Search results for " Nerve"

showing 10 items of 885 documents

Rare variants in the genetic background modulate cognitive and developmental phenotypes in individuals carrying disease-associated variants

2019

Purpose: To assess the contribution of rare variants in the genetic background toward variability of neurodevelopmental phenotypes in individuals with rare copy-number variants (CNVs) and gene-disruptive variants. Methods: We analyzed quantitative clinical information, exome sequencing, and microarray data from 757 probands and 233 parents and siblings who carry disease-associated variants. Results: The number of rare likely deleterious variants in functionally intolerant genes (“other hits”) correlated with expression of neurodevelopmental phenotypes in probands with 16p12.1 deletion (n=23, p=0.004) and in autism probands carrying gene-disruptive variants (n=184, p=0.03) compared with thei…

MaleParents0301 basic medicineProbandNeuronalGenetic Carrier Screening16p11.2 deletion030105 genetics & heredityCognitionFamily historyNeural Cell Adhesion MoleculesGenetics (clinical)Exome sequencingSequence DeletionGeneticsGenetic Carrier ScreeningPhenotypePenetrancePedigreePhenotypeAutistic Disorder/genetics; Autistic Disorder/physiopathology; Cell Adhesion Molecules Neuronal/genetics; Chromosomes Human Pair 16/genetics; Cognition/physiology; DNA Copy Number Variations/genetics; Female; Gene Expression Regulation/genetics; Genetic Background; Genetic Carrier Screening; Humans; Male; Methyltransferases/genetics; Nerve Tissue Proteins/genetics; Parents; Pedigree; Phenotype; Proteins/genetics; Sequence Deletion/genetics; Siblings; 16p11.2 deletion; CNV; autism; modifier; phenotypic variabilityFemaleGenetic BackgroundHumanDNA Copy Number VariationsCell Adhesion Molecules NeuronalCNVautismNerve Tissue ProteinsBiologyChromosomesArticle03 medical and health sciencesmental disordersmedicineHumansAutistic DisorderBiologyGenemodifierPair 16SiblingsCalcium-Binding ProteinsProteinsMethyltransferasesmedicine.disease16p11.2 deletion; autism; CNV; modifier; phenotypic variability; Genetics (clinical)Cytoskeletal Proteins030104 developmental biologyGene Expression Regulation[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human geneticsAutismphenotypic variabilityHuman medicine16p11.2 deletion; autism; CNV; modifier; phenotypic variability; Autistic Disorder; Cell Adhesion Molecules Neuronal; Chromosomes Human Pair 16; Cognition; DNA Copy Number Variations; Female; Gene Expression Regulation; Genetic Background; Humans; Male; Methyltransferases; Nerve Tissue Proteins; Parents; Pedigree; Phenotype; Proteins; Sequence Deletion; Siblings; Genetic Carrier ScreeningCell Adhesion MoleculesChromosomes Human Pair 16Transcription FactorsGenetics in Medicine
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Lipid composition of the human eye: are red blood cells a good mirror of retinal and optic nerve fatty acids?

2011

International audience; BACKGROUND: The assessment of blood lipids is very frequent in clinical research as it is assumed to reflect the lipid composition of peripheral tissues. Even well accepted such relationships have never been clearly established. This is particularly true in ophthalmology where the use of blood lipids has become very common following recent data linking lipid intake to ocular health and disease. In the present study, we wanted to determine in humans whether a lipidomic approach based on red blood cells could reveal associations between circulating and tissue lipid profiles. To check if the analytical sensitivity may be of importance in such analyses, we have used a do…

MalePathologyErythrocytes[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionBlood lipidsBiochemistryAnalytical Chemistrychemistry.chemical_compound0302 clinical medicineMolecular Cell BiologyPathologychemistry.chemical_classificationAged 80 and overLiquid Chromatography0303 health sciencesChromatographyMultidisciplinaryLipid ClassesQFatty AcidsRLipids3. Good healthChemistrymedicine.anatomical_structureDocosahexaenoic acidAlimentation et NutritionBlood ChemistryOptic nerveMedicineRetinal DisordersArachidonic acidFemalelipids (amino acids peptides and proteins)Fatty AlcoholsCellular TypesPolyunsaturated fatty acidResearch Articlemedicine.medical_specialtySpectrometry Mass Electrospray IonizationScienceGas Chromatography-Mass SpectrometryRetinaGlycerides03 medical and health sciencesDiagnostic MedicineLipidomicsLipid StructuremedicineFood and NutritionHumansBiology030304 developmental biologyAgedGas ChromatographyNutritionRetinaBlood CellsRetinalOptic NerveGlaucomaOphthalmologychemistryMacular Disorders030221 ophthalmology & optometry[SDV.AEN]Life Sciences [q-bio]/Food and NutritionBiomarkersGeneral Pathology
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Generalised sensory system abnormalities in amyotrophic lateral sclerosis: a European multicentre study.

2007

International audience; BACKGROUND: Amyotrophic lateral sclerosis (ALS) is defined as a disease of the motor neurones, although several studies indicate involvement of the sensory nervous system. AIM: To evaluate the sensory nerve conduction studies (NCS) in 88 patients with ALS as part of a European multicentre study. METHODS: Seven European clinical neurophysiologists examined consecutive series of ALS patients. The examinations were peer reviewed, and the diagnosis of ALS was confirmed clinically. RESULTS: 20 (22.7%) patients with ALS had sensory NCS abnormalities in at least one nerve. Of those, 11 (12.5% of all patients) obtained an additional peer review diagnosis of electrophysiologi…

MalePathologyNeural Conduction0302 clinical medicineMESH: Aged 80 and overDorsal root ganglionMESH: Neural ConductionAmyotrophic lateral sclerosisMESH: Amyotrophic Lateral SclerosisAged 80 and overMESH: Aged0303 health sciencesMESH: ElectrophysiologyMESH: Middle AgedMESH: Neurons AfferentMiddle AgedElectrophysiologyEuropePsychiatry and Mental healthmedicine.anatomical_structureMESH: Sensation DisordersSensation DisordersFemale[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]PolyneuropathySensory nerveAdultmedicine.medical_specialtyNeuromuscular diseaseShort ReportSensory systemCentral nervous system disease03 medical and health sciencesmedicineHumans[SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Neurons AfferentAged030304 developmental biologyMESH: Humansbusiness.industryAmyotrophic Lateral SclerosisMESH: Adultmedicine.diseaseMESH: MaleSurgeryNeurology (clinical)MESH: EuropebusinessMotor neurone diseaseMESH: Female030217 neurology & neurosurgery
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Complexity of the Hereditary Motor and Sensory Neuropathies

2015

Early-onset hereditary motor and sensory neuropathies are rare diseases representing a broad clinical and genetic spectrum. Without a notable familial history, the clinical diagnosis is complicated because acquired causes of peripheral neuropathy, such as inflammatory neuropathies, neuropathies with toxic causes, and nutritional deficiencies, must be considered. We examined the clinical, electrophysiological, and pathologic manifestations of a boy with an initial diagnosis of chronic inflammatory demyelinating polyneuropathy. The progression of the disease despite treatment led to a suspicion of hereditary motor and sensory neuropathy. Genetic testing revealed the presence of the MPZ p.D90…

MalePathologymedicine.medical_specialtyChronic inflammatory demyelinating polyneuropathySensory systemDiseaseBioinformaticsSural NervemedicineHumansGenetic testingmedicine.diagnostic_testbusiness.industrymedicine.diseasePhenotypePeripheral neuropathyChild PreschoolClinical diagnosisMutationPediatrics Perinatology and Child HealthMutation (genetic algorithm)Disease ProgressionNeurology (clinical)Hereditary Sensory and Motor NeuropathybusinessHereditary motor and sensory neuropathyMyelin P0 ProteinHeLa CellsJournal of Child Neurology
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Phagocytosis of erythrocytes in the subarachnoid space at spinal nerve exits

1982

After intracisternal injection of heparinised autologous blood in cats, spinal nerve exits (SNE) of the subarachnoid space (SAS) were examined by scanning and transmission electron microscopy. Phagocytes, erythrocytes and erythrophages (= macrophages which had phagocytosed red blood cells) were found at SNE. Some lining cells of the SAS had retracted from the adjacent cells and had rounded up. Cells which formed an integral part of the subarachnoid lining cells also had phagocytosed erythrocytes. Debris of an exhausted erythrophage was phagocytosed by other macrophages. Finally the observation has been made that erythrophages are capable of leaving the SAS actively by migrating through the …

MalePathologymedicine.medical_specialtyErythrocytesHistologyChemistryMacrophagesPhagocytosisAutologous bloodConnective tissueCell BiologyAnatomySubarachnoid SpacePathology and Forensic MedicineSpinal Nervesmedicine.anatomical_structurePhagocytosisSpinal nerveCatsMicroscopy Electron ScanningmedicineAnimalsFemaleSubarachnoid spaceCell and Tissue Research
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Upregulation of antibody response to heat shock proteins and tissue antigens in an ocular ischemia model.

2011

PURPOSE. The aim of this study was to characterize the serum antibody reactivities occurring after ocular ischemia reperfusion. The time course of serum antibody responses was examined. METHODS. Wistar rats were exposed to transient ocular ischemia by elevating intraocular pressure to 130 mm Hg for 60 minutes. Axonal damage was evaluated on optic-nerve sections 2 and 4 weeks later. Blood samples collected before and several times after ischemia were used for antibody detection via customized protein microarrays. Different tissue antigens, including heat shock proteins (HSPs) and crystallins, were selected based on previous identification of antibody reactivities in studies on ischemic event…

MalePathologymedicine.medical_specialtyIschemiaHSP27 Heat-Shock ProteinsProtein Array AnalysisVimentinBiologyAutoantigensDownregulation and upregulationAntigenRetinal DiseasesHeat shock proteinGlial Fibrillary Acidic ProteinmedicineAnimalsRats WistarEye ProteinsIntraocular PressureAutoantibodiesGlial fibrillary acidic proteinRetinal VesselsSpectrinMyelin Basic ProteinOptic Nervemedicine.diseaseAxonsRatsUp-RegulationMyelin-Associated GlycoproteinShock (circulatory)Immunoglobulin GReperfusion Injurybiology.proteinMyelin-Oligodendrocyte Glycoproteinmedicine.symptomAntibodyMyelin ProteinsInvestigative ophthalmologyvisual science
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Infantile neuroaxonal dystrophy: Diagnosis by skin biopsy

1991

A child who shows progressive motor and mental deterioration after the first year of life, who has pyramidal signs, marked muscle hypotonia, but no seizures, suggests to have infantile neuroaxonal dystrophy (INAD). Beyond the age of two years, the EEG also entails characteristic findings. Diagnosis may be obtained by an ultrastructural examination of biopsied skin. The respective clinical and morphological findings are recorded and illustrated from four patients in this report.

MalePathologymedicine.medical_specialtyMuscle HypotoniaBiopsySural nerveInfantile neuroaxonal dystrophyDegenerative diseaseDevelopmental NeuroscienceBiopsyHumansMedicineSkinmedicine.diagnostic_testMental deteriorationbusiness.industryLeukodystrophyInfantPeripheral Nervous System DiseasesNeuromuscular DiseasesGeneral Medicinemedicine.diseaseChild PreschoolPediatrics Perinatology and Child HealthSkin biopsyFemaleNeurology (clinical)businessBrain and Development
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Changes of sonographic, magnetic resonance tomographic, electromyographic, and histopathologic findings within a 2-month period of examinations after…

1998

This study compares sonographical, histopathological, magnetic resonance imaging (MRI), and electromyographical (EMG) findings following acute muscle denervation. We performed an experimental denervation of the supraspinatus and infraspinatus muscles on 35 New Zealand white rabbits by segment resection of the suprascapular nerve. The sonographical appearance of the supraspinatus muscle was followed and documented at short time intervals within a 2-month follow-up period. The sonographical, histopathological, and MRI changes due to denervation suggest a regular pattern. Apart from the reduction of the muscle diameter, there were considerable sonographical signs of denervation with an increas…

MalePathologymedicine.medical_specialtySupraspinatus muscleElectromyographySensitivity and Specificity030218 nuclear medicine & medical imaging03 medical and health sciences0302 clinical medicinemedicineAnimalsOrthopedics and Sports MedicinePeripheral NervesUltrasonographyDenervationMuscle Denervationmedicine.diagnostic_testElectromyographybusiness.industryMagnetic resonance imagingGeneral MedicineAnatomySuprascapular nerveMagnetic Resonance ImagingMuscle DenervationMuscle atrophy3. Good healthMuscular Atrophymedicine.anatomical_structureSurgeryRabbitsmedicine.symptombusiness030217 neurology & neurosurgeryFollow-Up StudiesReinnervationArchives of Orthopaedic and Trauma Surgery
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Further delineation of eye manifestations in homozygous 15q13.3 microdeletions including TRPM1: a differential diagnosis of ceroid lipofuscinosis.

2014

The 15q13.3 heterozygous microdeletion is a fairly common microdeletion syndrome with marked clinical variability and incomplete penetrance. The average size of the deletion, which comprises six genes including CHRNA7, is 1.5 Mb. CHRNA7 has been identified as the gene responsible for the neurological phenotype in this microdeletion syndrome. Only seven patients with a homozygous microdeletion that includes at least CHRNA7, and is inherited from both parents have been described in the literature. The aim of this study was to further describe the distinctive eye manifestations from the analysis in the three French patients diagnosed with the classical 1.5 Mb homozygous microdeletion. Patients…

MalePathologymedicine.medical_specialtygenetic structuresalpha7 Nicotinic Acetylcholine ReceptorEncephalopathyTRPM Cation ChannelsChromosome DisordersBiologyBlindnessEyePupilNeuronal Ceroid-LipofuscinosesNight BlindnessSeizuresIntellectual DisabilityRetinal DystrophiesGeneticsmedicineElectroretinographyMyopiaHumansEye AbnormalitiesChildGenetics (clinical)TRPM1Genetic Association StudiesCongenital stationary night blindnessGeneticsChromosomes Human Pair 15DystrophyEye Diseases HereditaryGenetic Diseases X-LinkedOptic NerveMicrodeletion syndromemedicine.diseasePenetranceChild PreschoolFemalesense organsDifferential diagnosisChromosome DeletionAmerican journal of medical genetics. Part A
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Free Bipedicled Radial Forearm and Posterior Interosseous Artery Perforator Flap Phalloplasty

2019

Abstract Introduction The free radial forearm (FRFA) flap is universally still considered as the gold standard technique in penile reconstruction. Typically, a considerably large flap is required, often involving almost the entire circumference of the forearm. Partial necrosis may occur at the distal-most (dorsoradial) part of the flap as a result of insufficient perfusion. Aim To describe a new technique using the posterior interosseous artery (PIOA) to supercharge FRFA phalloplasty. Methods In a 12-month period, all patients having FRFA flap phalloplasty were enrolled. Perioperative, after complete flap dissection, an indocyanine green perfusion scan was performed. In case of insufficient…

MalePhalloplastymedicine.medical_specialtyUrologyEndocrinology Diabetes and MetabolismFistula030232 urology & nephrologySettore MED/19 - Chirurgia PlasticaPerfusion scanningAnastomosisPenile Reconstruction03 medical and health sciencesPostoperative Complications0302 clinical medicineEndocrinologyUrethraForearmmedicine.arterymedicineSex Reassignment SurgeryHumansPenile Reconstruction; Phalloplasty; Sex Reassignment Surgery; Transmen030219 obstetrics & reproductive medicinebusiness.industryArteriesmedicine.diseaseSurgeryPosterior interosseous arteryForearmPsychiatry and Mental healthStenosisPosterior interosseous nervemedicine.anatomical_structureReproductive MedicineTransmenFemalePhalloplastybusinessPerforator FlapPenis
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