Search results for " N-methyl-D-aspartate"

showing 10 items of 94 documents

A repetitive intracortical microstimulation pattern induces long-lasting synaptic depression in brain slices of the rat primary somatosensory cortex.

2000

Repetitive intracortical microstimulation (ICMS) applied to the rat primary somatosensory cortex (SI) in vivo was reported to induce reorganization of receptive fields and cortical maps. The present study was designed to exam- ine the effect of such an ICMS pattern applied to layer IV of brain slices containing SI on the efficacy of synaptic in- put to layer II/III. Effects of ICMS on the synaptic strength was quantified for the first synaptic component ( s1) of cor- tical field potentials (FPs) recorded from layer II/III of SI. FPs were evoked by stimulation in layer IV. The pattern of ICMS was identical to that used in vivo. However, stimula- tion intensity had to be raised to induce an a…

MaleLong-Term PotentiationNeurotransmissionIn Vitro TechniquesInhibitory postsynaptic potentialBicucullineReceptors N-Methyl-D-AspartateGABA AntagonistsRats Sprague-DawleymedicineAnimalsReceptors AMPASynaptic potentialNeuronal PlasticityChemistryGeneral NeuroscienceLong-term potentiationSomatosensory CortexBicucullineElectric StimulationRatsElectrophysiologyembryonic structuresSynaptic plasticitySynapsesExcitatory postsynaptic potentialNeuroscienceMicroelectrodesmedicine.drugExperimental brain research
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Prenatal exposure to the CB1 receptor agonist WIN 55,212-2 causes learning disruption associated with impaired cortical NMDA receptor function and em…

2005

The aim of this study was to investigate whether prenatal exposure to the cannabinoid CB1 receptor agonist WIN 55,212-2 (WIN) at a daily dose devoid of overt signs of toxicity and/or gross malformations (0.5 mg/kg, gestation days 5-20), influences cortical glutamatergic neurotransmission, learning and emotional reactivity in rat offspring. Basal and K+-evoked extracellular glutamate levels were significantly lower in cortical cell cultures obtained from pups exposed to WIN during gestation with respect to those measured in cultures obtained from neonates born from vehicle-treated dams. The addition of NMDA to cortical cell cultures from neonates born from vehicle-treated dams concentration-…

MaleMarijuana AbuseCannabinoid receptoractive avoidance behaviour; basal and K+-evoked glutamate levels; cortical cell cultures; homing behaviour; maternal marijuana consumption; ultrasonic vocalizationEmotionsReceptor Cannabinoid CB1Pregnancyactive avoidance behaviourWIN 55212-2Cells CulturedCerebral CortexBehavior AnimalGlutamate receptorBraincortical cell culturesCalcium Channel Blockersactive avoidance behaviour; basal and k plus -evoked glutamate levels; basal and k+-evoked glutamate levels; cortical cell cultures; homing behaviour; maternal marijuana consumption; ultrasonic vocalizationPrenatal Exposure Delayed EffectsChloratesNMDA receptorbasal and K+-evoked glutamate levelsFemaleMicrotubule-Associated Proteinsmedicine.drugAgonistmedicine.medical_specialtyOffspringmedicine.drug_classCognitive NeuroscienceMorpholinesGlutamic Acidmaternal marijuana consumptionNeurotransmissionBiologyNaphthalenesReceptors N-Methyl-D-AspartateCellular and Molecular NeuroscienceGlutamatergicInternal medicinemedicineAvoidance LearningAnimalsRats WistarBenzoxazinesRatsultrasonic vocalizationEndocrinologyAnimals Newbornhoming behaviourVocalization AnimalExtracellular SpaceNeuroscience
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NMDA glutamate but not dopamine antagonists blocks drug-induced reinstatement of morphine place preference.

2004

The effects of dopaminergic and glutamatergic antagonists on the drug-induced reinstatement of a previously extinguished morphine conditioned place preference (CPP) in mice were evaluated. Following extinction of a place preference induced by morphine (40 mg/kg), a non-contingent injection of the dopaminergic antagonists SCH 23390 (0.125, 0.5 mg/kg), raclopride (0.3, 1.2 mg/kg), haloperidol (0.1, 0.2 mg/kg) and the dopamine (DA) release inhibitor CGS 10746B (1, 10 mg/kg) or glutamatergic NMDA antagonists memantine (10, 20, 40 mg/kg) and MK-801 (0.1, 0.2, 0.3 mg/kg) alone or with 10 mg/kg morphine was given. Neither the dopaminergic nor the glutamatergic antagonists alone reinstated the plac…

MaleMice Inbred StrainsPharmacologyReceptors N-Methyl-D-AspartateExtinction PsychologicalGlutamatergicMiceDopaminemedicineHaloperidolAnimalsDrug InteractionsRacloprideAnalysis of VarianceBehavior AnimalDose-Response Relationship DrugMorphineChemistryGeneral NeuroscienceDopaminergicMemantineConditioned place preferenceAnalgesics OpioidNMDA receptorConditioning OperantDopamine AntagonistsExcitatory Amino Acid Antagonistsmedicine.drugBrain research bulletin
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Bilobalide, a constituent of Ginkgo biloba , inhibits NMDA-induced phospholipase A 2 activation and phospholipid breakdown in rat hippocampus

2000

In rat hippocampal slices superfused with magnesium-free buffer, glutamate (1 mM) caused the release of large amounts of choline due to phospholipid breakdown. This phenomenon was mimicked by N-methyl-D-aspartate (NMDA) in a calcium-sensitive manner and was blocked by NMDA receptor antagonists such as MK-801 and 7-chlorokynurenate. The NMDA-induced release of choline was not caused by activation of phospholipase D but was mediated by phospholipase A2 (PLA2) activation as the release of choline was accompanied by the formation of lyso-phosphatidylcholine (lyso-PC) and glycerophospho-choline (GPCh) and was blocked by 5-[2-(2-carboxyethyl)-4-dodecanoyl-3,5-dimethylpyrrol-1-yl]pentano ic acid, …

MaleMicrodialysisN-MethylaspartateMicrodialysisGlycineCyclopentanesPharmacologyHippocampal formationHippocampusReceptors N-Methyl-D-AspartatePhospholipases ACholinechemistry.chemical_compoundPhospholipase A2BilobalideSeizuresAnimalsCholineRats WistarFuransCells CulturedPhospholipidsPharmacologyPlants MedicinalDose-Response Relationship DrugbiologyPhospholipase DGlutamate receptorGinkgo bilobaLysophosphatidylcholinesGeneral MedicineGlycerylphosphorylcholineRatsEnzyme ActivationPhospholipases A2Ginkgolideschemistrybiology.proteinNMDA receptorDiterpenesNaunyn-Schmiedeberg's Archives of Pharmacology
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Dose-dependent induction of CPP or CPA by intra-pVTA ethanol: Role of mu opioid receptors and effects on NMDA receptors.

2020

AbstractThe neurobiological mechanisms underlying alcohol motivational properties are still not fully understood, however, the mu-opioid receptors (MORs) have been evidenced as central elements in the manifestation of the alcohol reinforcing properties. Drug-associated environmental stimuli can trigger alcohol relapse and promote alcohol consumption whereby N-methyl-D-aspartate (NMDA) receptors play a pivotal role. Here we sought to demonstrate, for the first time, that ethanol induces conditioned place preference or aversion (CPP or CPA) when administered locally into the ventral tegmental area (VTA) and the associated role of MORs. We further analyzed the changes in the expression and mRN…

MaleMicroinjectionsReceptors Opioid muHippocampusNucleus accumbensPharmacologyReceptors N-Methyl-D-AspartateArticle03 medical and health scienceschemistry.chemical_compound0302 clinical medicineConditioning PsychologicalmedicineAvoidance LearningAnimalsRats WistarReceptorBiological Psychiatry030304 developmental biologyPharmacology0303 health sciencesEthanolDose-Response Relationship DrugEthanolChemistryVentral Tegmental AreaConditioned place preference030227 psychiatryRatsVentral tegmental areamedicine.anatomical_structureInfusions Intraventricularnervous systemNMDA receptorμ-opioid receptor030217 neurology & neurosurgeryProgress in neuro-psychopharmacologybiological psychiatry
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Involvement of NMDA glutamate receptors in the acquisition and reinstatement of the conditioned place preference induced by MDMA.

2015

Some 3,4-methylenedioxymethamphetamine (MDMA) users become dependent as a result of chronic consumption. A greater understanding of the neurobiological basis of the rewarding effects of MDMA could contribute to developing effective pharmacotherapies for MDMA-related problems. The present study evaluated the role of N-methyl-D-aspartate (NMDA) glutamate receptors (NMDARs) in the acquisition and reinstatement of conditioned place preference (CPP) induced by MDMA. Adolescent male mice were conditioned with 1 or 10 mg/kg MDMA and pretreated with 5 or 10 mg/kg of the NMDAR antagonist memantine during acquisition of conditioning (experiment 1), or before a reinstatement test (experiment 2). In ad…

MaleN-Methyl-34-methylenedioxyamphetamineMale miceSpatial BehaviorPharmacologyReceptors N-Methyl-D-AspartateMiceSerotonin AgentsMemantineMemorymental disordersConditioning PsychologicalAvoidance LearningMedicineAnimalsPharmacologyCacaoMotivationDose-Response Relationship Drugbusiness.industrymusculoskeletal neural and ocular physiologyGlutamate receptorMemantineAntagonistMDMAExtinction (psychology)Conditioned place preferencePsychiatry and Mental healthnervous systemNMDA receptorbusinessExcitatory Amino Acid Antagonistspsychological phenomena and processesmedicine.drugBehavioural pharmacology
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Low Density Lipoprotein Receptor-related Protein 1 (LRP1) Modulates N-Methyl-d-aspartate (NMDA) Receptor-dependent Intracellular Signaling and NMDA-i…

2013

The lipoprotein receptor LRP1 is essential in neurons of the central nervous system, as was revealed by the analysis of conditional Lrp1-deficient mouse models. The molecular basis of its neuronal functions, however, is still incompletely understood. Here we show by immunocytochemistry, electron microscopy, and postsynaptic density preparation that LRP1 is located postsynaptically. Basal and NMDA-induced phosphorylation of the transcription factor cAMP-response element-binding protein (CREB) as well as NMDA target gene transcription are reduced in LRP1-deficient neurons. In control neurons, NMDA promotes γ-secretase-dependent release of the LRP1 intracellular domain (LRP1-ICD). However, pul…

MaleN-MethylaspartateCell SurvivalBlotting WesternGene ExpressionMice Transgenicmacromolecular substancesAMPA receptorBiologyCREBReceptors N-Methyl-D-AspartateBiochemistryMiceNeurobiologyPostsynaptic potentialAnimalsMolecular BiologyCells CulturedMice KnockoutNeuronsReverse Transcriptase Polymerase Chain Reactionmusculoskeletal neural and ocular physiologyTumor Suppressor ProteinsMembrane ProteinsCell BiologyEmbryo MammalianLRP1Cell biologyProtein SubunitsReceptors LDLnervous systemSynapsesLDL receptorbiology.proteinNMDA receptorFemaleAmyloid Precursor Protein SecretasesSignal transductionDisks Large Homolog 4 ProteinGuanylate KinasesPostsynaptic densityLow Density Lipoprotein Receptor-Related Protein-1Protein BindingSignal TransductionSynaptosomesJournal of Biological Chemistry
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Memantine blocks sensitization to the rewarding effects of morphine

2009

Knowledge regarding the specific brain changes and neural plasticity processes produced by repeated drug exposure may be used to advance the understanding of the neurobiology of addiction in order to design appropriate medications. In the present study, the influence of N-methyl-d-aspartate (NMDA) glutamatergic receptors on sensitization to the motor and rewarding effects of morphine was evaluated. The effects of pre-exposure to saline or 20 mg/kg morphine plus the NMDA receptor antagonist memantine (10 or 20 mg/kg) on motor activity and place conditioning induced by a low dose of morphine (2 mg/kg) were assessed. The dose of 2 mg/kg of morphine was ineffective in mice pre-exposed to saline…

MaleNarcoticsMotor ActivityPharmacologyReceptors N-Methyl-D-AspartateMiceGlutamatergicRewardMemantineConditioning PsychologicalNeuroplasticityAnimalsMedicineMolecular BiologySensitizationAnalysis of VarianceMotivationDose-Response Relationship DrugMorphinebusiness.industryGeneral NeuroscienceGlutamate receptorMemantineConditioned place preferencemedicine.anatomical_structureMorphineNMDA receptorNeurology (clinical)businessExcitatory Amino Acid AntagonistsDevelopmental Biologymedicine.drugBrain Research
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D1/D5 modulation of synaptic NMDA receptor currents.

2009

Converging evidence suggests that salience-associated modulation of behavior is mediated by the release of monoamines and that monoaminergic activation of D1/D5receptors is required for normal hippocampal-dependent learning and memory. However, it is not understood how D1/D5modulation of hippocampal circuits can affect salience-associated learning and memory. We have observed in CA1 pyramidal neurons that D1/D5receptor activation elicits a bidirectional long-term plasticity of NMDA receptor-mediated synaptic currents with the polarity of plasticity determined by NMDA receptor, NR2A/B subunit composition. This plasticity results in a decrease in the NR2A/NR2B ratio of subunit composition. Sy…

MaleNeuronal PlasticityGeneral Neurosciencemusculoskeletal neural and ocular physiologyReceptors Dopamine D1Long-term potentiationAMPA receptorNeurotransmissionBiologyReceptors N-Methyl-D-AspartateSynaptic TransmissionArticleMice Inbred C57BLMicenervous systemSynaptic plasticitySynapsesNMDA receptorAnimalsReceptors Dopamine D5Nerve NetReceptorLong-term depressionNeuronal memory allocationNeuroscienceThe Journal of neuroscience : the official journal of the Society for Neuroscience
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Functional Synaptic Projections onto Subplate Neurons in Neonatal Rat Somatosensory Cortex

2002

Subplate neurons (SPn) play an important role in the formation of thalamocortical connections during early development and show glutamatergic and GABAergic spontaneous synaptic activity. We characterized these synaptic inputs by performing whole-cell recordings from SPn in somatosensory cortical slices of postnatal day 0-3 rats. At -70 mV, electrical stimulation of the thalamocortical afferents elicited in 68% of the SPn a monosynaptic CNQX-sensitive postsynaptic current (PSC). These fast PSCs were mediated by AMPA receptors, because they were prolonged by cyclothiazide and blocked by GYKI 52466. On membrane depolarization, thalamocortical stimulation elicited in 50% of the cells an additio…

MalePatch-Clamp TechniquesAction PotentialsStimulationAMPA receptorBiologyIn Vitro TechniquesSomatosensory systemReceptors N-Methyl-D-AspartateMembrane PotentialsGABA AntagonistsThalamusSubplatemedicineAnimalsReceptors AMPAARTICLERats Wistargamma-Aminobutyric AcidNeuronsAfferent PathwaysGeneral NeuroscienceLysineCell MembraneExcitatory Postsynaptic PotentialsDepolarizationSomatosensory CortexReceptors GABA-AElectric StimulationRatsmedicine.anatomical_structurenervous systemAnimals NewbornSynapsesGABAergicNMDA receptorCyclothiazideNeuroscienceExcitatory Amino Acid Antagonistsmedicine.drug
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