Search results for " Ly"

showing 10 items of 2487 documents

T lymphocyte-stimulating microbial toxins as ?superantigens?

1991

Infectious pathogens generally have to cope with the host's adaptive immune system, i.e., T and B lymphocytes. Common evasion mechanisms in this complex interaction are antigenic variations, the escape to immunologically priviledged sites or the use of immunosuppressive mechanisms. Many bacteria and other microorganisms eleborate soluble factors or toxins that act suppressively on cells of the immune system, such as pore-forming molecules or proteins that interfere with the function of G proteins. Gram-positive cocci and a mycoplasma have developed an extremely potent mechanism of T cell stimulation by closely mimicking recognition of specific antigen. From the functional similarity to anti…

Microbiology (medical)G proteinT-LymphocytesT cellBacterial ToxinsImmunologyBiologyLymphocyte Activationmedicine.disease_causeMicrobiologyMiceMycoplasmaImmune systemAntigenmedicineSuperantigenAnimalsImmunology and AllergyAntigens BacterialGeneral MedicineMycoplasmaT lymphocyteAcquired immune systemmedicine.anatomical_structureImmunologyMitogensMedical Microbiology and Immunology
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Human T helper cells reactive with somatic bacterial antigens belong to the Th1 subset

1994

The aim of this study was to characterize the cytokine secretion patterns of human T helper cells from healthy donors reactive with somatic antigens from various bacteria, the nematode Anisakis and tetanus toxoid. From the peripheral blood of four healthy donors we have established 70 T cell lines reactive with antigens from Yersinia, Salmonella, Morganella, Klebsiella, Serratia, Escherichia, Chlamydia, Shigella, Streptococcus, tetanus toxoid and Anisakis, respectively. Our results show that all T cells reactive with bacteria produce interferon-gamma (IFN-gamma) and tumor necrosis factor-alpha (TNF-alpha), but no interleukin (IL)-4 and no or very little IL-2 and IL-10 and, thus, belong to t…

Microbiology (medical)Interleukin 2T cellImmunologyBiologyCell LineMicrobiologyAntigenTetanus ToxoidmedicineAnimalsHumansImmunology and AllergyCells CulturedInterleukin 4Antigens BacterialToxoidGeneral MedicineT lymphocyteTh1 CellsAnisakisInterleukin 10medicine.anatomical_structureAntigens HelminthImmunologyCytokinesCytokine secretionmedicine.drugMedical Microbiology and Immunology
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Evaluation of the Architect Epstein-Barr Virus (EBV) Viral Capsid Antigen (VCA) IgG, VCA IgM, and EBV Nuclear Antigen 1 IgG Chemiluminescent Immunoas…

2014

ABSTRACTCommercial immunoassays for detecting IgG and IgM antibodies against Epstein-Barr virus (EBV), viral capsid antigens (VCA), and IgGs toward EBV nuclear antigen-1 (EBNA-1) are routinely used in combination to categorize EBV infection status. In this study, we evaluated the performances of the Architect EBV VCA IgG, VCA IgM, and EBNA-1 IgG chemiluminescent microparticle assays (CMIAs) in EBV serological analyses using indirect immunofluorescence assays and anticomplement immunofluorescence assays as the reference methods for VCA IgG, VCA IgM, and EBNA-1 IgG antibody detection, respectively. A total of 365 serum samples representing different EBV serological profiles were included in t…

Microbiology (medical)MaleEpstein-Barr Virus InfectionsHerpesvirus 4 HumanClinical BiochemistryImmunologyFluorescent Antibody Techniquemedicine.disease_causeImmunofluorescenceAntibodies ViralSensitivity and SpecificityImmunoglobulin GSerologyAntigenhemic and lymphatic diseasesDiagnostic Laboratory Immunologyotorhinolaryngologic diseasesImmunology and AllergyMedicineHumansChildAntigens ViralImmunoassaymedicine.diagnostic_testbiologybusiness.industryDiagnostic Tests RoutineInfantVirologyEpstein–Barr virusstomatognathic diseasesEpstein-Barr Virus Nuclear AntigensImmunoglobulin MImmunoglobulin MImmunoassayChild PreschoolImmunoglobulin GImmunologyLuminescent Measurementsbiology.proteinCapsid ProteinsFemaleAntibodybusiness
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Scrub typhus, acute respiratory distress, and hemophagocytic lymphohistiocytosis

2013

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Microbiology (medical)MalePediatricsmedicine.medical_specialtyHemophagocytic lymphohistiocytosisRespiratory Distress SyndromeScrub typhubusiness.industryScrub typhus; acute respiratory distress; hemophagocytic lymphohistiocytosis.General MedicineScrub typhusAcute respiratory distresshemophagocytic lymphohistiocytosis.Liver Failure Acutemedicine.diseaseInfectious Diseaseshemophagocytic lymphohistiocytosisScrub typhusmedicineHumansbusinessacute respiratory distreacute respiratory distressInternational Journal of Infectious Diseases
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Measles and Secondary Hemophagocytic Lymphohistiocytosis

2012

To the Editor: We found interesting the article by Lupo et al. about a case of fatal measles in an immunocompetent 29-year-old woman (Fatal measles without rash in immunocompetent adult, France; http://dx.doi.org/10.3201/eid1803.111300). Perhaps, however, the possible diagnosis of secondary hemophagocytic lymphohistiocytosis (HLH) should also have been considered in that setting. HLH is a potentially fatal hyperinflammatory syndrome characterized by histiocyte proliferation and hemophagocytosis. HLH may be inherited (i.e., primary, familial, generally occurring in infants) or may occur at any age secondary to infection, malignancy, or rheumatologic disease. Secondary HLH is determined accor…

Microbiology (medical)Secondary Hemophagocytic LymphohistiocytosisLetterEpidemiologylcsh:Medicinerashacute lymphocytic leukemiaMeaslesArticlelcsh:Infectious and parasitic diseasespancytopeniadeathhemic and lymphatic diseasesAcute lymphocytic leukemiamedicineHumanspneumoniaviruseslcsh:RC109-216Letters to the EditorHistiocyteCytopeniahemophagocytosisbusiness.industrylcsh:RExanthemaacute respiratory distress syndromemedicine.diseasesecondary hemophagocytic lymphohistiocytosisPancytopeniaPneumoniaInfectious DiseasesImmunologyFemaleFranceHemophagocytosisbusinessMeaslesEmerging Infectious Diseases
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Recombinant epidermolytic (exfoliative) toxin A of Staphylococcus aureus is not a superantigen

1992

The epidermolytic (exfoliative) toxins produced by Staphylococcus aureus cause epidermolysis and skin blistering. In addition, they have been implicated to belong to the group of T lymphocyte stimulating molecules known as "superantigens". Here we show that recombinant epidermolytic toxin A produced in S. aureus is not mitogenic for human and murine T lymphocytes. We discuss the possibility that minute contaminations of highly mitogenic exoproteins may cause the mitogenicity in several proteins that are reported to be superantigens.

Microbiology (medical)Staphylococcus aureusT-LymphocytesBlotting WesternImmunologyClostridium difficile toxin ABiologyLymphocyte Activationmedicine.disease_causeMonocytesMicrobiologylaw.inventionMicelawSuperantigenmedicineAnimalsHumansImmunology and AllergyCloning MolecularStaphylococcus aureus delta toxinCells CulturedAntigens BacterialMice Inbred BALB CToxinGeneral MedicineT lymphocyteRecombinant ProteinsExfoliatinsCytolysisStaphylococcus aureusRecombinant DNAInterleukin-2SpleenMedical Microbiology and Immunology
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Polymorphisms within the TNFSF4 and mapkapk2 loci influence the risk of developing invasive aspergillosis: A two-stage case control study in the cont…

2020

Here, we assessed whether 36 single nucleotide polymorphisms (SNPs) within the TNFSF4 and MAPKAPK2 loci influence the risk of developing invasive aspergillosis (IA). We conducted a twostage case control study including 911 high-risk patients diagnosed with hematological malignancies that were ascertained through the aspBIOmics consortium. The meta-analysis of the discovery and replication populations revealed that carriers of the TNFSF4rs7526628T/T genotype had a significantly increased risk of developing IA (p = 0.00022). We also found that carriers of the TNFSF4rs7526628T allele showed decreased serum levels of TNFSF14 protein (p = 0.0027), and that their macrophages had a decreased fungi…

Microbiology (medical)Thymic stromal lymphopoietinCiências Médicas::Ciências da Saúde:Ciências da Saúde [Ciências Médicas]lnfectious Diseases and Global Health Radboud Institute for Molecular Life Sciences [Radboudumc 4]Context (language use)Single-nucleotide polymorphismPlant ScienceCD38BiologyMonocytes03 medical and health sciencesAll institutes and research themes of the Radboud University Medical Center0302 clinical medicineGenotypeB cells; MAPKAPK2; TNFSF14; TNFSF4; TSLP; genetic susceptibility; invasive aspergillosis; monocytes; serum biomarkersB cells; Genetic susceptibility; Invasive aspergillosis; MAPKAPK2; Monocytes; Serum biomarkers; TNFSF14; TNFSF4; TSLPGenetic predispositionGenetic susceptibilityddc:610Allelelcsh:QH301-705.5Ecology Evolution Behavior and Systematics030304 developmental biology0303 health sciencesB cellsTNFSF14Science & TechnologyTNFSF4Case-control studyMAPKAPK2Serum biomarkers<i>TNFSF4</i>3. Good healthSettore MED/15 - MALATTIE DEL SANGUE<i>MAPKAPK2</i>lcsh:Biology (General)TSLPImmunologyInvasive aspergillosis030215 immunology
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The Expression and Prognostic Significance of VEGF and CXCR4 in Gastric Cancer: Correlation with Angiogenesis, Lymphangiogenesis and Progression

2022

The cellular response to hypoxia includes the expression of hypoxia-inducible factor-1 (HIF-1) and its target genes: vascular endothelial growth factor (VEGF) and CXC chemokine receptor 4 (CXCR4). The aim of this study was to investigate the expression and prognostic significance of VEGF and CXCR4, which are responsible for angiogenesis and progression in gastric cancer. Twenty-eight gastric cancer patients were analyzed. The mRNA expression was examined in primary tumors and corresponding normal gastric mucosa by RT-PCR. The protein level was examined by immunohistochemistry staining. The high expression of VEGF and CXCR4 was found in 71.0 and 64.0% of tumors, respectively. The mean levels…

Microbiology (medical)gastric cancer; VEGF; CXCR4; angiogenesis; lymphangiogenesis; lymph node metastasesGeneral MedicineMolecular BiologyMicrobiologyCurrent Issues in Molecular Biology
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Molecular signature of Epstein Barr virus-positive Burkitt lymphoma and post-transplant lymphoproliferative disorder suggest different roles for Epst…

2014

Epstein Barr virus (EBV) infection is commonly associated with human cancer and, in particular, with lymphoid malignancies. Although the precise role of the virus in the pathogenesis of different lymphomas is largely unknown, it is well recognized that the expression of viral latent proteins and miRNA can contribute to its pathogenetic role. In this study, we compared the gene and miRNA expression profile of two EBV-associated aggressive B non-Hodgkin lymphomas known to be characterized by differential expression of the viral latent proteins aiming to dissect the possible different contribution of such proteins and EBV-encoded miRNAs. By applying extensive bioinformatic inferring and an exp…

Microbiology (medical)lcsh:QR1-502Epstein Barr Virupost-transplant lymphoproliferative disorderBiologyEpstein Barr Virusmedicine.disease_causeMicrobiologylcsh:MicrobiologyVirusPost-transplant lymphoproliferative disorderhemic and lymphatic diseasesGene expressionmicroRNAmedicinegene expression profilingOriginal Research ArticleBurkitt lymphoma; Epstein Barr Virus; MicroRNA; gene expression profiling; latency; post-transplant lymphoproliferative disorderlatencyBurkitt lymphomaEpstein-Barr Virus PositiveMicroRNAmedicine.diseaseEpstein–Barr virusLymphomaGene expression profilingBurkitt lymphoma; Epstein barr virus; Gene expression profiling; Latency; microRNA; Post-transplant lymphoproliferative disorder; Microbiology; Microbiology (medical)ImmunologyBurkitt lymphoma Epstein Barr Virus MicroRNA gene expression profiling latency post-transplant lymphoproliferative disorder
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Visceral leishmaniasis, hypertriglyceridemia and secondary hemophagocytic lymphohistiocytosis

2016

cytokines, an upregulation of adhesion molecules and MHC I and II molecules on mono/macrophages, and an expansion of inflammatory monocytes. This exaggerated inflammatory response is responsible for necrosis and organ failure and results in uncontrolled proliferation and phagocytic activity of histiocytes [2]. Hypertriglyceridemia (fasting, greater than or equal to 265 mg/100 ml) is one of the current diagnostic criteria for HLH [2]. Several studies link hypertriglyceridemia to inhibition of lipoprotein lipase (LPL) by tumor necrosis factor-α (TNF-α), and TNF-α is a powerful autocrine and paracrine regulator of adipose tissue [3]. Indeed, many different sources of intense and prolonged T-ly…

Microbiology (medical); Infectious Diseases0301 basic medicineMicrobiology (medical)Secondary Hemophagocytic LymphohistiocytosisNecrosisSettore MED/17 - Malattie Infettive030106 microbiologyAdipose tissueTriglycerideSeverityLymphohistiocytosis Hemophagocytic03 medical and health sciences0302 clinical medicinemedicineHumansMacrophage030212 general & internal medicineVisceral leishmaniasisHypertriglyceridemiaLipoprotein lipasebusiness.industryAnemia; Severity; Triglyceride; Visceral leishmaniasisHypertriglyceridemiaAnemiaGeneral Medicinemedicine.diseaseInfectious DiseasesVisceral leishmaniasisImmunologyLeishmaniasis VisceralTumor necrosis factor alphamedicine.symptombusinessInfection
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