Search results for " DOPAMINE"

showing 10 items of 177 documents

Self-Regulation Therapy to Reproduce Drug Effects:A Suggestion Technique to Change Personality and theDRD3Gene Expression

2013

This study proposes a strategy, based on self-regulation therapy, to change personality and its biological substrate, the DRD3 gene expression. It has been demonstrated that acute doses of stimulating drugs, like methylphenidate, are able to change personality and the expression of certain genes in the short term. On the other hand, self-regulation therapy has been proven to reproduce the effects of drugs. Thus, it is feasible to hope that self-regulation therapy is equally effective as methylphenidate in changing personality and the gene expression. This is a preliminary study with a single-case experimental design with replication in which 2 subjects participated. The results and potentia…

MaleComplementary and Manual TherapyDrugHypnosisPsychotherapistPersonality InventoryC-Fosmedia_common.quotation_subjectGene ExpressionMessenger RNA expressionD-3 receptorGene expressionmedicineHumansPersonalityPeripheral blood lymphocytesSuggestionmedia_commonMethylphenidateReceptors Dopamine D3Middle AgedClinical PsychologyExpression (architecture)MethylphenidateCentral Nervous System StimulantsFemalesense organsDopamine receptor geneMATEMATICA APLICADAPsychologyPersonalitymedicine.drugInternational Journal of Clinical and Experimental Hypnosis
researchProduct

Clavines as antitumor agents. 3: Cytostatic activity and structure/activity relationships of 1-alkyl agroclavines and 6-alkyl 6-noragroclavines.

1986

The cytostatic potential of twenty antibiotic agroclavines has been examined in the L5178y mouse lymphoma cell system. Twelve of these compounds are described for the first time. It is shown that the substituent at N-1 of agroclavine is very important whereas the substituent at N-6 is of less influence if it is not hydrogen. Incorporation studies in the presence of 1-propylagroclavine suggest that DNA synthesis in the lymphoma cells is inhibited. The effect on the corresponding [3H]thymidine incorporation in murine spleen lymphocytes is comparably low. Neither a significant change of mRNA efflux nor of DNA polymerase alpha and beta activities was caused. The mechanism of action seems to be …

MaleDNA polymeraseDNA-Directed DNA PolymeraseLymphocyte ActivationReceptors DopamineMiceStructure-Activity RelationshipDrug DiscoverymedicineAnimalsRNA MessengerRNA NeoplasmErgolinesLeukemia L1210ReceptorAlkylPharmacologychemistry.chemical_classificationAntibiotics AntineoplasticDNA synthesisbiologyDNA NeoplasmIn vitroNeoplasm ProteinsErgolineMechanism of actionchemistryBiochemistryReceptors Serotoninbiology.proteinEffluxmedicine.symptommedicine.drugThe Journal of Antibiotics
researchProduct

rTMS of supplementary motor area modulates therapy-induced dyskinesias in Parkinson disease

2005

The neural mechanisms and circuitry involved in levodopa-induced dyskinesia are unclear. Using repetitive transcranial magnetic stimulation (rTMS) over the supplementary motor area (SMA) in a group of patients with advanced Parkinson disease, the authors investigated whether modulation of SMA excitability may result in a modification of a dyskinetic state induced by continuous apomorphine infusion. rTMS at 1 Hz was observed to markedly reduce drug-induced dyskinesias, whereas 5-Hz rTMS induced a slight but not significant increase.

MaleDyskinesia Drug-InducedApomorphinemedicine.medical_treatmentDopamineNeurological disorderNOCentral nervous system diseaseDegenerative diseasemental disordersNeural PathwaysmedicineHumansAgedSupplementary motor areaDyskinesiabusiness.industryDyskinesia Drug-Induced; Treatment Outcome; Male; Middle Aged; Female; Humans; Parkinson Disease; Motor Cortex; Recovery of Function; Apomorphine; Dopamine Agonists; Neural Pathways; Aged; Transcranial Magnetic Stimulation; DopamineMotor CortexParkinson DiseaseRecovery of FunctionMiddle Agedmedicine.diseaseSMA*Transcranial Magnetic Stimulationnervous system diseasesTranscranial magnetic stimulationApomorphinemedicine.anatomical_structureTreatment OutcomeDyskinesiaDrug-InducedDopamine AgonistsFemaleSettore MED/26 - NeurologiaNeurology (clinical)medicine.symptombusinessNeurosciencemedicine.drug
researchProduct

Association between neonatal temperament,SLC6A4,DRD4and a functional polymorphism located inTFAP2B

2011

Genetic studies on human personality have provided little satisfactory results to date mainly because of the complexity of this trait. Neonatal temperament using observational measures is an alternative phenotype to approach genetics to human behavior. An association study was conducted on 117 Caucasian newborns. Their temperament was evaluated using the Neonatal Behavior Assessment Scale 48 h after birth. Thirteen polymorphisms in the SLC6A4, DRD4 and TFAP2B genes were genotyped. Linear regression was performed to analyze data, and Bonferroni correction was applied. To check the functional effect of the TFAP2B Indel Intron 2 polymorphism, reporter gene luciferase assays using a mouse corti…

MaleGenotypemedia_common.quotation_subjectMinisatellite RepeatsBiologyBehavioral NeuroscienceExonGeneticsHumansAlleleTemperamentIndelGeneAllelesGenetic Association Studiesmedia_commonSerotonin Plasma Membrane Transport ProteinsGeneticsPolymorphism GeneticReceptors Dopamine D4Infant NewbornIntronVariable number tandem repeatReal-time polymerase chain reactionTranscription Factor AP-2NeurologyInfant BehaviorFemaleTemperamentGenes, Brain and Behavior
researchProduct

Modulation of high impulsivity and attentional performance in rats by selective direct and indirect dopaminergic and noradrenergic receptor agonists

2011

Rationale Impulsivity is associated with a number of psychiatric disorders, most notably attention deficit/hyperactivity disorder (ADHD). Drugs that augment catecholamine function (e.g. methylphenidate and the selective noradrenaline reuptake inhibitor atomoxetine) have clinical efficacy in ADHD, but their precise mechanism of action is unclear. Objective The objective of this study is to investigate the relative contribution of dopamine (DA) and noradrenaline (NA) to the therapeutic effects of clinically effective drugs in ADHD using rats selected for high impulsivity on the five-choice serial reaction time task (5CSRTT). Methods We examined the effects of direct and indirect DA and NA rec…

MaleImpulsivityQuinpiroleDopamineSerial LearningAtomoxetine HydrochlorideImpulsivityChoice BehaviorPiperazines03 medical and health sciences0302 clinical medicineQuinpiroleDopaminemental disordersAnimals Outbred StrainsReaction TimemedicineAnimalsAttentionOriginal InvestigationPharmacologyPropylaminesMethylphenidateDopaminergicAtomoxetineGBR-12909Adrenergic AgonistsGuanfacineRats030227 psychiatry3. Good healthGuanfacineSumaniroleFive-choice serial reaction time taskAtomoxetine; Dopamine; Five-choice serial reaction time task; GBR-12909; Guanfacine; Impulsivity; Methylphenidate; Noradrenaline; Quinpirole; Sumanirole; Adrenergic Agonists; Animals; Animals Outbred Strains; Atomoxetine Hydrochloride; Attention; Benzimidazoles; Choice Behavior; Dopamine Agonists; Guanfacine; Impulsive Behavior; Male; Methylphenidate; Piperazines; Propylamines; Quinpirole; Rats; Reaction Time; Serial Learning; PharmacologyAnesthesiaDopamine AgonistsImpulsive BehaviorNoradrenalineAtomoxetineMethylphenidateBenzimidazolesmedicine.symptomPsychologyNeuroscience030217 neurology & neurosurgerymedicine.drugAtomoxetine hydrochloride
researchProduct

Involvement of Dopamine D2 Receptors in Addictive-Like Behaviour for Acetaldehyde

2014

Acetaldehyde, the first metabolite of ethanol, is active in the central nervous system, where it exerts motivational properties. Acetaldehyde is able to induce drinking behaviour in operant-conflict paradigms that resemble the core features of the addictive phenotype: drug-intake acquisition and maintenance, drug-seeking, relapse and drug use despite negative consequences. Since acetaldehyde directly stimulates dopamine neuronal firing in the mesolimbic system, the aim of this study was the investigation of dopamine D2-receptors' role in the onset of the operant drinking behaviour for acetaldehyde in different functional stages, by the administration of two different D2-receptor agonists, q…

MaleIndoleslcsh:MedicinePharmacologyBehavioral Neurosciencechemistry.chemical_compoundquinpiroleMedicine and Health Scienceslcsh:ScienceNeuropharmacologyDrug DependenceMultidisciplinaryDopaminergicD2 dopamine receptorsAcetaldehyde; Operant self-administration; D2 dopamine receptors; quinpiroleNeurologyBehavioral PharmacologyDopamine AgonistsSignal TransductionResearch Articlemedicine.drugAlcohol DrinkingDrug-Seeking BehaviorAcetaldehydeAddictive-Like BehaviourNeuropharmacologyQuinpiroleDopamineDopamine receptor D2medicineAnimalsRats WistarAcetaldehyde; Addictive-Like Behaviour; Dopamine D2 ReceptorsPharmacologyOperant self-administrationEthanolReceptors Dopamine D2Neurotransmissionlcsh:RAcetaldehydeBiology and Life SciencesDopamine D2 ReceptorsRatsRopinirolePharmacodynamicschemistrySettore BIO/14 - FarmacologiaConditioning Operantlcsh:QNeurosciencePLoS ONE
researchProduct

Partial replication of a DRD4 association in ADHD individuals using a statistically derived quantitative trait for ADHD in a family-based association…

2007

Contains fulltext : 52515.pdf (Publisher’s version ) (Closed access) BACKGROUND: Previous research found an association between single nucleotide polymorphisms (SNPs) in the promoter region of DRD4 and statistically derived phenotypes generated from attention-deficit/hyperactivity disorder (ADHD) symptoms. We sought to replicate this finding by using the same methodology in an independent sample of ADHD individuals. METHODS: Four SNPs were genotyped in and around DRD4 in 2631 individuals in 642 families. We developed a quantitative phenotype at each SNP by weighting nine inattentive and nine hyperactive-impulsive symptoms. The weights were selected to maximize the heritability at each SNP. …

MaleLinkage disequilibriumGenetics and epigenetic pathways of disease [NCMLS 6]Databases FactualMedizinNeuroinformatics [DCN 3]Severity of Illness Index0302 clinical medicinePerception and Action [DCN 1]Determinants in Health and Disease [EBP 1]ChildPromoter Regions GeneticGenetics0303 health sciencesEuropePhenotypeChild PreschoolFemalemedicine.symptomPsychologyFunctional Neurogenomics [DCN 2]medicine.medical_specialtyAdolescentSingle-nucleotide polymorphismQuantitative trait locusImpulsivityMental health [NCEBP 9]Polymorphism Single NucleotideGenomic disorders and inherited multi-system disorders [IGMD 3]03 medical and health sciencesQuantitative Trait HeritableCognitive neurosciences [UMCN 3.2]Genetic modelmental disordersmedicineAttention deficit hyperactivity disorderSNPHumansGenetic Predisposition to Diseaseddc:610Medizinische Fakultät » Universitätsklinikum Essen » LVR-Klinikum Essen » Klinik für Psychiatrie Psychosomatik und Psychotherapie des Kindes- und JugendaltersPsychiatryBiological Psychiatry030304 developmental biologyFamily HealthReceptors Dopamine D4Heritabilitymedicine.diseaseGenetic defects of metabolism [UMCN 5.1]Attention Deficit Disorder with Hyperactivity030217 neurology & neurosurgeryBiological psychiatry
researchProduct

Effects of risperidone and SCH 23390 on isolation-induced aggression in male mice.

1998

In this study, the antiaggressive effects of risperidone and SCH 23390 have been explored. Using the paradigm of isolation-induced aggression, 150 albino male mice of the OF1 strain were allocated to control and experimental groups which received three doses of risperidone (0.01, 0.05 and 0.1 mg/kg) or two doses of SCH 23390 (0.05 and 0.1 mg/kg). Only the highest doses of risperidone decreased threat and attack behaviours but all doses significantly impaired motor behaviour. SCH 23390 decreased attack with the two doses used and also produced significant increases in immobility. Although both antipsychotics are antiaggressive, this action seems to be more specific in the case of risperidone…

MaleMale micePharmacologyNeurotransmissionMotor Activitychemistry.chemical_compoundMiceSexual Behavior AnimalDopaminemedicineAnimalsPharmacology (medical)Biological PsychiatryPharmacologySCH-23390RisperidoneAggressionReceptors Dopamine D1BenzazepinesRisperidoneGroomingAggressionPsychiatry and Mental healthDopamine D2 Receptor AntagonistsNeurologychemistryIsolation induced aggressionSocial IsolationDepression ChemicalExploratory BehaviorDopamine AntagonistsFemaleNeurology (clinical)Serotoninmedicine.symptomPsychologymedicine.drugEuropean neuropsychopharmacology : the journal of the European College of Neuropsychopharmacology
researchProduct

Tetrahydroisoquinolines as dopaminergic ligands: 1-Butyl-7-chloro-6-hydroxy-tetrahydroisoquinoline, a new compound with antidepressant-like activity …

2009

International audience; Three series of 1-substituted-7-chloro-6-hydroxy-tetrahydroisoquinolines (1-butyl-, 1-phenyl- and 1-benzyl derivatives) were prepared to explore the influence of each of these groups at the 1-position on the affinity for dopamine receptors. All the compounds displayed affinity for D(1)-like and/or D(2)-like dopamine receptors in striatal membranes, and were unable to inhibit [(3)H]-dopamine uptake in striatal synaptosomes. Different structure requirements have been observed for adequate D(1) or D(2) affinities. This paper details the synthesis, structural elucidation, dopaminergic binding assays, structure-activity relationships (SAR) of these three series of isoquin…

MaleModels MolecularStereochemistryDopamineClinical BiochemistryPharmaceutical ScienceMotor Activity010402 general chemistryLigands01 natural sciencesBiochemistryReceptors Dopaminechemistry.chemical_compoundMiceStructure-Activity RelationshipDopamineTetrahydroisoquinolinesDrug DiscoverymedicineStructure–activity relationshipAnimalsReceptorMolecular Biology010405 organic chemistryTetrahydroisoquinolineReceptors Dopamine D2Receptors Dopamine D1[SCCO.NEUR]Cognitive science/NeuroscienceOrganic ChemistryDopaminergicAntagonistAntidepressive AgentsCorpus Striatum3. Good health0104 chemical sciencesRatschemistryDopamine receptorHydroxyquinolinesMolecular MedicineLead compoundmedicine.drugProtein Binding
researchProduct

Effects of DA D1 and D2 antagonists on the sensitisation to the motor effects of morphine in mice

2002

Abstract Acute morphine administration produces hyperactivity in mice and repeated treatment induces an enhancement of this effect. In this experiment, we study the sensitisation to the hyperactivity induced by intermittent morphine administration (40 mg/kg) and the effects of dopamine (DA) antagonists on this phenomenon. Animals received three injections, separated by 48 h, and after each injection, their activity was registered between 30 and 60 min. In Experiment 1, animals were divided into two groups, which received saline and morphine (S–S–M) or only morphine (M–M–M). In Experiment 2, animals were divided into 12 groups. Half, which was designed to study the effects of DA antagonists …

MaleNarcoticsMotor ActivityPharmacologyMicechemistry.chemical_compoundDopamineAnimalsMedicineNeurotransmitterBiological PsychiatrySensitizationPharmacologyRacloprideSCH-23390Morphinebusiness.industryReceptors Dopamine D1AntagonistDopamine D2 Receptor Antagonistsmedicine.anatomical_structurechemistryToxicityMorphineDopamine Antagonistsbusinessmedicine.drugProgress in Neuro-Psychopharmacology and Biological Psychiatry
researchProduct