalol on blood pressure, heart rate, plasma renin, plasma noradrenaline and pressor effect of exogenous noradrenaline was investigated during long-term treatment. During the initial four weeks of treatment, labetalol at a dose of 400 mg/day showed a slight effect only on supine blood pressure, whereas upright blood pressure was already lowered effectively after the second week of treatment (p less than 0.01). An increase in the mean dose to 850 mg/day had an additional blood pressure-lowering effect (p less than 0.001), whereby a preferential decrease of the orthostatic blood pres…

Adultmedicine.medical_specialtySympathetic Nervous SystemSupine positionPostureAlpha (ethology)Blood PressureEssential hypertensionPlasma renin activityPlacebosNorepinephrineInternal medicineReceptors Adrenergic betaReninDrug DiscoveryHeart ratemedicineHumansDrug InteractionsLabetalolLabetalolGenetics (clinical)Plasma noradrenalineDose-Response Relationship Drugbusiness.industryGeneral MedicineMiddle AgedReceptors Adrenergic alphamedicine.diseaseReceptors AdrenergicEndocrinologyBlood pressureEthanolaminesChronic DiseaseHypertensionMolecular Medicinebusinesscirculatory and respiratory physiologymedicine.drugKlinische Wochenschrift
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Managing Adult-onset Still's disease: The effectiveness of high-dosage of corticosteroids as first-line treatment in inducing the clinical remission.…

2019

Abstract To assess the effectiveness of the treatment with high dosage of corticosteroids (CCSs), as first-line therapy, in inducing remission in naïve Adult-onset Still's disease (AOSD) patients compared with low dosage of CCSs, after 6 months. To further evaluate the rate of patients maintaining the remission and the rate of CCSs discontinuation, after additional 12 months of follow-up. A retrospective evaluation of patients prospectively followed was designed to compare the rate of clinical remission in naïve AOSD patients treated with high dosages of CCSs (0.8–1 mg/kg/day of prednisone-equivalent) or low dosage of CCSs (0.2–0.3 mg/kg/day of prednisone-equivalent), after 6 months. An add…

Adult-OnsetMalePediatricsAdult-onset Still's diseaseDiseaseAdrenal Cortex Hormonecorticosteroids0302 clinical medicinemonocyclic patternAdrenal Cortex HormonesRetrospective StudieMedicine030212 general & internal medicineProspective StudiesProspective cohort studyRemission InductionDisease ManagementGeneral MedicineMiddle AgedTreatment OutcomeHigh dosage030220 oncology & carcinogenesisFemaleDrugStill's Disease Adult-OnsetResearch ArticleHumanAdultcorticosteroidmedicine.medical_specialtyLow dosageObservational StudyAdult-onset Still's diseaseFollow-Up StudieDose-Response Relationship03 medical and health sciencesremissionAdult-onset Still's disease; corticosteroids; first-line therapy; monocyclic pattern; remission; Adrenal Cortex Hormones; Adult; Disease Management; Dose-Response Relationship Drug; Female; Follow-Up Studies; Humans; Male; Methotrexate; Middle Aged; Prospective Studies; Remission Induction; Retrospective Studies; Still's Disease Adult-Onset; Treatment Outcomefirst-line therapyHumansRetrospective StudiesDose-Response Relationship Drugbusiness.industry6900Retrospective cohort studyStill's DiseaseFirst line treatmentSettore MED/16 - ReumatologiaProspective StudieMethotrexateObservational studybusinessFollow-Up Studies
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A study of beta-adrenoceptors in rat lung parenchymal strip.

1989

Abstract The aim of the present study was to characterize the β-adrenoceptor population in rat lung strip. For this purpose, Schild plots were obtained for the β-adrenoceptor antagonists atenolol (β1-selective), butoxamine (β2-selective) and propranolol (nonselective), using three different agonists: isoprenaline (non-selective), salbutamol (β2-selective) and noradrenaline (β11-selective). The slopes of these Schild plots were close to the theoretical value of unity, and pA2 values determined with isoprenaline, salbutamol and noradrenaline as agonists were: for propranolol, 7·86 ± 0·22, 7·72 ± 0·15 and 7·89 ± 0·23; for atenolol, 5·19 ± 0·05, 5·33 ± 0·07 and 5·47 ± 0·22 and for butoxamine, 6…

AgonistMalemedicine.medical_specialtymedicine.drug_classPopulationPharmaceutical SciencePropranololPharmacologyIn Vitro TechniquesButoxamineNorepinephrineInternal medicineIsoprenalineReceptors Adrenergic betamedicineAnimalsAlbuterolBeta (finance)educationLungPharmacologyeducation.field_of_studyChemistryAntagonistIsoproterenolAtenololPropranololButoxamineRatsEndocrinologyAtenololmedicine.drugThe Journal of pharmacy and pharmacology
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Desensitization of inhibitory prejunctional alpha 2-adrenoceptors and putative imidazoline receptors on rabbit heart sympathetic nerves.

1993

To find out whether sympathetic nerves of the rabbit heart possess pharmacologically relevant prejunctional imidazoline receptors different from α-autoreceptors, the inhibition by oxymetazoline, aganodine and BDF 6143 (4-chloro-2-[2-imidazoline-2-ylamino]-isoindoline hydrochloride) of endogenous noradrenaline overflow evoked by stimulation of extrinsic postganglionic sympathetic nerves (0.66 Hz, 80 pulses) was investigated. In addition we wanted to find out whether either type of these prejunctional receptors undergoes desensitization upon pre-exposure to respective agonists. The α2-adrenoceptor agonist oxymetazoline inhibited the evoked noradrenaline overflow (2.9 nmol/l, IC50; about 90010…

Agonistmedicine.medical_specialtySympathetic Nervous Systemmedicine.drug_classReceptors DrugRauwolscineOxymetazolineImidazoline receptorStimulationMuscarinic agonistchemistry.chemical_compoundNorepinephrineReceptors Adrenergic alpha-2Internal medicinemedicinePrazosinAnimalsAdrenergic alpha-AntagonistsAutoreceptorsPharmacologyHeartGeneral MedicineEndocrinologychemistryAutoreceptorImidazoline ReceptorsRabbitsAdrenergic alpha-Agonistsmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Characterization of sensory neurotransmission and its inhibition via alpha 2B-adrenoceptors and via non-alpha 2-receptors in rabbit iris.

1993

To find out whether, and which type of, adrenoceptors mediate prejunctional inhibition of sensory neurotransmitter release from trigeminal fibres, the modulation of twitch response to electrical field stimulation on rabbit isolated iris was investigated. Evoked iris sphincter contractions consisted of a minor fast cholinergic and a large slow component. The latter was unaffected by atropine and guanethidine, hence nonadrenergic noncholinergic in nature (NANC), but nearly completely abolished by capsaicin pretreatment and by the neurokinin receptor antagonist spantide. The response was probably not mediated by NK2 receptors as SR 48,968, an NK2 selective nonpeptide antagonist, failed to redu…

Agonistmedicine.medical_specialtymedicine.drug_classRauwolscineOxymetazolineOxymetazolineImidazoline receptorIrisSubstance PKininsNeurotransmissionIn Vitro TechniquesSubstance Pchemistry.chemical_compoundInternal medicinemedicineAnimalsNeurons AfferentSympathomimeticsReceptorPharmacologyNeurotransmitter AgentsChemistryGeneral MedicineReceptors Adrenergic alphaReceptor antagonistElectric StimulationEndocrinologyCholinergic FibersRabbitsmedicine.drugMuscle ContractionSignal TransductionNaunyn-Schmiedeberg's archives of pharmacology
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Interoceptive Abilities in Inflammatory Bowel Diseases and Irritable Bowel Syndrome

2020

International audience; Alexithymia is usually described by three main dimensions difficulty identifying feelings (DIF), difficulty describing feelings (DDF), and externally oriented thinking (EOT). The most commonly used questionnaire investigating alexithymia, the Toronto Alexithymia Scale (TAS-20), supports this three-factor structure. One important assumption is that alexithymia severity is associated to vulnerability to somatic diseases, among them gastrointestinal disorders. However, the association between alexithymia and gastrointestinal disorders is not systematic, thus questioning the role of alexithymia as a vulnerability factor for those illnesses. A recent factor analysis sugge…

Alexithymialcsh:RC435-571DiseaseInflammatory bowel diseaseInflammatory bowel disease03 medical and health sciencesToronto Alexithymia Scale0302 clinical medicineAlexithymiainflammatory bowel diseaselcsh:PsychiatrymedicineIrritable bowel syndromeDepression (differential diagnoses)Original Researchirritable bowel syndromePsychiatry[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologymedicine.diagnostic_testbusiness.industryinteroceptive abilities[SDV.MHEP.HEG]Life Sciences [q-bio]/Human health and pathology/Hépatology and Gastroenterologymedicine.disease[SDV.MHEP.HEG] Life Sciences [q-bio]/Human health and pathology/Hépatology and Gastroenterology030227 psychiatryPsychiatry and Mental healthIrritable bowel syndromemedicine.anatomical_structureHypothalamic-pituitary adrenal axis[SCCO.PSYC]Cognitive science/Psychology[SCCO.PSYC] Cognitive science/PsychologyAnxietyInteroceptive abilitiesalexithymiahypothalamic-pituitary adrenal axismedicine.symptombusiness030217 neurology & neurosurgeryHypothalamic–pituitary–adrenal axis[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyClinical psychology
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