Search results for " Immediate-Early"

showing 10 items of 16 documents

Patchwork Pattern of Transcriptional Reactivation in the Lungs Indicates Sequential Checkpoints in the Transition from Murine Cytomegalovirus Latency…

1999

The lungs are a relevant organ site of primary and recurrent human cytomegalovirus (hCMV) disease (for overviews, see references 21, 22, 31, 34, 39, and 44). Murine CMV (mCMV) can serve us as a model for studying CMV pneumonia in acute infection (6, 27, 33, 37) as well as for studying viral latency, reactivation, and recurrence in the lungs (2, 17, 18, 42, 43). We have shown recently that transcription from the major immediate-early (MIE) transcription unit ie1-ie3 (hereafter referred to as ie1/3), which is driven by a strong MIE promoter-enhancer (MIEPE) (3), occurs during pulmonary latency of mCMV but fails to initiate the productive cycle (17). Notably, the paralogous MIEPE of hCMV can f…

Gene Expression Regulation ViralTranscriptional ActivationHuman cytomegalovirusvirusesImmunologyCytomegalovirusReplicationBiologyMicrobiologyMiceTransactivationTranscription (biology)VirologyGene expressionVirus latencymedicineAnimalsEnhancerGenes Immediate-EarlyLungTranscription factorMice Inbred BALB CEffectormedicine.diseaseVirologyVirus LatencyInsect ScienceCytomegalovirus InfectionsFemaleVirus ActivationTranscription FactorsJournal of Virology
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Murine Cytomegalovirus Major Immediate-Early Enhancer Region Operating as a Genetic Switch in Bidirectional Gene Pair Transcription

2007

ABSTRACT Enhancers are defined as DNA elements that increase transcription when placed in any orientation relative to a promoter. The major immediate-early (MIE) enhancer region of murine cytomegalovirus is flanked by transcription units ie1/3 and ie2 , which are transcribed in opposite directions. We have addressed the fundamental mechanistic question of whether the enhancer synchronizes transcription of the bidirectional gene pair (synchronizer model) or whether it operates as a genetic switch, enhancing transcription of either gene in a stochastic alternation (switch model). Clonal analysis of cytokine-triggered, transcription factor-mediated MIE gene expression from latent viral genomes…

GeneticsMice Inbred BALB CBase SequenceTranscription GeneticGeneral transcription factorImmunologyResponse elementCytomegalovirusEnhancer RNAsE-boxPromoterBiologyMicrobiologyGenome Replication and Regulation of Viral Gene ExpressionMiceEnhancer Elements GeneticVirologyInsect ScienceTAF2AnimalsEnhancer trapEnhancerGenes Immediate-EarlyDNA PrimersJournal of Virology
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The Complex Regulatory Role of Cytomegalovirus Nuclear Egress Protein pUL50 in the Production of Infectious Virus

2021

The regulation of the nucleocytoplasmic release of herpesviral capsids is defined by the process of nuclear egress. Due to their large size, nuclear capsids are unable to traverse via nuclear pores, so that herpesviruses evolved to develop a vesicular transport pathway mediating their transition through both leaflets of the nuclear membrane. This process involves regulatory proteins, which support the local distortion of the nuclear envelope. For human cytomegalovirus (HCMV), the nuclear egress complex (NEC) is determined by the pUL50-pUL53 core that initiates multicomponent assembly with NEC-associated proteins and capsids. Hereby, pUL50 serves as a multi-interacting determinant that recru…

Human cytomegalovirusGene Expression Regulation ViralProteomicsefficiency of infectious virus productionQH301-705.5Nuclear Envelope[SDV]Life Sciences [q-bio]virusesQuantitative proteomicsCytomegalovirusconditional expressionGenome Viralnuclear egress complex (NEC)Virus ReplicationArticleCell LineViral ProteinsCapsidNEC protein pUL50DNA PackagingmedicineHumansddc:610Biology (General)Nuclear poreNuclear membraneregulation of viral replicationGenes Immediate-EarlyCell Nucleusfunctional propertiesChemistryVirionGeneral MedicineFibroblastsmedicine.diseaseCell biologyVesicular transport protein[SDV] Life Sciences [q-bio]Kineticsmedicine.anatomical_structureLytic cycleCapsidhuman cytomegalovirusLamin
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In Vivo Replication of Recombinant Murine Cytomegalovirus Driven by the Paralogous Major Immediate-Early Promoter-Enhancer of Human Cytomegalovirus

1999

ABSTRACT Transcription of the major immediate-early (MIE) genes of cytomegaloviruses (CMV) is driven by a strong promoter-enhancer (MIEPE) complex. Transactivator proteins encoded by these MIE genes are essential for productive infection. Accordingly, the MIEPE is a crucial control point, and its regulation by activators and repressors is pertinent to virus replication. Since the MIEPE contains multiple regulatory elements, it was reasonable to assume that specific sequence motifs are irreplaceable for specifying the cell-type tropism and replication pattern. Recent work on murine CMV infectivity (A. Angulo, M. Messerle, U. H. Koszinowski, and P. Ghazal, J. Virol. 72:8502–8509, 1998) has do…

Human cytomegalovirusImmunologyReplicationCytomegalovirusBiologyVirus ReplicationRecombinant virusMicrobiologyMiceVirologymedicineAnimalsPromoter Regions GeneticEnhancerGenes Immediate-EarlyGeneIn Situ HybridizationTropismRecombination GeneticInfectivityMice Inbred BALB CPromotermedicine.diseaseVirologyEnhancer Elements GeneticLiverViral replicationInsect ScienceFemaleJournal of Virology
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Focal Transcriptional Activity of Murine Cytomegalovirus during Latency in the Lungs

1999

ABSTRACT Interstitial pneumonia is a frequent and critical manifestation of human cytomegalovirus (CMV) disease in immunocompromised patients, in particular in recipients of bone marrow transplantation. Previous work in the murine CMV infection model has identified the lungs as a major organ site of CMV latency and recurrence. It was open to question whether the viral genome is transcriptionally silent or active during latency. Transcription could be latency associated and thus be part of the latency phenotype. Alternatively, transcriptional activity could reflect episodes of reactivation. We demonstrate here that transcription of the immediate-early (IE) transcription unit ie1-ie3 selectiv…

Human cytomegalovirusMaleMuromegalovirusTranscription GeneticRNA SplicingImmunologyReplicationBiologyMicrobiologyTransactivationExonMiceMuromegalovirusTranscription (biology)Bone MarrowRecurrenceVirologyVirus latencyGene expressionmedicineAnimalsGeneGenes Immediate-EarlyLungExonsmedicine.diseasebiology.organism_classificationVirologyVirus LatencyInsect ScienceImmunologyDNA ViralFemale
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Tropism of human cytomegalovirus for endothelial cells is determined by a post-entry step dependent on efficient translocation to the nucleus.

2000

Marked interstrain differences in the endothelial cell (EC) tropism of human cytomegalovirus (HCMV) isolates have been described. This study aimed to define the step during the replicative cycle of HCMV that determines this phenotype. The infection efficiency of various HCMV strains in EC versus fibroblasts was quantified by immunodetection of immediate early (IE), early and late viral antigens. Adsorption and penetration were analysed by radiolabelled virus binding assays and competitive HCMV-DNA-PCR. The translocation of penetrated viral DNA to the nucleus of infected cells was quantified by competitive HCMV-DNA-PCR in pure nuclear fractions. The intracytoplasmic translocation of capsids …

Human cytomegalovirusUmbilical VeinsvirusesBlotting WesternActive Transport Cell NucleusCytomegalovirusChromosomal translocationBiologyAntibodies ViralTransfectionVirus ReplicationVirusImmediate-Early ProteinsViral ProteinsViral Envelope ProteinsViral entryVirologyGene expressionmedicineHumansEndotheliumPromoter Regions GeneticAntigens ViralGenes Immediate-EarlyTropismCells CulturedCell NucleusMembrane GlycoproteinsAntibodies MonoclonalGenetic VariationFibroblastsmedicine.diseaseVirologyMolecular biologyCell nucleusMicroscopy Electronmedicine.anatomical_structureOrgan SpecificityDNA ViralTrans-ActivatorsAdsorptionImmunostainingThe Journal of general virology
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Random, asynchronous, and asymmetric transcriptional activity of enhancer-flanking major immediate-early genes ie1/3 and ie2 during murine cytomegalo…

2001

ABSTRACT The lungs are a major organ site of cytomegalovirus (CMV) pathogenesis, latency, and recurrence. Previous work on murine CMV latency has documented a high load and an even distribution of viral genomes in the lungs after the resolution of productive infection. Initiation of the productive cycle requires expression of the ie1/3 transcription unit, which is driven by the immediate-early (IE) promoter P 1/3 and generates IE1 and IE3 transcripts by differential splicing. Latency is molecularly defined by the absence of IE3 transcripts specifying the essential transactivator protein IE3. In contrast, IE1 transcripts were found to be generated focally and randomly, reflecting sporadic P …

Lung DiseasesMuromegalovirusTranscription GeneticvirusesImmunologyReplicationEnhancer RNAsBiologyMicrobiologyImmediate early proteinImmediate-Early ProteinsTransactivationMiceViral ProteinsViral Envelope ProteinsTranscription (biology)VirologyVirus latencymedicineAnimalsEnhancerTranscription factorGenes Immediate-EarlyLungGeneticsMice Inbred BALB CMembrane Glycoproteinsvirus diseasesHerpesviridae Infectionsmedicine.diseaseUpstream EnhancerVirus LatencyEnhancer Elements GeneticInsect ScienceTrans-ActivatorsFemaleJournal of virology
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Measles virus enhances the expression of cellular immediate-early genes and DNA-binding of transcription factor AP-1 in lung epithelial A549 cells.

2002

In this work we investigated the effect of measles virus (MV) infection on the expression of immediate-early genes junB, c-jun and c-fos mRNA as well as AP-1 DNA-binding activity in the lung epithelial-like adenocarcinoma cell line A549. The transcription factor AP-1, which is a group of dimeric complexes of the Fos and Jun family proteins, is an important regulator in many cellular responses to different extracellular stimuli. Membrane cofactor protein CD46, which acts as a receptor for laboratory-adapted and vaccine strains of MV, has been reported to associate with beta1 integrin molecules, which are known to trigger signaling events and activate immediate-early genes. The expression of …

Lung NeoplasmsJUNBBiologyMeasles virusMembrane Cofactor Protein03 medical and health sciencesAntigens CDVirologyGene expressionTumor Cells CulturedHumansMononegaviralesTranscription factorGenes Immediate-Early030304 developmental biologyA549 cell0303 health sciencesMembrane GlycoproteinsCD46Interleukin-6030302 biochemistry & molecular biologyGeneral MedicineDNAbiology.organism_classificationVirology3. Good healthTranscription Factor AP-1Gene Expression RegulationMeasles virusImmediate early geneArchives of virology
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CB1 Cannabinoid Receptors and On-Demand Defense Against Excitotoxicity

2003

Abnormally high spiking activity can damage neurons. Signaling systems to protect neurons from the consequences of abnormal discharge activity have been postulated. We generated conditional mutant mice that lack expression of the cannabinoid receptor type 1 in principal forebrain neurons but not in adjacent inhibitory interneurons. In mutant mice,the excitotoxin kainic acid (KA) induced excessive seizures in vivo. The threshold to KA-induced neuronal excitation in vitro was severely reduced in hippocampal pyramidal neurons of mutants. KA administration rapidly raised hippocampal levels of anandamide and induced protective mechanisms in wild-type principal hippocampal neurons. These protecti…

MaleCannabinoid receptorReceptors Drugmedicine.medical_treatment2-ArachidonoylglycerolExcitotoxicityHippocampal formationmedicine.disease_causeHippocampusMicechemistry.chemical_compoundPiperidinesCannabinoid receptor type 1Excitatory Amino Acid AgonistsReceptors Cannabinoidgamma-Aminobutyric AcidMice KnockoutNeuronsKainic AcidMultidisciplinaryBrainEndocannabinoid systemNeuroprotective AgentsMitogen-Activated Protein KinasesRimonabantSignal Transductionmedicine.medical_specialtyKainic acidPolyunsaturated AlkamidesGlutamic AcidMice TransgenicArachidonic AcidsIn Vitro TechniquesBiologyGlyceridesProsencephalonInternal medicinemedicineAnimalsFuransGenes Immediate-EarlyEpilepsyCannabinoidsBrain-Derived Neurotrophic FactorExcitatory Postsynaptic PotentialsMice Inbred C57BLEndocrinologyGene Expression Regulationnervous systemchemistryMutationPyrazolesCannabinoidNeuroscienceEndocannabinoidsScience
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Stimulation of immediate early gene expression by desipramine in rat brain.

1997

The stimulation of immediate early gene expression in brain and neuronal cell culture systems has been reported after various experimental paradigms such as chemiconvulsant-provoked seizures or specific drug applications. In particular, the induction of immediate early genes by adrenergic model substances has been demonstrated by several investigators. This report demonstrates that a single dose of desipramine (10 or 25 mg/kg), a classical tricyclic antidepressant drug acting on the adrenergic system, induced c-fos and zif268 expression in rat hippocampus without affecting c-jun. The observed immediate early gene response might reflect part of a signal transduction cascade involved in long-…

MaleProto-Oncogene Proteins c-junAdrenergicStimulationPharmacologyBiologyAntidepressive Agents Tricyclicc-FosHippocampusPolymerase Chain ReactionImmediate-Early ProteinsRats Sprague-DawleyDesipraminemedicineAnimalsRNA MessengerGenes Immediate-EarlyBiological PsychiatryEarly Growth Response Protein 1Regulation of gene expressionBrain Chemistryc-junDesipramineStimulation ChemicalRatsDNA-Binding ProteinsGene Expression Regulationbiology.proteinLocus CoeruleusSignal transductionOligonucleotide ProbesImmediate early geneNeuroscienceProto-Oncogene Proteins c-fosmedicine.drugTranscription FactorsBiological psychiatry
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