Search results for " MHC Class I"

showing 10 items of 50 documents

Axolotl MHC architecture and polymorphism.

1999

The MHC of the urodele amphibian Ambystoma mexicanum consists of multiple polymorphic class I loci linked, so far as yet known, to a single class II B locus. This architecture is very different from that of the anuran amphibian Xenopus. The number of class I loci in the axolotl can vary from 6 to 21 according to the haplotypes as shown by cDNA analysis and Southern blot studies in families. These loci can be classified into seven sequence groups with features ranging from the class Ia to the class Ib type. All individuals express genes from at least three of the seven groups, and all individuals possess the class Ia-like type.

Protein ConformationImmunologyGenes MHC Class IIMolecular Sequence DataGenes MHC Class IMajor histocompatibility complexAmbystomaEvolution MolecularMajor Histocompatibility ComplexAxolotlPolymorphism (computer science)Complementary DNAHLA-A2 AntigenImmunology and AllergyAnimalsHumansAmino Acid SequenceAmbystoma mexicanumGeneConserved SequenceSouthern blotGeneticsPolymorphism GeneticbiologyHaplotypebiology.organism_classificationBlotting Southernbiology.proteinEuropean journal of immunology
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Structure Learning in Nested Effects Models

2007

Nested Effects Models (NEMs) are a class of graphical models introduced to analyze the results of gene perturbation screens. NEMs explore noisy subset relations between the high-dimensional outputs of phenotyping studies, e.g., the effects showing in gene expression profiles or as morphological features of the perturbed cell. In this paper we expand the statistical basis of NEMs in four directions. First, we derive a new formula for the likelihood function of a NEM, which generalizes previous results for binary data. Second, we prove model identifiability under mild assumptions. Third, we show that the new formulation of the likelihood allows efficiency in traversing model space. Fourth, we…

Statistics and ProbabilityTraverseComputer scienceMolecular Networks (q-bio.MN)Genes MHC Class IIPerturbation (astronomy)Genes InsectFeature selectionQuantitative Biology - Quantitative Methods03 medical and health sciences0302 clinical medicineGeneticsAnimalsheterocyclic compoundsQuantitative Biology - Molecular NetworksGraphical modelMolecular BiologyQuantitative Methods (q-bio.QM)Oligonucleotide Array Sequence Analysis030304 developmental biologyLikelihood Functions0303 health sciencesNanoelectromechanical systemsModels StatisticalModels GeneticGene Expression ProfilingGenomicsComputational MathematicsDrosophila melanogasterPhenotypeFOS: Biological sciencesBinary dataIdentifiabilityRNA InterferenceLikelihood functionAlgorithmAlgorithms030217 neurology & neurosurgery
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The use of clonal mRNA as an antigenic format for the detection of antigen-specific T lymphocytes in IFN-gamma ELISPOT assays.

2003

Abstract Most assay systems for the quantification of antigen-specific T-cell responses in infectious, malignant and autoimmune disease depend on the peptide antigen format and are therefore restricted to known epitopes and their presenting HLA molecules. Here we tested in ELISPOT assays the application of in vitro-transcribed clonal mRNA as an alternative antigen format covering all potential epitopes of a given antigen. As model antigens, we chose pp65 of human cytomegalovirus (HCMV) and human tyrosinase (hTyr). Antigen-presenting cells (APC) were K562 cells stably transfected with single HLA class I alleles and autologous dendritic cells (DC). As effectors, we applied in vitro-generated …

T-LymphocytesImmunologyAntigen-Presenting CellsEpitopes T-LymphocyteGenes MHC Class IHuman leukocyte antigenBiologyTransfectionEpitopeImmunoenzyme TechniquesViral Matrix ProteinsInterferon-gammaAntigenmedicineImmunology and AllergyHumansInterferon gammaRNA MessengerCloning MolecularMonophenol MonooxygenaseELISPOTTransfectionT lymphocyteDendritic CellsPhosphoproteinsVirologyMolecular biologyElectroporationK562 CellsCD8medicine.drugJournal of immunological methods
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Thymoma and paraneoplastic myasthenia gravis

2010

Paraneoplastic autoimmune diseases associate occasionally with small cell lung cancers and gynecologic tumors. However, myasthenia gravis (MG) occurs in at least 30% of all patients with thymomas (usually present at MG diagnosis). These epithelial neoplasms almost always have numerous admixed maturing polyclonal T cells (thymocytes). This thymopoiesis-and export of mature CD4(+)T cells-particularly associates with MG, though there are rare/puzzling exceptions in apparently pure epithelial WHO type A thymomas. Other features potentially leading to inefficient self-tolerance induction include defective epithelial expression of the autoimmune regulator (AIRE) gene and/or of major histocompatib…

ThymomaThymomaT-LymphocytesGenes MHC Class IIImmunologyCellThymus Glandmedicine.disease_causeAutoantigensAutoimmunityhemic and lymphatic diseasesMyasthenia GravisHumansImmunology and AllergyMedicineLymphopoiesisPolyendocrinopathies AutoimmuneAutoantibodiesMHC class IIbiologybusiness.industryLymphopoiesisFOXP3Epithelial Cellsmedicine.diseaseAutoimmune regulatorMyasthenia gravismedicine.anatomical_structureImmunoglobulin GImmunologybiology.proteinbusinessParaneoplastic Syndromes Nervous SystemTranscription FactorsAutoimmunity
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Release of dendritic cells from cognate CD4 + T-cell recognition results in impaired peripheral tolerance and fatal cytotoxic T-cell mediated autoimm…

2012

Resting dendritic cells (DCs) induce tolerance of peripheral T cells that have escaped thymic negative selection and thus contribute significantly to protection against autoimmunity. We recently showed that CD4 + Foxp3 + regulatory T cells (Tregs) are important for maintaining the steady-state phenotype of DCs and their tolerizing capacity in vivo. We now provide evidence that DC activation in the absence of Tregs is a direct consequence of missing DC–Treg interactions rather than being secondary to generalized autoimmunity in Treg-less mice. We show that DCs that lack MHC class II and thus cannot make cognate interactions with CD4 + T cells are completely unable to induce peripheral CD8 +…

TransgeneGenes MHC Class IIAutoimmunityMice Transgenicchemical and pharmacologic phenomenaAdaptive ImmunityLymphocyte Activationmedicine.disease_causeT-Lymphocytes RegulatoryAutoimmunityMicemedicineAnimalsCytotoxic T cellHomeodomain ProteinsMHC class IIMultidisciplinarybiologyPeripheral ToleranceBody WeightHistological TechniquesFOXP3Peripheral tolerancehemic and immune systemsDendritic CellsBiological SciencesFlow CytometryAcquired immune systemTamoxifenImmunologybiology.proteinCD8T-Lymphocytes CytotoxicProceedings of the National Academy of Sciences
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Relationship between HLA I surface expression and different cytopathic effects produced after herpes simplex virus infection in vitro.

1992

In the present study, we investigated the effects of herpes simplex virus (HSV) infection on the expression of HLA class I antigens and beta 2-microglobulin in human fibroblasts. The mRNA abundance for HLA class I was shown to be strongly reduced after infection with HSV strains either producing cell rounding or fusion from within (FFWI), however, HLA class I expression on the surface of cells is strongly reduced only after appearance of FFWI. Using a ts mutant (ts 78R) or CyA in combination with a fusion from without (FFWO) inducing strain of HSV, this loss of HLA class I antigens is assumed to be correlated to the rearrangement of the cell membrane during the fusion process itself as a la…

biologyCellGene ExpressionGenes MHC Class IGeneral MedicineHuman leukocyte antigenbiology.organism_classificationmedicine.disease_causeBlotting NorthernVirologyHerpesviridaeVirusIn vitroHerpes simplex virusmedicine.anatomical_structureAntigenCytopathogenic Effect ViralHLA AntigensVirologyAlphaherpesvirinaemedicineHumansSimplexvirusbeta 2-MicroglobulinCells CulturedArchives of virology
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Human γδ T-Cells: From Surface Receptors to the Therapy of High-Risk Leukemias

2018

γδ T lymphocytes are potent effector cells, capable of efficiently killing tumor and leukemia cells. Their activation is mediated by γδ T-cell receptor (TCR) and by activating receptors shared with NK cells (e.g., NKG2D and DNAM-1). γδ T-cell triggering occurs upon interaction with specific ligands, including phosphoantigens (for Vγ9Vδ2 TCR), MICA-B and UL16 binding protein (for NKG2D), and PVR and Nectin-2 (for DNAM-1). They also respond to cytokines undergoing proliferation and release of cytokines/chemokines. Although at the genomic level γδ T-cells have the potential of an extraordinary TCR diversification, in tissues they display a restricted repertoire. Recent studies have identified …

lcsh:Immunologic diseases. Allergy0301 basic medicineαβ T-cellChemokineB-cell depletion; hematopoietic stem cells; HLA-haploidentical transplantation; receptors; αβ T-cell; γδ T-cellsReceptors Antigen T-Cell alpha-betaMini ReviewHLA-haploidentical transplantationImmunologyGenes MHC Class Ichemical and pharmacologic phenomenaMajor histocompatibility complexCD19Mice03 medical and health sciencesγδ T-cellsAntigenReceptorsMHC class ImedicineAnimalsHumansImmunology and AllergyIntraepithelial LymphocytesB-LymphocytesLeukemiaB-cell depletionbiologyT-cell receptorHematopoietic Stem Cell Transplantationmedicine.diseaseNKG2DKiller Cells NaturalLeukemia030104 developmental biologySettore MED/38 - PEDIATRIA GENERALE E SPECIALISTICACytomegalovirus InfectionsImmunologybiology.proteinlcsh:RC581-607Hematopoietic stem cellsFrontiers in Immunology
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Analysis of HLA-DRB1,DQA1,DQB1 haplotypes in Sardinian centenarians

2008

Some genetic determinants of longevity might reside in those polymorphisms for the immune system genes that regulate immune responses. Many longevity association studies focused their attention on HLA (the human MHC) polymorphisms, but discordant results have been obtained. Sardinians are a relatively isolate population and represent a suitable population for association studies. Some HLA-DR and DQ alleles form very stable haplotypes with a strong linkage disequilibrium. In a previous study on Sardinian centenarians we have suggested that HLA-DRB1 *15 allele might be marginally associated to longevity. HLA-DR,DQ haplotypes are in strong linkage disequilibrium and well conserved playing a ro…

musculoskeletal diseasesAgingLinkage disequilibriummedia_common.quotation_subjectGenes MHC Class IILongevityPopulationBiologyBiochemistryArticleHLA-DQ alpha-ChainsLinkage Disequilibrium03 medical and health sciences0302 clinical medicineEndocrinologyGene FrequencyHLA-DQ AntigensGeneticsHLA-DQ beta-ChainsHumansskin and connective tissue diseaseseducationMolecular BiologyHLA-DRB1Allele frequencyComputingMilieux_MISCELLANEOUS030304 developmental biologyGenetic associationmedia_commonAged 80 and overGeneticsLikelihood Functions0303 health scienceseducation.field_of_studyPolymorphism GeneticHLA-DQB1HaplotypeLongevityHLA-DR AntigensCell BiologyHaplotypesItalyHLA Longevity SardiniaMedicineHLA-DRB1 Chains030215 immunologyExperimental Gerontology
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HLA class II haplotypes differentiate between the adult autoimmune polyglandular syndrome types II and III.

2013

Background: Genetics of the adult autoimmune polyglandular syndrome (APS) is poorly understood. Aim: The aim of this study was to gain further insight into the genetics of the adult APS types. Site: The study was conducted at a university referral center. Methods: The human leukocyte antigen (HLA) class II alleles, haplotypes, and genotypes were determined in a large cohort of patients with APS, autoimmune thyroid disease (AITD), and type 1 diabetes and in healthy controls by the consistent application of high-resolution typing at a four-digit level. Results: Comparison of the allele and haplotype frequencies significantly discriminated patients with APS vs AITD and controls. The HLA class…

musculoskeletal diseasesHla class iiAdultMaleendocrine system diseasesAdolescentEndocrinology Diabetes and MetabolismClinical BiochemistryGenes MHC Class IIHuman leukocyte antigenBiochemistryDiagnosis DifferentialYoung AdultEndocrinologyGene FrequencyAutoimmune Polyglandular SyndromeGenotypeMedicineHumansGenetic Predisposition to DiseaseTypingAlleleskin and connective tissue diseasesChildPolyendocrinopathies AutoimmuneType 1 diabetesbusiness.industryBiochemistry (medical)Haplotypenutritional and metabolic diseasesMiddle Agedmedicine.diseaseHaplotypesCase-Control StudiesImmunologyFemalebusinessThe Journal of clinical endocrinology and metabolism
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H2-M polymorphism in mice susceptible to collagen-induced arthritis involves the peptide binding groove.

1996

The ability to develop type II collagen (CII)-induced arthritis (CIA) in mice is associated with the major histocompatibilityI-A gene and with as yet poorly defined regulatory molecules of the major histocompatibility complex (MHC) class II antigen processing and presentation pathway. H2-M molecules are thought to be involved in the loading of antigenic peptides into the MHC class II binding cleft. We sequencedH2-Ma, H2-Mb1, andH2-Mb2 genes from CIA-susceptible and-resistant mouse strains and identified four differentMa andMb2 alleles and three differentMb1 alleles defined by polymorphic residues within the predicted peptide binding groove. Most CIA-resistant mouse strains share commonMa, M…

musculoskeletal diseasesImmunologyGenes MHC Class IIMolecular Sequence DataGenes MHC Class IPeptide bindingMice Inbred StrainsMajor histocompatibility complexEpitopeMiceAntigenMHC class IGeneticsAnimalsAmino Acid SequencePhylogenyDNA PrimersMHC class IIPolymorphism GeneticbiologyBase SequenceSequence Homology Amino AcidAntigen processingH-2 AntigensHistocompatibility Antigens Class IIMolecular biologyArthritis ExperimentalHistocompatibilityHaplotypesbiology.proteinCollagenSequence AlignmentImmunogenetics
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