Search results for " Linkage"

showing 10 items of 174 documents

A dominant gene for developmental dyslexia on chromosome 3.

2001

Developmental dyslexia is a neurofunctional disorder characterised by an unexpected difficulty in learning to read and write despite adequate intelligence, motivation, and education. Previous studies have suggested mostly quantitative susceptibility loci for dyslexia on chromosomes 1, 2, 6, and 15, but no genes have been identified yet. We studied a large pedigree, ascertained from 140 families considered, segregating pronounced dyslexia in an autosomal dominant fashion. Affected status and the subtype of dyslexia were determined by neuropsychological tests. A genome scan with 320 markers showed a novel dominant locus linked to dyslexia in the pericentromeric region of chromosome 3 with a m…

AdultMaleReading disabilityAdolescentLocus (genetics)Biologybehavioral disciplines and activitiesDyslexia03 medical and health sciences0302 clinical medicineGenetic linkageDCDC2Memorymental disordersGeneticsmedicineHumansChildGenetics (clinical)Finland030304 developmental biologyAgedGenes DominantGenetics0303 health sciencesAnalysis of VariancePsychological TestsRadiation Hybrid MappingReceptors Dopamine D2HaplotypeDyslexiaReceptors Dopamine D3Chromosome MappingOriginal ArticlesMiddle Agedmedicine.diseasePedigreeDevelopmental disorderChromosome 3HaplotypesReadingReceptors SerotoninFemaleChromosomes Human Pair 3Lod Score030217 neurology & neurosurgeryJournal of medical genetics
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GAA trinucleotide repeat expansion in variant Friedreich's ataxia families.

1997

Phenotypic variants in Friedreich's ataxia include late onset, preservation of the lower limbs tendon reflexes, and slow progression. We describe clinical and electrophysiological features from three families with Friedreichlike phenotypes. Friedreich's ataxia diagnosis was confirmed by finding two allelic expansions of the GAA trinucleotide repeat at the X25 gene. In family 1 both patients had a late-onset phenotype with preservation of knee and ankle jerks, lack of cardiomyopathy, and preserved H reflex. One of them did not have electrophysiologic evidence of sensory axonal neuropathy. Patients from family 2 showed variability in the age of onset, and 2 out of 3 affected children had hype…

AdultMaleReflex Stretchcongenital hereditary and neonatal diseases and abnormalitiesPathologymedicine.medical_specialtySensory axonal neuropathyAtaxiaPhysiologyGenetic LinkageAction PotentialsLate onsetBiologyH-ReflexCellular and Molecular NeuroscienceDegenerative diseaseTrinucleotide RepeatsPhysiology (medical)medicineHumansNeurons AfferentChildAllelesLegGenetic VariationDNACardiomyopathy Hypertrophicmedicine.diseasePedigreePeripheral neuropathyFriedreich AtaxiaReflexDisease ProgressionFemaleNeurology (clinical)medicine.symptomAge of onsetTrinucleotide repeat expansionMusclenerve
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Phenotypic variability in patients with generalised resistance to thyroid hormone.

1995

Genetic linkage of generalised resistance to thyroid hormone (GRTH) to the human thyroid receptor beta 1 gene has been identified. To date 38 different mutations in several kindreds have been documented. We report on a family with GRTH displaying an adenine for guanine substitution at nucleotide 1234 resulting in a threonine for alanine substitution at codon 317 of exon 9. This mutation has been described for different phenotypes, suggesting that the heterogeneity in GRTH may be the result of multiple genetic factors.

AdultMaleThyroid Hormone Resistance SyndromeThyroid Hormonesmedicine.medical_specialtyGenetic LinkageMolecular Sequence DataThyroid Function TestsBiologymedicine.disease_causeThyroid function testsGenetic HeterogeneityExonGenetic linkageInternal medicineGeneticsmedicineHumansPoint MutationAmino Acid SequenceChildGeneGenetics (clinical)GeneticsMutationReceptors Thyroid HormoneBase Sequencemedicine.diagnostic_testGenetic heterogeneityPoint mutationThyroidPedigreePhenotypemedicine.anatomical_structureEndocrinologyChild PreschoolFemaleResearch ArticleJournal of Medical Genetics
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Hereditary angioedema with normal C1-inhibitor activity in women.

2000

Summary Background Hereditary angioedema (HAE) is a well defined autosomal dominant disease (Mendelian Inheritance in Man #106100) that results from an inherited deficiency of C1 (the activated first component of complement) inhibitor function. We report an unusual variant of HAE with normal biochemical C1-inhibitor function, occurring only in women. Methods We screened 574 patients with recurrent angioedema of the skin for presence of HAE. 283 patients were selected, in whom angioedema was associated with abdominal pain attacks or recurrent life-threatening episodes of upper-airway obstruction, or both, rather than with urticaria. We measured C1-inhibitor concentration and functional activ…

AdultMaleX ChromosomeAdolescentGenetic LinkageComplement C1 Inactivator ProteinsC1-inhibitorEcallantideSex FactorsRecurrenceTerminology as TopicmedicineHumansHereditary Angioedema Type IIISex RatioFamily historyAngioedemaChildDominance (genetics)Genes DominantAngioedemabiologybusiness.industryAutosomal dominant traitComplement C4General MedicineMiddle Agedmedicine.diseaseAbdominal PainPedigreeAirway ObstructionImmunologyHereditary angioedemaMutationbiology.proteinFemalemedicine.symptombusinessmedicine.drugLancet (London, England)
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Methodological Aspects of the Application of the Naka-Rushton Equation to Clinical Electroretinogram

1993

The nonlinear relation between stimulus intensity and response amplitude of the electroretinogram (ERG) scotopic b wave can be described by a curve based on the Naka-Rushton (NR) equation. Up to now, the NR equation has been used to assess the features of the normal and pathological ERG, but the best approach for a correct evaluation of the parameters is still debatable. The parameters are thought to be related to the different conditions of retinal activities. The method is well known in experimental laboratories but is quite unusual at the clinical level. In the present paper the derivative analysis of the NR function is proposed as an easier approach to understand the variations of the N…

AdultMaleX ChromosomeAdolescentLightElectrodiagnosisGenetic LinkageVisual AcuityDark AdaptationCataractRetinaCellular and Molecular NeuroscienceExperimental LaboratoriesOpticsRetinal DiseasesElectroretinographymedicineHumansResponse AmplitudeApplied mathematicsScotopic visionChildAgedMathematicsmedicine.diagnostic_testbusiness.industryDerivative analysisGeneral MedicineMiddle AgedSensory SystemsOphthalmologyFemalebusinessRetinitis PigmentosaElectroretinographyOphthalmic Research
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The Long-Term Effects of Physical Loading and Exercise Lifestyles on Back-Related Symptoms, Disability, and Spinal Pathology Among Men

1995

Study design Historical cohort, including selected subgroups. Objectives To understand the long-term effects of exercise on back-related outcomes, back pain, sciatica, back-related hospitalizations, pensions, and magnetic resonance imaging findings were studied among former elite athletes. Summary of background data Exercise and sports participation have become increasingly popular, as have recommendations of exercises for back problems, but little is known about their long-term effects. Methods Questionnaires were returned by 937 former elite athletes and 620 control subjects (83% response rate). Identification codes allowed record linkage to hospital discharge and pension registers. Magne…

AdultMalemedicine.medical_specialtyAdolescentCohort StudiesDisability EvaluationPensionsSciaticaLumbarPhysical medicine and rehabilitationSurveys and QuestionnairesmedicineBack painHumansOrthopedics and Sports MedicineExercise physiologyExerciseLife Stylehealth care economics and organizationsAgedSciaticabiologybusiness.industryAthletesAge FactorsOdds ratioMiddle Agedequipment and suppliesbiology.organism_classificationMagnetic Resonance ImagingSpineHospitalizationPhysical therapyNeurology (clinical)medicine.symptombusinessLow Back Painhuman activitiesRecord linkageSportsCohort studySpine
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The reliability of the SADS-LA in a family study setting

1991

The joint-rater and test-retest reliability study of two translated versions of the SADS-LA (Schedule for Affective Disorders and Schizophrenia--Lifetime version--modified for the study of anxiety disorders), one in French and the other in German, have been tested in family study settings, in a sample of patients and first-degree relatives. The test-retest reliability study demonstrated that identification of major affective disorders and schizophrenia was performed with sufficient reliability; however, diagnoses of subtypes of major disorders (e.g. bipolar II disorder) and identification of minor disorders was less reliable. The implications of these findings in phenotype identification du…

AdultMalemedicine.medical_specialtyBipolar DisorderPsychometricsGenetic Linkagebehavioral disciplines and activitiesFamily studiesBipolar II disorderPrevalence of mental disordersmedicineHumansPharmacology (medical)Medical diagnosisPsychiatryBiological PsychiatryReliability (statistics)Psychiatric Status Rating ScalesDepressive DisorderReproducibility of ResultsGeneral Medicinemedicine.diseasePsychiatry and Mental healthPsychotic DisordersSchizophreniaStructured interviewSchizophreniaAnxietyFemaleSchizophrenic Psychologymedicine.symptomPsychologyFollow-Up StudiesClinical psychologyEuropean Archives of Psychiatry and Clinical Neuroscience
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Personality disorders among the relatives of schizophrenia patients.

1994

In light of current linkage studies in schizophrenia, research on the "schizophrenia spectrum" deserves increased attention for an exact determination of the affected phenotype: Those disorders that have a much higher prevalence among biological relatives of schizophrenia patients are supposed to share common etiological factors with "core" schizophrenia. However, there is controversy over which of the DSM-III-R personality disorders should be included in the spectrum. In a controlled family study of inpatients with a DSM-III-R diagnosis of schizophrenia (n = 101), schizophreniform and schizoaffective disorders (n = 69), and unipolar major depression (n = 160), familial rates of personality…

AdultMalemedicine.medical_specialtyGenetic Linkagemedia_common.quotation_subjectPersonality Assessmentbehavioral disciplines and activitiesPersonality DisordersSchizotypal Personality DisorderSchizoid personality disorderRisk Factorsmental disordersmedicinePersonalityHumansPsychiatrymedia_commonPsychiatric Status Rating ScalesDepressive DisorderModels Geneticmedicine.diseaseSchizotypal personality disorderPersonality disordersPsychiatry and Mental healthPhenotypePsychotic DisordersSchizophreniaParanoid personality disorderSchizophreniaFemaleSchizophrenic PsychologyPersonality Assessment InventoryPsychologyDiagnosis of schizophreniaClinical psychologySchizophrenia bulletin
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A Cohort Study of Childhood Cancer Incidence after Postnatal Diagnostic X-Ray Exposure

2009

Ionizing radiation is an established cause of cancer, yet little is known about the health effects of doses from diagnostic examinations in children. The risk of childhood cancer was studied in a cohort of 92.957 children who had been examined with diagnostic X rays in a large German hospital during 1976-2003. Radiation doses were reconstructed using the individual dose area product and other exposure parameters, together with conversion coefficients developed specifically for the medical devices and standards used at the radiology department. Newly diagnosed cancers occurring between 1980 and 2006 were determined through record linkage to the German Childhood Cancer Registry. The median ra…

AdultMalemedicine.medical_specialtyPediatricsNeoplasms Radiation-InducedAdolescentBiophysicsCohort StudiesGermanyNeoplasmsRadiation IonizingEpidemiologymedicineHumansRadiology Nuclear Medicine and imagingRegistriesChildChildhood Cancer RegistryRadiationbusiness.industryIncidenceX-RaysIncidence (epidemiology)InfantCancermedicine.diseaseLeukemia2nd malignant neoplasms; ionizing-radiation; computed-tomography; ultrasound exposure; young-children; risk-factors; in-utero; survivors; leukemia; irradiationChild PreschoolMultivariate AnalysisCohortFemalebusinessRecord linkageCohort studyRadiation Research
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IL 10.G microsatellites mark promoter haplotypes associated with protection against the development of reactive arthritis in Finnish patients.

2001

Objective To investigate the association of microsatellites and single-nucleotide promoter polymorphisms (SNPs) in the gene for the cytokine interleukin-10 (IL-10) with susceptibility to and outcome of reactive arthritis (ReA). Methods From genomic DNA, IL-10 microsatellites G and R and IL-10 promoter polymorphisms at positions −1087 and −524 were typed by polymerase chain reaction, automated fragment length analysis, and restriction fragment digestion in 85 Finnish patients with ReA and 62 HLA–B27–positive Finnish controls. ReA patients had been followed up for 20 years. Genotypes and haplotypes of IL-10 were correlated with distinct features of the disease course, such as triggering agent…

AdultMalemusculoskeletal diseasesGenetic LinkageImmunologySingle-nucleotide polymorphismArthritis ReactivePolymorphism Single NucleotideRestriction fragmentRheumatologyProhibitinsGenotypeHumansImmunology and AllergyPharmacology (medical)AllelePromoter Regions GeneticAllele frequencyFinlandGeneticsbiologyreactive arthritis IL-10 microsatellites polymorphismHaplotypeMiddle AgedInterleukin-10HaplotypesImmunologybiology.proteinMicrosatelliteFemaleRestriction fragment length polymorphismFollow-Up StudiesMicrosatellite Repeats
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