Search results for " Muscle"

showing 10 items of 1495 documents

The nature of host tissue destruction in tumor invasion

1986

The nature of host tissue destruction in tumor invasion was investigated in experimentally induced carcinomas and sarcomas, xenografted into skeletal muscle. By means of light and electron microscopy it was shown that in both carcinomas and sarcomas the confrontation of host tissue with the invading tumor cells does not result in immediate destruction of host tissue but in a transitory state of coexistence which gradually proceeds to progressive host tissue atrophy. This process of progressive atrophy, which finally results in the total disappearance of the invaded host tissue, is considered to be caused mainly by the increasing pressure and competitive withdrawal of oxygen and nutrients by…

Pathologymedicine.medical_specialtyRatónTransplantation HeterologousMice NudeAdenocarcinomaBiologyHost tissueExtracellular matrixMiceAtrophymedicineCarcinomaAnimalsNeoplasm InvasivenessProcess (anatomy)MusclesSkeletal musclemedicine.diseaseRatsMicroscopy ElectronMuscular Atrophymedicine.anatomical_structureRats Inbred LewSarcoma ExperimentalSarcomaNeoplasm TransplantationVirchows Archiv B Cell Pathology Including Molecular Pathology
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A novel in vitro model for the study of plaque development in atherosclerosis

2006

SummaryFor the study of atherogenesis in vitro, coculture systems have been devised, in which two or more cell types can be cultured in close contact to each other. Herein, we describe a novel in vitro model that aims at the simulation of the morphology ofa normal muscular artery allowing for the study of the initial events in atherosclerosis. Usinga modified fibrin gel as a scaffold for the coculture of endothelial cells (ECs) and smooth muscle cells (SMCs), we generated an autologous in vitro model with a multilayer growth of SMCs (intima-like structure) covered by an endothelium. The production of extracellular matrix (ECM) could be visualized histologically and verified by (i) ascorbic-…

Pathologymedicine.medical_specialtyTime FactorsEndotheliumCellular differentiationMyocytes Smooth MuscleMonocytesMuscle Smooth VascularCell LineExtracellular matrixCell MovementLamininCell AdhesionmedicineHumansFoam cellFibrinDose-Response Relationship Drugbiologybusiness.industryEndothelial CellsCell DifferentiationHematologyAtherosclerosisCoculture TechniquesIn vitroExtracellular MatrixCell biologyLipoproteins LDLmedicine.anatomical_structureCell culturebiology.proteinbusinessGelsFoam CellsLipoproteinThrombosis and Haemostasis
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Subendothelial infiltration of neutrophil granulocytes and liberation of matrix-destabilizing enzymes in an experimental model of human neo-intima.

2008

SummaryIt was the objective of this study to examine the role of human neutrophil granulocytes (PMN) in an in-vitro model of human neo-intima developed for the study of atherosclerosis. Human granulocytes were subjected to a co-culture model of human endothelial and smooth muscle cells. Subendothelial lipid accumulation was achieved by addition of native LDL to the culture medium. Tissue samples were analyzed by immunohistochemistry and scanning/transmission electron microscopy, and culture supernatants were examined for the presence of interleukin- 8 (IL-8), MCP-1, GRO-α, elastase and matrixmetalloproteinase-8 (MMP-8). Following addition of 2 mg/ml LDL, adherence, transmigration and infilt…

Pathologymedicine.medical_specialtyTime FactorsEndotheliumNeutrophilsChemokine CXCL1Myocytes Smooth MuscleApoptosisBiologyGranulocyteMuscle Smooth VascularmedicineMyocyteHumansSecretionLeukocyte RollingCells CulturedChemokine CCL2ElastaseInterleukin-8InterleukinEndothelial CellsHematologymedicine.diseaseAtherosclerosisMolecular biologyCoculture TechniquesCulture MediaExtracellular MatrixLipoproteins LDLmedicine.anatomical_structureMatrix Metalloproteinase 8Neutrophil InfiltrationApoptosisLeukocyte ElastaseTunica IntimaInfiltration (medical)Signal TransductionThrombosis and haemostasis
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The Participation of the Complement System in Atherosclerotic Vascular Disease

1991

Atherosclerosis is a vascular disease of large and medium-sized arteries wherein the tunica intima becomes thickened due to lipid accumulation, mostly cholesterol and its esters, smooth muscle cell proliferation, and increased deposition of connective tissue matrix. A major risk factor in the development of this disease is hypercholesterolemia arising from elevated levels of low density lipoproteins (LDL). The earliest recognizable lesion, which may be a precursor to the fibrofatty plaque, is the fatty streak. It is predominantly composed of monocyte-derived macrophage foam cells, i.e. cells ladened with intracellular lipid droplets. Hence, a fundamental aspect of atherogenesis is the insud…

Pathologymedicine.medical_specialtyVascular smooth muscleChemistryMonocyteFatty streakConnective tissueTunica intimaLesionmedicine.anatomical_structureLipid dropletmedicineMacrophagelipids (amino acids peptides and proteins)medicine.symptom
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Clinical spectrum of BICD2 mutations.

2020

Background and purpose Mutations in the BICD2 gene cause autosomal dominant lower extremity-predominant spinal muscular atrophy 2A (SMALED2A), a condition that is associated with a specific pattern of thigh and calf muscle involvement when studied by magnetic resonance imaging (MRI). Patients may present minor clinical sensory impairment, but objective sensory involvement has yet to be demonstrated. Methods We collected clinical data from 11 patients from five different families carrying mutations in BICD2. Genetic diagnosis was achieved using gene panel testing and skin biopsies were taken from two patients to study the epidermal nerve fiber density. Results In the studied patients, three …

Pathologymedicine.medical_specialtyWeaknessSensory systemNerve fiberBICD2 Charcot-Marie-Tooth hereditary motor neuropathy muscle magnetic resonance imaging spinal muscular atrophyThighmedicine.disease_causeMuscular Atrophy Spinal03 medical and health sciences0302 clinical medicineCharcot-Marie-Tooth DiseasemedicineHumans030212 general & internal medicineMuscle SkeletalMutationLegmedicine.diagnostic_testbiologybusiness.industryMagnetic resonance imagingSpinal muscular atrophymedicine.diseasebiology.organism_classificationMagnetic Resonance ImagingMediusmedicine.anatomical_structureNeurologyMutationNeurology (clinical)medicine.symptombusinessMicrotubule-Associated Proteins030217 neurology & neurosurgery
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The eye in thyroid autoimmunity – Autoimmunité et œil lors de la maladie de Basedow

2014

Thyroid eye disease or thyroid-associated orbitopathy (TAO) is the commonest cause of orbital disease with an inflammatory component. With a basic knowledge of both orbital anatomy and the pathogenesis of TAO, it is easy to understand how its typical constellation of features originates. These features can be considered as primary or secondary. Primary features are those due directly to the disease process affecting a tissue e.g. restrictive myopathy. Secondary features occur due to the interaction of the affected tissue and the anatomical configuration of the tissues e.g. corneal ulceration or optic neuropathy, and are less likely to be florid at presentation. TAO is characterized by an im…

Pathologymedicine.medical_specialtybusiness.industryEndocrinology Diabetes and MetabolismThyroidInflammationGeneral Medicinemedicine.diseaseExtraocular muscleseye diseasesProinflammatory cytokineLymphocytic InfiltrateOptic neuropathyPathogenesisEndocrinologymedicine.anatomical_structureImmunologymedicinemedicine.symptomMyopathybusinessAnnales d'Endocrinologie
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Primary desminopathies.

2007

•  Introduction •  Desmin is an essential component of the extrasarcomeric cytoskeleton in striated muscle cells •  Distal myopathy,cardiac arrhythmias,cardiomyopathy:classical criteria of primary desminopathies •  Sub-sarcolemmal and cytoplasmic desmin-positive protein aggregates:the morphological hallmark of primary and secondary desminopathies •  The spectrum of pathogenic desmin gene mutations •  The molecular pathogenesis of primary desminopathies: some answers gained,but even more questions raised •  Diagnostic work-up to distinguish primary from secondary desminopathies •  Treatment and clinical management of primary desminopathy patients Abstract Mutations of the human desmin gene o…

Pathologymedicine.medical_specialtyintermediate filamentsCardiomyopathyReviewsgranulofilamentous materialdesmininclusion bodiesmacromolecular substancesBiologymyofibrillar myopathyprotein aggregationdesmin-related myopathySarcolemmaMuscular DiseasesmedicineMyocyteAnimalsHumansIntermediate filamentMyopathyMuscle SkeletalCytoskeletonGenetic heterogeneityCardiac muscleCell Biologymedicine.diseasemusculoskeletal systemmutationsmedicine.anatomical_structuredesminopathyMutationMolecular MedicineDesminmedicine.symptomMyofibrilJournal of cellular and molecular medicine
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Capillaries within human skeletal muscle fibers.

1991

Internalized capillaries, i.e. capillaries within muscle fibers, represent a rare myopathological feature. This was systematically studied in 923 muscle biopsy specimens and found in 24, chiefly in the gastrocnemius muscle, more rarely in the biceps and quadriceps muscles affecting males more often than females and most frequently associated with juvenile spinal muscular atrophy or Becker's muscular dystrophy. Internalized capillaries, often multiple, ran along the long axis of the muscle fiber within an "internalized" extracellular space and were almost exclusively seen in type I myofibers. Internalization seems to start at the site of fiber splitting while penetration through the intact s…

Pathologymedicine.medical_specialtymedia_common.quotation_subjectBiopsyeducationBiologyBicepsPathology and Forensic MedicineMuscle hypertrophyGastrocnemius muscleAtrophymedicineHumansMuscular dystrophyInternalizationmedia_commonMuscle biopsySarcolemmamedicine.diagnostic_testHistocytochemistryMusclesCell BiologyAnatomyHypertrophymedicine.diseaseImmunohistochemistryCapillariesMicroscopy ElectronAtrophyPathology, research and practice
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Pterygium axillae as a rare manifestation of Poland syndrome

2016

Abstract Poland syndrome is characterized by a combination of absent pectoralis muscle, abnormalities of the rib cage, the breast, as well as brachy-syndactyly. We report a case of a 3 month old girl who was born with right sided axillary pterygium combined with flattening of the right anterior chest wall. Resection of a sclerotic band with reconstruction by Z-plasty was performed. Intraoperative and histopathological findings confirmed that the axillary pterygium developed on the basis of a scarred, hypoplastic pectoralis major muscle. Our case adds to the body of evidence that Poland syndrome may present in a heterogeneous fashion, including the rare finding of axillary pterygium with ass…

PediatricRib cagemedicine.medical_specialtybusiness.industryPoland syndromePectoralis major muscle030204 cardiovascular system & hematologymedicine.diseaseResectionRight Anterior ChestPterygiumSurgery03 medical and health sciences0302 clinical medicinePterygium axillaePoland syndrome030220 oncology & carcinogenesisPediatrics Perinatology and Child HealthMedicineSurgeryContracturemedicine.symptombusinessPectoralis MuscleJournal of Pediatric Surgery Case Reports
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Clinical and Genetic Aspects of Juvenile Onset Pompe Disease

2021

AbstractLittle is known about clinical symptomatology and genetics of juvenile onset Pompe disease (JOPD). The aims of this study were to analyze how these children are diagnosed, what clinical problems they have, and how phenotype is related to genotype. To accomplish this, we analyzed retrospectively data of 34 patients diagnosed after their first and before completion of their 18th birthday. Median age at diagnosis was 3.9 (range 1.1–17) years. Eight patients (23.5%) developed initial symptoms in the first year, 12 (35%) between 1 and 7 years, and 6 (18%) thereafter. Eight (23.5%) had no clinical symptoms at the time of diagnosis. Indications for diagnostics were a positive family histor…

Pediatricsmedicine.medical_specialtyGeneralized muscle weaknessDisease03 medical and health sciences0302 clinical medicineGenotypeHumansMedicineFamily historyRetrospective Studies030304 developmental biology0303 health sciencesGlycogen Storage Disease Type IIbusiness.industryHypertrophic cardiomyopathyMuscle weaknessalpha-GlucosidasesGeneral Medicinemedicine.disease3. Good healthPhenotypeJuvenile onsetMutationPediatrics Perinatology and Child HealthFailure to thriveNeurology (clinical)medicine.symptombusiness030217 neurology & neurosurgeryNeuropediatrics
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