Search results for " Regulator"

showing 10 items of 728 documents

High osmolarity glycerol (HOG) signalling in Magnaporthe oryzae: Identification of MoYPD1 and its role in osmoregulation, fungicide action, and patho…

2015

AbstractThis study comprises a first functional analysis of an YPD1-homologue in filamentous phytopathogenic fungi and its role in the HOG signalling pathway. We generated a gene deletion mutant of the gene MoYPD1 in Magnaporthe oryzae and characterized the resulting mutant strain. We have shown that MoYpd1p is a component of the phosphorelay system acting in the HOG pathway due to its Y2H protein interaction with the HKs MoHik1p and MoSln1p as well as with the response regulator MoSsk1p. Fungicidal activity of fludioxonil was reported to be based on the inhibition of MoHik1p resulting in hyperactivation of the HOG signalling pathway and lethality. Western analysis proved that both, osmotic…

GlycerolFilamentous fungiOsmotic shockMutantVirulenceFludioxonilDioxolesPlant ScienceFludioxonilBiologyMicrobiologyFungal ProteinsOsmoregulationOsmotic PressureGeneticsPyrrolesPhosphotransferGeneEcology Evolution Behavior and SystematicsPlant DiseasesVirulenceOsmolar ConcentrationOryzaHedgehog signaling pathwayFungicides IndustrialCell biologyMagnaportheResponse regulatorInfectious DiseasesPhosphorylationSignal TransductionEnvironmental signallingFungal Biology
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Rapid adaptation of signaling networks in the fungal pathogen Magnaporthe oryzae

2019

Abstract Background One fundamental question in biology is how the evolution of eukaryotic signaling networks has taken place. “Loss of function” (lof) mutants from components of the high osmolarity glycerol (HOG) signaling pathway in the filamentous fungus Magnaporthe oryzae are viable, but impaired in osmoregulation. Results After long-term cultivation upon high osmolarity, stable individuals with reestablished osmoregulation capacity arise independently from each of the mutants with inactivated HOG pathway. This phenomenon is extremely reproducible and occurs only in osmosensitive mutants related to the HOG pathway – not in other osmosensitive Magnaporthe mutants. The major compatible so…

GlycerolMagnaportheved/biology.organism_classification_rank.speciesMutantGenomeSalt StressTranscriptome0302 clinical medicineOsmoregulationLoss of Function MutationGene Expression Regulation FungalGene Regulatory NetworksSuppressorReestablishment of osmoregulation0303 health sciencesbiologyMagnaporthe oryzaeRewiringAdaptation PhysiologicalRapid adaptationCell biologyMagnaportheOsmoregulationEpigeneticsGenome FungalBiotechnologySignal TransductionResearch Articlelcsh:QH426-470lcsh:BiotechnologyDioxolesFungal Proteins03 medical and health sciencesDrug Resistance Fungallcsh:TP248.13-248.65GeneticsPyrrolesModel organismGene030304 developmental biologyPlant DiseasesOsmotic concentrationved/biologyGene Expression ProfilingEvolution of signaling networksHOG pathwayOryzabiology.organism_classificationlcsh:Genetics030217 neurology & neurosurgery
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Phenotype traits associated with different alleles at the RPS5 locus in Saccharomyces cerevisiae

1992

The RPS5 gene has been characterised through its ability to reduce invertase production by the SUC5 gene. In this paper we show that RPS5 acts by maintaining low levels of SUC5 mRNA. We also show that RPS5 acts on the SUC1 and SUC4 genes but not on SUC2 and SUC3, which are members of the SUC family. RPS5 also shows a pleiotropic effect on the amount of mitochondrial cytochromes.

Glycoside HydrolasesTranscription GeneticGenes FungalSaccharomyces cerevisiaeGenes RecessiveLocus (genetics)Saccharomyces cerevisiaeSaccharomycesGene Expression Regulation EnzymologicGene Expression Regulation FungalGenes RegulatorGene expressionGeneticsAlleleGeneAllelesGenes DominantRegulator geneGeneticsbeta-FructofuranosidasebiologyGeneral Medicinebiology.organism_classificationPhenotypePhenotypeCytochromesCurrent Genetics
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A distinct subset of HLA-DR+-regulatory T cells is involved in the induction of preterm labor during pregnancy and in the induction of organ rejectio…

2010

Regulatory T cells (Tregs) are known to suppress alloimmune responses during pregnancy and post organ transplantation. We demonstrate that a distinct subset of FoxP3(+)DR(+)-Tregs among the total CD4(+)CD127(low+/-)CD25(+)-Treg cell pool is critically involved in preterm labor induction and kidney transplant rejection as well. Compared to healthy pregnancies and non-rejecting kidney recipients, we found that the percentage of the FoxP3(+)DR(+)-Treg subset was not reduced, but that the level of HLA-DR expression of such Tregs was strongly diminished in preterm laboring women and in patients with acute renal allograft rejection. In addition, both patient collectives showed a significantly red…

Graft RejectionMalemedicine.medical_specialtyImmunologychemical and pharmacologic phenomenaT-Lymphocytes RegulatoryOrgan transplantationImmune toleranceInterleukin-7 Receptor alpha SubunitObstetric Labor PrematurePregnancyT-Lymphocyte SubsetsHLA-DRImmune ToleranceImmunology and AllergyMedicineHumansKidney transplantationbusiness.industryInterleukin-2 Receptor alpha SubunitFOXP3hemic and immune systemsForkhead Transcription FactorsHLA-DR Antigensmedicine.diseaseKidney TransplantationTransplant rejectionCD4 Lymphocyte CountTransplantationTolerance inductionImmunologyPremature BirthFemalebusinessClinical immunology (Orlando, Fla.)
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Genetic manipulation of longevity-related genes as a tool to regulate yeast life span and metabolite production during winemaking

2013

Abstract Background Yeast viability and vitality are essential for different industrial processes where the yeast Saccharomyces cerevisiae is used as a biotechnological tool. Therefore, the decline of yeast biological functions during aging may compromise their successful biotechnological use. Life span is controlled by a variety of molecular mechanisms, many of which are connected to stress tolerance and genomic stability, although the metabolic status of a cell has proven a main factor affecting its longevity. Acetic acid and ethanol accumulation shorten chronological life span (CLS), while glycerol extends it. Results Different age-related gene classes have been modified by deletion or o…

HST3GlycerolSaccharomyces cerevisiae ProteinsTranscription Genetic<it>HST3</it>Saccharomyces cerevisiaeLongevitylcsh:QR1-502SOD2BioengineeringApoptosisWinePUB1Saccharomyces cerevisiaeStressApplied Microbiology and Biotechnologylcsh:MicrobiologyHistone DeacetylasesStress granuleSirtuin 2<it>PUB1</it>Gene expressionChronological agingSirtuinsNADH NADPH OxidoreductasesRNA MessengerEthanol metabolismSilent Information Regulator Proteins Saccharomyces cerevisiaeAcetic AcidbiologyEthanolSuperoxide DismutaseResearchRNA-Binding Proteinsbiology.organism_classificationYeastYeastBiochemistryCaspasesFermentationMutationFermentationHistone deacetylaseGene DeletionBiotechnologyMicrobial Cell Factories
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Translating the Role of mTOR- and RAS-Associated Signalopathies in Autism Spectrum Disorder: Models, Mechanisms and Treatment

2021

Mutations affecting mTOR or RAS signaling underlie defined syndromes (the so-called mTORopathies and RASopathies) with high risk for Autism Spectrum Disorder (ASD). These syndromes show a broad variety of somatic phenotypes including cancers, skin abnormalities, heart disease and facial dysmorphisms. Less well studied are the neuropsychiatric symptoms such as ASD. Here, we assess the relevance of these signalopathies in ASD reviewing genetic, human cell model, rodent studies and clinical trials. We conclude that signalopathies have an increased liability for ASD and that, in particular, ASD individuals with dysmorphic features and intellectual disability (ID) have a higher chance for disrup…

Heart diseaseAutism Spectrum DisorderReviewQH426-47003 medical and health sciencesEpilepsy0302 clinical medicineIntellectual disabilitymedicineGeneticsAnimalsHumansGene Regulatory NetworksGenetics (clinical)PI3K/AKT/mTOR pathway030304 developmental biology0303 health sciencesbusiness.industryTOR Serine-Threonine KinasesCancermedicine.diseasePhenotype3. Good healthClinical trialDisease Models AnimalGene Expression RegulationAutism spectrum disorderintellectual disabilityMutationras ProteinsmTORbusinessNeuroscience030217 neurology & neurosurgerySignal TransductionRASGenes
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Hepatitis B protein HBx binds the DLEU2 lncRNA to sustain cccDNA and host cancer-related gene transcription.

2019

Objective: The HBV HBx regulatory protein is required for transcription from the covalently closed circular DNA (cccDNA) minichromosome and affects the epigenetic control of both viral and host cellular chromatin. Design: We explored, in relevant cellular models of HBV replication, the functional consequences of HBx interaction with DLEU2, a long non-coding RNA (lncRNA) expressed in the liver and increased in human hepatocellular carcinoma (HCC), in the regulation of host target genes and the HBV cccDNA. Results: We show that HBx binds the promoter region, enhances the transcription and induces the accumulation of DLEU2 in infected hepatocytes. We found that nuclear DLEU2 directly binds HBx…

Hepatitis B virusCarcinoma Hepatocellular2312HepatologyvirusesLiver NeoplasmsCell Culture Techniquesmacromolecular substanceshepatocellular carcinomaVirus Replicationliverdigestive system diseasesHepatocytesTrans-ActivatorsHumansEnhancer of Zeste Homolog 2 ProteinRNA Long NoncodingViral Regulatory and Accessory Proteins1506hepatitis BDNA CircularGut
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Virus replication and virion export in X-deficient hepatitis B virus transgenic mice

2002

The function of the X protein (pX) in the replication cycle of mammalian hepadnaviruses is enigmatic. Using tissue culture experiments it has been shown that the X gene product is not central to hepatitis B virus (HBV) replication and virion export. However, at present it is still unclear whether this also applies to the in vivo situation. Using a terminally redundant X-deficient HBV DNA construct, transgenic mice were established that exhibited high-level expression of the viral core protein in liver and kidneys. Importantly, replicative DNA intermediates and mature viral genomes could be detected in the liver and serum of these mice, respectively. These findings indicate that, in the in v…

Hepatitis B virusHepatitis B virus DNA polymerasevirusesTransgeneMice TransgenicBiologyVirus Replicationmedicine.disease_causeHepatitis B virus PRE betaGene productMicechemistry.chemical_compoundVirologymedicineAnimalsViral Regulatory and Accessory ProteinsHepatitis B virusVirionVirologyMolecular biologydigestive system diseasesMice Inbred C57BLHBxViral replicationchemistryMice Inbred DBATrans-ActivatorsDNAJournal of General Virology
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Evolutionary history of the OmpR/IIIA family of signal transduction two component systems in Lactobacillaceae and Leuconostocaceae

2011

15 pages, 3 tables, 7 figures.

Histidine KinaseEvolutionMolecular Sequence DataSignal transductionEvolution MolecularBacterial ProteinsPhylogeneticsQH359-425Lactic acid bacteriaAmino Acid SequenceGeneEcology Evolution Behavior and SystematicsPhylogenyGeneticsTwo component systemLeuconostocaceaebiologyPhylogenetic treeLactobacillalesfungiLactobacillaceaebiology.organism_classificationTwo-component regulatory systemResponse regulatorLactobacillaceaeMultigene FamilyLeuconostocaceaeProtein KinasesSequence AlignmentOmpR/IIIA familyResearch Article
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Fumarate regulation of gene expression in Escherichia coli by the DcuSR (dcuSR genes) two-component regulatory system.

1998

ABSTRACT In Escherichia coli the genes encoding the anaerobic fumarate respiratory system are transcriptionally regulated by C 4 -dicarboxylates. The regulation is effected by a two-component regulatory system, DcuSR, consisting of a sensory histidine kinase (DcuS) and a response regulator (DcuR). DcuS and DcuR are encoded by the dcuSR genes (previously yjdHG ) at 93.7 min on the calculated E. coli map. Inactivation of the dcuR and dcuS genes caused the loss of C 4 -dicarboxylate-stimulated synthesis of fumarate reductase ( frdABCD genes) and of the anaerobic fumarate-succinate antiporter DcuB ( dcuB gene). DcuS is predicted to contain a large periplasmic domain as the supposed site for C 4…

Histidine KinaseGenetics and Molecular Biologymedicine.disease_causeMicrobiologyAntiportersBacterial ProteinsFumaratesmedicineEscherichia coliDicarboxylic AcidsMolecular BiologyEscherichia coliRegulation of gene expressionDicarboxylic Acid TransportersbiologySuccinate dehydrogenaseEscherichia coli ProteinsHistidine kinaseMembrane ProteinsPeriplasmic spaceGene Expression Regulation BacterialFumarate reductaseTwo-component regulatory systemDNA-Binding ProteinsSuccinate DehydrogenaseResponse regulatorMutagenesis InsertionalBiochemistryGenes Bacterialbiology.proteinCarrier ProteinsProtein KinasesSignal TransductionTranscription FactorsJournal of bacteriology
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