Search results for " INFLAMMATION"

showing 10 items of 634 documents

OTUB1 inhibits CNS autoimmunity by preventing IFN-γ-induced hyperactivation of astrocytes.

2019

Astrocytes are critical regulators of neuroinflammation in multiple sclerosis (MS) and its animal model experimental autoimmune encephalomyelitis (EAE). Growing evidence indicates that ubiquitination of signaling molecules is an important cell‐intrinsic mechanism governing astrocyte function during MS and EAE. Here, we identified an upregulation of the deubiquitinase OTU domain, ubiquitin aldehyde binding 1 (OTUB1) in astrocytes during MS and EAE. Mice with astrocyte‐specific OTUB1 ablation developed more severe EAE due to increased leukocyte accumulation, proinflammatory gene transcription, and demyelination in the spinal cord as compared to control mice. OTUB1‐deficient astrocytes were hy…

Central Nervous SystemEncephalomyelitis Autoimmune ExperimentalNeuroimmunomodulationmedicine.medical_treatmentexperimental autoimmune encephalomyelitisAutoimmunityBiologymultiple sclerosisubiquitinationGeneral Biochemistry Genetics and Molecular BiologyProinflammatory cytokineneuroinflammationInterferon-gammaMice03 medical and health sciences0302 clinical medicineastrocytemedicineAnimalsMolecular BiologyCells CulturedNeuroinflammation030304 developmental biologyMice Knockout0303 health sciencesGeneral Immunology and MicrobiologySuppressor of cytokine signaling 1General NeuroscienceExperimental autoimmune encephalomyelitisArticlesmedicine.disease3. Good healthCell biologyMice Inbred C57BLCysteine EndopeptidasesCytokinemedicine.anatomical_structureAnimals NewbornAstrocytesSTAT proteinOTUB1FemaleNeurogenic InflammationJanus kinase030217 neurology & neurosurgeryAstrocyte
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Understanding the Role of T Cells in CNS Homeostasis.

2015

T cells within the central nervous system (CNS) have been generally considered pathogenic, especially in the context of neuroinflammatory disease. However, recent findings have revealed varied functions for T cells in the healthy CNS, as well as more complex roles for these cells in infection and injury than previously appreciated. Here we review evidence indicating important roles for different T cell subsets in the maintenance of CNS homeostasis. We examine the contribution of T cells in limiting inflammation and damage upon CNS injury, infection, and in neurodegeneration, and discuss the current understanding of the cellular and molecular mechanisms involved. Insight into these processes…

Central Nervous SystemT cellT-LymphocytesImmunologyCentral nervous systemContext (language use)InflammationDiseaseBiologyLymphocyte Depletion03 medical and health sciences0302 clinical medicineT-Lymphocyte SubsetsmedicineImmunology and AllergyAnimalsHomeostasisHumansNeurodegenerationmedicine.diseaseCns injurymedicine.anatomical_structureImmunologymedicine.symptomNeurogenic Inflammation030217 neurology & neurosurgeryHomeostasis030215 immunologyTrends in immunology
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Complex regional pain syndrome–up-to-date

2017

The pathophysiology of complex regional pain syndromes includes inflammation and central reorganisation. The treatment should be adjusted to the prevailing pathophysiology including possible psychosocial factors.

Central reorganisationmedicine.medical_specialtyMovement disorderslcsh:RD78.3-87.303 medical and health sciences0302 clinical medicinemedicineKetamine030212 general & internal medicineIntensive care medicinebusiness.industryChronic painPAIN Clinical Updates14Sensory lossmedicine.diseaseComplex regional pain syndrome3. Good healthTreatmentAnesthesiology and Pain MedicineNociceptionComplex regional pain syndromePosttraumatic inflammationlcsh:AnesthesiologyHyperalgesiaNeuroplasticitymedicine.symptombusinessPsychosocial030217 neurology & neurosurgerymedicine.drugPAIN Reports
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Inhibitory effects of N-acetylcysteine on the functional responses of human eosinophils in vitro

2007

Background Oxidative stress appears to be relevant in the pathogenesis of inflammation in allergic diseases like bronchial asthma. Eosinophils are oxidant-sensitive cells considered as key effectors in allergic inflammation. Objective The aim of this work was to study the effects of the clinically used antioxidant N-acetyl-L-cysteine (NAC) on the functional responses of human-isolated eosinophils. Methods Human eosinophils were purified from the blood of healthy donors by a magnetic bead separation system. The effects of NAC were investigated on the generation of reactive oxygen species (chemiluminescence and flow cytometry), Ca2+ signal (fluorimetry), intracellular glutathione (GSH; flow c…

Chemokine CCL11EotaxinLuminescenceImmunologyhuman eosinophilsBiologyPharmacologymedicine.disease_causeTranslocation GeneticAllergic inflammationAcetylcysteinechemistry.chemical_compoundmedicineHumansImmunology and Allergychemistry.chemical_classificationreactive oxygen speciesEosinophil cationic proteinReactive oxygen speciesCell DeathEosinophil Cationic ProteinNADPH OxidasesFree Radical ScavengersGlutathioneEosinophilPhosphoproteinsGlutathioneN-acetylcysteineAcetylcysteineEosinophilsN-Formylmethionine Leucyl-Phenylalaninemedicine.anatomical_structurechemistryBiochemistryChemokines CCeosinophil cationic proteinCalciumReactive Oxygen SpeciesOxidative stressp47(phox)medicine.drugp67(phox)
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Changes in Chemokines and Chemokine Receptors Expression in a Mouse Model of Alzheimer's Disease

2018

The amyloid precursor protein plus presenilin-1 (APP/PS1) mice are a frequently-used model for Alzheimer's disease studies (AD). However, the data relevant to which proteins are involved in inflammatory mechanism are not sufficiently well-studied using the AD mouse model. Using behavioral studies, quantitative RT-PCR and Western-blot techniques, significant findings were determined by the expression of proteins involved in inflammation comparing APP/PS1 and Wild type mice. Increased GFAP expression could be associated with the elevation in number of reactive astrocytes. IL-3 is involved in inflammation and ABDF1 intervenes normally in the transport across cell membranes and both were found …

ChemokineCCL3CCL1CCR8BiologyApplied Microbiology and BiotechnologyReceptors CCR8Mice03 medical and health sciencesChemokine receptorAlzheimer DiseaseGlial Fibrillary Acidic Proteinmental disordersmedicineAmyloid precursor proteinAnimalsChemokine CCL4Molecular BiologyEcology Evolution Behavior and SystematicsChemokine CCL3030304 developmental biologyInflammation0303 health scienceschemokine receptors chemotaxis inflammation behaviorHand StrengthChemotaxisChemotaxisCell BiologyAlzheimer's diseaseCell biologyGliosisbiology.proteinReceptors ChemokineChemokinesmedicine.symptomResearch PaperDevelopmental BiologyInternational Journal of Biological Sciences
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Tuning inflammation in tuberculosis: the role of decoy receptors

2009

Decoy receptors are "silent scavengers" of CC chemokines and cytokines, which play a key role in damping inflammation and tissue damage. In this review we discuss on recent findings demonstrating that these receptors set the balance between antimicrobial resistance, immune activation and inflammatory response in Mycobacterium tuberculosis infection.

ChemokineDecoy receptormedicine.medical_treatmentImmunologyInflammationMycobacterium tuberculosiImmunopathologyMicrobiologyMycobacterium tuberculosisMiceImmune systemmedicineAnimalsHumansTuberculosisDecoy receptorsReceptors CytokineReceptorCytokineDecoy receptors; TIR8/SIGIRR; D6; Mycobacterium tuberculosis; Cytokines; Chemokines; Immunopathology; InflammationInflammationSettore MED/04 - Patologia GeneraleAntiinfective agentbiologyMycobacterium tuberculosisbiology.organism_classificationTIR8/SIGIRRInfectious DiseasesCytokineChemokineImmunologybiology.proteinmedicine.symptomD6
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Atherosclerosis as an inflammatory disease.

2011

In many ways, atherosclerosis is a chronic inflammatory disorder and this issue is confirmed by recent investigations of that have focused on inflammation, providing new insight into mechanisms of disease. Several recent studies have addressed the role of chemokines in leukocyte accumulation in atherosclerosis, extending our knowledge and understanding of the complex and cell type-specific functions of chemokines in atherosclerosis. Activated T-lymphocytes within the atherosclerotic vessel wall express the CD40 ligand surface molecule, known to play a major role in several immunological pathways. In addition to activated T-lymphocytes, functional CD40 and CD40L are coexpressed by human vasc…

ChemokineSettore MED/09 - Medicina InternaEndotheliumT-LymphocytesInflammationDrug DiscoverymedicineLeukocytesAnimalsHumansCD154PharmacologyInflammationCD40Innate immune systembiologyCell adhesion moleculeMacrophagesAtherosclerosis inflammationAcquired immune systemAtherosclerosisPlaque Atheroscleroticmedicine.anatomical_structureDrug DesignImmunologybiology.proteinCancer researchCytokinesEndothelium Vascularmedicine.symptomChemokinesCurrent pharmaceutical design
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Role of the chemokine decoy receptor D6 in balancing inflammation, immune activation, and antimicrobial resistance in Mycobacterium tuberculosis infe…

2008

D6 is a decoy and scavenger receptor for inflammatory CC chemokines. D6-deficient mice were rapidly killed by intranasal administration of low doses of Mycobacterium tuberculosis. The death of D6(-/-) mice was associated with a dramatic local and systemic inflammatory response with levels of M. tuberculosis colony-forming units similar to control D6-proficient mice. D6-deficient mice showed an increased numbers of mononuclear cells (macrophages, dendritic cells, and CD4 and CD8 T lymphocytes) infiltrating inflamed tissues and lymph nodes, as well as abnormal increased concentrations of CC chemokines (CCL2, CCL3, CCL4, and CCL5) and proinflammatory cytokines (tumor necrosis factor alpha, int…

Chemokinedecoy receptor inflammation Mycobacterium tuberculosis infectionmedicine.medical_treatmentInterleukin-1betaImmunologyMice TransgenicInflammationReceptors CCR10BiologyModels BiologicalArticleCCL5Proinflammatory cytokineInterferon-gammaMiceImmune systemAnti-Infective AgentsDrug Resistance BacterialmedicineAnimalsImmunology and AllergyInterferon gammaInflammationTumor Necrosis Factor-alphaArticlesMycobacterium tuberculosisPhenotypeCytokineImmune SystemImmunologybiology.proteinTumor necrosis factor alphaLymph Nodesmedicine.symptommedicine.drug
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Immunological Aspects of Von Hippel-Lindau Disease: A Focus on Neuro-Oncology and Myasthenia Gravis

2022

Von Hippel-Lindau (VHL) disease is an autosomal dominant condition that predisposes affected individuals to a variety of malignant and benign neoplasms. The pathogenetic turning point of this illness is the accumulation of hypoxia-inducible factor (HIF)-1α, a transcription factor of several genes involved in oncogenesis, angiogenesis, tissue regeneration, metabolic regulation, hematopoiesis, and inflammatory responses. From an oncological perspective, increased awareness of the molecular pathways underlying this disease is bringing us closer to the development of specific and targeted therapies. Meanwhile, on the surgical side, improved understanding can help to better identify the patients…

Clinical BiochemistryAch receptors VHL disease VHL/HIF axis belzutifan immune system myasthenia gravis inflammation pazopanib
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Evaluation of intraocular pressure and other biomechanical parameters to distinguish between subclinical keratoconus and healthy corneas

2021

(1) Purpose: To assess the main corneal response differences between normal and subclinical keratoconus (SCKC) with a Corvis® ST device. (2) Material and Methods: We selected 183 eyes of normal patients, of a mean age of 33 ± 9 years and 16 eyes of patients with SCKC of a similar mean age. We measured best corrected visual acuity (BCVA) and corneal topography with a Pentacam HD device to select the SCKC group. Biomechanical measurements were performed using the Corvis® ST device. We carried out a non-parametric analysis of the data with SPSS software (Wilcoxon signed rank-test). (3) Results: We found statistically significant differences between the control and SCKC groups in some corneal b…

Corvis® STmedicine.medical_specialtyKeratoconusIntraocular pressureWilcoxon signed-rank testgenetic structuresCorvis<sup>®</sup> STArticle03 medical and health sciencesocular inflammation0302 clinical medicineDiscriminant function analysisOphthalmologymedicine030304 developmental biologySubclinical infectionBest corrected visual acuity0303 health sciencesmedicine.diagnostic_testcornea biomechanicsbusiness.industryRMean ageGeneral Medicinemedicine.diseaseCorneal topographyeye diseasessubclinical keratoconus030221 ophthalmology & optometryMedicinesense organsbusinessintraocular pressure
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