Search results for " Cell Line"

showing 10 items of 238 documents

Chemical and biological evaluation of cross-linked halloysite-curcumin derivatives

2020

Abstract Well designed and safe nano drug carrier systems are an important tool in biomedical applications. The combination of two or more drugs has been used in medicine both to enhance the therapeutic effect and to decrease the side effects of drugs. Biocompatible halloysite nanotubes, that possess two different surfaces, are a suitable nanomaterial for a simultaneous carrier and release of two drugs that can exert a synergistic effect against cancer cells. In this study, three curcumin derivatives and doxorubicin were loaded by supramolecular and covalent linkage at the lumen and external surface of the halloysite nanotubes. The obtained multifunctional systems were characterized by seve…

Drugmedia_common.quotation_subjectNanoparticle020101 civil engineering02 engineering and technologyengineering.materialHalloysite0201 civil engineeringchemistry.chemical_compoundGeochemistry and PetrologymedicineDoxorubicinCytotoxicitymedia_commonSettore CHIM/02 - Chimica FisicaHalloysite nanotubes Curcumin derivatives Dual drug delivery Antiproliferative activity Breast cancer cell lines and acute myeloid leukemia cell linesChemistryGeologySettore CHIM/06 - Chimica Organica021001 nanoscience & nanotechnology3. Good healthCancer cellBiophysicsengineeringCurcuminSettore BIO/14 - Farmacologia0210 nano-technologyDrug carriermedicine.drug
researchProduct

GDNF reverses priming for dyskinesia in MPTP-treated, L-DOPA-primed common marmosets

2001

Parkinson's disease (PD) is associated with a progressive loss of dopamine neurons in the substantia nigra and degeneration of dopaminergic terminals in the striatum. Although L-DOPA treatment provides the most effective symptomatic relief for PD it does not prevent the progression of the disease, and its long-term use is associated with the onset of dyskinesia. In rodent and primate studies, glial cell line-derived neurotrophic factor (GDNF) may prevent 6-OHDA- or MPTP-induced nigral degeneration and so may be beneficial in the treatment of PD. In this study, we investigate the effects of GDNF on the expression of dyskinesia in L-DOPA-primed MPTP-treated common marmosets, exhibiting dyskin…

DyskinesiaParkinson's diseaseL-DOPASettore BIO/14 - FarmacologiaSettore MED/26 - NeurologiaGlial cell line-derived neurotrophic factorMPTPMarmoset
researchProduct

The Binomial “Inflammation-Epigenetics” in Breast Cancer Progression and Bone Metastasis: IL-1β Actions Are Influenced by TET Inhibitor in MCF-7 Cell…

2022

The existence of a tight relationship between inflammation and epigenetics that in primary breast tumor cells can lead to tumor progression and the formation of bone metastases was investigated. It was highlighted how the induction of tumor progression and bone metastasis by Interleukin-1 beta, in a non-metastatic breast cancer cell line, MCF-7, was dependent on the de-methylating actions of ten-eleven translocation proteins (TETs). In fact, the inhibition of their activity by the Bobcat339 molecule, an inhibitor of TET enzymes, determined on the one hand, the modulation of the epithelial-mesenchymal transition process, and on the other hand, the reduction in the expression of markers of bo…

Epithelial-Mesenchymal TransitionDNA methylation; bone metastasis; inflammation; Interleukin-1β; ten-eleven translocation proteins; MCF-7 cell lineInterleukin-1betaBreast NeoplasmsBone NeoplasmsMCF-7 cell lineCatalysisEpigenesis GeneticInorganic ChemistrySettore BIO/13 - Biologia ApplicataCell Line TumorHumansPhysical and Theoretical ChemistryMolecular BiologySpectroscopybone metastasisDNA methylationten-eleven translocation proteinsOrganic ChemistryGeneral MedicineInterleukin-1βComputer Science ApplicationsSettore BIO/18 - GeneticainflammationMCF-7 CellsFemaleInflammatory Breast NeoplasmsInternational Journal of Molecular Sciences
researchProduct

Monitoring Human Neutrophil Differentiation by Digital Holographic Microscopy

2021

We report on the usefulness of digital holographic microscopy (DHM) for the assessment of human neutrophil differentiation from myeloid cells. The cell and nuclear regions have been designated by image segmentation of the optical phase function, and the changes of the cell nucleus morphology in relation to the whole cell morphology have been examined during the process of granulocytic differentiation into mature neutrophils in PLB-985 cell line. Nucleus phase volume and circularity and the ratios between the nucleus and the cell projected area and volume provide a reliable set of parameters to characterize the maturation process. As control, cell differentiation has been monitored in parall…

Fluorescence-lifetime imaging microscopyHuman neutrophilMaterials Science (miscellaneous)Cellular differentiationQC1-999CellBiophysicsPLB-985 cell lineGeneral Physics and Astronomy02 engineering and technology01 natural scienceslabel-free010309 opticsNeutrophil differentiationneutrophil differentiation0103 physical sciencesmedicinePhysical and Theoretical ChemistryMathematical Physicscell and nucleus morphologyChemistryPhysics021001 nanoscience & nanotechnologyCell biologyStainingmedicine.anatomical_structureDigital holographic microscopydigital holography microscopy0210 nano-technologyNucleusFrontiers in Physics
researchProduct

Gold(I)-Coumarin-Caffeine-Based Complexes as New Potential Anti-Inflammatory and Anticancer Trackable Agents.

2018

Three new gold(I)-coumarin-based trackable therapeutic complexes and two non-trackable analogues have been synthesised and fully characterised. They all display anti-proliferative properties on several types of cancer cell lines, including those of colon, breast, and prostate. Two complexes displayed significant anti-inflammatory effects; one displayed pro-inflammatory behaviour; this highlights the impact of the position of the fluorophore on the caffeine scaffold. Additionally, the three coumarin derivatives could be visualised in vitro by two-photon microscopy.

Fluorophoremedicine.drug_classUltraviolet RaysAnti-Inflammatory AgentsAntineoplastic Agents010402 general chemistry01 natural sciencesBiochemistryAnti-inflammatoryFluorescencechemistry.chemical_compoundCoordination ComplexesCoumarinsCaffeineCell Line TumorDrug DiscoverymedicineHumansGeneral Pharmacology Toxicology and PharmaceuticsFluorescent DyesPharmacology010405 organic chemistryOrganic ChemistryCancerCoumarinmedicine.diseaseCombinatorial chemistryIn vitro0104 chemical sciencesHEK293 CellsMicroscopy Fluorescence MultiphotonchemistryMolecular MedicineGoldCancer cell linesCaffeineChemMedChem
researchProduct

Expression of retinoic acid nuclear receptors in the mouse embryonal carcinoma cell line PCC7-Mz1

1992

Mouse embryonal carcinoma cell line PCC7-Mz1 can serve as a model of mammalian neural development [1989, J. Cell. Biol. 109, 2481-2493]. Upon exposure to all-trans retinoic acid (RA), Mz1 cells differentiate into a stable pattern of neurons, astroglia and fibroblasts whereas variants of the parental cell line either are restricted in their patterns of derivatives or do not respond at all to RA. Using gene probes specific for the alpha 1, alpha 2 and beta 2 isoforms of the retinoic acid nuclear receptor, we have studied by Northern blot analysis the expression of these transcription factors in uninduced and induced cells of clone Mz1 and in variants with different developmental potential. al…

Gene isoformmedicine.medical_specialtyTranscription GeneticReceptors Retinoic AcidCellular differentiationBiophysicsRetinoic acidTretinoinExpressionBiologyEmbryonic carcinoma cell line PCC7-MzBiochemistryEmbryonal carcinomaMicechemistry.chemical_compoundStructural BiologyInternal medicineTumor Cells CulturedGeneticsmedicineAnimalsRNA MessengerNorthern blotMolecular BiologyCell NucleusdbcAMPTeratomaRetinoic acid receptorCell DifferentiationCell BiologyBlotting Northernmedicine.diseaseCell biologyRetinoic acid receptorEndocrinologyBucladesineNuclear receptorchemistryCell cultureRNACarrier ProteinsPoly AIsoformsFEBS Letters
researchProduct

Non-small cell lung cancer (NSCLC), EGFR downstream pathway activation and TKI targeted therapies sensitivity: Effect of the plasma membrane-associat…

2017

Adenocarcinoma of Non-Small Cell Lung Cancer (NSCLC) is a severe disease. Patients carrying EGFR mutations may benefit from EGFR targeted therapies (e.g.: gefitinib). Recently, it has been shown that sialidase NEU3 directly interacts and regulates EGFR. In this work, we investigate the effect of sialidase NEU3 overexpression on EGFR pathways activation and EGFR targeted therapies sensitivity, in a series of lung cancer cell lines. NEU3 overexpression, forced after transfection, does not affect NSCLC cell viability. We demonstrate that NEU3 overexpression stimulates the ERK pathway but this activation is completely abolished by gefitinib treatment. The Akt pathway is also hyper-activated upo…

Genetics and Molecular Biology (all)0301 basic medicineOncologyMAPK/ERK pathwayLung NeoplasmsColorectal cancerCell Membraneslcsh:Medicinenon-small cell lung cancer (NSCLC)BiochemistryLung and Intrathoracic TumorsAntineoplastic Agent0302 clinical medicineProtein-Tyrosine KinaseCarcinoma Non-Small-Cell LungMedicine and Health SciencesPost-Translational ModificationPhosphorylationNon-Small-Cell Lunglcsh:ScienceTumorMultidisciplinaryBlottingGefitinibTransfectionProtein-Tyrosine KinasesBIO/10 - BIOCHIMICAErbB ReceptorsOncology030220 oncology & carcinogenesisAdenocarcinomaPhosphorylationHyperexpression TechniquesElectrophoresis Polyacrylamide GelCellular Structures and OrganellesWesternReceptorHumanmedicine.drugSignal TransductionResearch ArticleElectrophoresismedicine.medical_specialtyBlotting WesternNeuraminidaseAntineoplastic AgentsReal-Time Polymerase Chain ReactionTransfectionResearch and Analysis MethodsCell Line03 medical and health sciencesGefitinibInternal medicineCell Line TumormedicineGeneticsGene Expression and Vector TechniquesHumansPoint MutationMolecular Biology TechniquesMolecular BiologyPI3K/AKT/mTOR pathwayColorectal CancerMolecular Biology Assays and Analysis TechniquesPolyacrylamide GelBiochemistry Genetics and Molecular Biology (all)Epidermal Growth Factorbusiness.industryCarcinomalcsh:RCell MembraneQuinazolineCancers and NeoplasmsBiology and Life SciencesProteinsCell Biologymedicine.diseaserespiratory tract diseasesNon-Small Cell Lung CancerLung Neoplasm030104 developmental biologyAgricultural and Biological Sciences (all)MutationQuinazolineslcsh:QReceptor Epidermal Growth FactorAntineoplastic Agents; Blotting Western; Carcinoma Non-Small-Cell Lung; Cell Line Tumor; Cell Membrane; Electrophoresis Polyacrylamide Gel; Humans; Lung Neoplasms; Neuraminidase; Protein-Tyrosine Kinases; Quinazolines; Real-Time Polymerase Chain Reaction; Receptor Epidermal Growth Factor; Signal Transduction; Biochemistry Genetics and Molecular Biology (all); Agricultural and Biological Sciences (all)businessPloS one
researchProduct

C1q acts in the tumour microenvironment as a cancer-promoting factor independently of complement activation

2015

Complement C1q is the activator of the classical pathway. However, it is now recognized that C1q can exert functions unrelated to complement activation. Here we show that C1q, but not C4, is expressed in the stroma and vascular endothelium of several human malignant tumours. Compared with wild-type (WT) or C3- or C5-deficient mice, C1q-deficient (C1qa−/−) mice bearing a syngeneic B16 melanoma exhibit a slower tumour growth and prolonged survival. This effect is not attributable to differences in the tumour-infiltrating immune cells. Tumours developing in WT mice display early deposition of C1q, higher vascular density and an increase in the number of lung metastases compared with C1qa−/− mi…

Genetics and Molecular Biology (all)0301 basic medicinePROTEINGeneral Physics and AstronomyMELANOMAApoptosisInbred C57BLBiochemistryDISEASEAnimals; Apoptosis; Cell Line Tumor; Cell Movement; Cell Proliferation; Complement Activation; Complement C1q; Complement C3; Complement C5; Humans; Mice; Mice Inbred C57BL; Mice Knockout; Neoplasms; Biochemistry Genetics and Molecular Biology (all); Chemistry (all); Physics and Astronomy (all)Micefluids and secretionsCell Movementimmune system diseasesNeoplasmsIMMUNE-RESPONSEskin and connective tissue diseasesComplement ActivationComplement C1qMice KnockoutComplement component 5TumorMultidisciplinaryQChemistry (all)Complement C5Complement C33. Good healthCell biologyMultidisciplinary SciencesDEFICIENCYmedicine.anatomical_structureScience & Technology - Other TopicsHumanKnockoutSciencechemical and pharmacologic phenomenaBiologyArticleGeneral Biochemistry Genetics and Molecular BiologyTROPHOBLAST INVASIONMECHANISMSCell LinePhysics and Astronomy (all)03 medical and health sciencesClassical complement pathwayImmune systemINFLAMMATIONCell Line TumormedicineAnimalsHumansCell ProliferationScience & TechnologyBiochemistry Genetics and Molecular Biology (all)AnimalCell growthEFFECTOR SYSTEMComplement C1qApoptosiGeneral ChemistryComplement systemMice Inbred C57BL030104 developmental biologyCancer cellNeoplasmBone marrowANTIBODY THERAPYNature Communications
researchProduct

A BMP7 Variant Inhibits Tumor Angiogenesis In Vitro and In Vivo through Direct Modulation of Endothelial Cell Biology

2015

Bone morphogenetic proteins (BMPs), members of the TGF-β superfamily, have numerous biological activities including control of growth, differentiation, and vascular development. Using an in vitro co-culture endothelial cord formation assay, we investigated the role of a BMP7 variant (BMP7v) in VEGF, bFGF, and tumor-driven angiogenesis. BMP7v treatment led to disruption of neo-endothelial cord formation and regression of existing VEGF and bFGF cords in vitro. Using a series of tumor cell models capable of driving angiogenesis in vitro, BMP7v treatment completely blocked cord formation. Pre-treatment of endothelial cells with BMP7v significantly reduced their cord forming ability, indicating …

Genetics and Molecular Biology (all)MaleVascular Endothelial Growth Factor AFibroblast Growth FactorAngiogenesisBone Morphogenetic Protein 7Nudelcsh:MedicineSmad ProteinsFibroblast growth factorBiochemistryNeovascularizationMiceCell Movementlcsh:ScienceBMP7 Angiogenesis TumorTumorMultidisciplinaryCell DeathNeovascularization PathologicMedicine (all)Cell migrationCell biologyEndothelial stem cellSettore MED/26 - NEUROLOGIAVascular endothelial growth factor ADrug CombinationsAdipose TissueAdipose Tissue; Animals; Bone Morphogenetic Protein 7; Cell Death; Cell Line Tumor; Cell Movement; Cell Proliferation; Collagen; Drug Combinations; Endothelial Cells; Fibroblast Growth Factor 2; Glioblastoma; Human Umbilical Vein Endothelial Cells; Humans; Laminin; Male; Mice Nude; Neoplastic Stem Cells; Neovascularization Pathologic; Neovascularization Physiologic; Proteoglycans; Receptor Fibroblast Growth Factor Type 1; Signal Transduction; Smad Proteins; Vascular Endothelial Growth Factor A; Vascular Endothelial Growth Factor Receptor-2; Xenograft Model Antitumor Assays; Biochemistry Genetics and Molecular Biology (all); Agricultural and Biological Sciences (all)Neoplastic Stem CellsFibroblast Growth Factor 2ProteoglycansCollagenmedicine.symptomReceptorType 1Research ArticleSignal TransductionMice NudeNeovascularization PhysiologicBMP7BiologyCell LineSettore MED/04 - PATOLOGIA GENERALECell Line TumormedicineHuman Umbilical Vein Endothelial CellsAnimalsHumansAgricultural and Biological Sciences (all); Biochemistry Genetics and Molecular Biology (all); Medicine (all)Receptor Fibroblast Growth Factor Type 1PhysiologicNeovascularizationCell ProliferationPathologicMatrigelBiochemistry Genetics and Molecular Biology (all)lcsh:REndothelial CellsKinase insert domain receptorVascular Endothelial Growth Factor Receptor-2Xenograft Model Antitumor AssaysAgricultural and Biological Sciences (all)lcsh:QAngiogenesisLamininGlioblastomaPLoS ONE
researchProduct

Role and importance of polymorphisms with respect to DNA methylation for the expression of CYP2E1 enzyme

2014

Different individuals possess slightly different genetic information and show genetically-determined differences in several enzyme activities due to genetic variability. Following an integrated approach, we studied the polymorphisms and methylation of sites contained in the 5' flanking region of the metabolizing enzyme CYP2E1 in correlation to its expression in both tumor and non-neoplastic liver cell lines, since to date little is known about the influence of these (epi)genetic elements in basal conditions and under induction by the specific inductor and a demethylating agent. In treated cells, reduced DNA methylation, assessed both at genomic and gene level, was not consistently associate…

Genotype5' Flanking RegionCell Survival5' flanking regionMinisatellite RepeatsBiologyGene Expression Regulation EnzymologicGenotypeGeneticsCYP2E1 gene polymorphismTumor Cells CulturedHumansGeneHepatocellular carcinoma cell lines; CYP2E1 gene polymorphisms; DNA methylation; 5-Azacytidine; Ethanol;GeneticsPolymorphism GeneticEthanolLiver cellHaplotype5-AzacytidineCytochrome P-450 CYP2E1General MedicineMethylationHep G2 CellsHepatocellular carcinoma cell lineDNA MethylationMolecular biologySettore BIO/18 - GeneticaDNA methylationAzacitidineRestriction fragment length polymorphismPolymorphism Restriction Fragment Length
researchProduct