Search results for " REPLICATION"

showing 10 items of 406 documents

Correlation of virus replication, cytokine (TNF-? and IL-1) producing cells, neuronal necrosis and inflammation after intranasal infection of mice wi…

1995

The number of TNF-alpha and IL-1 beta producing cells was investigated during the acute replication phase of herpes simplex virus (HSV) in trigeminal ganglia after intranasal infection with strains of different virulence. The highly virulent strain WAL replicated strongly and induced many cytokine producing cells early in the ganglia. The low virulent strain HFEM replicated less, only few cytokine producing cells were detected late. The thymidine-kinase negative (TK-) virus 1301 did not replicate but produced some lymphocytic inflammation. The higher the virulence of strains of HSV-1 or -2 was, the stronger was the extent of histopathological lesions; moreover, a dissociation in time betwee…

MaleTime Factorsmedicine.medical_treatmentVirulenceInflammationBiologyVirus Replicationmedicine.disease_causeHerpesviridaeVirusMiceNecrosisT-Lymphocyte SubsetsVirologymedicineAnimalsSimplexvirusAdministration IntranasalNeuronsMice Inbred BALB CTumor Necrosis Factor-alphaHerpes SimplexGeneral MedicineVirologyCytokineHerpes simplex virusTrigeminal GanglionViral replicationmedicine.symptomCD8Interleukin-1Archives of Virology
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Perlecan-Induced Suppression of Smooth Muscle Cell Proliferation Is Mediated Through Increased Activity of the Tumor Suppressor PTEN

2004

We were interested in the elucidation of the interaction between the heparan sulfate proteoglycan, perlecan, and PTEN in the regulation of vascular smooth muscle cell (SMC) growth. We verified serum-stimulated DNA synthesis, and Akt and FAK phosphorylation were significantly reduced in SMCs overexpressing wild-type PTEN. Our previous studies showed perlecan is a potent inhibitor of serum-stimulated SMC growth. We report in the present study, compared with SMCs plated on fibronectin, serum-stimulated SMCs plated on perlecan exhibited increased PTEN activity, decreased FAK and Akt activities, and high levels of p27, consistent with SMC growth arrest. Adenoviral-mediated overexpression of cons…

MaleVascular smooth musclePhysiology:CIENCIAS MÉDICAS ::Farmacodinámica [UNESCO]Aorta ThoracicBasement MembraneCulture Media Serum-FreeMuscle Smooth VascularRats Sprague-DawleyMicePhosphorylationCells CulturedGlycosaminoglycansbiologyProtein-Tyrosine KinasesCell cycle:CIENCIAS MÉDICAS [UNESCO]musculoskeletal systemUNESCO::CIENCIAS MÉDICAS ::FarmacodinámicaUNESCO::CIENCIAS MÉDICAScardiovascular systemPhosphorylationSmooth muscle cell proliferationCardiology and Cardiovascular MedicineCell DivisionDNA ReplicationBasement membraneRecombinant Fusion ProteinsPerlecanProtein Serine-Threonine KinasesVascular injurySmooth muscle cell proliferation ; Restenosis ; Vascular injury ; Vascular development ; Basement membraneCatheterizationProto-Oncogene ProteinsAnimalsPTENProtein kinase BRestenosisCell growthVascular developmentOligonucleotides AntisenseFibronectinsRatsFibronectinFocal Adhesion Kinase 1Focal Adhesion Protein-Tyrosine Kinasesbiology.proteinCancer researchHeparitin SulfateCarotid Artery InjuriesProtein Processing Post-TranslationalProto-Oncogene Proteins c-aktHeparan Sulfate ProteoglycansCirculation Research
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Transmissible gastroenteritis virus (TGEV)-based vectors with engineered murine tropism express the rotavirus VP7 protein and immunize mice against r…

2011

A coronavirus vector based on the genome of the porcine transmissible gastroenteritis virus (TGEV) expressing the rotavirus VP7 protein was constructed to immunize and protect against rotavirus infections in a murine model. The tropism of this TGEV-derived vector was modified by replacing the spike S protein with the homologous protein from mouse hepatitis virus (MHV). The rotavirus gene encoding the VP7 protein was cloned into the coronavirus cDNA. BALB/c and STAT1-deficient mice were inoculated with the recombinant viral vector rTGEVS-MHV-VP7, which replicates in the intestine and spreads to other organs such as liver, spleen and lungs. TGEV-specific antibodies were detected in all the in…

MaleViral vectorsRotavirusSwinevirusesRecombinant virusmedicine.disease_causeAntibodies ViralVirus ReplicationMice0302 clinical medicinefluids and secretionsRotavirusAntigens ViralCoronavirus0303 health sciencesMice Inbred BALB CProtectionvirus diseases3. Good healthAnimals SucklingSTAT1 Transcription FactorRNA ViralFemaleGenetic EngineeringGene Expression Regulation ViralDiarrheaBiologyTropismArticleRotavirus InfectionsMicrobiologyViral vectorCell Line03 medical and health sciencesMouse hepatitis virusVirologymedicineAnimalsTropism030304 developmental biologyTransmissible gastroenteritis virusRotavirus Vaccinesbiology.organism_classificationVirologyImmunizationViral replicationCapsid ProteinsImmunity Maternally-Acquired030215 immunology
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Management of chronic hepatitis C in childhood: The impact of therapy in the clinical practice during the first 2 decades

2011

Background and aim: Treatment of chronic hepatitis C in children is controversial and its role in the clinical practice is unknown. We retrospectively investigated the impact of treatment in a large cohort of children with chronic hepatitis C over the past 20years. Methods: 376 hepatitis C virus RNApositive children were recruited consecutively in five Italian centres since 1990and followed for1–17years. Results: 86 (23%)subjects were treated: 73 with recombinant interferon alone and 13 with pegylated-interferon and ribavirin. Sustained clearance of hepatitis C virus RNA was observed in 25%of the former, in 92%of the latter and in 9% of untreated cases(p < 0.001). Loss of viraemia was re…

Malemedicine.medical_specialtyAdolescentGenotypeCombination therapyHepatitis C virusNatural historyCHILDRENHepacivirusInterferon alpha-2medicine.disease_causeAntiviral AgentsTHERAPYPolyethylene Glycolschemistry.chemical_compoundChronic hepatitisHepatitis C virus RNAInternal medicineRibavirinmedicineHumansChildRetrospective StudiesHepatologyHepatitis C virusbusiness.industryRibavirinGastroenterologyInfantInterferon-alphaCHRONIC HEPATITISHepatitis C ChronicRecombinant ProteinsTreatmentNatural historyClinical PracticeSustained virological responseChildren; Hepatitis C virus; Natural history; Sustained virological response; TreatmentchemistryViral replicationChild PreschoolHCVImmunologyRNA ViralDrug Therapy CombinationFemaleInterferonsbusinessDigestive and Liver Disease
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Significance of IgG and IgM HCV antibody secretion in vitro in patients with chronic hepatitis C: correlation with disease activity and response to i…

1994

Hepatitis C virus antibodies are found in the serum of most patients with chronic hepatitis C. However, the significance of the humoral response is still uncertain. In this study, in vitro IgG and IgM anti-hepatitis C virus secretion by peripheral blood mononuclear cells of patients with chronic hepatitis C was analyzed. Peripheral-blood mononuclear cells from 21 of 36 patients (58.3%) secreted IgG anti-hepatitis C virus in vitro, as demonstrated with anti-hepatitis C virus—specific enzyme immunoassays and recombinant immunoblot assays. Ten of the 36 patients (27.8%) showed both IgG and IgM anti-hepatitis C virus core in vitro. In 9 of these 10 patients, IgM anti-hepatitis C virus was also …

Malemedicine.medical_specialtyHepatitis C virusHepacivirusInterferon alpha-2medicine.disease_causeVirus ReplicationPeripheral blood mononuclear cellVirusInterferonInternal medicinemedicineHumansHepatitis AntibodiesLymphocytesInterferon alfaCells CulturedHepatitisHepatologybiologybusiness.industryInterferon-alphaAlanine TransaminaseHepatologyHepatitis C AntibodiesMiddle Agedmedicine.diseaseVirologyHepatitis CRecombinant ProteinsImmunoglobulin MLiverImmunoglobulin GImmunologyChronic Diseasebiology.proteinFemaleAntibodybusinessmedicine.drugFollow-Up StudiesHepatology (Baltimore, Md.)
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Slow and fast evolving endosymbiont lineages: positive correlation between the rates of synonymous and nonsynonymous substitution

2015

The availability of complete genome sequences of bacterial endosymbionts with strict vertical transmission to the host progeny opens the possibility to estimate molecular evolutionary rates in different lineages and understand the main biological mechanisms influencing these rates. We have compared the rates of evolution for non-synonymous and synonymous substitutions in nine bacterial endosymbiont lineages, belonging to four clades (Baumannia, Blochmannia, Portiera, and Sulcia). The main results are the observation of a positive correlation between both rates with differences among lineages of up to three orders of magnitude and that the substitution rates decrease over long endosymbioses.…

Microbiology (medical)GeneticsDNA ReplicationNatural selectionfood.ingredientGeneration timeendosymbiosisEndosymbiosisObligateDNA RepairDNA repair[SDV]Life Sciences [q-bio]BlochmanniaDNA replicationlcsh:QR1-502BiologyEvolutionary rateMicrobiologyGenomelcsh:MicrobiologyfoodGeneration timePerspectiveComputingMilieux_MISCELLANEOUSnucleotide substitutionFrontiers in Microbiology
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Mode of selection and experimental evolution of antiviral drugs resistance in vesicular stomatitis virus

2004

Abstract The possession of an antiviral resistance mutation benefits a virus when the corresponding antiviral is present. But does the resistant virus pay a fitness cost when the antiviral is absent? Would an evolutionary history of association between a genotype and a resistance mutation overcome this cost by changes compensating the harmful side-effect of resistance mutations? Are combined therapies more effective against the rise of resistant viruses or against evolutionary compensations? To explore all these questions, we took an experimental evolution approach. After selecting vesicular stomatitis virus (VSV) populations able to replicate under increasing concentrations of ribavirin an…

Microbiology (medical)GenotypeBiologyVirus ReplicationAntiviral AgentsMicrobiologyVirusVesicular stomatitis Indiana virusEvolution Molecularchemistry.chemical_compoundGenotypeDrug Resistance ViralRibavirinGeneticsMolecular BiologyEcology Evolution Behavior and SystematicsGeneticsExperimental evolutionDose-Response Relationship DrugRibavirinAntiviral therapyInterferon-alphaDrug SynergismResistance mutationbiology.organism_classificationVirologyInfectious DiseaseschemistryVesicular stomatitis virusMutationFitness costInfection, Genetics and Evolution
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Overcoming drug resistance in HSV, CMV, HBV and HCV infection.

2015

Although vaccination has provided as a very efficient preventive tool, antiviral therapy is still needed to control viral infections not avoidable by prophylaxis with vaccines; those caused by viruses for which a vaccine is available, but vaccination is not universally implemented or does not result in complete, long-term protection; and in immunocompromised individuals with reduced immune control of viral replication. After more than 50 years of the first licensing for an antiherpetic drug, novel compounds for herpes-simplex viruses and human cytomegalovirus will open new strategies for better control and management of these two recurrent viral infections. Besides, the development and use…

Microbiology (medical)Human cytomegalovirusHepatitis B virusvirusesHepacivirusCytomegalovirusDrug resistanceHepacivirusmedicine.disease_causeMicrobiologyAntiviral AgentsDrug DiscoveryDrug Resistance ViralmedicineHumansSimplexvirusHepatitis B virusbiologybusiness.industryHepatitis Bmedicine.diseasebiology.organism_classificationVirologyVaccinationViral replicationImmunologybusinessViral loadFuture microbiology
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Chronic hepatitis B: who to treat and which choice of treatment?

2009

The goal of antiviral therapy in patients with chronic hepatitis B is to prevent, through persistent suppression of HBV replication, cirrhosis and hepatocellular carcinoma. Currently, seven drugs are available: IFN-alpha, pegylated interferon, lamivudine, adefovir dipivoxil, entecavir, telbivudine and tenofovir. The choice of the drugs should always take into consideration the clinical features of patients, the antiviral efficacy of each drug, the risk of developing resistance, the long-term safety profile, the method of administration and the cost of therapy. Ideal candidates for treatment are hepatitis B e antigen-positive patients with a prolonged phase of immune clearance and hepatitis …

Microbiology (medical)Liver Cirrhosismedicine.medical_specialtyHepatitis B virusCarcinoma Hepatocellularmedicine.disease_causeVirus ReplicationMicrobiologyGastroenterologyAntiviral AgentsDrug Administration ScheduleHepatitis B ChronicPegylated interferonVirologyTelbivudineInternal medicineDrug Resistance ViralmedicineAdefovirHumansRandomized Controlled Trials as TopicHepatitis B virusbusiness.industryNucleotidesLamivudineNucleosidesEntecavirHepatitis Bmedicine.diseaseVirologyInfectious DiseasesPractice Guidelines as TopicHepatitia BbusinessViral hepatitisantiviral Therapymedicine.drugExpert review of anti-infective therapy
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Evolutionary history conditions the timing of transmission in vesicular stomatitis virus.

2001

It has been postulated that early transmitted viruses would evolve to be more virulent than late transmitted ones. The reason for this prediction is that early transmission selects for rapid viral replication and, consequently, rapid host death, whereas late transmission would select for slow-replicating viruses that permit longer survival to the host. To test this prediction, experimental lineages of vesicular stomatitis virus (VSV) had been adapted to three different transmission dynamics during more than 100 generations. Transmission dynamic differed in the stage of infection at which transmission took place: early, intermediate or late. Regardless the timing of transmission imposed duri…

Microbiology (medical)Time FactorsVirulenceVesicular stomatitis Indiana virusBiologyVirus ReplicationMicrobiologyModels BiologicalVirusVesicular stomatitis Indiana viruslaw.inventionlawRhabdoviridae InfectionsGeneticsHumansMolecular BiologyEcology Evolution Behavior and SystematicsGeneticsExperimental evolutionVirulenceHost (biology)biology.organism_classificationVirologyBiological EvolutionInfectious DiseasesTransmission (mechanics)Viral replicationVesicular stomatitis virusInfection, genetics and evolution : journal of molecular epidemiology and evolutionary genetics in infectious diseases
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