Search results for " STRESS"

showing 10 items of 3936 documents

Insulin-activated Akt rescues Aβ oxidative stress-induced cell death by orchestrating molecular trafficking

2011

Increasing evidence indicates that Alzheimer's disease, one of the most diffused aging pathologies, and diabetes may be related. Here, we demonstrate that insulin signalling protects LAN5 cells by amyloid-β42 (Aβ)-induced toxicity. Aβ affects both activation of insulin receptors and the levels of phospho-Akt, a critical signalling molecule in this pathway. In contrast, oxidative stress induced by Aβ can be antagonized by active Akt that, in turn, inhibits Foxo3a, a pro-apoptotic transcription factor activated by reactive oxygen species generation. Insulin cascade protects against mitochondrial damage caused by Aβ treatment, restoring the mitochondrial membrane potential. Moreover, we show t…

AgingbiologyAmyloid betaInsulinmedicine.medical_treatmentCell BiologyMitochondrionmedicine.disease_causeCell biologyInsulin receptormedicinebiology.proteinPhosphorylationSignal transductionProtein kinase BOxidative stressAging Cell
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Antioxidants as a Potential Therapy Against Age-Related Neurodegenerative Diseases: Amyloid Beta Toxicity and Alzheimers Disease

2006

Alzheimer's disease (AD) is a progressive age-related neurodegenerative disorder with distinct neuropathological features. Extracellular plaques, consisting of aggregated amyloid peptides of 39-43 amino acids are one of the most prominent pathological hallmarks of this disease. Although the exact neurochemical effector mechanism of Abeta aggregation is not yet elucidated, age-associated disturbances of metal ion metabolism have been proposed to promote the formation of aggregates from soluble Abeta. Oxidative stress is postulated to be a downstream effect of Abeta-metal ion interactions. Therefore, the modulation of brain metal metabolism and attenuation of oxidative stress by antioxidant m…

Agingmedicine.medical_specialtyAntioxidantAmyloidAmyloid betamedicine.medical_treatmentPharmacologymedicine.disease_causeAntioxidantsNeurochemicalDegenerative diseaseAlzheimer DiseaseInternal medicinemental disordersDrug DiscoverymedicineAnimalsHumansPharmacologyAmyloid beta-PeptidesMetal metabolismbiologyChemistryNeurodegenerative Diseasesmedicine.diseaseEndocrinologybiology.proteinAlzheimer's diseaseOxidative stressCurrent Pharmaceutical Design
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Lifelong soya consumption in males does not increase lifespan but increases health span under a metabolic stress such as type 2 diabetes mellitus.

2021

Soya consumption can decrease oxidative stress in animal models. Moreover, phytoestrogens such as genistein, present in soya, can mimic some of the beneficial effects of estrogens and are devoid of significant side effects, such as cancer. In this study, we have performed a controlled lifelong study with male OF1 mice that consumed either a soya-free diet or a soya-rich diet. We show that, although we found an increase in the expression and activity of antioxidant enzymes in soya-consuming mice, it did not increase lifespan. We reasoned that the soya diet could not increase lifespan in a very healthy population, but perhaps it could extend health span in stressed animals such as type 2 diab…

Agingmedicine.medical_specialtyAntioxidantGoto Kakizaki ratsmedia_common.quotation_subjectmedicine.medical_treatmentLongevityGenisteinPhytoestrogensDiseasemedicine.disease_causeAntioxidants03 medical and health scienceschemistry.chemical_compoundfluids and secretions0302 clinical medicineStress PhysiologicalInternal medicineMedicineAnimals030304 developmental biologymedia_common2. Zero hunger0303 health sciencesbusiness.industryLongevityfood and beveragesType 2 Diabetes MellitusIsoflavonesAnimal FeedIsoflavones3. Good healthRatsOxidative StressEndocrinologychemistryDiabetes Mellitus Type 2Soya030220 oncology & carcinogenesisAntioxidant enzymesPhytoestrogensSoybeansbusinessOxidative stressDevelopmental BiologyMechanisms of ageing and development
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Molecular bases of the treatment of Alzheimer's disease with antioxidants: prevention of oxidative stress

2004

Alzheimer's disease is associated with a systemic oxidative stress situation which can be followed in vivo by determining biomarkers such as plasma lipoperoxides and TBARS levels and the oxidation degree of glutathione in red blood cells. It has been observed that Alzheimer's patients show an increased level of plasma TBARS, which indicates a higher free radical oxidation of plasma unsaturated phospholipids, and an increased oxidation of red blood cells glutathione, which indicates oxidative stress in peripheral cells. This latter, glutathione oxidation, was found to correlate statistically with the cognitive status of the patients. Treatment with vitamin E resulted in an improved cognitive…

Agingmedicine.medical_specialtyAntioxidantmedicine.medical_treatmentClinical BiochemistryDiseasemedicine.disease_causeBiochemistryAntioxidantschemistry.chemical_compoundAlzheimer DiseaseIn vivoInternal medicineTBARSmedicineHumansVitamin ETocopherolMolecular BiologyChemistryVitamin EGeneral MedicineGlutathioneGlutathioneMitochondriaOxidative StressEndocrinologyImmunologyMolecular MedicineOxidative stressMolecular Aspects of Medicine
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Age-Related Changes of Liver Antioxidant Enzymes and 8-Hydroxy-2-Deoxyguanosine During Fetal–Neonate Transition and Early Rat Development

2000

We have studied the pro-antioxidant status of the rat liver on the last day of gestation and at 1, 15, and 30 days of extrauterine life. Representative variables, such as activities of superoxide dismutase (SOD), catalase, and glutathione peroxidase and concentrations of reduced glutathione and 8-hydroxy-2'-deoxyguanosine, were determined in liver to assess the degree of birth-associated oxidative stress during the fetal-neonatal transition and early development of the rat. Percentages by which liver Cu/ZnSOD activity increased over the basal value of the fetal liver were 54%, 95%, and 127% at neonatal days 1, 15, and 30, respectively. There was a lack of induction in the development profil…

Agingmedicine.medical_specialtyAntioxidantmedicine.medical_treatmentClinical Biochemistrymedicine.disease_causeBiochemistryAntioxidantsSuperoxide dismutasechemistry.chemical_compoundFetusPregnancyInternal medicineGeneticsmedicineAnimalsDeoxyguanosineRats WistarMolecular Biologychemistry.chemical_classificationGlutathione PeroxidaseFetusbiologySuperoxide DismutaseGlutathione peroxidaseDeoxyguanosineCell BiologyGlutathioneCatalaseGlutathioneRatsOxidative StressEndocrinologyAnimals NewbornLiverchemistry8-Hydroxy-2'-DeoxyguanosineCatalasebiology.proteinFemaleOxidative stressDNA DamageIUBMB Life (International Union of Biochemistry and Molecular Biology: Life)
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Being the Family Caregiver of a Patient With Dementia During the Coronavirus Disease 2019 Lockdown

2021

Background: Family caregivers of patients with dementia are at high risk of stress and burden, and quarantine due to the coronavirus disease 2019 (COVID-19) pandemic may have increased the risk of psychological disturbances in this population. The current study was carried out during the national lockdown declared in March 2020 by the Italian government as a containment measure of the first wave of the coronavirus pandemic and is the first nationwide survey on the impact of COVID-19 lockdown on the mental health of dementia informal caregivers.Methods: Eighty-seven dementia centers evenly distributed on the Italian territory enrolled 4,710 caregiver–patient pairs. Caregivers underwent a tel…

Agingmedicine.medical_specialtyCognitive NeurosciencePopulationPsychological interventionProtective factorNeurosciences. Biological psychiatry. Neuropsychiatryburden03 medical and health sciencesstress0302 clinical medicinestremedicineDementiaburden; caregiver; COVID-19; dementia; stressPsychiatryeducationcaregiverOriginal ResearchMED/26 - NEUROLOGIAeducation.field_of_study030214 geriatricsburden caregiver COVID-19 dementia stressFamily caregiversbusiness.industryCOVID-19COVID-19; burden; caregiver; dementia; stressmedicine.diseaseMental healthSettore MED/26 - NEUROLOGIAAnxietyCaregiver stressM-PSI/08 - PSICOLOGIA CLINICAmedicine.symptombusiness030217 neurology & neurosurgeryNeuroscienceRC321-571dementia
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The Effect of Moderate- Versus High-Intensity Resistance Training on Systemic Redox State and DNA Damage in Healthy Older Women

2018

This study investigated effects of a 16-week progressive resistance training program (RTP) with elastic bands at two different intensities on systemic redox state, DNA damage, and physical function in healthy older women. Methods: Participants were randomly assigned to the high-intensity group (HIGH; n = 39), moderate-intensity group (MOD; n = 31), or control group (CG; n = 23). The exercise groups performed an RTP twice a week with three to four sets of 6 (HIGH) or 15 (MOD) repetitions of six overall body exercises at a perceived exertion rate of 8–9 on the OMNI-Resistance Exercise Scale for use with elastic bands. Thiol redox state was determined by reduced glutathione (GSH), oxidized gl…

Agingmedicine.medical_specialtyDNA damageStrength trainingEstrès oxidatiuUrinemedicine.disease_causeRedox03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicinemedicineHumansDeoxyguanosineAgedAged 80 and overResearch and TheoryResistance trainingResistance Training030229 sport sciencesGlutathioneMiddle AgedEntrenament (Esport)GlutathioneHealthy VolunteersExercise TherapyEndocrinologychemistryFemaleOxidation-Reduction030217 neurology & neurosurgeryOxidative stressDNA Damage
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Impaired calcium homeostasis in aged hippocampal neurons

2009

Abstract Development of neurodegenerative diseases such as Alzheimer's and Parkinson's disease is strongly age-associated. The impairment of calcium homeostasis is considered to be a key pathological event leading to neuronal dysfunction and cell death. However, the exact impact of aging on calcium homeostasis in neurons remains largely unknown. In the present work we have investigated intracellular calcium levels in cultured primary hippocampal neurons from young (2 months) and aged (24 months) rat brains. Upon stimulation with glutamate or hydrogen peroxide aged neurons in comparison to young neurons demonstrated an increased vulnerability to these disease-related toxins. Measurement of c…

Agingmedicine.medical_specialtyNeurotoxinsGlutamic Acidchemistry.chemical_elementHippocampusBiologyCalciumHippocampusCalcium in biologyRats Sprague-DawleyInternal medicinemedicineAnimalsCalcium SignalingOrganic ChemicalsCells CulturedCellular SenescenceNeuronsCalcium metabolismCalpainGeneral NeuroscienceNeurodegenerationGlutamate receptorCalpainHydrogen PeroxideOxidantsmedicine.diseaseRatsOxidative Stressmedicine.anatomical_structureEndocrinologynervous systemchemistryNerve Degenerationbiology.proteinCalciumNeuronNeuroscienceNeuroscience Letters
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Effects of Nrf2 deficiency on bone microarchitecture in an experimental model of osteoporosis

2014

Objective. Redox imbalance contributes to bone fragility. We have evaluated the in vivo role of nuclear factor erythroid derived 2-related factor-2 (Nrf2), an important regulator of cellular responses to oxidative stress, in bone metabolism using a model of postmenopausal osteoporosis. Methods. Ovariectomy was performed in both wild-type and mice deficient in Nrf2 (Nrf2-/-). Bone microarchitecture was analyzed by CT. Serum markers of bone metabolism were also measured. Reactive oxygen species production was determined using dihydrorhodamine 123. Results. Sham-operated or ovariectomized Nrf2 -/- mice exhibit a loss in trabecular bone mineral density in femur, accompanied by a reduction in co…

Agingmedicine.medical_specialtycytoarchitectureArticle SubjectNF-E2-Related Factor 2MedicinaOsteoporosisOsteoclastsBone Marrow Cellsprotein deficiencymedicine.disease_causeenvironment and public healthBiochemistryBone resorptionBone remodelingMiceIn vivoInternal medicinemedicineAnimalsFemurcontrolled studyFemurlcsh:QH573-671Cells CulturedMice Knockoutchemistry.chemical_classificationReactive oxygen specieslcsh:CytologyChemistrybone densityCell DifferentiationCell BiologyGeneral Medicinerespiratory systemmedicine.diseaseosteoporosisMice Inbred C57BLDisease Models AnimalOxidative StressEndocrinologyOvariectomized ratReactive Oxygen SpeciesTomography X-Ray ComputedBiomarkersOxidative stressResearch Article
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A low cortisol response to acute stress is related to worse basal memory performance in older people

2014

Age-related memory decline has been associated with a faulty regulation of the hypothalamus-pituitary-adrenal axis (HPA-axis). The aim of this study was to investigate whether the magnitude of the stress-induced cortisol increase is related to memory performance when memory is measured in non-stressful conditions. To do so, declarative and working memory performance were measured in 31 men and 35 women between 55 and 77 years of age. On a different day, the magnitude of their cortisol response to acute psychosocial stress was measured. The relationship between the cortisol response and memory performance was U shaped: a low cortisol response to stress was related to poorer declarative and w…

Agingmedicine.medical_specialtyendocrine systemCognitive NeuroscienceEffects of stress on memoryAudiologycortisolMemory performanceelderlyworking memoryDevelopmental psychologylcsh:RC321-571older peopleBasal (phylogenetics)Low cortisolmedicineOriginal Research Articlelcsh:Neurosciences. Biological psychiatry. NeuropsychiatryWorking memoryStressorMiddle agedeclarative memoryHPA-axisSDG 1 - No Povertymiddle-agePsychologyOlder people/dk/atira/pure/sustainabledevelopmentgoals/no_povertyhormones hormone substitutes and hormone antagonistsNeuroscienceacute psychosocial stress
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