Search results for " Smooth"

showing 10 items of 455 documents

JAK2 mediates lung fibrosis, pulmonary vascular remodelling and hypertension in idiopathic pulmonary fibrosis: an experimental study

2018

BackgroundPulmonary hypertension (PH) is a common disorder in patients with idiopathic pulmonary fibrosis (IPF) and portends a poor prognosis. Recent studies using vasodilators approved for PH have failed in improving IPF mainly due to ventilation (V)/perfusion (Q) mismatching and oxygen desaturation. Janus kinase type 2 (JAK2) is a non-receptor tyrosine kinase activated by a broad spectrum of profibrotic and vasoactive mediators, but its role in PH associated to PH is unknown.ObjectiveThe study of JAK2 as potential target to treat PH in IPF.Methods and resultsJAK2 expression was increased in pulmonary arteries (PAs) from IPF (n=10; 1.93-fold; P=0.0011) and IPF+PH (n=9; 2.65-fold; P<0.00…

0301 basic medicinePulmonary and Respiratory Medicinemedicine.medical_specialtyHypertension PulmonaryBlotting WesternMyocytes Smooth MuscleFluorescent Antibody TechniqueVasodilationVascular RemodelingReal-Time Polymerase Chain ReactionVascular remodelling in the embryo03 medical and health sciencesIdiopathic pulmonary fibrosisTransforming Growth Factor betaRight ventricular hypertrophyInternal medicinepulmonary hypertensionAnimalsHumansMedicineRNA Small InterferingRats WistarCells CulturedCell ProliferationBKCaJanus kinase 2biologybusiness.industryEndothelial CellsJanus Kinase 2idiopathic pulmonary fibrosismedicine.diseaseImmunohistochemistryPulmonary hypertensionIdiopathic Pulmonary FibrosisTriterpenesRatsPhenotype030104 developmental biologyJAK2biology.proteinCardiologyAnimal studiesJanus kinasebusinessSignal TransductionPulmonary artery smooth muscle cells Pulmonary artery endothelial cells.Thorax
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Large strain stimulation promotes extracellular matrix production and stiffness in an elastomeric scaffold model

2016

Mechanical conditioning of engineered tissue constructs is widely recognized as one of the most relevant methods to enhance tissue accretion and microstructure, leading to improved mechanical behaviors. The understanding of the underlying mechanisms remains rather limited, restricting the development of in silico models of these phenomena, and the translation of engineered tissues into clinical application. In the present study, we examined the role of large strip-biaxial strains (up to 50%) on ECM synthesis by vascular smooth muscle cells (VSMCs) micro-integrated into electrospun polyester urethane urea (PEUU) constructs over the course of 3 weeks. Experimental results indicated that VSMC …

0301 basic medicineScaffoldVascular smooth muscleMaterials scienceIn silico0206 medical engineeringMyocytes Smooth MuscleBiomedical Engineering02 engineering and technologyECM (extracellular matrix)ArticleMuscle Smooth VascularBiomaterialsExtracellular matrix03 medical and health sciencesTissue engineeringmedicineAnimalsMechanical conditioningCells CulturedTissue EngineeringTissue ScaffoldsRational designStiffnessModels Theoretical020601 biomedical engineeringBiomaterialElasticityExtracellular MatrixPolyesterElastomeric scaffold030104 developmental biologyElastomersRats Inbred LewMechanics of MaterialsBiophysicsCollagenStress Mechanicalmedicine.symptomMechanical propertieBiomedical engineering
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Resveratrol and the Interaction between Gut Microbiota and Arterial Remodelling

2020

Arterial remodelling refers to the alteration in the structure of blood vessel that contributes to the progression of hypertension and other cardiovascular complications. Arterial remodelling is orchestrated by the crosstalk between the endothelium and vascular smooth muscle cells (VSMC). Vascular inflammation participates in arterial remodelling. Resveratrol is a natural polyphenol that possesses anti-oxidant and anti-inflammatory properties and has beneficial effects in both the endothelium and VSMC. Resveratrol has been studied for the protective effects in arterial remodelling and gut microbiota, respectively. Gut microbiota plays a critical role in the immune system and inflammatory pr…

0301 basic medicineVascular smooth muscleEndotheliumMyocytes Smooth MusclePopulationlcsh:TX341-641InflammationReviewresveratrolVascular Remodeling030204 cardiovascular system & hematologyResveratrolGut floraPharmacologydigestive systemAntioxidantsMuscle Smooth VascularVascular remodelling in the embryo03 medical and health scienceschemistry.chemical_compound0302 clinical medicinemedicineAnimalsHumanseducationCell ProliferationNeointimal hyperplasiaeducation.field_of_studyNutrition and Dieteticsgut microbiotabiologybusiness.industryanti-oxidantSIRT1 arterial remodellingArteriesmedicine.diseasebiology.organism_classificationGastrointestinal Microbiome030104 developmental biologymedicine.anatomical_structurechemistryinflammationcardiovascular systemmedicine.symptombusinesslcsh:Nutrition. Foods and food supplyFood ScienceNutrients
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Junctional adhesion molecules JAM-B and JAM-C promote autoimmune-mediated liver fibrosis in mice

2018

Fibrosis remains a serious health concern in patients with chronic liver disease. We recently reported that chemically induced chronic murine liver injury triggers increased expression of junctional adhesion molecules (JAMs) JAM-B and JAM-C by endothelial cells and de novo synthesis of JAM-C by hepatic stellate cells (HSCs). Here, we demonstrate that biopsies of patients suffering from primary biliary cholangitis (PBC), primary sclerosing cholangitis (PSC) or autoimmune hepatitis (AIH) display elevated levels of JAM-C on portal fibroblasts (PFs), HSCs, endothelial cells and cholangiocytes, whereas smooth muscle cells expressed JAM-C constitutively. Therefore, localization and function of JA…

0301 basic medicine[SDV]Life Sciences [q-bio]Cholangitis SclerosingMyocytes Smooth MuscleeducationImmunologyImmunoglobulinsAutoimmune hepatitisVascular RemodelingChronic liver diseaseMural cellPrimary sclerosing cholangitisFatty Acids MonounsaturatedMice03 medical and health sciencesFibrosisCell AdhesionmedicineAnimalsHumansImmunology and AllergyMyofibroblastsCells CulturedInflammationMice KnockoutFibrous capsule of GlissonLiver Cirrhosis Biliarybusiness.industryfungiEndothelial Cellsmedicine.diseaseFibrosishumanities3. Good healthMice Inbred C57BLDisease Models AnimalHepatitis Autoimmune030104 developmental biologyLiverVasoconstrictioncardiovascular systemCancer researchHepatic stellate cellFemaleHepatic fibrosisbusinessCell Adhesion MoleculesJournal of Autoimmunity
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Age‐related ultrastructural changes of the basement membrane in the mouse blood‐brain barrier

2018

Abstract The blood‐brain barrier (BBB) is essential for a functional neurovascular unit. Most studies focused on the cells forming the BBB, but very few studied the basement membrane (BM) of brain capillaries in ageing. We used transmission electron microscopy and electron tomography to investigate the BM of the BBB in ageing C57BL/6J mice. The thickness of the BM of the BBB from 24‐month‐old mice was double as compared with that of 6‐month‐old mice (107 nm vs 56 nm). The aged BBB showed lipid droplets gathering within the BM which further increased its thickness (up to 572 nm) and altered its structure. The lipids appeared to accumulate toward the glial side of the BM. Electron tomography …

0301 basic medicineblood‐brain barrierAgingElectron Microscope TomographyMyocytes Smooth Muscleelectron tomographyBlood–brain barrierMuscle Smooth Vascular03 medical and health sciencesMice0302 clinical medicineMicroscopy Electron TransmissionLipid dropletmedicineAnimalsBasement membraneChemistryNeurodegenerationBrainLipid metabolismBiological TransportCell BiologyOriginal ArticlesLipid Dropletsmedicine.diseaseLipid Metabolismbasement membraneCell biologyCapillariesMice Inbred C57BL030104 developmental biologymedicine.anatomical_structureElectron tomographyAgeingageingBlood-Brain Barrier030220 oncology & carcinogenesisAstrocytesUltrastructureMolecular MedicineOriginal ArticleNeuroglia
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Resveratrol and Vascular Function

2019

Resveratrol increases the production of nitric oxide (NO) in endothelial cells by upregulating the expression of endothelial NO synthase (eNOS), stimulating eNOS enzymatic activity, and preventing eNOS uncoupling. At the same time, resveratrol inhibits the synthesis of endothelin-1 and reduces oxidative stress in both endothelial cells and smooth muscle cells. Pathological stimuli-induced smooth muscle cell proliferation, vascular remodeling, and arterial stiffness can be ameliorated by resveratrol as well. In addition, resveratrol also modulates immune cell function, inhibition of immune cell infiltration into the vascular wall, and improves the function of perivascular adipose tissue. All…

0301 basic medicineendotheliumEndotheliumAdipose tissueBlood PressureReviewresveratrol030204 cardiovascular system & hematologyResveratrolsirtuin 1AntioxidantsMuscle Smooth VascularCatalysisvascular functionNitric oxidelcsh:ChemistryInorganic Chemistry03 medical and health scienceschemistry.chemical_compound0302 clinical medicinecardiovascular diseaseEnosmedicineAnimalsHumansPhysical and Theoretical ChemistryReceptorlcsh:QH301-705.5Molecular BiologyAntihypertensive AgentsSpectroscopybiologySirtuin 1Cell growthChemistryOrganic Chemistryfood and beveragesGeneral Medicinebiology.organism_classificationComputer Science ApplicationsCell biology030104 developmental biologymedicine.anatomical_structurelcsh:Biology (General)lcsh:QD1-999biology.proteinEndothelium Vascularendothelial nitic oxide synthaseInternational Journal of Molecular Sciences
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Smooth muscle and neural dysfunction contribute to different phases of murine postoperative ileus.

2015

Background Postoperative ileus (POI) is characterized by a transient inhibition of gastrointestinal (GI) motility after abdominal surgery mediated by the inflammation of the muscularis externa (ME). The aim of this study was to identify alterations in the enteric nervous system that may contribute to the pathogenesis of POI. Methods Gastrointestinal transit, contractility of isolated smooth muscle strips and inflammatory parameters were evaluated at different time points (1.5 h to 10 days) after intestinal manipulation (IM) in mice. Immune-labeling was used to visualize changes in myenteric neurons. Key Results Intestinal manipulation resulted in an immediate inhibition of GI transit recove…

0301 basic medicinemedicine.medical_specialtyCarbacholIleusPhysiologyInflammationBiologyEnteric Nervous SystemProinflammatory cytokineContractility03 medical and health sciencesMiceIleusOrgan Culture TechniquesPostoperative ComplicationsInternal medicinemedicineAnimalsEndocrine and Autonomic SystemsGastroenterologyMuscle SmoothSmooth muscle contractionmedicine.diseaseMice Inbred C57BL030104 developmental biologyEndocrinologyAnesthesiaCholinergicEnteric nervous systemFemalemedicine.symptomInflammation MediatorsGastrointestinal Motilitymedicine.drugNeurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society
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Cardiovascular disease in aging: an overview on thoracic aortic aneurysm as an emerging inflammatory disease

2017

Medial degeneration associated with thoracic aortic aneurysm and acute aortic dissection was originally described by Erdheim as a noninflammatory lesion related to the loss of smooth muscle cells and elastic fibre fragmentation in the media. Recent evidences propose the strong role of a chronic immune/inflammatory process in aneurysm evocation and progression. The coexistence of inflammatory cells with markers of apoptotic vascular cell death in the media of ascending aorta with aneurysms and type A dissections raises the possibility that activated T cells and macrophages may contribute to the elimination of smooth muscle cells and degradation of the matrix. On the other hand, several infla…

0301 basic medicinemedicine.medical_specialtyPathologyAgingImmunologyMyocytes Smooth MuscleThoracic aortic aneurysm; acute aortic dissection; inflammatory pathwayReview Article030204 cardiovascular system & hematologyThoracic aortic aneurysmLesion03 medical and health sciencesAortic aneurysm0302 clinical medicineAneurysmmedicine.arteryInternal medicineThoracic aortic aneurysmAscending aortamedicinelcsh:PathologySettore MED/05 - Patologia ClinicaMyocyteHumansAortic dissectionInflammationAortaacute aortic dissectionAortic Aneurysm Thoracicbusiness.industrySettore MED/23 - Chirurgia CardiacaCell Biologymedicine.diseaseinflammatory pathway030104 developmental biologyCardiovascular DiseasesCardiologycardiovascular systemmedicine.symptombusinesslcsh:RB1-214
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Cannabinoid type 1 receptor modulates intestinal propulsion by an attenuation of intestinal motor responses within the myenteric part of the peristal…

2007

Cannabinoid-1 (CB1) receptor activation affects gastrointestinal propulsion in vivo. It was our aim to further characterize the involved myenteric mechanisms in vivo and in vitro. In CB1(-/-) mice and wild-type littermates we performed in vivo transit experiments by charcoal feeding and in vitro electrophysiological recordings in mouse small intestinal smooth muscle. Ascending neuronal contraction (ANC) following electrical field stimulation was studied in rat ileum in a partitioned organ bath separating the aboral stimulation site from the oral recording site. The knockout animals displayed an accelerated upper gastrointestinal transit compared to control animals. The CB1 receptor antagoni…

AM251Malemedicine.medical_specialtyCannabinoid receptorPhysiologyPolyunsaturated Alkamidesmedicine.medical_treatmentNeuromuscular JunctionMotilityStimulationArachidonic AcidsBiologyNeuromuscular junctionMembrane PotentialsMiceOrgan Culture TechniquesPiperidinesReceptor Cannabinoid CB1Internal medicineCannabinoid Receptor ModulatorsReflexmedicineAnimalsRNA MessengerIntestinal MucosaRats WistarReceptorMice KnockoutMyoelectric Complex MigratingEndocrine and Autonomic SystemsReverse Transcriptase Polymerase Chain ReactionGastroenterologyMuscle SmoothEndocannabinoid systemElectric StimulationRatsIntestinesEndocrinologymedicine.anatomical_structurePyrazolesPeristalsisCannabinoidmedicine.drugEndocannabinoidsNeurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society
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Pharmacological analysis of intrinsic neural control of rat duodenum motility in vitro

1988

Adenosine monophosphatemedicine.medical_specialtyDuodenumMotilitychemistry.chemical_compoundAdenosine TriphosphateAdenine nucleotideInternal medicinemedicineAnimalsPharmacologyAdenine NucleotidesMuscle SmoothAdenosine MonophosphateElectric StimulationIn vitroRatsAdenosine DiphosphateAdenosine diphosphateEndocrinologymedicine.anatomical_structurechemistryDuodenummedicine.symptomGastrointestinal MotilityAdenosine triphosphateMuscle ContractionMuscle contractionPharmacological Research Communications
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